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Science Magazine: “Taking race out of human genetics”
2500 words
I always love these. Refuting race-denialists has become sort of a past time for me. It’s interesting to see either the same things all the time (more likely), or something new, but still bullshit (Chanda Chisala’s attempt to put the cause for low IQ and intellectual achievement for blacks to redneck whites). But most of what is said by race-denialists and the egalitarian Left are easily refutable.
Taking race out of human genetics
In the wake of the sequencing of the human genome in the early 2000s, genome pioneers and social scientists alike called for an end to the use of race as a variable in genetic research. Unfortunately, by some measures, the use of race as a biological category has increased in the postgenomic age. Although inconsistent definition and use has been a chief problem with the race concept, it has historically been used as a taxonomic categorization based on common hereditary traits (such as skin color) to elucidate the relationship between our ancestry and our genes. We believe the use of biological concepts of race in human genetic research—so disputed and so mired in confusion—is problematic at best and harmful at worst. It is time for biologists to find a better way.
Race is a great variable in genetic research. Just because the HGP says human races differ by .1 percent of the genome doesn’t mean anything. The genetic distance between species isn’t what matters, what matters is how those genes that differ are EXPRESSED, and not how much genetic distance is between them. The use of race as a biological category has increased because it is a useful indicator of certain diseases and other things. Inconsistent definitions don’t mean anything as self-identified ancestry was correct 99.86 percent of the time in this study by Risch et al. Self-identified ancestry is good enough to show that what “has an inconsistent definition” has a basis in reality. Skin color is a good proxy for race, but not the only factor. What other better way is there?
Racial research has a long and controversial history. At the turn of the 20th century, sociologist and civil rights leader W. E. B. Du Bois was the first to synthesize natural and social scientific research to conclude that the concept of race was not a scientific category. Contrary to the then-dominant view, Du Bois maintained that health disparities between blacks and whites stemmed from social, not biological, inequality. Evolutionary geneticist Theodosius Dobzhansky, whose work helped reimagine the race concept in the 1930s at the outset of the evolutionary synthesis, wrestled with many of the same problems modern biologists face when studying human populations—for example, how to define and sample populations and genes (5). For much of his career, Dobzhansky brushed aside criticism of the race concept, arguing that the problem with race was not its scientific use, but its nonscientific misuse. Over time, he grew disillusioned, concerned that scientific study of human diversity had “floundered in confusion and misunderstanding”. His transformation from defender to detractor of the race concept in biology still resonates.
And these all don’t matter at all. Race does clearly exist in the biological sense.
Today, scientists continue to draw wildly different conclusions on the utility of the race concept in biological research. Some have argued that relevant genetic information can be seen at the racial level and that race is the best proxy we have for examining human genetic diversity.
Correct.
Others have concluded that race is neither a relevant nor accurate way to understand or map human genetic diversity
Incorrect. What is not relevant or accurate about seeing the genetic distances between populations that evolved separately for tens of thousands of years?
Several meetings and journal articles have called attention to a host of issues, which include (i) a proposed shift to “focus on racism (i.e., social relations) rather than race (i.e., supposed innate biologic predisposition) in the interpretation of racial/ethnic ‘effects’”; (ii) a failure of scientists to distinguish between self-identified racial categories and assigned or assumed racial categories ; and (iii) concern over “the haphazard use and reporting of racial/ethnic variables in genetic research” and a need to justify use of racial categories relative to the research questions asked and methods used. Several academic journals have taken up this last concern and, with mixed success, have issued guidelines for use of race in research they publish . Despite these concerns, there have been no systematic attempts to address these issues and the situation has worsened with the rise of large-scale genetic surveys that use race as a tool to stratify these data .
To see if there is a prevalence of certain disease in certain races/ethnicities seems pretty important to me. If it will better diagnose people and give faster care, that seems like a good thing to me.
It is important to distinguish ancestry from a taxonomic notion such as race. Ancestry is a process-based concept, a statement about an individual’s relationship to other individuals in their genealogical history; thus, it is a very personal understanding of one’s genomic heritage. Race, on the other hand, is a pattern-based concept that has led scientists and laypersons alike to draw conclusions about hierarchical organization of humans, which connect an individual to a larger preconceived geographically circumscribed or socially constructed group.
Seems to be the implication that there are no taxonomic differences between races. That’s funny. Hierarchal organizations of humans only exist really when you focus on certain traits, as all humans have strengths and weaknesses depending on the environment they evolved in.
Unlike earlier disagreements concerning race and biology, today’s discussions generally lack clear ideological and political antipodes of “racist” and “nonracist.” Most contemporary discussions about race among scientists concern examination of population-level differences between groups, with the goal of understanding human evolutionary history, characterizing the frequency of traits within and between populations, and using an individual’s self-identified ancestry to identify genetic risk factors of disease and to help determine the best course of medical treatments.
Population-level differences, race differences, whatever you want to call them, the effect is the same. Understanding human evolutionary history is understanding how and why races and ethnicities are so distinct from one another. As shown above in the Risch cite, self-identified ancestry is a good proxy to identify genetic risk factors to determine best medical treatments.
As a result, racial assumptions are not the biological guide-posts some believe them to be, as commonly defined racial groups are genetically heterogeneous and lack clear-cut genetic boundaries
They aren’t too heterogeneous. Ethnicities/races are homogenous enough to have enough as we evolved a level of genetic similarity to have us favor those more genetically similar to ourselves. Ah, the old ‘continuum fallacy’. Disregarded.
For example, hemoglobinopathies can be misdiagnosed because of the identification of sickle-cell as a “Black” disease and thalassemia as a “Mediterranean” disease
This is true. SCA isn’t just specifically an African disease. I covered SCA a bit on my disease, nutrition and parasitic load post. SCA comes up in populations in wet and warm climates. It’s from mosquitoes mostly. So those that live in those areas, for instance, Southern Italy, will be more susceptible to the disease. Though, these diseases are a pretty good proxy for racial identification.
Popular misinterpretations of the use of race in genetics also continue to fuel racist beliefs, so much so that, in 2014, a group of leading human population geneticists publicly refuted claims about the genetic basis of social differences between races.
A Troublesome Inheritance is a fine book.
Scientific journals and professional societies should encourage use of terms like “ancestry” or “population” to describe human groupings in genetic studies and should require authors to clearly define how they are using such variables. It is preferable to refer to geographic ancestry, culture, socioeconomic status, and language, among other variables, depending on the questions being addressed, to untangle the complicated relationship between humans, their evolutionary history, and their health. Some have shown that substituting such terms for race changes nothing if the underlying racial thinking stays the same.
The last sentence is right. you can call races ANYTHING you want. That doesn’t change the underlying reality of what is being spoken about. Call them red, blue and green. Call them any kind of weird name you can come up with, the underlying biological components do not change. This is what they don’t get. We can give you your definitions to certain words, but that doesn’t change the physiological/biological nature of HBD.
Having journals rationalize the use of classificatory terminology in studying human genetic diversity would force scientists to clarify their use and would allow researchers to understand and interpret data across studies. It would help avoid confusing, inconsistent, and contradictory usage of such terms.
Seeing as we have researchers like Risch et al doing the above, it’s clear that, no matter what you would like to call these clusters after DNA is sampled, that genetic variation among humans is 1) great and 2) extremely significant.
Phasing out racial terminology in biological sciences would send an important message to scientists and the public alike: Historical racial categories that are treated as natural and infused with notions of superiority and inferiority have no place in biology. We acknowledge that using race as a political or social category to study racism and its biological effects, although fraught with challenges, remains necessary. Such research is important to understand how structural inequities and discrimination produce health disparities in socioculturally defined groups.
It doesn’t matter!! Change the ‘historical racial categories’ if it will save your feelings, the fact that human biodiversity is still great among humans says otherwise. If you don’t want to group humanity into one of the things that make the most sense, it is you who’s being dishonest.
Biological effects between races are real and significant. I have covered them on this blog. How could ‘structural inequalities’ and ‘discrimination’ lead to health disparities?
The U.S. National Academies of Sciences, Engineering, and Medicine should convene a panel of experts from biological sciences, social sciences, and humanities to recommend ways for research into human biological diversity to move past the use of race as a tool for classification in both laboratory and clinical research. Such an effort would bring stakeholders together for a simple goal: to improve the scientific study of human difference and commonality. The committee would be charged with examining current and historical usage of the race concept and ways current and future technology may improve the study of human genetic diversity; thus, they could take up Dobzhansky’s challenge that “the problem that now faces the science of man [sic] is how to devise better methods for further observations that will give more meaningful results”. Regardless of where one stands on this issue, this is an opportunity to strengthen research by thinking more carefully about human genetic diversity.
The past and current identification of humans is clearly good enough for what we are discussing. Calling them anything else doesn’t matter and still doesn’t change anything in terms of this debate, whether it’s a Blank Slate argument, or anything like that, the repercussions of what is being discussed is still the same. The results we currently get are meaningful enough. Race clearly correlates with certain diseases and other mental and physical characteristics well enough to denote distinct racial categories.
Need I show what Sewall Wright, creator of the Fst (fixation index) has to say?
From my Race Is A Social Construct article:
Regardless of the method used in the analyses, all researchers reached estimated very close to that obtained by Lewontin: The differences observed by the subdivisions (populations, groups of populations, races) represented 10 to 15 percent of the total genetic variation found within the human species. Formally, these findings demonstrate, first, that the species is indeed subdivided into genetically definable groups of individuals and, second, that atleast some of these groups correspond to those defined by anthropologists as races on the basis of physical characters. They do not however, settle the arguments regarding the methods of racial classification. Unfortunately, Lewontin did not specify before initiating his analysis how large the difference has to be in order to call the groups “races”.
Consequently, the results of the studies have led population geneticists to two diametrically opposite conclusions. Lewontin called the observed differences trivial, and proclaimed that “racial classification is now seen to be of no genetic or taxonomic significance” so that “no justification can be offered for its continuance.” This view is echoed by authors of similar studies, who seem to be surprised that genetic variation within populations is greater than that between them. By contrast, Sewell Wright who can hardly be taken for a dilettante in questions of population genetics, has stated emphatically that if differences of this magnitude were observed in any other species, the groups they distinguish would be called subspecies.
One can extend Wright’s argument even further. The more than 200 species of haplochromine fishes in Lake Victoria differ from each other much less than the human races in their neural genes, although they are presumably distinguished by genes that control differences in their external appearances. The same can be said about atleast some of the currently recognized species of Darwin’s finches and other examples of recent adaptive radiations. In all these cases, reproductively isolated groups are impossible to tell apart by the methods used to measure differences in human races. Obviously, human races are not reproductively isolated (interracial marriages are common and the progenies of such marriages are fully fertile) but the external differences between them are comparable to cichlid fishes and Darwin’s finches. Under these circumstances, to claim that the genetic differences between the human races are trivial is a more political statement than a scientific argument. Trivial by what criterion? How much difference would Lewontin and those who side with him consider non-trivial?
By mixing science with politics, geneticists and anthropologists are committing the same infraction of which they are accusing other scientists, who they themselves label as racist. Even worse, by labelling the genetic differences as insignificant, they play into the hand of genuine racists who can demolish this claim and so further their own agenda. It is intellectually more honest to acknowledge and then point out that by no means imply supremacy of one race over others. This can be done by demonstrating that the differences are in genes that cannot be linked to any features that would be required for the preeminence of a particular race.
It’s clear that racial classification does exist. The creator of Fst, Sewall Wright, says that a Fst distance of .15 is more than enough for speciation (differing racial classifications). It directly refutes Lewontin, who put his political ideology of Marxism over science. Those cichlids in Lake Victoria are a perfect example though the definition of ‘species’ does change depending on which researcher you speak to, it doesn’t discount that there are real and physical genetic differences between races and ethnicities.
You can call race anything you’d like, the fact of the matter does not change that HBD is a real thing.
Refuting Agabond on Scientific Racism
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To set myself apart from other HBD bloggers, I decided to start a few series, one on Afrocentrism, HBD and Sports and refutations on how race isn’t a social construct. Agabond has a lot of untrue things to say, and I am here to refute them. Agabond, you can expect a lot of pingbacks from me on a lot of your posts, so get used to this.
Agabond wrote an article on the cons of Scientific Racism. The whole article is wrong.
According to race realism and the field of human biodiversity (HBD), the scientific racism of our day, the following are true:
- Race is genetic.
- Race affects intelligence.
- The races in order of intelligence are: Asians, Whites, Hispanics/Mixed, Blacks.
All of these are true.
Why this is wrong:
- Race is a social construct, created by the rules of society not by genetics.
My favorite saying from race-denialists. Yes, everything is a social construct. The Universe is a social construct, but that doesn’t mean that it doesn’t actually physically exist. We have social constructions for EVERYTHING, does that mean that NOTHING exists because they are social constructs?

The above picture is a PCA graph showing how the different races cluster on the graph. This is actually scientific evidence that what we call ‘race’ and ‘ethnicity’ has an actual biological basis. You say that “it’s created by the rules of society not genetics”, but you can clearly see here that GENETICS shows that humanity CLUSTERS into distinct groups.
2. The differences between people from different parts of the world are too new and too slight to account for differences in IQ.
Not true at all. They are not “too new”, Agabond. Due to genetic isolation between populations for thousands of years, i.e., no population migrations between people with different genomes, this caused evolution to happen faster.
Faster evolution means more ethnic differences. This is due to, as I alluded to before, no migration of others with differing genes. Due to us moving genetically AWAY from each other and not genetically CLOSER from each other, shows that evolution is speeding up in the OPPOSITE WAY.
How are they too new and too slight to account for differences in IQ? Selection pressures varied depending on where those populations evolved. The more cognitively demanding Northern part of Europe, as well as the cognitively demanding parts of Asia that East Asians evolved in led to cooperation, which in turn led to evolutionary selection pressures, ala altruism. They also had to be smart with their food in regards to rationing. Along with more variables, over tens of thousands of years of genetic isolation, populations evolved to have distinct physical, as well as biological differences due to those certain selection pressures in those environments.
The less cognitively demanding parts of the world, along the equator, doesn’t foster high intellectual capabilities due to the lifestyle of the peoples there. They lived a mostly hunter-gatherer lifestyle (which was extremely difficult in Eurasia during the Ice Age). Evolution dictated the traits that it would enhance with Africans, e.g., longer limbs for better throwing and to cover more ground while running to get food, darker skin due to the climate, as well as their hair.
There are evolutionary reasons for everything. You should have done a bit more research here before writing this.
Your intelligence is determined not by your race but by where you were born, when you were born, a bit from your parents’s genes and the rest from what you make of it.
Yes, it is determined by your race, but I’ll get back to that later. Depending on where you’re born? Sure. If you’re born in a place with high rates of disease, parasitic load and bad nutrition, this will retard IQ. But for those born in first world countries, their IQ is at their genetic limit due to better nutrition. You said “where you were born”, do you even realize that “where you were born” means that you’re giving credence to the hereditarian hypothesis?
When you were born? Is this an epigenetic argument?
A bit from your parents genes, well more than a bit. Funny you say that. We know that the mother’s IQ is the best predictor of the child’s IQ. That being said, in studies of racially mixed black and white children, those with white mothers have higher IQs than those with black mothers. So yes, it is your parents genes. I assume you were going for the “individual variation in IQ is greater than between the races” is what you were going for. Not going to work.
The rest is what you make of it. I agree here. Even those with high IQs won’t be automatically successful, personality variables have a lot to do with it, which I will cover in a future post.
1. Correlation does not equal causation
Just because blacks in America have a higher crime rate or a lower average IQ does not necessarily mean the cause is mainly genetic. To come to such a conclusion you would have to assume that racism is pretty much dead, that American society is just so gosh-darn fair to everyone that genetics is pretty much all that remains to account for the differences.
I can’t count how many times I’ve heard this since I jumped into the black-white IQ debate. Yes, we know that correlation does not equal causation, but with retesting what you tested before with the same variables and lab conditions, if you come to the same conclusions, it is not a correlation does not equal causation argument.
Higher crime rates and lower average IQs are inversely related. There is a negative correlation there. We can see here that the low IQ being correlated with crime argument holds up in studies.
Deborah Denno analyzed data from 987 African American school children in Philadelphia. Her data contained multiple measures of intelligence collected at ages four, seven, and thirteen as well as officially recorded criminal offenses. Chronic, violent offenders consistently had low IQ scores. For example, female chronic offenders were almost four times less likely to be in the top third of verbal-IQ test scores than female nonoffenders. Similarly, male violent offenders scored 10 to 17 percentile points lower on measures of vocabulary, reading, and language than nonoffenders.
In addition to finding a robust IQ-crime correlation, studies have turned up two other empirical regularities worth noting. The first regards two different types of IQ measures: performance IQ (PIQ) versus verbal IQ (VIQ). Performance IQ is measured with nonverbal tests of attention to detail, manual design construction, and visual puzzle solving. Verbal IQ is measured with tests of general factual knowledge, abstract reasoning, mental arithmetic, and vocabulary. Studies have consistently found that criminals have PIQ scores close to the general population but VIQ scores substantially lower. This PIQ > VIQ finding holds even when controlling for race, class, and reading ability (Moffitt), suggesting that verbal intelligence is a more important correlate of criminal behavior than other types of intelligence.
I also touched on black crime and correlates for crime here. No matter what you can say about this, low IQ is correlated with crime. There are countless studies on this. Juvenile criminals average 92, whereas adult offenders are at IQ 85, right at the black average in America.
Genetics does account for the difference though you seem dishonest enough to believe any other reasons for it.
2. Confirmation bias
Scientific racists notice the cases that prove their ideas while overlooking those that disprove them. But in science it is the exceptions that disprove the rule. That is why the law of gravity is still a part of science: no known exceptions!
Care to tell me what they overlooked that disproved them? Why did you not use any examples?
3. Lack of expertise
Look at the race realists and HBDers you hear about most:
- Steve Sailer, journalist/computer salesman.
His occupation says something about his knowledge on these subjects?
- J. Philippe Rushton, psychologist.
OK? What about him? He’s a Ph.D. in psychology. He cites what he says, obviously. Rushton is one of the most important psychologists of this generation. Have you ever read any of his books, or have you only read attack articles on him? He first began researching the study of altruism. This gives him credibility, in your eyes, to talk about racial differences, as well as HBD. Hell, altruism is how our complex societies formed. That is a part of HBD as well, but you seem to talk about things you don’t know.
- Francis Fukuyama, political economist.
First I’ve heard of him. Seems to get heat for his book arguing that we have almost reached the end of history. He says people have misunderstood his thesis, that “the French and American revolutions, and their underlying principles of liberty and equality, were the final resting point for human ideological evolution. So we need to consider whether Hegel, when he declared the end of history in 1806, was not right. My argument is concerned less with the world of real events and more with the world of ideas. Essentially the question I was trying to pose is whether there are any systematical ideological competitors left to modern liberalism.”
- Steve Hsu, astrophysicist.
I love Steve Hsu. It seems that, according to Agabond, you can’t have other interests outside of your field, and if you do have interests outside of your field and speak on them, that no one should listen to you. Things don’t work like that. Steve Hsu is actively looking for intelligence genes. He also shows the reality of human genetic variation.
Just because someone is interested in things outside of their field, does that mean they can’t become an authority on it?
- Richard Herrnstein, psychologist.
Same with Rushton. Are you telling me that psychologists can’t study IQ differences and behavioral differences between races? Hernnstein knows damn well what he’s talking about in terms of IQ. Are you trying to use Hernnstein and Murray as examples of people who talk about actual human genetics? Well, they don’t. They talk about IQ, and yea they talk about genetic causes for it, but they’re mostly concerned with the policy of the country that we are no noticing because we are ‘IQ blind’ so to speak.
- Charles Murray, political scientist.
Same as above. Murray has defended himself multiple times with attacks him and Hernnstein got on The Bell Curve. 22 years later, and the book is still not refuted.
- Arthur Jensen, psychology professor.
My personal favorite. He is THE AUTHORITY ON IQ as well as RACE DIFFERENCES IN IQ. Sure Rushton and Jensen bring up genetics sometimes, but they always cite where they got their information from.
Really bad arguments here.
You notice anything strange? No biologists or anthropologists, much less geneticists.
>some of the biggest names in HBD aren’t biologists, anthropologists (LOL), and geneticists
>means there are no HBD biologists, anthropologists (LOL) and geneticists
Nice fallacy.
Why in the world should we trust these people over biologists and anthropologists, the very people who study these things for a living? To leave no stone unturned, some biologists and anthropologists have even written books about race for the general public:
This is going to be good. Let’s see what “has been written for the general public by biologists and anthropologists”.
- “Guns, Germs, and Steel”, Jared Diamond, biologist
!!!! People still cite this?!?!??!?!?
I’ll say something on G,G&S then let Rushton take over:
So different levels of civilizations can be traced to environmental differences and not innate differences in races? Because physical environment can explain civilization differences does that mean all human brains are the same on average? Horrible strawman. No one says environment doesn’t matter.
We can look at 2 countries within Sub-Saharan Africa. Look at South Africa. Still one of the wealthiest countries in Africa. Economic freedom isn’t the only source of wealth, human capital and natural resources are important. The lack of proper resources for civilizations in the past isn’t why Africa is poor today since we can see actual African countries that are better of by simply having more economic freedom.
The poverty today in Congo isn’t dated back to the dawn of time. Diamond says New Guineans are probably smarter than white Europeans. So does he accept that all races are the same in the brain except New Guineans? So does he then accept that human brains can differ in environments? Jared Diamond’s work is irrelevant and does nothing to explain why the various races perform differently in Europe. You can say racism or lingering effects of oppression, but the reasons for Africa’s poverty is not relevant to the racial gaps in Europe and America.
If you think it’s caused by environmental poverty in the past, you still have to argue the facts on racial differences today, the evidence still exists.
Jared Diamond is a man who spent a lot of time in Papua New Guinea. I guess he grew to like the natives there and befriended some of them so he makes ridiculous leaps in logic to actually say they may be more intelligent than Europeans. Hilarious. Any intelligent person can see the ridiculousness of what he claims. I can’t even begin to think how, when faced with all of this evidence of differences, that you can possibly believe in some warped view of equality or egalitarianism.
Here is Rushton’s dismantling of G,G&S.
- “The Mismeasure of Man”, Stephen Jay Gould, biologist
Arthur Jensen refutation here.
- “Genes, Peoples, and Languages”, Luigi Luca Cavalli-Sforza, geneticist
What about it?
- “Man’s Most Dangerous Myth: The Fallacy of Human Race”, Ashley Montagu, anthropologist
Never heard of this book but from the Amazon review:
It presented a revolutionary theory for its time; breaking the link between genetics and culture, it argued that race is largely a social construction and not constitutive of significant biological differences between people.
Discarded.
These books are good to help you overcome the racist brainwashing of American society.
Let’s see. G,G,&S is trash. It has some cool tidbits of information, but it’s largely not true and is an egalitarians way of attempting to say that Europeans “got lucky” in regards to geography.
Mismeasure of Man is full of sidestepping and not addressing points, as well as wishful thinking mixed with lying about the truth and not telling his audience certain things. Sure, if anything, this book will make you MORE BRAINWASHED.
No idea why you cited Cavalli-Sforza.
A book that says race doesn’t exist. Wow, that’s a new one.
4. Conspiracy theories
The reason scientific racists give for trusting, say, Steve Sailer, a computer salesman, over Cavalli-Sforza, a professor of human genetics who has, like, studied race, is, wait for it, that people like Cavalli-Sforza secretly agree with them but are too afraid to say so in public! Have they gone mad?
What are you talking about? Are you talking about how Cvalli-Sforza uses terms like “population clusters”? That doesn’t matter. You can call the things what you’d like, but that doesn’t change the underlying biological reality of genetics, race and ethnicity.
If you’d have understood Cvalli-Sforza, you’d get that.
I know you got this pingback, Agabond. I’d love a response from you though I doubt it.
Towards a Theory of Everyone: Chanda Chisala Rebuttal on the Nature of the Black-White IQ Gap
3700 words
Chanda Chisala has been writing a series of articles for the Unz Review for almost a year now. They are on the nature of the black-white IQ gap. I’ve been eagerly awaiting his theory on the cause of the gap, as I always welcome any and all new information concerning this. Well, I was pretty underwhelmed by his theory.
Sowell has always used two arguments to cast doubt on the genetic hypothesis: the first one is the Flynn Effect or prior versions of it that he had noted himself, which shows that IQs have been rising with time for blacks and other people all over the world.
The “Flynn Effect” is rubbish. PumpkinPerson says:
It turned out Rushton was one of those “The Flynn effect is irrelevant” people. He found it prima facie absurd that we could have been a nation of mentally disabled people a century ago. It simply didn’t make any sense to him, given the outstanding achievements of early 20th century society. But it didn’t make any sense to me why the same tests that were culture reduced enough to measure the intelligence of South Africans could be so wrong when measuring Victorian intelligence. I needed an explanation. The Flynn effect is unrelated to g (general intelligence) and that was enough for him to just dismiss it and move on.
So even though Rushton and Jensen rebutted Flynn, as well as Flynn and Dickens, Chisala still chooses to use the Flynn Effect argument. Here is why it is irrelevant:
Let’s say Flynn is right. The average black now is as intelligent as the average white in 1945. That’s supposed to show that the race difference in IQ is environmentally caused because there hasn’t been that much genetic change in the white population and the IQ has allegedly gone up 15 points. So, you can have a 15 point difference created by just an environmental change, no one knows why. Some think better nutrition or malnourished brain, etc. That’s also a fallacy. Just because a change in one group over time is due to an environmental change, doesn’t mean, or even make it probable, that a difference between 2 groups at the same time is due to an environmental change. The Flynn Effect make’s that highly unlikely and here’s why.
The Flynn Effect, assuming it’s real, has been acting completely uniformly in every population. Any country you ask, the rate of increase is 3 per decade. That means it’s an environmental factor that affects whites and blacks the same way as well as the whole world. And as a result of this uniform environmental factor, you have a difference in IQ that’s being preserved. That would suggest that the response on the parts of blacks and whites is due to some non-environment factors, a genetic factor, which is making the difference in IQ remain constant as the Flynn Effect goes into effect.
What makes it even more unlikely, in the last 60 years, their environments have become very similar since segregation. These differences don’t exist now, they go to the same schools by court order, same TV shows, same movies, basically same environment for both, and yet, that increasing similarity in the environment, the Flynn Effect, the IQ gap has remained intact. Which means whatever counts for the gap is genetic and not environmental. The more and more similar the environment, the less and less of the difference can be due to the environment and the more and more it must be due to genes. So this 15 point gap surviving these changes in the environment, seems more and more likely to be genetic in origin.
So because this ‘Effect’ is the same across all populations and the gap didn’t close, that means it’s genetic. If the gap persisted even when IQs were rising 3 points per year, the B-W gap has still persisted, proving that it’s genetic.
That is why the Flynn Effect is irrelevant. This “Effect”, has been a slight upward trend in IQ, around 3 points per decade, which, in my opinion, has to do with the advent of better nutrition and an industrialized society. The rise in IQ started around 1880, almost perfectly coinciding with the industrial revolution in America. Along with a more industrialized society, it’s possible to give most citizens in the country good enough nutrition to where they are not iodine deficient (adding iodine to our salt boosted Americans IQs), as well as being deficient in zinc, iron, protein and certain B vitamins which the effects of not getting enough leads to the brain not growing to its full potential, which in turn leads to a lower IQ.
One more point on the Flynn Effect. The Flynn Effect does not occur on g, as it is not a Jensen Effect. Rushton defines Jensen Effect as follows:
Significant correlations occurring between g-factor loadings and other variables have been dubbed “The Jensen effect”.
…
Thus the secular increase in test scores (the “Lynn±Flynn effect”) is not a “Jensen effect” nor is this the first time the discriminating power of the Jensen effect has been shown.
The Flynn Effect is not on actual g. The black-white IQ gap is most heritable on those sub-tests that correlate highly with g. Through correlations on scores on inbreeding depression, Rushton and Jensen (2005) conclude that the magnitude of the black-white IQ gap is 80 percent genetic and 20 percent environmental.
Now to get to this other part of his theory.
The second very unique and original argument he has used is the differential IQ performance of black males and females, which seems to favor the females. He charges that the genetic hypothesis can not explain this, but it is explainable under an environmental hypothesis.
Sowell’s second argument is much stronger than the Flynn Effect argument because it is very difficult for hereditarians to explain why there should be a gender difference in African American IQ, especially one favoring females (let’s call this the “Sowell Effect,” to avoid repetition). This is very problematic for hereditarians, particularly since the trend is normally for male IQ to exceed female IQ, especially at the higher levels of the IQ distribution curve. We can see this unique trend among blacks even in the applications to medical school, a field that is considered a good metric for group intellectual comparisons.
This is very simply explained. Occam’s Razor anyone?
Even today in Africa, the women did the hunting and gathering, giving them more selective power. The same holds true for Eurasian men, who have a slight advantage in IQ over Eurasian women. Because of the colder climate in Eurasia, meat was one of the staples they had. So that shifted selection pressure from women over to men. Since men had the food, and the ability to hunt for it for that matter, men had more selection power to select the best possible mates. This led to Eurasian women being selected for beauty, whereas this led to African men being selected for physical attractiveness.
To quote from Erectus Walks Amongst Us:
In Africa, the women, even today, farm and gather food, so they have more selection power, but in the colder climates more of the food was meat, especially in the winter, and hunting was done by men, shifting some selection power to men. (Miller, 1994a). As a result of selection by men, Eurasian women have become more beautiful and, as a result of selection by women, Eurasian men have become workaholics and slightly more intelligent than Eurasian women (more intelligence = a better provider in Eurasia). African women have become slightly more intelligent than African men, however, who have become the more physically attractive sex.
So more intelligence led to a better provider. Being able to farm for and or hunt for food gave those who did it the selection ability to be able to sexually select to their liking.
Sowell (2013) claims this empirical victory in Intellectuals and Race (page 79):
Further evidence that the male-female difference in IQs among blacks is cultural is that black orphans raised by white families show no such female superiority in IQs, in addition to both sexes having higher average IQs than other black children.
Chisala says the Sandra Scarr data from the Minnesota Study does not back up this claim.
There are other studies that could possibly back Sowell up if he is right and we should check those too. For example, there is the well-known Eyferth Study in Germany which monitored the IQs of illegitimate children of black and white American soldiers who were stationed there at the end of the Second World War.
Wikipedia got its data from The g Factor, a book by Arthur Jensen (1998) that is probably the most cited in the racial intelligence debate. I went to the cited page and indeed found that Wikipedia had correctly reported Jensen’s data. The Sowell Effect had apparently disappeared among the black children born in Germany and the strong culture hypothesis seemed to be vindicated.
Arthur Jensen explains the cause for the mixed race children (and at the same time the cause for black female children having a higher IQ) on pp 483 of The g Factor: The Science of Mental Ability:
Finally, heterosis (the outbreeding effect; see Chapter 7, p. 196) probably enhanced the IQ level of the interracial children, thereby diminishing the IQ difference between the interracial children and the white children born to German women. A heterotic effect equivalent to about + 4 IQ points was reported for European-Asian interracial offspring in Hawaii.
This means that we can also resolve the debate about whether the black soldiers in this experiment were more selected than the white soldiers. It appears that the hereditarians were probably right on this point: the black soldiers had to have been significantly more intelligent than the white soldiers because the presence of a Sowell Effect indicates that the IQ of the black children has received extra depression (through an abnormal lowering of the male IQ, as usual.) However, it’s another Pyrrhic victory for hereditarians: the continued existence of apparent extra depression for black male IQ makes their simple models impotent, just as it does for standard environmentalist models.
Yet another point that Rushton and Jensen shoot down in their magnum opus paper:
Second, 20% to 25% of the “Black” fathers were not African Americans but French North Africans (i.e., largely Caucasian or “Whites” as we have defined the terms here). Third, there was rigorous selection based on IQ score in the U.S. Army at the time, with a rejection rate for Blacks on the preinduction Army General Classification Test of about 30%, compared with 3% for Whites (see Davenport, 1946, Tables I and III).
Huge error. About one-quarter of the ‘black fathers’ were French North Africans! Because North Africans have a higher genetic potential for IQ (nowhere near that of SSA), this is not a true representation of black fathers and white mothers.
Thus, racial hereditarians can not explain why the race of the mother matters
Is he being serious right now? It’s easily explained. We know that the mother’s IQ is the most important predictor of the child’s IQ. The prenatal environment is better in the white mother than in the black mother. Due to the mother’s IQ being the most important predictor of a child’s IQ, doesn’t that end the black-white IQ debate right there? Due to the fact that mixed race black and white children with white mothers show higher IQs than those with a black mother and white father, doesn’t that end the black-white IQ debate?
We racial hereditarians can definitely explain why the race of the mother matters. You should have done a bit more research into this matter.
And now on to my favorite part of this article. It’s so out there in its propensity for being a possibility for the cause of this gap between the races.
In Black Rednecks and White Liberals, Sowell (2006) theorizes that the modern ghetto culture of black Americans came from their association with white rednecks during the time of slavery and he believes it is the preservation of this detrimental culture – preserved with the intellectual help of “white liberals” – that keeps the black IQ low due to its anti-educational, anti-intellectual disposition. Sowell convincingly demonstrates some very uncanny similarities between ghetto black culture today and some aspects of white redneck culture that was more dominant in the South in the past than it is today, as more and more whites have decided to abandon it.
One huge problem with this. If that’s the case, if the cause of lower intellectual achievement is due to black Americans association with white rednecks during the time of slavery, all we need to do is look at Africa to see how they did without the “association with white rednecks during the time of slavery”! We can also look at those countries never touched by colonialism to see that they’re the same backwards countries.
Of course there will be “uncanny similarities”. When you have two groups who have lived amongst each other for a certain period of time, traits of both groups will rub off on each other. This is not a genetic cause, but an environmental cause. The similarities come down to being around other groups.
And here it is, here is the kicker:
Although I agree that the case for a cultural transfer from some groups of Southern whites is very strong, I think it is more likely that this “culture” was actually passed to blacks genetically rather than through mere influence and imitation. If that is the case, then it was in fact the presence of relatively strong mutations in that sub-population of whites that was affecting the stranger aspects of their behavior and intelligence, and they passed on the same genetic condition to blacks through mating with the black women.
THIS is his big reveal? No. Way. This has to be one of the funniest things I’ve heard in the black-white IQ debate. Hey, Chanda, there is something called Regression to the Mean (nice post, Jayman), which throws your theory out of the water.
blacks in fact had more stable families and even had less out-of-wedlock children than whites. He uses this to show that if slavery was the root of these problems, they could have started much earlier.
Wrong.
In this paper by Steven Ruggles, he says that analysis confirms that the high incidence of black Americans of single parenthood and children residing without their parents is not a recent phenomenon. Data shows that from 1880 through 1960, black children were two to three times more likely to reside without one or both children than white parents. This directly goes in the face of what liberals say is the cause of the demise of the black family structure. Ever since blacks have been free from slavery has this begun to happen.
What explains this perfectly, is Rushton’s r-K Selection Theory (now known as Life History Theory). Those who are more r selected (Africans), will have more children but spend less energy caring for them. Conversely on the other side, those more K-selected (Orientals and whites in the middle of K and r), will have fewer children but show more attention to them.
Some of Sowell’s strongest critics on this theory also suffer from the same progressional problem. Scholar and investigative journalist, Steve Sailer, for example,argued that much of the negative behavioral tendencies in black ghetto culture must have come with them from Africa. His theory is also unlikely to be true if the statistics about marriage and out-of-wedlock births etc are true. If their culture came with them from Africa they would not have had a long period where that culture seems to have been almost absent only to forcefully show up much later, in generations that had the least connection to or memory of Africa.
Sailer is correct. See my above cite showing that from 1880 through 1960 black children were two to three times more likely to reside with one or both children than white parents.
So we can see that it’s not a recent phenomenon.
Our theory thus explains a paradox that is difficult to explain by present environmental or hereditarian models: when blacks from Africa, the Caribbean and the US are compared, it is the least white-admixed black group that apparently performs best (the Africans), followed by Caribbean blacks who are in between; the most white-admixed group, the native black Americans, do worst. And yet within these communities, it is not necessarily true that the more white-admixed individuals perform worse; they may actually be over-represented on the highest levels of academic or social performance.
Dr. James Thompson says the sample for the Caribbean blacks in the UK is not a representative sample. Also, the hereditarian theory does not say that ALL Africans and African-descended peoples have a lower average IQ. It’s perfectly within the hereditarian hypothesis to have some African countries, as well as peoples, descended from African countries around the world, show a genetically higher IQ.
The evidence of such deleterious mutations still existing among modern day poor whites can be seen, not just from their low intellectual performance (going even lower than poor Caribbean boys), but even from their violent reactions against their fellow well-performing students, a culture that is also seen among ghetto black Americans, which is further evidence of a mutational rather than an imitational cause.
Wow, you mean to tell me that American whites aren’t a monolith and that there are some white groups in America with a lower average IQ? News to me!!
This solves one of the stronger challenges raised against the Unzian Asian Exception conjecture, asking why it was not East Asians who produced the greatest epochs of human intellectual achievements in history if it is true that their average IQs have consistently been stubbornly high for most of modern human history. It would be because the same canalization that protected them from low intelligence also “protected” them from producing the numbers of super-creative intellects that would be required for such revolutionary achievements in a concentrated period of time. They have a small creative smart fraction, in short.
The cause for lack of East Asian creativity is due to conforming in East Asian societies, which Rushton says in Race, Evolution and Behavior that it’s a genetic trait. Rushton did say that a larger average brain size means more creativity and that with social restrictions lifted, that East Asians may possibly become more creative than whites.
From time to time Lynn notes anomalies in his theory that require explanations. One of these is that Europeans made most of the great intellectual discoveries, while the East Asians, despite having a higher IQ, made relatively few—a paradox extensively documented by Charles Murray in his 2003 book, Human Accomplishment. Lynn proposes an explanation for this: it may be that East Asians are more conformist than Europeans and this inhibits creative achievement. (In Race, Evolution, and Behavior, I presented evidence that this personality trait has genetic roots.)
Winters Are Good For Your Genes: Lynn Book Finds World Average IQ 90, Declining From North To South
And yet the same hereditarians admit the conspicuous paucity of highly significant originators and innovators among East Asians, despite showing over-representation in high intellectual aptitude, sometimes very precociously so. East Asian women, who have the highest canalization coming from gender and race, are the most exemplary of this contrast. The shortage of such super-creative phenotypes can not be because they lack the numbers of people with the right genotype, but because the genotype is “buffered” from phenotypic expression by canalization.
See above.
Ashkenazi Jews, on the other hand, may be the most over-represented at the top of creative achievements in different intellectual fields (from chess to physics to literature, etc) simply because they happen to also be quite lowly canalized.
No. No way. Ashkenazi Jews are over-represented at the top of creative achievements in different intellectual fields because they mated with Roman women thousands of years ago. I have already noted about the mother being the best predictor of child’s intelligence. That’s the cause for high Ashkenazi IQ, not canalization.
Lower canalization also means that their improvement will be more rapid when such environmental conditions positively change (as can also be seen among recent black African immigrants, whose radical improvements begin even in children who were born under bad conditions in Africa, thus defying all kinds of hereditarian limitations.)
This is a case of super-selection. Only the most intelligent peoples leaving the country to immigrate.
In short, there is basically false assortative mating among black elites on average. This also explains why the mixed black male children have lower IQ when their mother is black than when their mother is white, as we demonstrated above.
I went over this earlier. Black mothers have a worse prenatal environment than do white mothers.
This obviously would not mean that the usual theories of environmentalists are correct either, since it should also not make a difference to them if the boys are included or excluded from the black American samples, especially in elite families. However, as we have faithfully acknowledged, both environmentalists and hereditarians also have some empirically confirmed arguments. Our present hypothesis, taking account of differential gender and racial canalization in human populations, can hopefully help to unify the valid aspects of the environmental and hereditarian frameworks.
I’ve noticed that Chisala used a hybrid environmentalist-hereditarian position to explain his theory on the black-white IQ gap.
I refuted the “Flynn Effect”, as well as the part of the Eyferth Study that talks about higher black female IQ, refuted the section about Caribbean blacks in the UK, and finally, I refuted his claim that we hereditarians “have no explanation for a mother’s IQ being the best predictor of the child’s IQ”.
In conclusion, this is just an extremely long-winded way of saying “whites are the cause of low black achievement, crime, IQ and anything else negative that affects blacks in Western countries”.
If that’s the case, Mr. Chisala, why is Africa so backwards?
HBD and Sports: Football
2000 words
In my first post in this series, I talked about HBD and Baseball. With the Super Bowl being tomorrow, I figured I’d talk about HBD and how it fits in to football.
According to TIDES (The Institute for Diversity and Ethics in Sports), in 2014, the racial mix of the NFL was 68.7 percent black, 28.6 percent white, “Latinos” at .7 percent, Asian at 1.1 percent and Pacific Islanders at .9 percent. International players made up the last 1 percent.
That’s basically the reverse ethnicity for baseball which is 75 percent white, 23 percent black and 2 percent Asian in 2014. As you see, when we start talking about sports with more athleticism involved, the number of blacks increases. With a more timing-based sport, such as baseball, they will be a lower portion of the racial mix in baseball, seeing as timing based (reaction time) sports are geared more towards people with high IQs, seeing as there is a high correlation between IQ and reaction time.
Blacks are over-represented in the NFL due to evolutionary selection pressures in the sub-Saharan desert. Africans have longer limbs, can sprint for longer distances and have higher stamina. This works out with what they had to do in Sub-Saharan Africa. They had to chase food, chuck spears etc.
Those longer limbs help them get them more space from defenders, allowing them to catch the ball at the highest point. Evolution also gave an advantage for jumping as well.

The above chart gives a racial breakdown of positions by race in the NFL, and what I will be referencing for the rest of this article.
The Center position (the one who snaps the ball to the QB) is majority white. That position requires the use of type I muscle fibers. Those fibers, which use oxygen to fire, which takes longer to get going, can go for a longer period of time before tiring out. The force per contraction of the muscle is spread out over time. So because those fibers take longer to get going, the white Center has a physiological advantage, on average, over black Centers. Their muscle fiber typing helps them
Conversely, for the speed positions (RB, WR, somewhat CB), those fibers fire without oxygen (so they fire anaerobically), they fire extremely quickly, and also tire out just as fast as they fire off. Because the muscle generates so much force in such a small period of time, this is better for those positions that take near pure speed, agility and quickness (there is a difference). Agility is defined as the ability to be quick and graceful. Speed is defined as the rate at which someone or something is able to move or operate. Quickness is defined as a high rate of movement or performance.
This is why CB is majority black. It takes an extreme amount of athleticism to be able to do what CBs do. They need to be quick off the snap, stay with the WR and then be able to react quickly for an interception or a tackle.
DE is another position that is majority black. That position is used to pressure the QB into throwing the ball before he’s ready. DEs also have longer limbs, which is helpful when attempting to maneuver around the Tackle to get to the QB. This is another position where those fast twitch muscle fibers come in handy.
DTs need to be at least 260 pounds. They need to be quick off the ball and have good vision to see above and around the Offensive Tackles. This is where the height advantage of blacks is again useful. Their longer limbs also has theme excel at this position.
Fullbacks are equally white and black. You need to be a big body to be able to block, you don’t need to be too quick, but be able to exert force for a small amount of time so you can block the defender who’s attempting to tackle the RB or another player who has the ball.
Guards, again are equally black and white. The same intangibles that hold for Centers also hold true for guards. All players on the offensive line are pretty damn big, long limbs again is a positive trait for those positions, with blacks obviously having the advantage there. But as I noted with Centers, they need to be able to hold a block, so the slow twitch muscle fibers of the white players help them there. The type II fibers of blacks also help on the offensive line, as their muscle fibers allow them to exert more force in a shorter amount of time, pushing back the defender so the one with the ball has a lane to get through.
Kickers are all white. This makes sense. Again, as this whole post is basically about, this comes down to differing amounts of type I muscle fibers between the races. Punters are needed to kick a ball, at times over 55 yards. This is where those slow twitch muscle fibers come in handy, and the reason why there are no black kickers.
Linebackers are majority black. They are called “the QBs of the defense”. You can see this where the MLB (the one who is the “QB” of the defense) directing calls and sometimes calling audibles. It doesn’t give a breakdown of Middle and Outside linebackers by race, but I’d assume that more MLBs are white, and more OLBs are black. The OLBs use their speed and agility to attempt a sack on the QB while the MLB needs to know where the ball is at all times.
Long snappers are all white. The long snapper is a Center who snaps the ball more than 15 yards during punts and 7 to 8 yards during field goals and extra point attempts.
The NT position is all black. A NT is the middle man on the defensive line in a 3-4 defense (3 down linemen, 4 linebackers as opposed to a 4-3 defense with 4 down linemen and 3 linebackers). The position is called “nose tackle” because the linemen lines up over the nose of the ball. The NT is usually the biggest player on the defense and pushes through the line to get to the QB to put pressure on him.
The same things that apply to punters, also apply to kickers.
Just what I was waiting for. QB. 65 white QBs compared to 14 black QBs. Why? Because the QB is, on average, the most intelligent player in the offense. He needs to remember calls, plays, audibles, needs to be calm under pressure and be analytical in where the ball is placed when there is a defender right on top of his receiver. Most all of the good QBs in the NFL are white. Those QBs in the NFL who are black, mostly are running QBs. That gets one dimensional over time, and the defense can better cue in to what the QB is doing so it gets stopped more often. The QB position is a proxy, IMO, for racial differences in intelligence because intelligence is a pretty big factor in regards to the QB position. To be able to think slightly into the future on where you need to throw the ball so your receiver can get to the ball away from the defender, to changing plays when you see a defensive alignment that doesn’t look right with the offensive alignment you already have called, the QB needs to be a highly intelligent person. Most black QBs are only liked because they make the game more exciting by 1) keeping the defense on their toes and 2) they break off huge runs when all players are covered and there is no one to tackle the QB.
Running backs are majority black. They need agility, quickness and speed to be able to shake defenders. Long limbs also help with covering more ground per step. This is why those players, such as Chris Johnson, had a 40 time of 4.24 seconds. Those with the fast twitch fibers are West African descended while those blacks with slow twitch fibers are East African descended. Most power backs are white in the NFL, which again comes back to slow twitch muscle fibers. They are usually stronger than those speed backs.
Safetys are majority black. As is the case with CB, safetys need the athleticism, long limbs, speed, quickness, agility and vision to be able to anticipate where the ball is, and then they need to be able to have the speed and agility to get to where the ball is on time, before a member of the opposing team has a chance to get to the ball.
Tackles, again are basically even, the same things apply for Guards and Centers.
Tight end is another position that’s about even. You need a bigger body to be able to position yourself better than the defender that’s covering you. Along with that bigger body comes longer limbs to be able to catch the ball at its highest point so the defender can’t tip or intercept the ball. You have some tightends, such as Vernon Davis, who are tall and have good speed. But he’s a rarity in tight ends in the NFL. The tightend is usually another blocker for the QB, so he needs the slow twitch muscle fibers. As I have alluded to above, the slow twitch muscle fibers have tightends able to hold blocks for a longer amount of time.
Finally, wide receiver is majority black. The same things as the other skill positions (RB, TE, QB), you need to have a big body to catch the ball at its highest point. They also need speed, quickness and agility to be able to get separation from the defender. You have some wide receivers who are short, but extremely fast like Washington Redskins receiver Desean Jackson, with a 4.35 40 time.
In this study by Wagner and Heyward, they note that biological differences exist between blacks and whites. They reviewed the literature on the differences between blacks and whites in fat free body mass (water, mineral and protein) fat patterning and body dimensions and proportions. Blacks, in general, have greater bone mineral density and body protein content than do whites, resulting in lower fat-free bone density. They also note racial differences in the differences of subcutaneous body fat, which is the body fat that’s just below the skin, as opposed to visceral body fat which is found in the peritoneal cavity, which can be measured with calipers to give a rough estimate of total body fat adiposity. The conclusion reached in the study was that differences in FFB (fat free body) was statistically significant between blacks and whites. They also have a greater BMC (bone mineral content) and BMD (bone mineral density) than do whites. They also argue that for a given BMI (body mass index), blacks might have less adiposity because they tend to be more mesomorphic. Researchers push for the development of racial-specific equations to better see differences in FFB.
The two races also differ in the width of hips, which less wide hips are better for more speed production in comparison to whites who have wider hips on average.
Differences in body type (somatype) are also linked to race. As I noted above, blacks skew more towards mesomorphy. Whites do as well, but it’s more prevalent in blacks. Endomorphs skew more towards Asians and whites. Ectomorphs skew more towards Asian populations.
In conclusion, football proves HBD right as well. The racial mix of differing positions, as well as strengths and weaknesses with them, show the reasons for certain races performing better than other races in certain positions. Innate physiological differences in blacks and whites show why there are racial disparities in all sports. Because of lower average fat mass in blacks, this allows for more speed because there is less body fat to weigh them down, on average. Somatypes also, roughly correlate with race.
We know and accept physical differences between the races that lead to over or underrepresentation in sports, but once someone brings up intelligence differences, you get shunned. So we all have the ability to be as intelligent as any other individual? Blacks have this innate advantage to be good at sports, but whites and Asians don’t get to say they are more intelligent on average?
Football is one of the 4 major sports in America that proves HBD.
How Did Man Evolve to Eat?
1300 words
How did we evolve to eat? I’ve been through Paleo Diet two times, and briefly touched on intermittent fasting in the second Paleo refutation. That is how man, no matter where he evolved in the world, ate. We didn’t know when we would get our next meal, therefore, evolutionary mechanisms evolved in us to have intermittent fasting be beneficial to us.
Intermittent fasting is halting the consumption of food for at least 14 hours for women and 16 hours for men. We evolved gathering and eating our food intermittently. Ability to function at a high level, physically and mentally, during extended periods without food, were crucial to human evolution. Your body performs best in a fasted state.
While you’re in a fasted state, the amount of catecholamines (adrenaline and noradrenaline) increase. That is your fight or flight mechanism. Catecholamines also increase fat burning, binding to fat cells putting them out of the cell to be burned off.
With increased catecholamine production from being in a fasted state which burns off body fat, you are also more alert as well. This is another evolutionary advantage. When Man was hunting for food he was in a fasted state, as he didn’t have access to food like we do today. Due to being in a fasted state with increased catecholamine production, this was a great advantage to being better prepared to be ready in case of a surprise attack by a predator or to always be on the ready to attack prey when seen. The scientific literature, though new and growing, supports this thesis with the amount of catecholamine production increased 24 hours after a fast.
Intermittent fasting leads to a dramatic increase in neural autophagy. Autophagy deals with the destruction of cells in the body, controlled digestion of damaged organelles in the cell. The conclusion of the study is, fasting is a simple, easy way to increase neural autophagy. It’s good for keeping the brain healthy, by destroying bad cells, letting the body rejuvenate them.
In conjunction with the evolutionary advantages of intermittent fasting, along with Genetic Similarity Theory, which has those care for others more because they share more alleles in common, and are therefore more closely related, the two evolved hand in hand to better make sure humans survive extended periods of time without food.
Which brings me to ‘starvation mode’. When people bring up starvation mode, they completely misrepresent the Minnesota Starvation Experiment. When they say that metabolic slowdown occurs when food is not consumed for a certain amount of time, they are correct, but they’re only half right. It’s a process called adaptive thermogenesis. Adaptive thermogenesis is the regulated production of heat in response to environmental changes in temperature and diet, which lead to metabolic inefficiency. When they talk about ‘starvation mode’, they talk about not having consumed food within the past, say 2 or 3 hours. That is a completely ignorant statement, as there are no deleterious effects of no food consumption until around 24 hours of fasting. ‘Metabolic slow down’ is not significant enough to prevent weight loss.
Now you may be thinking “What about breakfast, isn’t that the most important meal of the day?” Yes, “breakfast” is the most important meal of the day, but it doesn’t need to be had in the morning, immediately upon waking. The average person sleeps for about 8 hours, during that time, the body enters the fasted state. The “breakfast is the most important meal of the day myth” was originally pushed by Kellog’s in the 50s. The problem with any of these studies in regards to weight loss and or any other conclusions is that they are observational studies. The actual cause cannot be quantified. The slogan was created to obviously give Kellog’s more business, which was based on observational studies. The main hormone behind post-breakfast hunger is cortisol. The term ‘breakfast’ means “break your fast”, therefor “breakfast” can be had anytime AFTER your body goes into the fasted state.
Cortisol is secreted in response to a stressor, in order to help you cope with that stressor efficiently. Exercise (hunting for our ancestors), disrupts homeostasis because of the stressors that are put on the body. The stressors then require an adaptive response, which is cortisol. Most anything our ancestors did disrupted homeostasis, causing cortisol to be secreted. Because of increased cortisol levels during times of need, you can push through certain things than if you didn’t have that cortisol increase due to the stressor that made your body secrete the extra cortisol.
The stressors that our ancestors had to survive in the past, though, had a clear-cut line in beginning and end. Therefore, the fight or flight mechanism (catecholamine production) was easily secreted to elicit the needed response in order to survive, get food, and ultimately what evolution is about, making sure your shared genes pass on to the next generation. All of these responses in regards to intermittent fasting increase Man’s success on the planet, as well as evolutionary fitness. The strong selection pressures then select for those traits which are more advantageous, which pass down through the generations, getting better or becoming obsolete through non-use, e.g. migrating to a new area where those selection pressures that had certain traits arise weren’t in the new area.
To talk about another hormone, there is a hormone called ghrelin, which is secreted by the stomach in anticipation for a meal. It decreases after meal consumption and also stimulates the release of the growth hormone.
An evolutionary advantage for ghrelin is that it let Man know when the last time he ate was, as our bodies are pretty much like clocks and tell us certain things when it needs them, e.g. releasing ghrelin when it needs nutrients and sustenance, so they can then go and hunt for food, ensuring that their genetic lineage survives.
The drive for food, the drive to make sure genes pass on to the next generation, intermittent fasting and evolution of man, all intertwine with each other to tell the story of how we got to where we are today. There are way more health benefits to restricting periods of being in a fed state, and the evolution of those adaptive processes in our bodies from thousands of years of eating intermittently which our bodies had to evolve the traits that had us succeed in order to pass our genes on to the next generation. Everything we do in life is, at the most basic level, driven by our biology and the release of certain chemicals/hormones that make us seek out or want certain things.
In summary, the release of catecholamines in response to lack of food after a certain amount of time is one such example that shows that those evolutionary processes evolved to better protect us from extended periods of time without food, as well as giving us great benefits due to how our bodies evolved in response to those adaptations. This also leads to increased altruism for those with close genetic similarity, e.g. more alleles in common than with other peoples. The increased catecholamine production leads one to be more alert of their surroundings, which is an evolutionary advantage due to the release of ghrelin making man hungry, which in turn led to searching for food. That then led to an increase in the catecholamines to increase those adrenaline hormones to make man better prepared for any attack by a predator, and to be ready for any perspective prey he saw. Intermittent fasting, including all hormonal advantages involved with it, evolved closely together with altruism for one’s own people. This led to an increase in genetic fitness, as well as having a better chance to pass your genes on to the next generation.
Black Crime, Black Physical Attractiveness and Testosterone
I touched on black crime a bit a few months ago. I will now go more in depth on reasons for black crime.
First, what is the caused of all crime, no matter the race?
People have always said that parental socialization was the cause of black crime. But, genetic confounding was never considered in those studies. Kevin Beaver et al found in an analysis of adoptees that once genetic confounds were properly controlled, the effects of parenting on criminal justice measures were nonsignificant. That’s weird. I thought parental socialization was the cause of a kid turning out bad or good.
What is the cause of rape?
In a 37 year nationwide study in Sweden, it was found that sex offending runs in families. They reported strong evidence of familial clustering of sex offenses, mostly accounted for by genes and NOT shared environmental influences (ie, the same thing happening to both people in the family, therefore being the environmental cause of rape).
It’s of course genetics. We know that there is huge variation between individuals of the same race/ethnicity, how about between races? It also follows Rushton’s Rule of Three, Asians on top, whites intermediate and blacks on the bottom.
There are testosterone differences between blacks and whites. On top of those testosterone differences, men who stay and raise children have, on average, lower testosterone. This was noted in the West, but non-Western samples gave differing numbers. The study was conducted in two East African populations from Tanzania, Hadza foragers and Datoga pastoralists. They predicted that high levels of paternal care by the Hadza fathers would be associated with lower testosterone and that no difference would be found in non-fathers and fathers of Datoga men, who provide direct paternal care. The measurements in both populations confirmed the hypothesis, as well as adding further support that decreased testosterone leads to more paternal care.
What does that have to do with crime? Not having a father is detrimental to a young child, studies have shown in the UK that those from broken homes are more likely to commit crime than those from stable families. Also, seven out of ten offenders come from broken homes.
In this paper by Steven Ruggles, he says that analysis confirms that the high incidence of black Americans of single parenthood and children residing without their parents is not a recent phenomenon. Data shows that from 1880 through 1960, black children were two to three times more likely to reside without one or both children than white parents. This directly goes in the face of what liberals say is the cause of the demise of the black family structure. Ever since blacks have been free from slavery has this begun to happen.
Why did black father rates jump so much after the Civil Rights Movement?
I have my own theories.
Today, the black single mother rate is at 72 percent. In 1965, 24 percent of black infants were born without a father. What is the cause for that?
Blacks weren’t allowed to marry whites, and the CRA changed that. So with them being legally allowed to court white women, they obviously shied away from attempting to court black women. We can see that the trends show huge changes in 50 years. The cause is desegregation obviously, but what exactly is that environmental cause? Being able to court white women.
So with those men who DO stay around to raise children who show a decrease in testosterone, it’s extremely safe to say that those men who leave, regardless of race, have higher testosterone than those men who stay. This is reflected in racial single parent stats that show, again as for many more variables, Rushton’s Rule of Three.
What is wrong with black women? How come when segregation got lifted, black single motherhood rose exponentially?
Satoshi Kanazawa knows why. He noted in this study on why black women are not as attractive as other races, that women, on average are more physically attractive than men. EXCEPT BLACK WOMEN. The data shows black women as not being statistically different from the average, and far less attractive than whites, Asians and Native American women.
The cause is higher testosterone. Kanazawa says that the cause for this is due to the mean BMI of black women being 28.5 among black women and 26.1 among non-black women. Though he says that’s not the reason why black women are less physically attractive. Black women have lower levels of intellect net of BMI. Even the racial differences in intelligence don’t account for these differences. Black women are STILL less physically attractive, net of BMI and intelligence. Kanazawa says: “Net of intelligence, black men are significantly more physically attractive than nonblack men.”
Why is that? He says the reason is testosterone. He ends up concluding that testosterone differences are the only cause for these differences. With blacks having the highest testosterone out of all the races, black women obviously have higher rates of testosterone than white women. Those women with higher levels of testosterone have more masculine features, therefore making them less attractive.
Previous research has suggested that perceived attractiveness and personality are affected by the race such that White faces are more attractive but less masculine than Black faces. Such studies, however, have been based on very small stimulus sets. The current study investigated perceived attractiveness and personality for 600 Black, White and mixed-race faces. Many of the investigated personality traits were correlated with race when rated by White participants. Attractiveness specifically was greater for Black male faces than White male faces and among mixed-race faces. Blackness correlated with increased attractiveness. A reverse pattern was found for female faces with Whiteness being associated with attractiveness. The results are discussed in terms of the sexual dimorphism demonstrated in skin color. (Lewis 2010)
That’s the same as was noted in the Kanazawa paper. Darker-skinned males were more attractive, lighter-skinned women were more attractive.
Rushton wrote, in his last paper before his death, that skin color as a multi-generational adaptation to differences in climate over the past 70,000 years. He proposed life history theory (formerly known as r K selection theory) to explain the covariation found between human and non-human pigmentation and variables such as birth rate, infant mortality, longevity, the rate of HIV/AIDs and violent crime.
Dark skin is correlated with these things because of evolution.
High testosterone is linked to crimes of violence and sex. Men who gave saliva samples in prison were shown to violate more rules in prison, especially rules involving overt confrontation. Dabbs et al say: “the variety of rule violations suggests the behavior of high testosterone individuals reflects intractability, unmanageability, and lack of docility as well as aggression and violence.” Higher testosterone levels were associated with more violent crimes, parole board decisions against release, and more prison rule violations. This same thing was noticed in women, high-testosterone women had more crimes of unprovoked violence, increased number of prior charges, and decisions against parole.
For chronic adult offenders, the average IQ is 85, 1 SD below the mean of 100. Why do criminals have low IQ? Lack of abstract thought for one. Those with low IQ have a lack of ability to think of what their actions will do. Along with the lack of ability to delay gratification, paired with high testosterone and low IQ, that is the cause for higher than average crime in the black community in America.
Men with lower intelligence may be more likely to resort to evolutionarily familiar means of competition for resources and mating opportunities, and not to fully comprehend the consequences of their criminal behavior.
Even just a few points of IQ matter to criminality. There is a lot of data on the link between crime and low IQ. Who just so happens to have the lowest IQ in America on average?
It was noted in this study that states with lower IQs have more property crime and violent crime.
Increased intelligence is also linked to lower sex drive. I would reason that as shown above, those with higher IQs will be able to resist natural urges better than those with lower IQs. This shows in NLSY data with lower IQ people having more kids, higher IQ people fewer kids.
All of this data piles up, and the only thing that jumps out, is that lower IQ people have more testosterone, lower IQ people commit more crimes, therefore, high testosterone and low IQ is the cause of black criminality.
HBD and Diet Advice: Anglin Paleo Refutation Part 2
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A lot of people seem to have wrong views on nutrition. It’s not really taught in school, people think that it doesn’t matter so they do no independent research of their own and they believe anything and everything that comes out in the MSM as gospel. The thing is, the average person doesn’t read studies, or anything nutrition related for that matter, and believes most everything they read and hear in the MSM. I have talked about nutrition a bit here. I refuted Andrew Anglin’s atrocious writing and arguments for the Paleo Diet here and wrote on obesity and ethnicity including genetic and environmental causes. I also wrote on how nutrition is important prenatally as well as postnatally in developing children. I will also touch on comments in that Dailystormer article that jump out to me that need refuting.
Today I will talk about HBD and diet advice.
Steve Sailer wrote an article on HBD and Diet Advice back in September. He claims a few things that need to be disproven.
It’s common for nutrition scientists to give advice to white Americans based on studies done of what is good for nonwhites to eat. For example, in the 1980s, one of the most fashionable studies was of Japanese in Hawaii. The first generation ate mostly rice with little fat, and they had relatively few heart attacks. The next generation ate cheeseburgers and had higher rates of coronary disease than their parents.
I have covered this in the Dailystormer refutation.
Noted in this study are:
- High interpersonal variability in post-meal glucose observed in 800-person cohort
- Using personal and microbiome features enable accurate glucose response prediction
- Prediction is accurate and superior to common practice in an independent cohort
- Short-term personalized dietary interventions successfully lower post-meal glucose
You can see from the above bullet points that there is high interpersonal variability in post-meal glucose. What that means is, that between each individual in the cohort, there were different glucose spikes in each person.
They can accurately predict glucose response with certain tools. They devised a machine-learning algorithm that uses blood parameters, dietary habits, anthropometrics, physical activity, and gut microbiota measured in the cohort and showed that it accurately predicted personalized postprandial (post-meal) glycemic response to real-life meals.
They validated the prediction using a 100 person cohort.
Personalized dietary interventions showed interventions successfully lowered post-meal glucose. (Emphasis mine). This shows that each person should be on an individual diet and not on a one-size-fits-all diet.
Of course the next generation had higher rates of coronary disease than their parents. High carb, high fat diets lead to coronary blockage, leading to heart attacks and other coronary implications.
That is due to the demonization of fat starting in the 70s. We were told that fat is bad and carbs were fine. That turned out not to be the case. That’s what led to the obesity explosion. People think that eating fat “makes you fat”. Well if that’s the case, eating protein leads to kidney failure and eating carbs leads to Diabetes Mellitus. It’s stupid to think of it that way. Anything in excess is bad for you.
The RDA (Recommended Daily Values) for women is as follows:
69 grams of fat, which comes out to 585 kcal, 300 grams CHO which comes out to 1200 kcal and 53 grams of protein which comes out to 215 kcal. For men, it’s 80 grams of fat which comes out to 720 kcal, 375 grams CHO which comes out to 1500 kcal and 70 grams of protein which comes out to 280 kcal. This is data from the FDA on dietary recommendations for the average America.
Protein is nowhere near high enough. Protein is the main macronutrient you want to eat if you want to stay fuller longer as it has a higher TEF (Thermic Effect of Food). In the linked study, they come to the conclusion that TEF contributed to the satiating power of foods. Protein has the highest TEF of all of the macros, and because of this, some researchers have lobbied to have protein count as 3.2 kcal instead of 4 kcal. So if you want to stay fuller, eat more protein, fewer carbs and more fat. Carbs spike your insulin leading to insulin spikes, which lead to you feeling hungry sooner, as most people ingest fast digesting carbohydrates.
Sailer then cites this NYT article that says:
Today, at least 10 percent of Americans regularly take fish oil supplements. But recent trials have failed to confirm that the pills prevent heart attacks or stroke. And now the story has an intriguing new twist.
Wrong. So, so wrong. Controlled studies clearly show that omega-3 consumption had a positive influence on n-3 (fatty acid) intake. N-3 has also been recognized as a modulator of inflammation as well as the fact that omega-3 fatty acids down-regulate genes involved in chronic inflammation, which show that n-3 is may be good for atherosclerosis.
Studies have shown an increase in omega-3 consumption leads to decreased damage from heart attacks.
Omega-3 may also reduce damage after a stroke.
Dietary epidemiology has also shown a link between n-3 and mental disorders such as Alzheimers and depression. N-3 intake is also linked to intelligence, vision and mood. Infants who don’t get enough n-3 prenatally are at risk for developing vision and nerve problems. Other studies have shown n-3’s effects on tumors, in particular, breast, colon and prostate cancer.
Omega-3’s are also great for muscle growth. Omega-3 intake in obese individuals along with exercise show a speed up in fat-loss for that individual.
Where do these people get their information from? Not only are omega-3’s good for damage reduction after a stroke and a heart attack, they’re also good for muscle growth, breast, colon and prostate tumor reduction, infants deficient in omega-3 prenatally are at risk for developing nerve and vision problems. Increase in omega-3 consumption is also linked to increases in cognition, reduces chronic inflammation and is linked to lower instances of depression.
Omega-3’s are fine. As I said with the Anglin refutation, do not listen to those with no background in nutrition as they most likely have no idea what they are talking about.
Rasmus Nielsen, a geneticist at the University of California, Berkeley, and an author of the new study, said that the discovery raised questions about whether omega-3 fats really were protective for everyone, despite decades of health advice. “The same diet may have different effects on different people,” he said.
See above links on omega-3 intake and all of the positive/negative factors.
In the future, maybe you’ll be able to get your DNA analyzed and be given a list of diets in rank order of their likelihood that they will work for you. But, right now, you can still try different diets. In particular, ask your relatives about what has worked and not worked for them.
That doesn’t matter, as diets should be tailored to the individual, as seen in the Cell study.
Oh, wow. I just found that Anglin wrote a refutation to those who deny the Paleo Diet. Let’s see what that’s about.
And maybe these people have scientific research and/or personal experience to back up what they’re saying. I’m not insulting them for disagreeing with me on diet, that is clearly their right.
The evolutionary argument for Paleo does not line up with your statements. As I noted in my previous article, if you want to eat Paleo because it works with what you like to eat, good for you. But doing it for any magic benefits is stupid, as there are none.
I know for a fact that at least 9 out of 10 people who dare to take this challenge will report back positively if they follow it properly for a month, and this means a whole lot more than someone’s opinion about what it might or might not do, theoretically.
Want to know why people will report back positively? Any time you begin a new diet, especially one on a kcal restriction, your body will drop weight quickly. That’s what piranha personal trainers use on unknowing people. Telling them that they’re doing a “great job”, when in actuality, that happens to everyone who begins a new diet.
Instead, they are arguing theoretically, making highly debatable statements like “White people have evolved to be able to consume dairy products.”

The above map shows lactose intolerance for countries around the world. Ancient Europeans began dairying around 7500 ya and were lactose intolerant when starting to drink milk. But along with faster evolution, which includes no gene flow from other parts of the world, that led to Europeans evolving to, on average, have lower rates of lactose intolerance.
People should really learn what they’re talking about before they say it.
The way to know whether or not they are beneficial is to quit them for a period and see how you feel.
Placebo effect.
Currently, because the scientific literature on these topics is so convoluted and debated on, there is no other conceivable way to prove it one way or another than through our own testing.
The science is pretty solid on this. Anecdotes don’t mean anything to studies.
Many have referred to paleo as a “fad diet.” And it may be a diet that is a fad, but it is also a diet with a thousands upon thousands of years long precedent. One might even suggest that it is the consumption of grains and dairy that are the “fad,” as it is a relatively new trend, in the scope of things.
It IS A FAD DIET ; with NO basis in science that Europeans should eat that way.
If you feel as if you are at peak physical health eating grains and dairy, and have no desire to spend time trying to improve on this, than by all means skip the challenge.
Is he implying that the Paleo Diet is the only diet that doesn’t allow grains and dairy? Not true at all. The Slow Carb Diet is the same, as well as any other high fat, high protein low-carb diet.
We should also note that the definitions of vegetarianism were different then, and Hitler did eat eggs and probably wasn’t completely meat-free.
That’s vegetarianism. Veganism is the more extreme one you’re thinking of where absolutely no animal products are consumed at all.
Vegetarian diets are shown to lead to vitamin inadequacies such as zinc, calcium, iron, manganese, selenium, and copper. Vegetarianism works, it just has to be well-planned. You need to make sure you get the right amount of essential as well as non-essential amino acids, high amounts of protein and make sure you’re not nutrient deficient.
Last time I wrote that White rice and potatoes are good carbs, but I want to be clear that they are not necessary unless you are both already at 5% body weight and you are highly active.
They are not ‘good carbs’. They are white carbs, which are bad for us if we don’t go to the gym to utilize the CHO being ingested. Five percent body-fat? That’s for competition bodybuilders and marathon and distance runners. The average person will never cut down to the level of body-fat. CHO is extremely useful if you’re highly active and go to the gym.
Even if you are not active, you need to consume a decent amount of carbs once a week in order to keep your metabolism from slowing down too much. However, it is probably preferable to use fruits for this purpose, as they contain more micronutrients.
Correct. If you eat a low-carb diet, you need a CHO refeed once a week to keep metabolism high. Though, using fruits is stupid. I know the Paleo thing, but there are many reasons why fructose (the sugar found in fruit) is bad for you. Sugar is just as addictive as cocaine. So telling the average person to ‘use fruits for this purpose’ is stupid, as the average person doesn’t know when to stop eating.
Yes, this diet will technically cost more than a processed foods and grain-based diet, all things being equal. The only reason anyone ever ate grains in the first place is because they were cheap, and processed foods were invented for the same reason. Any natural and healthy diet is going to cost more, all things being equal. However, things don’t have to be equal.
Wrong. Whole foods are not more expensive. The conclusion that was (obviously) reached is that there is expensive and non-expensive junk food as well as whole foods. I personally spend 70 dollars a week on food for myself, with all of my meals planned out. Natural diets will not cost more, all things being equal. If you know how to eat and how to buy food, you will avoid spending too much money.
I tried to answer most of the questions people had in the last thread, but it was so filled up with denialism I could have missed something. So ask here.
I hope you answer this, as well as my other refutation of your horrible nutrition article. I doubt it though.
How did we really evolve to eat?
The most common form of eating, 3 meals a day, is abnormal from an evolutionary perspective. We didn’t evolve eating 3 times a day. We evolved eating intermittently. The study says that intermittent energy restriction periods of up to 16 hours are fine. Long-term calorie restriction is highly effective in reducing the risk for atherosclerosis in humans. Again, another huge benefit for intermittent fasting. As the data comes out on human cohorts, we will be able to see all of the great effects that IF has for us, because that’s how any human population, no matter where they evolved, evolved eating.
There are beneficial effects to IF including reduced oxidative damage and increased cellular stress resistance. Rats put on an IF diet show heightened life-spans. IF is also extremely useful to keep a youthful brain as you age.
There are a mountain of studies that show how beneficial IF is to us and is the TRUE way humans evolved to eat, not any specialized diets. We evolved eating intermittently, and with our hedonistic society we live in now, along with low ability to delay gratification, as well as other factors I have covered in my previous nutrition articles, have led to the effects we see in America, and around the world today.
In conclusion, don’t listen to people who have no background in nutrition. They tell clearly wrong information, and those who aren’t privy to new information in the nutrition world, won’t know that they are being lied to and or manipulated into believing things based on shoddy evidence.
“Race is a Social Construct”: Part 1
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“Race is a social construct”. You may hear that a lot from uneducated people. They may say that since the definition of race is ‘ever-changing’, that race doesn’t exist and that it only exists in our minds. They obviously have no understanding of genetics and how we came to be today. If you want to get technical, everything is a social construct. The Universe is a social construct. We’re only giving definitions to what we perceive something to be, so with the logic of ‘race being a social construct’, then everything is a social construct. With that logic, the Universe doesn’t exist because it’s a social construct.
I will look at 3 articles in the first of many articles on this subject. One from Angela Onwuachi-Willig, Bill Nye and Ta-Nehisi Coates. All 3 have extremely wrong views on the biological reality of race, and I will prove that here. I will quote from each article and show how they are wrong with scientific studies as well as point out their bad logic.
I will begin with Angela Onwuachi-Willig. In her article for The New York Times, ‘Race and Racial Identities Are Social Constructs‘, she says that because of the ever-changing definition of the term race, that it is a social construct and not a biological one.
Race is not biological. It is a social construct. There is no gene or cluster of genes common to all blacks or all whites. Were race “real” in the genetic sense, racial classifications for individuals would remain constant across boundaries. Yet, a person who could be categorized as black in the United States might be considered white in Brazil or colored in South Africa.
Race is not biological, it is a construct. There are no clusters of genes or one gene that is common in blacks or whites. That is correct, but her statement about race being social and not a biological construct is clearly ignorant as I will show below.

You can see in the picture above that races clearly do cluster in different clusters from other races. She is right about the changing definitions, especially Brazil, but Brazil is a special case. So much mixing has gone on in Brazil that there is evidence of skin color becoming independent of ancestry. One outlier example doesn’t make race a ‘social construct’. South Africa is also another one. They classify race in South Africa with four categories: black, colored, Indian/Asian or white. Obama would have been called ‘colored’ in South Africa today. But, again, just because there are changing definitions of race throughout the globe, doesn’t mean that race doesn’t exist.
Like race, racial identity can be fluid. How one perceives her racial identity can shift with experience and time, and not simply for those who are multiracial. These shifts in racial identity can end in categories that our society, which insists on the rigidity of race, has not even yet defined.
Is she making an argument for being ‘trans-racial’? I bet Rachel Dolezal would be happy.
In a society where being white (regardless of one’s socioeconomic class background or other disadvantages) means living a life with white skin privileges — such as being presumed safe, competent and noncriminal — whites who begin to experience discrimination because of their intimate connection with someone of another race, or who regularly see their loved ones fall prey to racial discrimination, may begin to no longer feel white. After all, their lived reality does not align with the social meaning of their whiteness.
I always hear about ‘white privilege’ but never get an actual definition of what it means. People complain about ‘white privilege’ because they, of course, don’t understand the biological reality of race. Anything that may prove innate differences between individuals or races they just can’t imagine exists because of what they’ve been taught their whole lives. She is talking about those whites who are in the BLM movement. The false ideals of egalitarianism are the cause of this.
More than 50 years ago, Congress enacted the most comprehensive antidiscrimination legislation in history, the Civil Rights Act of 1964. Half a century later in 2015, the same gaps in racial inequality remain or have grown deeper. Today, the unemployment rate for African-Americans remains more than double that for whites, public schools are more segregated now than they were in the 1950s and young black males are 21 times more likely to be shot and killed by the police than their white male peers. Even a white fourth-grade teacher in Texas, Karen Fitzgibbons, openly advocated for the racial segregation of the 1950s and 1960s on her Facebook page.
Right. IQ is the cause of the unemployment rate of African Americans. Not any imaginary forces such as ‘white privilege’. Public schools are more segregated today due to people wanting to be with others genetically similar to themselves. Blacks cause themselves to get shot and killed by police due to their actions during altercations with police officers. Oh no, someone has not politically correct opinions!! She should lose her job and never work a good job again!
That’s what the Left does. They attempt to shout you down with buzzwords so you can’t calmly and intellectually prove your case.
She is clearly wrong. Good thing this is called an ‘opinion piece’, there were few actual facts in it.
Now to touch on Bill Nye’s views on race. It’s funny. I loved his show when I was a kid. Now, knowing the truth about racial differences, hearing him say that made me lose all respect for him. He’s a mechanical engineer with a Bs from Cornell University. People only take what he says because he is ‘The Science Guy’ when he has no training in what he is talking about.
“We obsess about whether our dog is a pug-Jack Russell terrier mix with corgi overtones and an oaky finish. ‘An approachable little dog,’ whatever. They’re all dogs, okay? And so the idea of a purebred is just a human construct. There’s no such thing – in a sense there’s no such thing as a purebred dog.”
That right there is a fallacy. As with the woman’s article above, they both use the ‘continuum fallacy‘. The continuum fallacy is when someone rejects a vague claim because it’s not as precise as they want it to be. ‘There are no pure races’ or ‘there are no pure breeds of dog’, that doesn’t mean that genes don’t cluster differently, showing genetic differentiation.
“If a Papua New Guinean hooks up with a Swedish person all you get is a human. There’s no new thing you’re going to get. You just get a human. Japanese woman jumping the African guy, all you get is a human. They’re all humans. So this is a lesson to be learned. There really is, for humankind there’s really no such thing as race. There’s different tribes but not different races. We’re all one species.”
Right. That doesn’t mean there is no such thing as race. Grizzly bears and polar bears can mate to create a prizzly bear. Does that mean species doesn’t exist? (I will touch on speciation at the end of this article.) Once again, that statement doesn’t deny the biological reality of race, as you can see from the picture above.

Researchers have proven, scientifically, that humans are all one people. The color of our ancestors’ skin is a consequence of ultra-violet light, of latitude and climate.
We all belong to the same genus, Homo, but again, that doesn’t disprove race. He’s correct in saying that our ancestors’ skin is a consequence of UV light of latitude and climate, and right there he proves us correct in stating that sunlight differences depending on where your ancestors evolved in the world are the cause of racial differences.
Despite our recent sad conflicts here in the U.S., there really is no such thing, scientifically, as race. We are all one species. Each one of us more alike than different.
In this statement, he is saying the first sentence because of recent racial relations in the U.S. A clear politically-motivated statement.
“Each one of us more alike than different.’ That is correct, but, yet again, doesn’t disprove the reality of race. Geneticists estimate that humans will differ, on average, at 3 million base pairs in their DNA. That’s more than enough for distinct racial classification, as well as enough to differentiate us.
Is that supposed to mean anything? Cats have 90 percent homologous genes with humans, 82% with dogs, 80% with cows, 79% with chimpanzees, 69% with rats and 67% with mice.
90% of the mouse genome could be lined up with a region on the human genome.
99% of mouse genes turn out to have analogues in humans.
As you can see from the links above, we are all extremely genetically related to animals that are clearly extremely physically different from us humans. This shows, that the differences in humans are down to not how genetically distant we are from other animals in the animal kingdom, but how the differing genes we have are expressed.
We all came from Africa. We’re all made from the same star dust. We’re all going to live and die on the same planet – a pale blue dot in the vastness of space. We have to work together!
And finally, you can see his way of saying that racial differences mean nothing because ‘we all come from Africa’ and ‘ we’re all made from star dust’. That may be true, but that doesn’t do anything to acknowledge, or even show that racial differences are meaningless.
Bill Nye has absolutely no authority to speak on this matter. Liberals then eat this up and cite Bill Nye as proof that race doesn’t exist, which is clearly untrue as I have shown.
Finally, I will get to Ta-Nehisi Coates’ ‘The Social Construction of Race‘. He cites my favorite blogger/geneticist Razib Khan, so this should be good.
Ancestry — where my great-great-great-great grandparents are from — is a fact. What you call people with that particular ancestry is not. It changes depending on where you are in the world, when you are there, and who has power.
Right with the first sentence, and with the second. It seems he’s attempting to use what the first article I cited says: that due to ever-changing racial definitions that race doesn’t exist as we believe it to be. He says that ancestry is a fact, well wouldn’t that same ancestry be your racial classification? I am not following his logic. Just because there are differing views on the definition of race throughout the globe, doesn’t mean that there is no biological reality of race.
He cites someone else who states:
“Race” as a term is very nebulous. But human subgroups with similar ancestries can have group differences in DNA — and intelligence is highly unlikely to have no genetic basis at all (although most now believe its impact is greatly qualified by cultural and developmental differences).
Cultural and developmental differences. The cultural differences are thrown out. According to the editorial ‘Mainstream Science on Intelligence’, which came out shortly after The Bell Curve was published, one of their points is that IQ tests are not culturally biased if the individual speaks English. If they are not English speakers, they will either get a test in their native language or get Raven’s Progressive Matrices, which is a ‘culture free’ IQ test as it’s based on pattern recognition and has no writing involved. Developmental differences, yes. White mothers have a better prenatal environment then do black mothers, which is biological. Developmental differences are innate within the two populations.
I do not know. Andrew is more inclined to believe that there is some group-wide genetic explanation for the IQ difference. I am more inclined to believe that the difference lies in how those groups have been treated. One thing that I am not convinced by is controlling for income and education.
Oh, the old ‘Stereotype Threat‘ canard. The paper, which was cited more than 5000 times, states that African-Americans do worse on tests in which they are told that they are being judged on their race. Well, a meta-analysis of 55 published and unpublished studies came out and what was found that the it shows clear publication bias. Either due to people not knowing how to read scientific papers or more insidious tactics. The effect varies across studies and is small. Though elite university undergrads may underperform on tests of cognitive tests due to Stereotype Threat, this effect doesn’t generalize to non-adapted standardized tests, high-stakes settings and less academically gifted test takers. Stereotype Threat cannot explain the difference in mean cognitive test performance between African Americans and European Americans. (pg 68)
In the mid-20th century, as we have been documenting, it was the policy of this country to deny African-Americans access to the same methods of wealth-building, that it was making available to whites.
This is not merely a problem for your local diversity and sensitivity workshop. It is a problem of wealth and power. When you create a situation in which a community has a disproportionate number of poor people, and then you hyper-segregate that community, you multiply the problems of poverty for the entire community–poor or not. That is to say that black individuals are not simply poorer and less wealthier than white individuals. Because of segregation, black individuals and white individuals of the same income and same wealth, do not live in communities of equal wealth.
What bearing does segregation have on IQ differential? I don’t know. My skepticism of genetics is rooted in the fact that arguments for genetic inferiority among people of African ancestry are old, and generally have not fared well. My skepticism is also rooted in the belief that power generally seeks to justify itself. The prospect of actual equality among the races is frightening. If black and white people truly are equal on a bone-deep level, then the game might really be rigged, and we might actually have to do something about it. I think there’s much more evidence of that rigging, then there is evidence of cognitive deficiency .
I must add that I can not pretend to be a dispassionate, nor impartial observer. I come from a particular place. I’ve now been out in the world, and seen how other people in other places live. They don’t strike me as more intelligent. They strike me as better armed. There’s nothing scientific about that. But I think we all have core faiths. These are mine. You’ve been warned.
Regardless of the method used in the analyses, all researchers reached estimated very close to that obtained by Lewontin: The differences observed by the subdivisions (populations, groups of populations, races) represented 10 to 15 percent of the total genetic variation found within the human species. Formally, these findings demonstrate, first, that the species is indeed subdivided into genetically definable groups of individuals and, second, that atleast some of these groups correspond to those defined by anthropologists as races on the basis of physical characters. They do not however, settle the arguments regarding the methods of racial classification. Unfortunately, Lewontin did not specify before initiating his analysis how large the difference has to be in order to call the groups “races”.
Consequently, the results of the studies have led population geneticists to two diametrically opposite conclusions. Lewontin called the observed differences trivial, and proclaimed that “racial classification is now seen to be of no genetic or taxonomic significance” so that “no justification can be offered for its continuance.” This view is echoed by authors of similar studies, who seem to be surprised that genetic variation within populations is greater than that between them. By contrast, Sewell Wright who can hardly be taken for a dilettante in questions of population genetics, has stated emphatically that if differences of this magnitude were observed in any other species, the groups they distinguish would be called subspecies.
One can extend Wright’s argument even further. The more than 200 species of haplochromine fishes in Lake Victoria differ from each other much less than the human races in their neural genes, although they are presumably distinguished by genes that control differences in their external appearances. The same can be said about atleast some of the currently recognized species of Darwin’s finches and other examples of recent adaptive radiations. In all these cases, reproductively isolated groups are impossible to tell apart by the methods used to measure differences in human races. Obviously, human races are not reproductively isolated (interracial marriages are common and the progenies of such marriages are fully fertile) but the external differences between them are comparable to cichlid fishes and Darwin’s finches. Under these circumstances, to claim that the genetic differences between the human races are trivial is a more political statement than a scientific argument. Trivial by what criterion? How much difference would Lewontin and those who side with him consider non-trivial?
By mixing science with politics, geneticists and anthropologists are committing the same infraction of which they are accusing other scientists, who they themselves label as racist. Even worse, by labelling the genetic differences as insignificant, they play into the hand of genuine racists who can demolish this claim and so further their own agenda. It is intellectually more honest to acknowledge and then point out that by no means imply supremacy of one race over others. This can be done by demonstrating that the differences are in genes that cannot be linked to any features that would be required for the preeminence of a particular race.
It’s clear that racial classification does exist. The creator of Fst, Sewall Wright, says that a Fst distance of .15 is more than enough for speciation (differing racial classifications). It directly refutes Lewontin, who put his political ideology of Marxism over science. Those cichlids in Lake Victoria are a perfect example that though the definition of ‘species’ does change depending on which researcher you speak to, it doesn’t discount that there are real and physical genetic differences between races and ethnicities.
In conclusion, the term “race is a social construct” is a deliberately intellectually dishonest statement, or a statement used to hide the truth for more insidious things to happen due to the non-acknowledgement of race.
BEHAVIOR=GENES+ENVIRONMENT NEEDS TO DIE: Vindication!
1800 words
So I was at Barnes N Noble a few weeks ago and picked up this book called ‘This Idea Must Die‘. It has a ton of researchers who wrote for it. Each person writes a short, 3 to 4 page writing on what idea must die and why. it’s a great read so far. Jared Diamond, Steven Pinker and Richard Dawkins wrote for it.
Now, I just came to this section in the book where Steven Pinker talks about behavior=genes+environment. To quote Pinker from the book:
Would you say that the behavior of your computer or smartphone is determined by an interaction between its inherent design and the way it is influenced by the environment? It’s unlikely; such a statement would not be false, but it would be obtuse. Complex adaptive systems have a nonrandom organization , and they have inputs. But speaking of inputs as “shaping” the system’s behavior, or pitting its design against its input, would lead to no insight as to how the system works. The human brain is far more complex, and processes its input in more complex ways, than human-made devices, yet many people analyze it in ways that are too simplistic for our far simpler toys. Every term in the equation is suspect.
Behavior: More than half a century after the cognitive revolution, people still ask whether a behavior is genetically or environmentally determined. Yet neither the genes nor the environment can control the muscles indirectly. The cause of the behavior is the brain. While it’s sensible to ask how emotions, motives, or learning mechanisms have been influenced by genes, it makes no sense to ask this of behavior itself. (Emphasis mine.)
Genes: Molecular biologists have appropriated the term “gene” to refer to the stretches of DNA that code for a protein. Unfortunately, this sense differs from the one used in population genetics, behavioral genetics and evolutionary theory – namely, any information carried that’s transmittable across generations and has sustained effects on the phenotype. This includes any aspect of DNA that can affect gene expression, and is closer to what is meant by “innate” than genes in the molecular biologists’ narrow sense. The confusion between the two leads to innumerable red herrings in discussions of out makeup, such as the banality that the expression of genes (in the sense of protein-coding stretches of DNA) is regulated by signals from the environment. How else could it be? The alternative is that every cell synthesizes every protein all the time! The epigenetics bubble inflated by the science media is based on a similar confusion. (Emphasis mine.)
Environment: This term for the inputs to an organism is also misleading. Of all the energy impinging on an organism, only a subset, processed and transformed in complex ways, has an effect on its subsequent information processing. Which information is taken in, how it’s transformed, and how it affects the organism (that is, the way the organism learns) all depend on the organisms innate organization. To speak of the environment “determining” or “shaping” behavior is unperspicuous.(Emphasis mine.)
Even the technical sense of “environment”used in quantitative behavioral genetics is perversely confusing. Now, there’s nothing wrong with partitioning phenotypic variance into components that correlate with genetic variation (heritability) and with variation among families (“shared environment”). The problem comes from so-called “nonshared” or “unique” environmental influences. This consists of all the variance attributable to neither genetic nor familiar variation. In most studies, it’s calculated as 1 – (heritability + shared environment). Practically, you can think of it as the differences between identical twins who grow up in the same home. They share their genes, parents, older and younger siblings, school, peers, and neighborhood. So what could make them different? Under the assumption that behavior is a product of genes plus environment, it must be something in the environment of one that is not in the environment of the other.
But this category really should be called “miscellaneous/unknown,” because is has nothing necessarily to do with any measurable aspect of the environment, such as one sibling getting the top bunk and the other the bottom, or a parent unpredictably favoring one child, or one sibling getting chased by a dog, coming down with a virus, or being favored by a teacher. These influences are purely conjectural, and studies looking for them have failed to find them.The alternative is that this component actually consists of the effects of chance – new mutations, quirky prenatal effects, noise in brain development, and events in life with unpredictable effects. (Emphasis mine.)
Stochastic effects in development are increasingly being recognized by epidemiologists, frustrated by such recalcitrant phenomena such as nonagenarian pack-a-day smokers and identical twins discordant for schizophrenia, homosexuality, and disease outcomes. They’re increasingly forced to acknowledge that God plays die with our traits.(Emphasis mine.) Developmental biologists have come to similar conclusions. The bad habit of assuming that anything not classically genetic must be “environmental” has blinkered behavioral geneticists (and those who interpret their findings) into the fool’s errand of looking for environmental effects foe what may be randomness in the developmental processes. (Emphasis mine.)
A final confusion in the equation is the seemingly sophisticated add-on of “gene-environment interactions.” This is also designed to confuse. Gene-environment interactions do not refer to the fact that the environment is necessary for genes to do their thing (which is true of all genes). It refers to a flipflop effect inn which the genes affect a person one way in one environment but another way in another environment, whereas an alternative gene has a different patter. For example, if you inherit allele 1, you are vulnerable: a stressor makes you neurotic. If you inherit allele 2, you are resilient: a stressor leaves you normal. With either gene, if you are never stressed, you are normal.
Gene-environment interactions in this technical sense, confusingly, go into the “unique environmental” component, because they’re not the same (on average) in siblings growing up in the same family. Just as confusingly, “interactions” in the commonsense – namely, that a person with a given genotype is predictably affected by the environment – goes into the “heritability” component, because the quantitative genetics measures only correlations. This confound is behind the finding that the heritability of intelligence increases, and the effects of shared environment decrease, over a person’s lifetime. One explanation is that genes have effects late in life, but another is that people with a given genotype place themselves in environments that indulge their inborn tastes and talents. The “environment” increasingly depends on their genes, rather than being the cause of exogenous behavior. (Emphasis mine.) (pg 188-191 This Idea Must Die)
I’m pretty excited about this. I have said for a while, to quote Douglas Whitman:
Race is not a social construct. Society is a racial construct. Society and culture derive from race/biology
You can see the effects from race/biology anywhere in the world you look and see the majority racial/ethnic mix of a country and/or area. A place is ONLY as good as its majority population.
We can see the kinds of effects this will have on our society as a whole. The way all of these 3rd worlders are flooding in to our countries. They leave their countries, to come to ours (in the West), and they don’t realize that once they become the majority and displace the native populations of the countries, that the place they will be living in will be just as bad, or even worse than where they came from.
Now, what Pinker said about the nonshared environment was really interesting. Especially at the end where he says ‘The “environment” increasingly depends on their genes, rather than being the cause of exogenous behavior.’
What does that mean? You can see this, for the most part, whenever there are new immigrants to an area. They still act how they did back in their home countries. The native peoples of the country have a certain way they act, and so do the immigrants to that country. The native peoples environment is an expression of their genetics. The new environment that the new immigrants bring to the country is also an expression of genetics. So, we can see just with what Pinker explained above, that environment itself doesn’t dictate behavior, but GENES DICTATE BEHAVIOR AND ALSO THE ENVIRONMENT THAT GETS CREATED.
So, it seems that those with certain genes place themselves in environments that indulge their inborn tastes and talents. “Environment” INCREASINGLY DEPENDS ON THEIR GENES RATHER THAN BEING THE CAUSE OF EXOGENOUS BEHAVIOR.
We know that genes are the driving force in life, and with this new ‘epigenetic’ field of genetics, they will attempt to say that environment is the main cause of molding genetics because one may have different things happen to them in one environment and not the other. But, since we have just seen here that those with certain genes put themselves into their environments, what does this say about certain people you may know? Does this mean that their genes put them in their situation? Well, for the most part, yes.
If one chooses their environment based on their genetic makeup, wouldn’t that throw out any and all environmental interactions?
For instance, how leftists say that negros can’t help it and are ‘born in to poverty’. Negros, as well as other low IQ peoples, choose their environment based on their genetics.
THE ENVIRONMENT YOU CHOOSE IS BASED ON YOUR GENETICS. THE PEOPLE DICTATE THE ENVIRONMENT OF A PLACE.
Growing up in a bad environment does not make you a bad person, sorry to say, but your genes make you a bad person. People seem to have the wrong idea about how environment interactions work with genes. I’m glad this came out.
This is inferred from what Pinker said. This is also explicitly said in the Whitman quote.
This new “epigenetic revolution” needs to die. It is saying that gene x environment interactions matter, when it really is the opposite. What is really happening is that individuals group up, due to genetic similarity theory, the theory proposed by Rushton after noticing that each ethnic group basically stayed with each other, along with their genetics that dictates their environment to live in. By putting so many genetically similar people in the same environment, in this case, immigrants, you will, therefore, get the same situation of how the conditions in their countries were, due to genetic similarity theory having them be with peoples of similar genetics, as well as their genes dictating their environments.
BEHAVIOR=GENES+ENVIRONMENT NEEDS TO DIE!!
Genetic Reasons for Human Migration
800 words
It’s always talked about in race realist circles what caused the migration Out of Africa and what gives us an urge and sense of wonder for adventure. Well, like with most things in life, there is a genetic reason behind it.
So I use this website called StumbleUpon and I was Stumbling in the Genetics section, and I came across this article, and as I was reading it, I knew that it was going to be rare in Africans.
And what do you know, I was right.
Seems like the gene developed around 40,000 years ago. It’s obviously not just a coincidence or ‘correlation doesn’t equal causation’ argument, as a whole bunch of research backs this gene, and specific alleles, to certain traits in humans, compared to migration, or lack thereof, around the world.
From some research, I found that the abstract of the article says: it’s linked to hyperactivity, novelty-seeking and risk-taking behaviors. Consistent with findings in animal studies was: having more exploratory behavior as well as increased speed and locomotion. Populations with a history of migration in the past 1 to 30,000 years will show the highest instance of having the DRD4 alleles. Those that migrated more miles have a higher rate than those that migrated less. Sedentary populations show a lower instance of the DRD4 allele.
The abstract also says “After compiling existing data on DRD4 allele frequencies of 2,320 individuals from 39 populations and on the migration pattern of these groups, we found that, compared to sedentary populations, migratory populations showed a higher proportion of long alleles for DRD4.”
A correlation of .85 was found with km traveled and rate of DRD4 allele frequency distributions, which support the hypothesis. Nomadic populations had a 10.4 percent higher rate of DRD4 long alleles than sedentary ones.
Here is a blog post talking about the DRD4-7 genes and how they are involved with dopamine levels in the brain and have to do with motivation and behavior.
It cites a researcher saying that the gene occurs in populations that migrated first and furthest out of Africa are more likely to have the gene.
Another blog talks about how Africans with the 4R allele stayed in Africa whereas those with the 7R left and explored the world.
Research has found that it’s easier for the 2R allele to mutate into a 7R allele than for a 7R allele to mutate back to a 4R allele.
So it’s thought that those who emigrated but remained closer to Africa, such as Asia, lost the behavior to explore and settled rather than explore the Americas (which Siberians did do, who eventually became Indians. I would assume that they would have a higher prevalence of this gene, being as they migrated to the Americas while the Africans stayed in Africa, as well as Indians being the furthest genetically from Africans, it also makes sense there). Also saying that those who emigrated to Asia had the 7R, but stayed and it mutated to the 2R over time.
It says looking at the global prevalence of certain allele frequencies, the 4R is more common in Africa, the 2R in Asia, the 7R in the Americas (mostly South), supporting the theory for the 7R allele being the cause for global expansion and positive selection.
Found this article talking about how the DRD4 allele is a role “in the escalation of substance use during adolescence and potential for an enhanced understanding of early-onset substance use.” and those with the 7R allele “Supporting the differential susceptibility to parenting hypothesis, the results suggest a greater preventive effect for youths carrying a 7-repeat allele”.
This NatGeo article says “Most provocatively, several studies tie 7R to human migration. The first large genetic study to do so, led by Chuansheng Chen of the University of California, Irvine in 1999, found 7R more common in present-day migratory cultures than in settled ones. A larger, more statistically rigorous 2011 study supported this, finding that 7R, along with another variant named 2R, tends to be found more frequently than you would expect by chance in populations whose ancestors migrated longer distances after they moved out of Africa. Neither study necessarily means that the 7R form of the gene actually made those ancestors especially restless; you’d have to have been around back then to test that premise with certainty. But both studies support the idea that a nomadic lifestyle selects for the 7R variant.”
All in all, this shows a genetic reason WHY the migration OoA began. It’s very telling that the DDR7 allele first arose when we migrated OoA.