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Explaining the Black-White Prostate Cancer Gap

800 words

The black-white prostate cancer gap: genetic? Environmental? Both? Over the years, countless studies have been carried out—mostly on testosterone—to find the cause of the disparity between American blacks and whites.  Numerous research has shown that black Americans’ test is substantially higher than white Americans (Ross et al, 1986; Winters et al, 2001; etc). However, studies with larger samples showed this was not the case. In a sample of 3,654 whites and 585 black Vietnam veterans shows there is a 3 percent difference favoring blacks (Ellis and Nyborg, 1992). The gold standard—a meta-analysis on 14 relevant studies (which I will discuss tonight) shows that the difference in (free) testosterone is nowhere near what Ross et al (1986) say, but was substantially lower at 2.5 to 4.9 percent (Richard et al, 2014). The meta-analysis, which was done due to the conflicting figures in each study showed that the gap in testosterone.

Obviously these people weren’t genotyped for racial ancestry, so the authors included studies that only included racial descriptors:

If races/ethnicities included in the study were referred to as ‘black’, ‘African-American’, ‘non-Hispanic black’, ‘white’, ‘non-Hispanic white’ or ‘Caucasian’. We did not include men of Hispanic or Asian origin.

Since we know that self-identified race is an almost perfect predictor of genetic ancestry, and the meta-analysis included studies that used the only the descriptors for race/ethnicity then the fact that this is done on American sample shows that testosterone is not as high as commonly thought in blacks when compared to whites (13 percent higher free testosterone).

In this sphere, one of the most common things said is that testosterone is one of the biggest causes of the black-white crime gap. High levels of testosterone ARE linked to higher crime rates, and  blacks commit the most crimes, therefore blacks MUST have substantially higher levels of testosterone to account for the difference, right? Wrong. As I’ve shown above, Richard et al (2014) show that even after controlling for age, the difference in free testosterone was 2.5 to 4.9 percent. Black American males have an annual death rate from prostate cancer 2.4 times higher than whites (Taksler, Keating, and Cutler, 2012). If testosterone—one of the main possible culprits—does not explain the higher rate of prostate cancer mortality in American blacks in comparison to whites, what does?

Diet/environment and smaller genetic effects. People who have lower income cannot afford high-quality food, so they, therefore, have to buy low-quality, high carb, highly processed foods which lead to nutrient deficiencies. Drewnowski and Specter (2004) showed that 1) the highest rates of obesity are found in populations with the lowest incomes and education (correlated with IQ); 2) an inverse relationship between energy density and energy cost; 3) sweets and fats have higher energy density and are more palatable; and 4) poverty and food insecurity are associated with lower food expenditures, lower fruit and vegetable intake, and lower-quality diet. All of these data points show that those who are poor are more likely to be obese due to more energy-dense food being cheaper and fats and sugars being more palatable.

One important nutrient that people are often deficient in is vitamin D. Due to the name, people assume it’s a vitamin. It’s really not.  It’s a steroid hormone. Vitamin D promotes calcium absorption, maintains normal calcium and phosphate levels, promotes bone and cell growth, and reduces imflamation. Black Americans have numerous ailments that are associated with low vitamin D intake.

Black Americans have a lower intake of vitamin D in comparison to white Americans. This is due to low dietary intake of vitamin D and less sun exposure. Dark skin pigmentation reduces vitamin D production in the skin, as dark skin requires more sunlight in order to produce vitamin D. Rickets is a common a common problem for blacks as when the mother is pregnant, she doesn’t get sufficient vitamin D so when the baby is born, it is deficient.

One variable that Dr. Joseph Mercola brings up is that black women don’t breastfeed as much as white women (though the gap is beginning to close a bit) causing rickets (as well as the lack of availability of vitamin D for the baby due to darker skin needing more sunlight to acquire adequate vitamin D).

Pretty much a great case for why race and geography should inform vitamin D intake. This is pivotal for our understanding for racial/ethnic differences in disease acquisition, why these differences occur and what can be done to prevent them.

Elevated levels of testosterone in black men comparison to white men are supposed to explain the higher rate of mortality in black men compared to white men. Except Richard et al (2014) showed that the testosterone gap wasn’t as high as previously thought (at 2.4 to 5.9 percent higher). The deficiency in vitamin D explains this phenomenon. Low vitamin D is linked to aggressive prostate cancer. This is the cause for the disparity, not higher rates of testosterone.


Are There Race Differences in Penis Size? Part II

1000 words

I haven’t completely discredited the notion that Rushton and Lynn may be correct on this variable, but I’m highly skeptical. Hormonal data doesn’t show it. Hormones like IGF-1 and androgen don’t show the differences between races that would lead you to believe that Rushton’s Rule applies here.

PP is at it again, citing the same studies, not providing primary sources, and not addressing what I say to him about hormones in regards to penis size. Hormones affect the body in different ways, and different races have different levels of hormones. This is what I will discuss today.

Insulin-like growth factor 1 (IGF-1) is a hormone that, as it’s name implies, is structurally similar to the hormone insulin. IGF-1 is “partly responsible for systemic GH activities although it possesses a wide number of own properties (anabolic, antioxidant, anti-inflammatory and cytoprotective actions).” Laron and Klinger (1998) showed that children with Laron syndrome who stopped receiving IGF-1 injections showed reductions in penile and testicular size and they returned to pretreatment serum levels. This shows the effects of IGF-1 on sexual organ size.

Knowing this about IGF-1, for Rushton’s theory to be plausible, Blacks would have higher levels, Asians the lowest, and whites in the middle, skewing towards Asians. Platz et al (1999) investigated whether there were racial differences in circulating IGF-1 and insulin-like growth factor-binding protein 3 (IGFBP-3). IGFBP-3 binds IGF-1 and 2, with a dysregulation of IGFBP-3 correlating with cancer. IGFBP-3 is the main transporter of IGF-1 and 2 in the blood stream. The researchers tested men whose self-described ancestry (we know that self-describer ancestry is a great proxy for race, having a 99.86 percent success rate) African American (63) a random sample of Asians and Caucasians (75 respectively) aged 45 to 78 years old. Caucasians had the highest levels of IGF-1 (224 ng/ml), Asians (208 ng/ml), and African Americans (205 ng/ml). The IGF-1:IGFBP-3 ratio was greatest in Caucasians and lowest in Asians. This study was carried out to see if IGF-1 had an effect on prostate cancer. The 13 percent difference in IGFBP-3 between blacks and whites may account for the higher levels of prostate cancer, as IGFBP-3 can control IGF-1 bioavailabilty.

PP also cites Ross et al (1986) showing that blacks have “19 percent higher testosterone”, attempting to use this as evidence for the theory in favor of an inverse relationship between brain size and penis size. He seems to think that total testosterone matters, when what matters is free testosterone.It’s also 15 percent circulating testosterone, 13 percent free testosterone in that one study.  Free testosterone is biologically active, and is able to exert its effect by passing through a cell and activating its receptor. Speaking of free testosterone, in this meta-analysis of 23 studies on black-white differences in testosterone, Richard et al (2014) showed a 2.5 to 4.9 percent difference in free testosterone and concluded that that difference was not enough to account for the racial disparity in prostate cancer. So it’s either black Americans have lower levels of IGFBP-3 or diet/environmental factors that cause this racial disparity in prostate cancer, not testosterone.

Rohrmann et al (2007) showed that testosterone differences between blacks (n=363) and whites (n=674) did not noticeably differ (5.29 ng/ml and 5.11 ng/ml respectively). Mexican Americans (n=376) , on the other hand, showed a higher average rate (5.48 ng/ml) over both cohorts. Blacks had higher levels of estradiol than whites (40.80 pg/nl and 35.46 pg/nl respectively). Blacks also had a higher level of sex hormone-binding globulin (SHGB) (36.49 nmol/liter) than whites (34.91 nmol/liter) and Mexican Americans (34.91 nmol/liter). That may account for some of the racial disparity in prostate cancer, but it’s not testosterone (which shows that ‘higher levels of testosterone’ as PP says, isn’t proof of any racial differences in penis size).

The Kinsey data is nonrepresentative and nonrandom. We have comparative sizes for certain ethnies, and the only statistical difference is between Nigerians and Koreans and Czechs. Rushton and Boegart didn’t mention that blacks danced less than white college students, blacks are more prudish regarding nudity, more likely to have a prostitute as a sexual partner and less likely to want large families (Weizmann et al, 1990). A study on certain CAG repeats shows that Africans cluster with East Asians on two measures, contradicting Lynn’s hypothesis. French Army Surgeon, lol (see Weizman et al 1990 from above):

This work is filled with internal contradictions. For example, an average African Negro penis is said to be 7 3/4 to 8 inches long on p. 56, while on p. 242 it is stated that it “generally exceeds” 9 inches. Similarly, while the French Army surgeon announces on p. 56 that he once discovered a 12-inch penis, an organ of that size becomes “far from rare” on p. 243. As one might presume from such a work, there is no indication of the statistical procedures used to compute averages, what terms such as “often” mean, how subjects were selected, how measurements were made, what the sample sizes were, etc.

I think I’ve shown that there are no “””racial””” differences in size with the Veale et al 2014 study and the Orakwe and Ebuh (2007) study. As far as I see, two statistical differences exist between Nigerians and Koreans and Czechs. But there’s not enough “””quality data””” to say “this race bigger than that race”. To believe there are racial differences in penis size or that there is even an inverse relationship between penis size and brain size takes a huge leap of faith to believe.

There are, without a doubt, average differences in a lot of things between races; hormones being one of them. Any differences  between races in IGF-1 have no effect on penis size (IGF-1 is, however, one reason why black girls reach menarche at a younger age than white girls. Will write more on that in the future.). Africans were more similar to Asians that Caucasians on two of the five androgen indicators that Dutton (2015) tested. The Kinsey data is nonrepresentative and nonrandom and that is what PP continuously references. I’m highly skeptical leading towards no based on my knowledge of hormones and how they work in the human body. Testosterone does not explain any racial differences in penis size, and does not explain any differences in prostate cancer acquisition (though, other hormones do).

Are There Racial Differences in Penis Size?

1800 words

Do racial differences in penis size exist? The average person may say yes, due to viewing porn and hearing ‘stories’ from their friends, ie anecdotal accounts. But is this true? JP Rushton was at the helm of this resurrected idea, stating that an inverse relationship existed between penis size and brain size. He cites a WHO study on condom size showing that African countries get the biggest condoms, yet I cannot find the paper discussing it. PP wrote an article, Non-black men are so jealous of black penis sizeciting the same study Rushton (1997) cited in his book Race, Evolution, and Behavior. I will discuss PP’s musings on racial differences in penis size.

A pet peeve of mine is that a lot of non-black men in the HBD blogosphere believe everything that professor J. Philippe Rushton said about ethnic differences in IQ and brain size, but when it comes to black men having the largest penis size, they suddenly turn into HBD deniers, ranting and raving about how the research is wrong. Well, I’m sorry but HBD is not here to serve your racist supremacy, HBD is here to celebrate ALL RACES, NOT YOURS ONLY!!!! If the data shows that black men have the largest penis size, then shut up and get over it. What kind of man gets jealous over another man’s penis size anyway? A sick, twisted, perverted sinful one. Healthy well adjusted normal guys don’t even know how large their penis is, and couldn’t care less, because they have other things going for them like a girlfriend, a career, and sports, and their mind’s not in the gutter. The only legitimate reason to care is for science, so here are the FACTS:

Exactly, the only reason to care is for science. And the science shows no differences. What data shows differences in penis size? Something you can directly access right now without relying on a secondhand source to prove your claim?

According to data from the World Health Organization Global Programme on AIDS Specification and Guidelines for Condom Procurement (1991, p 33, Table 5), assuming a normal distribution, I estimate the average white American man has a penis length of 162 mm (SD = 19) , and the average African American man has a penis length of 170 mm (SD = 19). (since 5% of black men are longer that 200 mm, and 2% of white men are, while 27% of white men have penises shorter than 151 mm, while 15% of black men do).

No hyperlink to the study? Why do you rely on a secondhand account? Why didn’t you show the table?

Just found some information on the WHO study he cited. The information from which the WHO data is derived is derived from American blacks and Caucasians, not any subjects from Europe or Africa. He also asked 150 people on a Toronto shopping mall what their penis size was (back to garbage self-reports), and finally, who is this French army surgeon he brings up? So many questions. 

Also, PP, asking the questions and questioning his data and what he wrote isn’t “disagreeing on him on this and agreeing with him on everything else”, as you of all people know how critical o am of Lyn  and Rushton. Yet you seem to eat up everything they write, thinking they can never be wrong. Why is that? 

Rushton (1997) writes on pg 169 of Race, Evolution, and Behavior:

Data provided by the Kinsey Institute have confirmed the black-white difference in penis size (Table 8.2, and items 70-72 of Table 8.4). Alfred Kinsey and his colleagues instructed their respondents on how to measure their penis along the top surface, from belly to tip. The respondents were given cards to fill out and return in preaddressed stamped envelopes. Nobile (1982) published the first averages of these data finding the length and circumferences of the penis for the white samples was smaller than for the black sample. (Flaccid length = 3.86 inches [9.80 cm] vs. 4.34 inches [11.02 cm]; erect length = 6.15 inches [15.62 cm] vs. 6.44 inches [16.36 cm]; erect circumference = 4.83 inches [12.27 cm] vs. 4.96 inches [12.60 cm] respectively.)

Self-reports? Please. Self-reports are notoriously embellished. Moreover, the amount of black Americans in the Kinsey study was in double digits.

I can never find certain papers online, so that really hinders any further discussion on this.

If anyone can find any of these papers, leave a comment.

Harvey, P. H., & May, R. M. (1989). Out for the sperm count. Nature, 337, 508-9.

Short, R. V. (1979). Sexual selection and its component parts, somatic and genital selection, as illustrated by man and the great apes. In J. S. Rosenblatt, R. A. Hinde, C. Beer, & M-C Busnel (Eds.), Advances in the Study of Behavior, Vol. 9. New York: Academic

When it comes to scrotal circumference, I don’t have exact figures, but Rushton cites scholars showing that Africans exceed Europeans: Short, 1979; Ajmani, Jain & Saxena, 1985.

I found Ajmani et al (1985), they state that in 320 healthy Nigerians, average penile length was 3.21 inches, while scrotal circumference was 8.37 inches. PP says he didn’t have exact figures, but they were in Rushton’s references (which I see he didn’t check). Rushton doesn’t cite data on European scrotal circumference so how is that saying that ‘Africans exceed Europeans’ when only Nigerians were examined? Remember: studies are only applicable to the demographic tested. Extrapolating that data on to the whole of Africa makes no sense. Moreover, any data on Europeans is only for that specific ethny tested. Unless the whole of the European continent is averaged out, you cannot say that these differences exist.

This doesn’t even touch on Lynn’s “data” on penis size:

  Lynn attempts to justify his belief that there are differences between races in penis length on the basis that European and Asian males have lower levels of testosterone than Africans and that the “reduction of  testosterone had the effect of reducing penis length, for which evidence is given by Widodsky and Greene (1940).” Widodsky and Greene (1940) is actually a study of the effects of sex hormones on the penises of rats. This is hardly convincing evidence that there are racial differences in testosterone levels or that a reduction in penis length ever occurred in human history.

Lynn’s claims about differences in penis length between races build on earlier claims by Rushton and Bogaert (1987). The Rushton and Boagert paper is striking for its use of non-scholarly sources (Weizmann, Wiener, Wiesenthal, & Ziegler, 1991). These include a book of semi-pornographic “tall tales” by an anonymous nineteenth century French surgeon that makes wildly inconsistent claims about genital sizes in people of different races. Lynn also refers to this book without mentioning any problems with this as a source of information. Another odd data source cited by Rushton and Bogaert is an article authored by a certain “P. Nobile” published in Forum: International Journal of Human Relations. This publication is better known to the public as “The Penthouse Forum”, a popular men’s magazine. [This is pretty well known and embarrassing that Rushton did that.]

The data sources that Lynn uses in his recent paper are hardly much better. One of them is a book by Donald Templer (another self-professed race realist[1]) called Is Size Important? Templer is not a urologist but a psychologist so why he would claim to be an authority on this subject is unclear.[2] Lynn’s other source is the world penis size website. These are both self-published sources that have not been independently verified. A blogger named Ethnic Muse has carefully examined this site’s references and found that a number of articles listed on the site either do not exist under the name given or do not discuss penis size at all. There are also numerous discrepancies between the values provided by the website and the actual values given by the references.[3] Therefore, the information on this website cannot be trusted and no conclusions should be drawn from it.

Pretty embarrassing for race-realism, to be honest. Looks like the data from “P. Nobile” is from a Penthouse webforum that he ‘published’ his ‘results’ in, instead of a scholarly journal. Rushton did state ‘forum’ (not stating it was the ‘Penthouse forum’, though), but why should we take this as proof of anything?

The sadder one is multiple ‘references’ of Lynn’s that either don’t exist or don’t discuss penis size. Why trust these ‘sources’, when they aren’t controlled scientific studies?

Veale et al (2014) write:

It is not possible from the present meta-analysis to draw any conclusions about any differences in penile size across  different races. Lynn [31] suggest that penis length and girth are greatest in Negroids (sub-Saharan Africans), intermediate in Caucasoids (Europeans, South Asians and North African), and smallest in Mongoloids (East Asians), but this is baseupon studies that did not meet our present inclusion and

The greatest proportion of the participants in the present meta-analysis were Caucasoids. There was only one study of 320 men in Negroids and two studies of 445 men in Mongoloids. There are no indications of differences in

racial variability in our present study, e.g. the study from Nigeria was not a positive outlier. The question of racial variability can only be resolved by the measurements with large enough population being made by practitioners following the same method with other variables that mainfluence penis size (such as height) being kept constant. Future studies should also ensure they accurately report the race of their participants and conduct inter-rater reliability.

This meta-analysis was only done on Caucasians, but from the previous study on 320 Nigerians cited from Rushton (Ajmani et al, 1985) and 445 Mongoloids, no racial differences were found. When an actual study gets carried out on this, I doubt that there would be any differences between races.

Orakwe and Ebuh (2007) again test Nigerians (5.2 inches), then compare them with Italians (4.92 inches), Greeks (4.79 inches), Koreans (3.78), British (5.11 inches), and American Caucasians (4.9 inches). The only statistical difference was between Nigerians and Koreans. They conclude:

There is the possibility of racial differences in penile sizes, but there is no convincing scientific background to support the ascription of bigger penile dimensions to people of the Black race.

I wouldn’t say there is a ‘possibility’ that it’s true, based on a population of Nigerians. We can say that ethnic differences may exist between Koreans and Nigerians, but to extrapolate that to all five races and say that racial differences exist and that it fits neatly into Rushton’s outdated 3-way racial model is incorrect. It wasn’t a  representative sample of Nigerians (and I obviously don’t have access to the methods of the other papers, I will update this in the future when I find more data), and the bigger sample of the two samples that I cited showed a smaller size.


Do racial differences in penis size exist? We can’t really come to a conclusion based on the data we currently have. Using “””data””” from the Penthouse webforum and a self-reported survey online is embarrassing and the data shouldn’t be used in the discussion of whether these differences exist. The only reliable data on Africans, as far as I know, is on Nigerians; the study with the higher n showed a smaller length. The data so far, shows that no difference exist exists (Veale et al, 2014: 983).