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HBD and Diet Advice: Anglin Paleo Refutation Part 2

2300 words

A lot of people seem to have wrong views on nutrition. It’s not really taught in school, people think that it doesn’t matter so they do no independent research of their own and they believe anything and everything that comes out in the MSM as gospel. The thing is, the average person doesn’t read studies, or anything nutrition related for that matter, and believes most everything they read and hear in the MSM. I have talked about nutrition a bit here. I refuted Andrew Anglin’s atrocious writing and arguments for the Paleo Diet here and wrote on obesity and ethnicity including genetic and environmental causes. I also wrote on how nutrition is important prenatally as well as postnatally in developing children. I will also touch on comments in that Dailystormer article that jump out to me that need refuting.

Today I will talk about HBD and diet advice.

Steve Sailer wrote an article on HBD and Diet Advice back in September. He claims a few things that need to be disproven.

It’s common for nutrition scientists to give advice to white Americans based on studies done of what is good for nonwhites to eat. For example, in the 1980s, one of the most fashionable studies was of Japanese in Hawaii. The first generation ate mostly rice with little fat, and they had relatively few heart attacks. The next generation ate cheeseburgers and had higher rates of coronary disease than their parents.

I have covered this in the Dailystormer refutation.

Noted in this study are:

  1. High interpersonal variability in post-meal glucose observed in 800-person cohort
  2. Using personal and microbiome features enable accurate glucose response prediction
  3. Prediction is accurate and superior to common practice in an independent cohort
  4. Short-term personalized dietary interventions successfully lower post-meal glucose

You can see from the above bullet points that there is high interpersonal variability in post-meal glucose. What that means is, that between each individual in the cohort, there were different glucose spikes in each person.

They can accurately predict glucose response with certain tools. They devised a machine-learning algorithm that uses blood parameters, dietary habits, anthropometrics, physical activity, and gut microbiota measured in the cohort and showed that it accurately predicted personalized postprandial (post-meal) glycemic response to real-life meals.

They validated the prediction using a 100 person cohort.

Personalized dietary interventions showed interventions successfully lowered post-meal glucose. (Emphasis mine). This shows that each person should be on an individual diet and not on a one-size-fits-all diet.

Of course the next generation had higher rates of coronary disease than their parents. High carb, high fat diets lead to coronary blockage, leading to heart attacks and other coronary implications.

That is due to the demonization of fat starting in the 70s. We were told that fat is bad and carbs were fine. That turned out not to be the case. That’s what led to the obesity explosion. People think that eating fat “makes you fat”. Well if that’s the case, eating protein leads to kidney failure and eating carbs leads to Diabetes Mellitus.  It’s stupid to think of it that way. Anything in excess is bad for you.

The RDA (Recommended Daily Values) for women is as follows:

69 grams of fat, which comes out to 585 kcal, 300 grams CHO which comes out to 1200 kcal and 53 grams of protein which comes out to 215 kcal. For men, it’s 80 grams of fat which comes out to 720 kcal, 375 grams CHO which comes out to 1500 kcal and 70 grams of protein which comes out to 280 kcal. This is data from the FDA on dietary recommendations for the average America.

Protein is nowhere near high enough. Protein is the main macronutrient you want to eat if you want to stay fuller longer as it has a higher TEF (Thermic Effect of Food). In the linked study, they come to the conclusion that TEF contributed to the satiating power of foods. Protein has the highest TEF of all of the macros, and because of this, some researchers have lobbied to have protein count as 3.2 kcal instead of 4 kcal. So if you want to stay fuller, eat more protein, fewer carbs and more fat. Carbs spike your insulin leading to insulin spikes, which lead to you feeling hungry sooner, as most people ingest fast digesting carbohydrates.

Sailer then cites this NYT article that says:

Today, at least 10 percent of Americans regularly take fish oil supplements. But recent trials have failed to confirm that the pills prevent heart attacks or stroke. And now the story has an intriguing new twist.

Wrong. So, so wrong. Controlled studies clearly show that omega-3 consumption had a positive influence on n-3 (fatty acid) intake. N-3 has also been recognized as a modulator of inflammation as well as the fact that omega-3 fatty acids down-regulate genes involved in chronic inflammation, which show that n-3 is may be good for atherosclerosis.

Studies have shown an increase in omega-3 consumption leads to decreased damage from heart attacks.

Omega-3 may also reduce damage after a stroke.

Dietary epidemiology has also shown a link between n-3 and mental disorders such as Alzheimers and depression. N-3 intake is also linked to intelligence, vision and mood. Infants who don’t get enough n-3 prenatally are at risk for developing vision and nerve problems. Other studies have shown n-3’s effects on tumors, in particular, breast, colon and prostate cancer.

Omega-3’s are also great for muscle growth. Omega-3 intake in obese individuals along with exercise show a speed up in fat-loss for that individual.

Where do these people get their information from? Not only are omega-3’s good for damage reduction after a stroke and a heart attack, they’re also good for muscle growth, breast, colon and prostate tumor reduction, infants deficient in omega-3 prenatally are at risk for developing nerve and vision problems. Increase in omega-3 consumption is also linked to increases in cognition, reduces chronic inflammation and is linked to lower instances of depression.

Omega-3’s are fine. As I said with the Anglin refutation, do not listen to those with no background in nutrition as they most likely have no idea what they are talking about.

Rasmus Nielsen, a geneticist at the University of California, Berkeley, and an author of the new study, said that the discovery raised questions about whether omega-3 fats really were protective for everyone, despite decades of health advice. “The same diet may have different effects on different people,” he said.

See above links on omega-3 intake and all of the positive/negative factors.

In the future, maybe you’ll be able to get your DNA analyzed and be given a list of diets in rank order of their likelihood that they will work for you. But, right now, you can still try different diets. In particular, ask your relatives about what has worked and not worked for them.

That doesn’t matter, as diets should be tailored to the individual, as seen in the Cell study.

Oh, wow. I just found that Anglin wrote a refutation to those who deny the Paleo Diet. Let’s see what that’s about.

And maybe these people have scientific research and/or personal experience to back up what they’re saying.  I’m not insulting them for disagreeing with me on diet, that is clearly their right.

The evolutionary argument for Paleo does not line up with your statements. As I noted in my previous article, if you want to eat Paleo because it works with what you like to eat, good for you. But doing it for any magic benefits is stupid, as there are none.

I know for a fact that at least 9 out of 10 people who dare to take this challenge will report back positively if they follow it properly for a month, and this means a whole lot more than someone’s opinion about what it might or might not do, theoretically.

Want to know why people will report back positively? Any time you begin a new diet, especially one on a kcal restriction, your body will drop weight quickly. That’s what piranha personal trainers use on unknowing people. Telling them that they’re doing a “great job”, when in actuality, that happens to everyone who begins a new diet.

Instead, they are arguing theoretically, making highly debatable statements like “White people have evolved to be able to consume dairy products.”

Global-Lactose-Intolerance

The above map shows lactose intolerance for countries around the world. Ancient Europeans began dairying around 7500 ya and were lactose intolerant when starting to drink milk. But along with faster evolution, which includes no gene flow from other parts of the world, that led to Europeans evolving to, on average, have lower rates of lactose intolerance.

People should really learn what they’re talking about before they say it.

The way to know whether or not they are beneficial is to quit them for a period and see how you feel.

Placebo effect.

Currently, because the scientific literature on these topics is so convoluted and debated on, there is no other conceivable way to prove it one way or another than through our own testing.

The science is pretty solid on this. Anecdotes don’t mean anything to studies.

Many have referred to paleo as a “fad diet.”  And it may be a diet that is a fad, but it is also a diet with a thousands upon thousands of years long precedent.  One might even suggest that it is the consumption of grains and dairy that are the “fad,” as it is a relatively new trend, in the scope of things.

It IS A FAD DIET ; with NO basis in science that Europeans should eat that way.

If you feel as if you are at peak physical health eating grains and dairy, and have no desire to spend time trying to improve on this, than by all means skip the challenge.

Is he implying that the Paleo Diet is the only diet that doesn’t allow grains and dairy? Not true at all. The Slow Carb Diet is the same, as well as any other high fat, high protein low-carb diet.

We should also note that the definitions of vegetarianism were different then, and Hitler did eat eggs and probably wasn’t completely meat-free.

That’s vegetarianism. Veganism is the more extreme one you’re thinking of where absolutely no animal products are consumed at all.

Vegetarian diets are shown to lead to vitamin inadequacies such as zinc, calcium, iron, manganese, selenium, and copper. Vegetarianism works, it just has to be well-planned. You need to make sure you get the right amount of essential as well as non-essential amino acids, high amounts of protein and make sure you’re not nutrient deficient.

Last time I wrote that White rice and potatoes are good carbs, but I want to be clear that they are not necessary unless you are both already at 5% body weight and you are highly active.

They are not ‘good carbs’. They are white carbs, which are bad for us if we don’t go to the gym to utilize the CHO being ingested. Five percent body-fat? That’s for competition bodybuilders and marathon and distance runners. The average person will never cut down to the level of body-fat. CHO is extremely useful if you’re highly active and go to the gym.

Even if you are not active, you need to consume a decent amount of carbs once a week in order to keep your metabolism from slowing down too much.  However, it is probably preferable to use fruits for this purpose, as they contain more micronutrients.

Correct. If you eat a low-carb diet, you need a CHO refeed once a week to keep metabolism high. Though, using fruits is stupid. I know the Paleo thing, but there are many reasons why fructose (the sugar found in fruit) is bad for you. Sugar is just as addictive as cocaine. So telling the average person to ‘use fruits for this purpose’ is stupid, as the average person doesn’t know when to stop eating.

Yes, this diet will technically cost more than a processed foods and grain-based diet, all things being equal.  The only reason anyone ever ate grains in the first place is because they were cheap, and processed foods were invented for the same reason.  Any natural and healthy diet is going to cost more, all things being equal.  However, things don’t have to be equal.

Wrong. Whole foods are not more expensive. The conclusion that was (obviously) reached is that there is expensive and non-expensive junk food as well as whole foods. I personally spend 70 dollars a week on food for myself, with all of my meals planned out. Natural diets will not cost more, all things being equal. If you know how to eat and how to buy food, you will avoid spending too much money.

I tried to answer most of the questions people had in the last thread, but it was so filled up with denialism I could have missed something.  So ask here.

I hope you answer this, as well as my other refutation of your horrible nutrition article. I doubt it though.

How did we really evolve to eat?

The most common form of eating, 3 meals a day, is abnormal from an evolutionary perspective. We didn’t evolve eating 3 times a day. We evolved eating intermittently. The study says that intermittent energy restriction periods of up to 16 hours are fine. Long-term calorie restriction is highly effective in reducing the risk for atherosclerosis in humans. Again, another huge benefit for intermittent fasting. As the data comes out on human cohorts, we will be able to see all of the great effects that IF has for us, because that’s how any human population, no matter where they evolved, evolved eating.

There are beneficial effects to IF including reduced oxidative damage and increased cellular stress resistance. Rats put on an IF diet show heightened life-spansIF is also extremely useful to keep a youthful brain as you age.

There are a mountain of studies that show how beneficial IF is to us and is the TRUE way humans evolved to eat, not any specialized diets. We evolved eating intermittently, and with our hedonistic society we live in now, along with low ability to delay gratification, as well as other factors I have covered in my previous nutrition articles, have led to the effects we see in America, and around the world today.

In conclusion, don’t listen to people who have no background in nutrition. They tell clearly wrong information, and those who aren’t privy to new information in the nutrition world, won’t know that they are being lied to and or manipulated into believing things based on shoddy evidence.

Solutrean Hypothesis: Were the first peoples of the Americas from Spain?

Just like Afrocentrists have cockamamie theories, so do Eurocentrists. Some people believe that the first peoples of the Americas were the Solutreans, a stone age people from France and Spain who existed around 25 kya, and ended 16,500 ya. They would have had to cross 3000 miles of ice and water, does that seem possible 20 kya? Not at all.

The Solutrean Hypothesis first came about in the 30s by archaeologist Frank Hibben. He noted that the style of the spear tips the Solutreans used was like that of the Clovis culture in the Americas, who are the oldest culture on the continent. He said the tips bore an extremely strong resemblance to those from the Solutreans in Europe.

The points of the tips are different (diamond shaped, non-fluted vs concave bottoms, fluting). They also didn’t have boats to cross the ocean to reach the Americas.

From this Nature article, we can see that the genome of the Clovis people had direct ancestry to Native Americans:

An alternative, Solutrean, hypothesis posits that the Clovis predecessors emigrated from southwestern Europe during the Last Glacial Maximum4. Here we report the genome sequence of a male infant (Anzick-1) recovered from the Anzick burial site in western Montana. The human bones date to 10,705±3514C yearsBP (approximately 12,707–12,556 calendar yearsBP) and were directly associated with Clovis tools. We sequenced the genome to an average depth of 14.4×and show that the gene flow from the Siberian Upper Palaeolithic Mal’ta population5 into Native American ancestors is also shared by the Anzick-1 individual and thus happened before 12,600 yearsBP.

Consistent with the population migration from Siberia into the Americas, as noted in this study. This study I will quote below also shows why some of the same alleles are found in both Europeans and Native Americans:

Y chromosomal DNA polymorphisms were used to investigate Pleistocene male migrations to the American continent. In a worldwide sample of 306 men, we obtained 32 haplotypes constructed with the variation found in 30 distinct polymorphic sites. The major Y haplotype present in most Native Americans was traced back to recent ancestors common with Siberians, namely, the Kets and Altaians from the Yenissey River Basin and Altai Mountains, respectively. Going further back, the next common ancestor gave rise also to Caucasoid Y chromosomes, probably from the central Eurasian region. This study, therefore, suggests a predominantly central Siberian origin for Native American paternal lineages for those who could have migrated to the Americas during the Upper Pleistocene.

And what do you know? There was this study proves the bolded text from the above study. That the Caucasoid Y chromosome comes from the central Eurasian region, which is where the Yamnaya came from, who populated Europe around 4500 years ago.

A study of the genetics of Kennewick man from Nature says:

We therefore conclude based on genetic comparisons that Kennewick Man shows continuity with Native North Americans over at least the last eight millennia.

That proves that Kennewick man was of Siberian origins, not European.

To round this up, some people like to say how some of the Natives have myths that a bearded white man came from across the Atlantic and gave them knowledge, who then left, promising to return. Then on the same day he promised he would return, was the day that Hernan Cortez landed at the Yucatan Penninsula. From their myths, they knew that the man was a god, because of his description.

People like to say that this myth proves Europeans crossing the ocean thousands of years ago to give knowledge to Native peoples of the Americas. Ridiculous, and here is why.

When the Aztec Empire fell, they needed a way to explain how their once great empire fell. So they then thought of the myth I described above. That the only way their civilization could have possibly fallen is only because of a god.

In conclusion, the Solutrean Hypothesis has no basis in genetics, paleoanthropology, and even basic common sense. Those who say that the Hypothesis is true are uneducated, and just like Afrocentrists, trying to push an untrue agenda.

Refuting Richard Nisbett

3100 words

The environmental model on IQ in the debate on not only IQ as a whole, but in racial differences in intelligence has led to horrible policies throughout the world. Thinking that we are all equal and everyone is the same and has the same potential as the next person has really hurt us as a society.

Richard Nisbett is of the egalitarian (nurture) side of the debate on the black-white IQ gap. His main research has focused primarily on how laypeople reason and make presumptions about the world. His more recent work has focused on comparing East Asians and Westerners, and how and if they think differently. He says that the arguments have been made that Westerners learn analytically, focusing on the object and its attributes, use its attributes to categorize it and apply rules based on the category to explain and predict behavior. He says that East Asians reason holistically, focusing on the object in its surrounding field. There is little concern with categories or universal rules and behavior is explained on the basis of the forces presumed to be operative for the individual case at that particular time. This goes with white’s higher verbal IQ, as well as going with East Asian’s higher visio-spatial IQ. He says that his lab has found evidence for both.

Now that there is some background on Nisbett and his research interests, let’s turn to the B-W IQ gap.

Rushton and Jensen have refuted him multiple times. In their paper Race and IQ: A Theory-Based Review of the Research in Richard Nisbett’s Intelligence and How to Get It, they provide more than sufficient evidence that the B-W IQ gap, as well as other racial differences in intelligence, are genetic in origin. They propose that the hereditarian model (50/50 genetics/culture) better explains group differences than the culture-only model (0/100 genetics environment). They review 14 topics of contention which are:  (1) data to be explained; (2) malleability of IQ test scores; (3) culture-loaded versus g-loaded tests; (4) stereotype threat, caste, and “X” factors; (5) reaction-time measures; (6) within-race heritability; (7) between-race heritability; (8) subSaharan African IQ scores; (9) race differences in brain size; (10) sex differences in brain size; (11) trans-racial adoption studies; (12) racial admixture studies; (13) regression to the mean effects; and (14) human origins research and life-history traits. They conclude that the preponderance of evidence concludes that differences in intelligence, as well as other life-history traits between the races, are genetic (50 to 80 percent) in origin.

I will be quoting from this article Nisbett wrote for the New York Times, All Brains Are the Same Color.

JAMES WATSON, the 1962 Nobel laureate, recently asserted that he was “inherently gloomy about the prospect of Africa” and its citizens because “all our social policies are based on the fact that their intelligence is the same as ours — whereas all the testing says not really.”

Ah yes. What a great day that was when Watson said that. It needed to be said. The conversation on exactly why Africans are starving needs to be had. No, the answer is not colonialism. It’s intelligence. I’ve touched on how Kanazawa got attacked for stating that Africa’s woes are due to low intelligence, where the average is 70. Just as how Watson got attacked. Except since Watson is more well-known than Kanazawa, the PC crowd attempted to run him out of town. Watson is 100 percent correct with that statement.

Dr. Watson’s remarks created a huge stir because they implied that blacks were genetically inferior to whites, and the controversy resulted in his resignation as chancellor of Cold Spring Harbor Laboratory. But was he right? Is there a genetic difference between blacks and whites that condemns blacks in perpetuity to be less intelligent?

Blacks are genetically inferior to whites in intelligence, on average, just as West African blacks and their descendants are genetically superior in sprinting competitions and East Africans and their descendants are superior in distance running (the same as whites as both have the same muscle fiber type that allows for endurance running). Are we really to think that East Asians are superior nowhere? Clearly ridiculous. We know of Rushton’s Rule of Three, which holds through most all of the variables between races. Yes, there is a genetic difference in intelligence that will condemn blacks in perpetuity to be less intelligent.

The first notable public airing of the scientific question came in a 1969 article in The Harvard Educational Review by Arthur Jensen, a psychologist at the University of California, Berkeley. Dr. Jensen maintained that a 15-point difference in I.Q. between blacks and whites was mostly due to a genetic difference between the races that could never be erased. But his argument gave a misleading account of the evidence. And others who later made the same argument — Richard Herrnstein and Charles Murray in “The Bell Curve,” in 1994, for example, and just recently, William Saletan in a series of articles on Slate — have made the same mistake.

One of my favorite papers, the one that reignited the B-W IQ debate. Titled How Much Can We Boost IQ and Scholastic Achievement, he argues that scholastic achievement and IQ can’t be boosted to any meaningful level. He says prenatal effects such as nutrition, the length of pregnancy, maternal stress and environment within the uterus has an effect on IQ, whereas any postnatal (environmental) explanations have not been found to show a lowered IQ, except those kept in isolation. Let’s see what the ‘misleading account of the evidence’ he is talking about.

In fact, the evidence heavily favors the view that race differences in I.Q. are environmental in origin, not genetic.

See the Rushton and Jensen paper, as well as the Jensen paper I linked above. Any environmental explanation for racial differences in IQ can easily be explained away due to bad study design, or simply not testing the children again at adulthood, as any instances where blacks showed they had higher IQs than whites, they were never tested again at adulthood. Just so happens, that around age 10-12, where most of these tests get administered, is when the racial gap starts to become extremely noticeable. To think that race differences in IQ are environmental and not genetic is laughable.

The hereditarians begin with the assertion that 60 percent to 80 percent of variation in I.Q. is genetically determined. However, most estimates of heritability have been based almost exclusively on studies of middle-class groups. For the poor, a group that includes a substantial proportion of minorities, heritability of I.Q. is very low, in the range of 10 percent to 20 percent, according to recent research by Eric Turkheimer at the University of Virginia. This means that for the poor, improvements in environment have great potential to bring about increases in I.Q.

Rushton and Jensen state in the refutation of Nisbett I linked that non-white samples show the same heritabilities as white samples.There is little evidence of any cultural, or hardship differences, such as being raised as a visible minority, in one group and not the other. If blacks had heritabilities that were consistently lower than white heritabilities, then we could conclude that racism and poverty were responsible for a lower IQ in blacks. Though when a researcher compared black and white IQs on 3 tests, he found that heritabilities were 50 percent in each group. “The heritabilities in the Basic, Primary, and Cattell tests were, respectively: Whites—.61, .37, and .71; Blacks—.75, .42, and .19.”

On Turkheimer, they say that he was right that he did find gene x environment interactions that made genetic influences weaker and shared environment stronger for those from poorer homes in comparison to those from more affluent homes. Though most studies show no interaction effects, or interactions vary significantly.

Other studies have shown that heritabilities are the same both within as well as between white and black samples. That led Jensen to label this the ‘default hypothesis’. Researchers analyzed full and half siblings from the NLSY on three Peabody Achievement Tests. 161 black full siblings, 106 pairs of black half siblings, 314 pairs of full white siblings and 53 pairs of white half-siblings. with measures in math and reading. The best fitting model for all of the data was by which the sources of the sources of the differences between those within race and the differences between races were the same, at 50 percent genetic and environmental. The combined model (50/50)  best explains it, whereas the culture-only and genetics-only models are inadequate.

In any case, the degree of heritability of a characteristic tells us nothing about how much the environment can affect it. Even when a trait is highly heritable (think of the height of corn plants), modifiability can also be great (think of the difference growing conditions can make).

Skewed perception on only working with young children. Heritabilities range from 20 percent to 90 percent from the time someone is a toddler to adulthood. He wouldn’t see the same in adult populations.

There is, for example, the evidence that brain size is correlated with intelligence, and that blacks have smaller brains than whites. But the brain size difference between men and women is substantially greater than that between blacks and whites, yet men and women score the same, on average, on I.Q. tests. Likewise, a group of people in a community in Ecuador have a genetic anomaly that produces extremely small head sizes — and hence brain sizes. Yet their intelligence is as high as that of their unaffected relatives.

Men and women don’t score the same on average. Rushton and Jackson found that on average, men had an edge on IQ tests, scoring 3.63 points higher than women. The people from Ecuador are dwarfs, encephalization quotient explains that, as well as brain size to non-fat mass being most important. Back to Rushton’s Rule of Three, East Asians have bigger brains and more neurons, whites intermediate and blacks last.

About 25 percent of the genes in the American black population are European, meaning that the genes of any individual can range from 100 percent African to mostly European. If European intelligence genes are superior, then blacks who have relatively more European genes ought to have higher I.Q.’s than those who have more African genes. But it turns out that skin color and “negroidness” of features — both measures of the degree of a black person’s European ancestry — are only weakly associated with I.Q. (even though we might well expect a moderately high association due to the social advantages of such features).

Funny. Rushton and Jensen refuted this 2 years before Nisbett wrote this article. Rushton and Jensen say that in certain areas of the Deep South, black IQ is around 70, consistent with the hereditarian explanation of effects of hybridization. An average IQ of 71 was found for all black children in one district, whereas it was 101 for whites (Jensen, 1977). Also, Lynn (2002) and Rowe (2002) analyzed IQ scores for those that are mixed black and white and found scores in between 100 and 85, around 93. Though they do say that evidence isn’t conclusive on the matter. They say Cape Coloreds and African Americans may have better nutrition, or are treated better in society. The Minnesota Study held many variables constant, and still the mixed race children had higher IQs. That supports the genetic hypothesis.

During World War II, both black and white American soldiers fathered children with German women. Thus some of these children had 100 percent European heritage and some had substantial African heritage. Tested in later childhood, the German children of the white fathers were found to have an average I.Q. of 97, and those of the black fathers had an average of 96.5, a trivial difference.

Nope. Doesn’t work that way, Nisbett.

If European genes conferred an advantage, we would expect that the smartest blacks would have substantial European heritage. But when a group of investigators sought out the very brightest black children in the Chicago school system and asked them about the race of their parents and grandparents, these children were found to have no greater degree of European ancestry than blacks in the population at large.

Strawman. No one says there aren’t any smart blacks.

Most tellingly, blood-typing tests have been used to assess the degree to which black individuals have European genes. The blood group assays show no association between degree of European heritage and I.Q. Similarly, the blood groups most closely associated with high intellectual performance among blacks are no more European in origin than other blood groups.

Correct. Though, the studies failed to choose genetic markers with large allele frequencies between Europeans and Africans (Jensen, 1998b pg 480).

A superior adoption study — and one not discussed by the hereditarians — was carried out at Arizona State University by the psychologist Elsie Moore, who looked at black and mixed-race children adopted by middle-class families, either black or white, and found no difference in I.Q. between the black and mixed-race children. Most telling is Dr. Moore’s finding that children adopted by white families had I.Q.’s 13 points higher than those of children adopted by black families. The environments that even middle-class black children grow up in are not as favorable for the development of I.Q. as those of middle-class whites.

“Superior”. I touched on Moore here in my ‘Blank Slate’ article:

Another is the Moore study, which tested 23 black adopted children and 23 black children adopted by middle class black families. Their findings indicated that blacks adopted to black families scored at 104 compared to the blacks adopted by white families who scored at 117. People may point to this and say “Well, they didn’t differ in their environment and not their genes, so therefor the B-W IQ gap is 100 percent environmental.” Ridiculous. As with the other 2 studies, they were not tested again at adulthood. To say that any of these 3 studies mentioned above prove a 100 percent environmental cause is intellectually dishonest.

Not worth talking about either.

Important recent psychological research helps to pinpoint just what factors shape differences in I.Q. scores. Joseph Fagan of Case Western Reserve University and Cynthia Holland of Cuyahoga Community College tested blacks and whites on their knowledge of, and their ability to learn and reason with, words and concepts. The whites had substantially more knowledge of the various words and concepts, but when participants were tested on their ability to learn new words, either from dictionary definitions or by learning their meaning in context, the blacks did just as well as the whites.

Nothing strange here. Blacks have a high verbal IQ in comparison to their visio-spatial. Rushton thought that black rappers and entertainers had high verbal IQs.Also, people from the same Community College would have, on average, around the same intelligence.

Whites showed better comprehension of sayings, better ability to recognize similarities and better facility with analogies — when solutions required knowledge of words and concepts that were more likely to be known to whites than to blacks. But when these kinds of reasoning were tested with words and concepts known equally well to blacks and whites, there were no differences. Within each race, prior knowledge predicted learning and reasoning, but between the races it was prior knowledge only that differed.

That environment can markedly influence I.Q. is demonstrated by the so-called Flynn Effect. James Flynn, a philosopher and I.Q. researcher in New Zealand, has established that in the Western world as a whole, I.Q. increased markedly from 1947 to 2002. In the United States alone, it went up by 18 points. Our genes could not have changed enough over such a brief period to account for the shift; it must have been the result of powerful social factors. And if such factors could produce changes over time for the population as a whole, they could also produce big differences between subpopulations at any given time.

In fact, we know that the I.Q. difference between black and white 12-year-olds has dropped to 9.5 points from 15 points in the last 30 years — a period that was more favorable for blacks in many ways than the preceding era. Black progress on the National Assessment of Educational Progress shows equivalent gains. Reading and math improvement has been modest for whites but substantial for blacks.

Nope. The data that Murray uses only shows those of the same age, which is the accurate model. Others use those from all age groups, skewing the findings due to different heritabilities at different ages.

Most important, we know that interventions at every age from infancy to college can reduce racial gaps in both I.Q. and academic achievement, sometimes by substantial amounts in surprisingly little time. This mutability is further evidence that the I.Q. difference has environmental, not genetic, causes. And it should encourage us, as a society, to see that all children receive ample opportunity to develop their minds.

Yes, interventions from infancy all the way to the end of grade school help, as genes explained only 22 percent of the variance in those at age 5, 40 percent at age 7 and 82 percent at age 18. IQ differences can be changed in younger children, as they are more malleable at younger ages as I have just noted. But as they age, to quote Jensen, their genes “turn on” and fall to the average for that racial grouping.

People like us need to refute these lies that attempt to say that we are all the same and that any and all intelligence and scholastic achievements are environmental in origin and not largely genetic, as Rushton and Jensen have argued vehemently over the years. I will close with the final paragraph of the paper that Rushton and Jensen refuted Nisbett on sums up this situation perfectly:

There is no value in denying reality. While improving opportunities and removing arbitrary barriers is a worthy ethical goal, we must realize that equal opportunity will result in equitable, though unequal outcomes. Expanding on the application of his “default hypothesis” that group differences are based on aggregated individual differences, themselves based on both genetic and environmental contributions, Jensen [59] proposed “two laws of individual differences”—(1) individual differences in learning and performance increase as task complexity increases, and (2) individual differences in performance increase with practice and experience (unless there is a low ceiling on proficiency). We must recognize that the more environmental barriers are ameliorated and everybody’s intellectual performance is improved, the greater will be the relative influence of genetic factors (because the environmental variance is being removed). This means that equal opportunity will result in unequal outcomes, within-families, between-families, and between population groups. The fact that we have learned to live with the first, and to a lesser degree the second, offers some hope we can learn to do so for the third.

People like us need to defend men who are no longer here to defend their work, in Rushton and Jensen. The environmental explanation for IQ, as well as racial differences in IQ, is preposterous. To think that a full environmental causality actually means anything to intelligence is clearly a pipe dream.

“Race is a Social Construct”: Part 1

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“Race is a social construct”. You may hear that a lot from uneducated people. They may say that since the definition of race is ‘ever-changing’, that race doesn’t exist and that it only exists in our minds. They obviously have no understanding of genetics and how we came to be today. If you want to get technical, everything is a social construct. The Universe is a social construct. We’re only giving definitions to what we perceive something to be, so with the logic of ‘race being a social construct’, then everything is a social construct. With that logic, the Universe doesn’t exist because it’s a social construct.

I will look at 3 articles in the first of many articles on this subject. One from Angela Onwuachi-Willig, Bill Nye and Ta-Nehisi Coates. All 3 have extremely wrong views on the biological reality of race, and I will prove that here. I will quote from each article and show how they are wrong with scientific studies as well as point out their bad logic.

I will begin with Angela Onwuachi-Willig. In her article for The New York TimesRace and Racial Identities Are Social Constructs, she says that because of the ever-changing definition of the term race, that it is a social construct and not a biological one.

Race is not biological. It is a social construct. There is no gene or cluster of genes common to all blacks or all whites. Were race “real” in the genetic sense, racial classifications for individuals would remain constant across boundaries. Yet, a person who could be categorized as black in the United States might be considered white in Brazil or colored in South Africa.

Race is not biological, it is a construct. There are no clusters of genes or one gene that is common in blacks or whites. That is correct, but her statement about race being social and not a biological construct is clearly ignorant as I will show below.

i-6505999ec389c9cb434f204f598809d8-race

You can see in the picture above that races clearly do cluster in different clusters from other races. She is right about the changing definitions, especially Brazil, but Brazil is a special case. So much mixing has gone on in Brazil that there is evidence of skin color becoming independent of ancestry. One outlier example doesn’t make race a ‘social construct’. South Africa is also another one. They classify race in South Africa with four categories: black, colored, Indian/Asian or white. Obama would have been called ‘colored’ in South Africa today. But, again, just because there are changing definitions of race throughout the globe, doesn’t mean that race doesn’t exist.

Like race, racial identity can be fluid. How one perceives her racial identity can shift with experience and time, and not simply for those who are multiracial. These shifts in racial identity can end in categories that our society, which insists on the rigidity of race, has not even yet defined.

Is she making an argument for being ‘trans-racial’? I bet Rachel Dolezal would be happy.

In a society where being white (regardless of one’s socioeconomic class background or other disadvantages) means living a life with white skin privileges — such as being presumed safe, competent and noncriminal — whites who begin to experience discrimination because of their intimate connection with someone of another race, or who regularly see their loved ones fall prey to racial discrimination, may begin to no longer feel white. After all, their lived reality does not align with the social meaning of their whiteness.

I always hear about ‘white privilege’ but never get an actual definition of what it means. People complain about ‘white privilege’ because they, of course, don’t understand the biological reality of race. Anything that may prove innate differences between individuals or races they just can’t imagine exists because of what they’ve been taught their whole lives. She is talking about those whites who are in the BLM movement. The false ideals of egalitarianism are the cause of this.

More than 50 years ago, Congress enacted the most comprehensive antidiscrimination legislation in history, the Civil Rights Act of 1964. Half a century later in 2015, the same gaps in racial inequality remain or have grown deeper. Today, the unemployment rate for African-Americans remains more than double that for whites, public schools are more segregated now than they were in the 1950s and young black males are 21 times more likely to be shot and killed by the police than their white male peers. Even a white fourth-grade teacher in Texas, Karen Fitzgibbons, openly advocated for the racial segregation of the 1950s and 1960s on her Facebook page.

Right. IQ is the cause of the unemployment rate of African Americans. Not any imaginary forces such as ‘white privilege’. Public schools are more segregated today due to people wanting to be with others genetically similar to themselves. Blacks cause themselves to get shot and killed by police due to their actions during altercations with police officers. Oh no, someone has not politically correct opinions!! She should lose her job and never work a good job again!

That’s what the Left does. They attempt to shout you down with buzzwords so you can’t calmly and intellectually prove your case.

She is clearly wrong. Good thing this is called an ‘opinion piece’, there were few actual facts in it.

Now to touch on Bill Nye’s views on race. It’s funny. I loved his show when I was a kid. Now, knowing the truth about racial differences, hearing him say that made me lose all respect for him. He’s a mechanical engineer with a Bs from Cornell University. People only take what he says because he is ‘The Science Guy’ when he has no training in what he is talking about.

“We obsess about whether our dog is a pug-Jack Russell terrier mix with corgi overtones and an oaky finish. ‘An approachable little dog,’ whatever. They’re all dogs, okay? And so the idea of a purebred is just a human construct. There’s no such thing – in a sense there’s no such thing as a purebred dog.”

That right there is a fallacy. As with the woman’s article above, they both use the ‘continuum fallacy‘. The continuum fallacy is when someone rejects a vague claim because it’s not as precise as they want it to be. ‘There are no pure races’ or ‘there are no pure breeds of dog’, that doesn’t mean that genes don’t cluster differently, showing genetic differentiation.

“If a Papua New Guinean hooks up with a Swedish person all you get is a human.  There’s no new thing you’re going to get. You just get a human. Japanese woman jumping the African guy, all you get is a human. They’re all humans. So this is a lesson to be learned. There really is, for humankind there’s really no such thing as race. There’s different tribes but not different races. We’re all one species.”

Right. That doesn’t mean there is no such thing as race. Grizzly bears and polar bears can mate to create a prizzly bear. Does that mean species doesn’t exist? (I will touch on speciation at the end of this article.) Once again, that statement doesn’t deny the biological reality of race, as you can see from the picture above.

355d5b456573a2f5552037a86d5fab5b

Researchers have proven, scientifically, that humans are all one people. The color of our ancestors’ skin is a consequence of ultra-violet light, of latitude and climate.

We all belong to the same genus, Homo, but again, that doesn’t disprove race. He’s correct in saying that our ancestors’ skin is a consequence of UV light of latitude and climate, and right there he proves us correct in stating that sunlight differences depending on where your ancestors evolved in the world are the cause of racial differences.

Despite our recent sad conflicts here in the U.S., there really is no such thing, scientifically, as race. We are all one species. Each one of us more alike than different.

In this statement, he is saying the first sentence because of recent racial relations in the U.S. A clear politically-motivated statement.

“Each one of us more alike than different.’ That is correct, but, yet again, doesn’t disprove the reality of race. Geneticists estimate that humans will differ, on average, at 3 million base pairs in their DNA. That’s more than enough for distinct racial classification, as well as enough to differentiate us.

Is that supposed to mean anything? Cats have 90 percent homologous genes with humans, 82% with dogs, 80% with cows, 79% with chimpanzees, 69% with rats and 67% with mice.

90% of the mouse genome could be lined up with a region on the human genome.

99% of mouse genes turn out to have analogues in humans.

As you can see from the links above, we are all extremely genetically related to animals that are clearly extremely physically different from us humans. This shows, that the differences in humans are down to not how genetically distant we are from other animals in the animal kingdom, but how the differing genes we have are expressed.

We all came from Africa. We’re all made from the same star dust. We’re all going to live and die on the same planet – a pale blue dot in the vastness of space. We have to work together!

And finally, you can see his way of saying that racial differences mean nothing because ‘we all come from Africa’ and ‘ we’re all made from star dust’. That may be true, but that doesn’t do anything to acknowledge, or even show that racial differences are meaningless.

Bill Nye has absolutely no authority to speak on this matter. Liberals then eat this up and cite Bill Nye as proof that race doesn’t exist, which is clearly untrue as I have shown.

Finally, I will get to Ta-Nehisi Coates’ ‘The Social Construction of Race‘. He cites my favorite blogger/geneticist Razib Khan, so this should be good.

Ancestry — where my great-great-great-great grandparents are from — is a fact. What you call people with that particular ancestry is not. It changes depending on where you are in the world, when you are there, and who has power.

Right with the first sentence, and with the second. It seems he’s attempting to use what the first article I cited says: that due to ever-changing racial definitions that race doesn’t exist as we believe it to be. He says that ancestry is a fact, well wouldn’t that same ancestry be your racial classification? I am not following his logic. Just because there are differing views on the definition of race throughout the globe, doesn’t mean that there is no biological reality of race.

He cites someone else who states:

“Race” as a term is very nebulous. But human subgroups with similar ancestries can have group differences in DNA — and intelligence is highly unlikely to have no genetic basis at all (although most now believe its impact is greatly qualified by cultural and developmental differences).

Cultural and developmental differences. The cultural differences are thrown out. According to the editorial ‘Mainstream Science on Intelligence’, which came out shortly after The Bell Curve was published, one of their points is that IQ tests are not culturally biased if the individual speaks English. If they are not English speakers, they will either get a test in their native language or get Raven’s Progressive Matrices, which is a ‘culture free’ IQ test as it’s based on pattern recognition and has no writing involved. Developmental differences, yes. White mothers have a better prenatal environment then do black mothers, which is biological. Developmental differences are innate within the two populations.

I do not know. Andrew is more inclined to believe that there is some group-wide genetic explanation for the IQ difference. I am more inclined to believe that the difference lies in how those groups have been treated. One thing that I am not convinced by is controlling for income and education.

Oh, the old ‘Stereotype Threat‘ canard. The paper, which was cited more than 5000 times, states that African-Americans do worse on tests in which they are told that they are being judged on their race. Well, a meta-analysis of 55 published and unpublished studies came out and what was found that the it shows clear publication bias. Either due to people not knowing how to read scientific papers or more insidious tactics. The effect varies across studies and is small. Though elite university undergrads may underperform on tests of cognitive tests due to Stereotype Threat, this effect doesn’t generalize to non-adapted standardized tests, high-stakes settings and less academically gifted test takers. Stereotype Threat cannot explain the difference in mean cognitive test performance between African Americans and European Americans. (pg 68)

In the mid-20th century, as we have been documenting, it was the policy of this country to deny African-Americans access to the same methods of wealth-building, that it was making available to whites.
African-Americans can’t build the same type of wealth due to mean genetic differences in IQ.
This is not merely a problem for your local  diversity and sensitivity workshop. It is a problem of wealth and power. When you create a situation in which a community has a disproportionate number of poor people, and then you hyper-segregate that community, you multiply the problems of poverty for the entire community–poor or not. That is to say that black individuals are not simply poorer and less wealthier than white individuals.  Because of segregation, black individuals and white individuals of the same income and same wealth, do not live in communities of equal wealth.
Wealth and power are directly related to IQ. Communities have a disproportionate amount of poor people due to them being low-IQ, blacks, whites or Asians, you can’t get away from low-IQ being the cause of poverty. People hyper-segregate themselves, see my Genetic Similarity article. Again, segregation is due to GST, not any insidious plot to hold anyone down. I’ve spoken multiple time on how oxytocin is responsible for ‘racism’ (ethnocentrism), and everything he is saying is just proving my point.
What bearing does segregation have on IQ differential? I don’t know. My skepticism of genetics is rooted in the fact that arguments for genetic inferiority among people of African ancestry are old, and generally have not fared well. My skepticism is also rooted in the belief that power generally seeks to justify itself. The prospect of actual equality among the races is frightening. If black and white people truly are equal on a bone-deep level, then the game might really be rigged, and we might actually have to do something about it. I think there’s much more evidence of that rigging, then there is evidence of cognitive deficiency .
There is no evidence for racial/ethnic equality. Any ‘evidence’ is shoddy and has NOT and NEVER WILL BE replicated because any studies that show racial equality are either badly administered or they never do a follow-up study at adulthood.  Blacks and whites are not equal ‘on a bone-deep level’. What kind of statement is that? There is WAY MORE evidence for cognitive deficiency than for ‘rigging’ of the game to hold blacks down in America.
I must add that I can not pretend to be a dispassionate, nor impartial observer. I come from a particular place. I’ve now been out in the world, and seen how other people in other places live. They don’t strike me as more intelligent. They strike me as better armed. There’s nothing scientific about that. But I think we all have core faiths. These are mine. You’ve been warned.
“My personal anecdotal experiences mean more to scientific studies on race and intelligence.” Where have we all heard that before? Too bad your ‘core faiths’ are just that Mr. Coates: FAITH.

Regardless of the method used in the analyses, all researchers reached estimated very close to that obtained by Lewontin: The differences observed by the subdivisions (populations, groups of populations, races) represented 10 to 15 percent of the total genetic variation found within the human species. Formally, these findings demonstrate, first, that the species is indeed subdivided into genetically definable groups of individuals and, second, that atleast some of these groups correspond to those defined by anthropologists as races on the basis of physical characters. They do not however, settle the arguments regarding the methods of racial classification. Unfortunately, Lewontin did not specify before initiating his analysis how large the difference has to be in order to call the groups “races”.

Consequently, the results of the studies have led population geneticists to two diametrically opposite conclusions. Lewontin called the observed differences trivial, and proclaimed that “racial classification is now seen to be of no genetic or taxonomic significance” so that “no justification can be offered for its continuance.” This view is echoed by authors of similar studies, who seem to be surprised that genetic variation within populations is greater than that between them. By contrast, Sewell Wright who can hardly be taken for a dilettante in questions of population genetics, has stated emphatically that if differences of this magnitude were observed in any other species, the groups they distinguish would be called subspecies.

One can extend Wright’s argument even further. The more than 200 species of haplochromine fishes in Lake Victoria differ from each other much less than the human races in their neural genes, although they are presumably distinguished by genes that control differences in their external appearances. The same can be said about atleast some of the currently recognized species of Darwin’s finches and other examples of recent adaptive radiations. In all these cases, reproductively isolated groups are impossible to tell apart by the methods used to measure differences in human races. Obviously, human races are not reproductively isolated (interracial marriages are common and the progenies of such marriages are fully fertile) but the external differences between them are comparable to cichlid fishes and Darwin’s finches. Under these circumstances, to claim that the genetic differences between the human races are trivial is a more political statement than a scientific argument. Trivial by what criterion? How much difference would Lewontin and those who side with him consider non-trivial?

By mixing science with politics, geneticists and anthropologists are committing the same infraction of which they are accusing other scientists, who they themselves label as racist. Even worse, by labelling the genetic differences as insignificant, they play into the hand of genuine racists who can demolish this claim and so further their own agenda. It is intellectually more honest to acknowledge and then point out that by no means imply supremacy of one race over others. This can be done by demonstrating that the differences are in genes that cannot be linked to any features that would be required for the preeminence of a particular race.

It’s clear that racial classification does exist. The creator of Fst, Sewall Wright, says that a Fst distance of .15 is more than enough for speciation (differing racial classifications). It directly refutes Lewontin, who put his political ideology of Marxism over science. Those cichlids in Lake Victoria are a perfect example that though the definition of ‘species’ does change depending on which researcher you speak to, it doesn’t discount that there are real and physical genetic differences between races and ethnicities.

In conclusion, the term “race is a social construct” is a deliberately intellectually dishonest statement, or a statement used to hide the truth for more insidious things to happen due to the non-acknowledgement of race.

Refuting Afrocentrism Part 2: Are Italians Black?

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Afrocentrists like to say things like ‘Italians were black’ and ‘the Romans were black’ and ‘The Moors were black’. All of this is based on shoddy evidence and uneducated people not knowing what they’re talking about.

In this article by an Afrocentrist, he claims that ‘Italians were black’ and talks about ‘dark-skinned Sicilians’.

Southern Italians were considered “black” in the South and were subjected to the Jim Crow laws of segregation. They weren’t allowed to marry “whites.” It was difficult, damn near impossible.

They were designated as “black” on census forms if they lived in the South and that is because the majority of them were dark-skinned Sicilians.

No idea what he’s talking about. In America at the time, Northern Italians said that Southern Italians were of a different race due to a slightly different look. Well, genetic testing shows similarities between the Northern and Southern Italians which I will get to later.

First off, it’s not only Sicilians who are ‘dark-skinned’. It’s all of Southern Europe.

UV rays

The map seen above is a map of UV rays that Europe and parts of North Africa get. Notice how North Africa and Southern Europe get the same amount of UV rays. That’s the cause of the difference in appearance between the North and South of Italy.

Mass lynchings happened to them often.

Mass lynchings happened to everyone often, not just blacks and Italians. Lynchings happened to anyone who raped, murdered, or did any other heinous crime. 27 percent of those lynched between the years of 1882-1968 were white. It wasn’t only a ‘black problem’.

One of the biggest mass lynchings happened to Italians in New Orleans when they thought that a Italian immigrant had killed a “white” police officer.

Right. It was the biggest mass lynching ever in the history of the US, 11 Italians got lynched. But, what he says about the cause being ‘killing a “white” police officer’ is unfounded. They got lynched for killing the police officer, not because they were of ‘another race’.

The very few Northern Italians that immigrated here perpetuated the myth that Southern Italians and Greeks were of a different race than them in order to save their own asses. This wasn’t true, and there are actually dark-skinned Italians all over Italy, not just in the South, as well as light-skinned Italians all over Italy.

But it is true. The differences between Northern and Southern Italians are embellished due to political reasons. There are dark-skinned Italians who live in Italy but are not genetically Italian/Greek. Yes, light-skinned ‘all over Italy’, those in the North are more Germanic, while the South has slight admixture  from North Africa and sub-Saharan Africa. Though, some from the South migrated to the North and vice versa. The amount of non-white admixture in Italians is less than that of the average for Europe:

Combined data from two large mtDNA studies provides an estimate of non-Caucasoid maternal ancestry in Italians. The first study sampled 411 Italians from all over the country and found five South Asian M and East Asian D sequences (1.2%) and eight sub-Saharan African L sequences (1.9%). The second study sampled 465 Sicilians and detected ten M sequences (2.2%) and three L sequences (0.65%). This makes a total of 3% non-white maternal admixture (1.3% Asian and 1.7% African), which is very low and typical for European populations, since Pliss et al. 2005, e.g., observed 1.8% Asian admixture in Poles and 1.2% African admixture in Germans. (Plaza et al. 2003; Romano et al. 2003)

Similar data from the Y-chromosome reveals Italians’ even lower non-Caucasoid paternal admixture. Both studies obtained samples from all over the mainland and islands. No Asian DNA was detected anywhere, but a single sub-Saharan African E(xE3b) sequence was found in the first study’s sample of 416 (0.2%), and six were observed in the second study’s sample of 746 (0.8%). The total is therefore a minuscule 0.6%, which decreases to 0.4% if only Southern Italians are considered and 0% if only Sicilians are considered. Again, these are normal levels of admixture for European populations (e.g. Austrians were found to have 0.8% E(xE3b) by Brion et al. 2004). (Semino et al. 2004; Cruciani et al. 2004)

An analysis of 10 autosomal allele frequencies in Southern Europeans (including Italians, Sicilians and Sardinians) and various Middle Eastern/North African populations revealed a “line of sharp genetic change [that] runs from Gibraltar to Lebanon,” which has divided the Mediterranean into distinct northern and southern clusters since at least the Neolithic period. The authors conclude that “gene flow [across the sea] was more the exception than the rule,” attributing this result to “a joint product of initial geographic isolation and successive cultural divergence, leading to the origin of cultural barriers to population admixture.” (Simoni et al. 1999)

These studies show the opposite of what Afrocentrists, and even Nordicists say.

The reason I say very few is because over 80% of Italian immigrants were from Southern Italy (Sicily, Abruzzo, Calabria, Campania, Sardinia, Naples, etc.)

Correct, and as seen above with those 3 studies, neither of them are ‘African’. My grandmother was born in Calabria. She looked like any other normal Italian woman you see on the street. These people take their ideas from movies, take genetics information from a movie like True Romance and attempt to say that all Italians are ‘black’ or ‘African’ or ‘Moorish’.

It was highly unlikely (damn near impossible) for a Southern Italian to own a slave because they were seen as the same as blacks, and at the time, they were the second (right behind blacks) most discriminated against group.

Too bad Italians started coming to America in the 1870s. What he states doesn’t even make sense because blacks owned slaves way disproportionately than whites. 4.8 percent of Southern whites, along with the North, being 1.3 percent of all whites in America in 1860 owned slaves. There are reports from New Orleans from their 1860 census that showed 3000 freed blacks owned slaves, accounting for 28 percent of the city’s population.

In 1860 Louisiana, at least 6 blacks owned more than 65 slaves, with the biggest number of slaves being 165 slaves who worked on a sugar plantation. So even if Italians were looked at as ‘black’, as you can see, blacks themselves had no problem owning slaves, and actually did it more than whites did right before slavery ended.

A lot of this confusion comes from the race of the Moors. The Moors are a Caucasoid Muslim group from North Africa. People hear ‘Africa’ and automatically think sub-Saharan Africa. Well, the Moors were Cacausoid for one. 2, as I have shown above, the amount of Moorish/Berber admixture is minute in Italians. 3rd, I will show now that the Berbers are not sub-Saharan African.

You have other Afrocentrist websites who talk about so-called ‘black Moors’. Well, the Moors were Berbers and Arabs, who are Caucasian.

Berbers live in groups scattered across NorthAfrica whose origins and genetic relationships with their neighbours are not well established. The first hypervariablesegment of the mitochondrial DNA (mtDNA) control region was sequenced in a total of 155 individuals from three Tunisian Berber groups and compared to other North Africans. The mtDNA lineages found belong to a common set of mtDNA haplogroups already described in NorthAfrica. Besides the autochthonous North African U6 haplogroup, a group of L3 lineages characterized by the transition at position 16041 seems to be restricted to North Africans, suggesting that an expansion of this group of lineages took place around 10500 years ago in NorthAfrica, and spread to neighbouring populations. Principal components and the coordinate analyses show that some Berber groups (the Tuareg, the Mozabite, and the Chenini-Douiret) are outliers within the NorthAfrican genetic landscape. This outlier position is consistent with an isolation process followed by genetic drift in haplotypefrequencies, and with the high heterogeneity displayed by Berbers compared to Arab samples as shown in the AMOVA. Despite this Berber heterogeneity, no significant differences were found between Berber and Arab samples, suggesting that the Arabization was mainly a cultural process rather than a demographic replacement.
berbers
berberchildwh1
Above are pictures of modern day Berbers.
Here is what Afrocentrists would like you to believe they looked like:
Moors3
Which has no basis in genetics or history. The Moors were Caucasoid and not African.
In conclusion, Italians aren’t black, nor were they ever looked at as black. Southern Europe getting the same UV rays as Northern Africa explains the slight appearance differences between Northern and Southern Italians as well as being greatly embellished due to political reasons. Genetic testing shows lower non-European admixture than average for Europe.
On the Moors: Genetic testing shows North African admixture, as well as Arabization being a cultural process and not a demographic replacement.

Response to Daily Stormer article “Black Africans are Genetically Closer to Bonobos Than to White Humans”

UPDATE:

I emailed one of the researchers and I got this response:

Hello,

you are of course absolutely right. Bonobos are equally distant to
people from Africa, Europe or anywhere else in the world.

The X/A ratio measures something completely different: It compares
individuals from a certain group (bonobos, chimpanzees, or human
populations) and compares how different the X autosomes in this group are from the autosomes (the non-sex-chromosomes, i.e. everything that is not X and Y). Since each generation you have three X chromosomes per four autosomes (XX for the mother + XY from the dad = three X), you would
expect that the ratio should be 3/4 (thats why there is a dashed line at
0.75 in the plot). But there are many ways in which this measure could be nudged off this expected value. That Europeans look different in this measure could for instance be explained by later waves of primarily male migrants out of Africa that mixed with people in Europe, but there are other ideas as well.

I am really sorry to see that the plot is misconstrued as evidence for racist ideas. Hope this helped to clear up what is meant with this plot.

Cheers

 

 

lolindeed

‘lol’ indeed. Learn how to read scientific papers

800 words

Seems to be a bit of misinformation going on because of this piece so I thought I’d clear it up.

Andrew Anglin claims that the Nature article says that blacks are genetically close to bonobos than to Europeans.

That couldn’t be more wrong.

First off, the article doesn’t talk about any type of actual genetics being closer to anything that is mentioned in the article. What the article is talking about is social and sexual behaviors.

Although they are similar in many respects, bonobos and chimpanzees differ strikingly in key social and sexual behaviours and for some of these traits they show more similarity with humans than with each other.

It also says:

We find that more than three per cent of the human genome is more closely related to either the bonobo or the chimpanzee genome than these are to each other.

And? Is that supposed to mean anything? Cats have 90 percent homologous genes with humans; 82% with dogs; 80% with cows; 79% with chimpanzees; 69% with rats and 67% with mice.

90% of the mouse genome could be lined up with a region on the human genome.

99% of mouse genes turn out to have analogues in humans.

We  share 97.5 percent of our DNA with mice.

So we see here that we share a lot of DNA with other animals, as well as animals also sharing similar amounts of DNA, which shows that it comes down to how genes are expressed and not the amount of genetic distance between the 2 animals being tested.

Looking to the image on top of the article, it shows the X/A ratio between Europeans, Africans, the Pan Ancestor and Bonobos.

The X/A ratio is the ratio between the number of X chromosomes and the number of sets of autosomes in an organism. It’s used primarily to determine sex in some species, such as the drosophila flies.

A simple reading of the text above and below this chart that was referenced to supposedly show that Africans are genetically closer to Bonobos will show you that it’s talking about the X/A ratio, not about genetics.

 

Differences in female and male population history, for example, with respect to reproductive success and migration rates, are of special interest in understanding the evolution of social structure. To approach this question in the Pan ancestor, we compared the inferred ancestral population sizes of the X chromosome and the autosomes. Because two-thirds of X chromosomes are found in females whereas autosomes are split equally between the two sexes, a ratio between their effective population sizes (X/A ratio) of 0.75 is expected under random mating. The X/A ratio in the Pan ancestor, corrected for the higher mutation rate in males, is 0.83.

Similarly, we estimated an X/A ratio of 0.85 (0.79–0.93) for present-day bonobos using Ulindi single nucleotide polymorphisms in 200-kb windows.

Under the assumption of random mating, this would mean that on average two females reproduce for each reproducing male. The difference in the variance of reproductive success between the sexes certainly contributes to this observation, as does the fact that whereas bonobo females often move to new groups upon maturation, males tend to stay within their natal group.

Here’s the main point:

Because both current and ancestral X/A ratios are similar to each other and also to some human groups (Fig. 4), this suggests that they may also have been typical for the ancestor shared with humans.

Talking about the X/A ratio, not genetics.

Here is the text below Fig. 4:

The X/A ratios for Ulindi (bonobo), an African human and a European human were inferred from heterozygosity, and that for the Pan ancestor was inferred from ILS. The low X/A ratio for the European has been suggested to be due to demographic effects connected to migrating out of Africa30. Errors, 95% confidence interval

I hope this clears up anything about this article.

What it’s saying is, is that the X/A ratios for the bonobo, African and European were gathered from heterozygosity, for the Pan ancestor is was gathered from incomplete lineage shortages (ILS).

Low X/A ratio for the European doesn’t mean more genetic distance from Africans or bonobos.

Response to Daily Stormer article on the Paleo Diet

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So I came across this Daily Stormer article a while ago and had a nice laugh. The article is full of misconceptions, half-truths and other things that will have the person who is not knowledgeable about nutrition think that what he says in that article is right. This will be a pretty lengthy article, and I will also touch up on 5 reasons why the Paleo diet is pathetic.

For those who do not know, the paleo diet comes from the word “paleolithic.” The idea is that our bodies are evolved to eat the hunter-gatherer diet, and that the introduction of grain (though necessary for the development of civilization) was a negative health development which our bodies have never fully adapted to.

Nothing really wrong here, except where he says “our bodies are evolved to eat hunter-gatherer diet”. As far as I know, there is no literature that Europeans “evolved to eat a hunter-gatherer diet” and to say that a certain group of people needs to eat these types of food has no foundation in science. Actually, there is WAY more variability in the way that individuals process healthy and unhealthy foods. Noted in this study are:

  • High interpersonal variability in post-meal glucose observed in 800-person cohort
  • Using personal and microbiome features enable accurate glucose response prediction
  • Prediction is accurate and superior to common practice in an independent cohort
  • Short-term personalized dietary interventions successfully lower post-meal glucose

In today’s issue of Cell, two groups led by Eran Elinav and Eran Segal have presented a stunning paper providing startling new insight into the personal nature of nutrition. The Israeli research teams have demonstrated that there exists a high degree of variability in the responses of different individuals to identical meals, and through the elegant application of machine learning, they have provided insight into the diverse factors underlying this variability.

This right here refutes what he says, but I’ll just go through the article and pick it apart.

In particular, grains contain gluten and antinutrients which can irritate your digestive system.

This is so wrong that I don’t even know where to start.

According to this RD (someone who I would listen to, over some blogger), she says that they have “incredible health benefits” and “In many cases, they’re the very same components that are thought to give beans, lentils, whole grains, vegetables, and fruits their well-documented disease-fighting powers. In fact, you may know these “antinutrients” by another name – “phytonutrients,” the highly-prized, health-boosting compounds that we celebrate in whole foods.”

In the end, she says you don’t need to worry about antinutrients as long as you eat a varied diet of nutrient-rich whole foods.

Now on to gluten. In this study, it was found that each treatment diet, whether it contained gluten or not, prompted subjects to report a worsening of gastrointestinal symptoms to similar degrees. Even when the placebo diet was identical to the baseline diet, the people still reported a worsening of symptoms. They could find absolutely no specific response to gluten.

Basically, you want to think if something would have been available to you in the natural habitat of Europe, where we are tuned to live.  Or if it is nutritionally identical or very similar to something that would have been in our natural environment in Europe.  If it is not, it is probably no good.

Source?

There is no limit to how much you can eat.  You should eat until you are full, even if you are overweight.

Asking for eating disorders and weight gain here.

If you are using this diet to lose weight and so are going very low carb, you still must once a week eat carbs to let your body know you are not in a state of starvation. If you go more than a week without eating any carbs, your metabolism will slow drastically, which causes you to keep the weight on.

Half right. No idea where he gets “you still must once a week eat carbs to let your body know you are not in a state of starvation”, but you do need to have a carb refeed once a week.

Lowering carb intake does lower leptin, which is a fat-burning hormone. But it won’t be enough to cause “your metabolism to slow drastically, which causes you to keep the weight on.”

The number one thing here is to avoid all grains.  These include bread, pasta, cereals and so on.  Even if you believe I am too extreme in believing these things are negative, there are, even based on nutritional information everyone agrees on, options far better than these.

Good advice, but you don’t need to consistently avoid them. Pareto Principle (80/20 rule). Just be on point 80 percent of the time and you’re fine.

Pork is the second worst, but also the most delicious.  I eat a lot of pork.  It is a solid meat.  One thing to beware of is that sausages often contain filler which contains gluten.  As you become more natural in your eating habits, even small amounts of grain can cause a serious reaction, so one should be aware of traces.

No.

One can also live on cold cuts alone, though this could get pricey.

Please. Talks about health, yet tells people to eat processed cold cuts.

Fish is good, though larger fish can contain mercury, which you may or may not be concerned about.

“May or may not be concerned about.”

You should be concerned about it. A 200-pound man should eat no more than 3 cans of tuna per week.

Beans are not a vegetable even though some of them get called vegetables, and in my opinion there is no reason to eat them, ever. They are difficult to digest and can cause stomach complications which cause your body to use up energy dealing with them. Energy that would best be spent elsewhere. They also contain antinutrients which leach nutrients from other foods in your digestive system.

Already went over antinutrients above, but he says they “leach nutrients from other foods in your digestive system” which is bullshit. Beans are not difficult to digest (because of fiber). Good thing his “opinion” doesn’t mean anything.

Black beans are a super food and cooking beans remove most of the supposedly bad antinutrients.

To quote Dr. John Berardi:

Research suggests that the benefits of legumes outweigh their anti-nutrient content.Cooking eliminates most anti-nutrient effects, and some anti-nutrients (like lectins) may even be good for us.

Benefits far outweigh the (supposed) negatives of beans.

And there is no reason to eat them.  Yes, they do contain protein, but it is a protein much inferior to meat protein, so there is just no logical point in indulging in beans. 

Seriously? By “inferior to meat protein” I assume that he means that beans aren’t a complete protein. Combining beans and rice makes a complete protein, complimenting the other food with the missing essential amino acids to make the complete protein.

Currently, our markets are filled with fruits which are imported from tropical climates, and are thus not natural to us.

No basis in science.

something that Southeast Asians and Americans Indians are adapted to but we are not.

What?

I suppose it should go without saying, but I will say it anyway: all processed foods should be avoided completely.  This includes not only grain-based processed foods such as breads, cereals and pastas, and obviously sugary or salty processed snacks, but also processed meats such as hot dogs and processed “microwave dinners.”

>Eat cold cuts

>Don’t eat processed foods

What?

Alcohol is generally unhealthy if consumed in high amounts, but I understand well that for most men a certain amount is necessary.

Consuming alcohol once a week is fine. See here.

For the same reason it makes you tired, it also have a devastating effect on your metabolism and testosterone levels.

Nope. From the above link:

In a 3 week study, which had men and women consume 30-40 grams of alcohol per day showed a 6.8 percent test reduction in men and none for women. That’s 3 beers a day for 3 weeks, one night of drinking is not harmful.

120 grams of alcohol (10 beers) will lower test levels by 23 percent for 16 hours:

For alcohol to significantly lower testosterone, you need to do some serious drinking. ~120 g alcohol, the equivalent of 10 beers, will lower testosterone by 23% for up to 16 hours after the drinking binge. If you drink so goddamn much that you are admitted to the hospital, you get a similar effect with a reduction of about -20%.

By “have a devastating effect on your metabolism”, I assume he means that when you consume alcohol, your body takes priority to metabolize the alcohol (alcohol is the 4th macronutrient, which the general public doesn’t know about) and fat burning is put on hold until all alcohol is metabolized from the body.

On the Paleo Diet as a whole, I trust John Berardi, Ph.D. and CSCS (Certified Strength and Conditioning Specialist). He says that the Paleo diet “has some flaws”, such as the evolutionary arguments not holding up. He says that incorporating the foods that Anglin is telling you not to eat would likely be a big improvement.

For most people to be on a strict diet where they are barred certain foods, they will most likely not stick to it.

I could have just linked that PNAS article along with the individualized diets are best and supported by the study that came out the other day, but debunking most of the stuff he said had to be done, because people see things like this and automatically think it’s right (I noticed he included no references in his article).

Listening to people who have no background in nutrition is a recipe for failure. Listening to what he says as a basis for dietary habits is ignorant. His reasoning is ignorant. If you want to eat Paleo, go ahead, but if it’s for the reasons included in his article, his main argument does not hold up as seen in the PNAS and Cell articles.

“Differences in brain structure development may explain test score gap for poor children” Maybe not….

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Summary: They would have to explain why whites in poorer families score higher than blacks in all other income brackets except blacks in families making over 200,000 dollars per year, which even then blacks only score 3 points higher in the 200,000 dollar plus per year income bracket. They say that frontal and temporal lobes are smaller in poorer children, which whites have bigger frontal and temporal lobes on average as well as having more activity in the frontal lobes, which is thought to be the seat of intelligence. Blacks having smaller brains than whites on average explains the size differences between the differences in the different parts of the brain mentioned.

Low-income children had atypical structural brain development and lower standardized test scores, with as much as an estimated 20 percent in the achievement gap explained by development lags in the frontal and temporal lobes of the brain, according to an article published online by JAMA Pediatrics.

If true, only 20 percent of the achievement gap affected by poverty. I doubt it seeing as poor whites in families making less than 20,000 dollars a year still have 180 points over blacks in the same income bracket and blacks in families making more than 200,000 dollars per year have a 981 score, only 3 points higher than whites in families making less than 20,000 dollars per year. Whites in families making less than 20,000 dollars per year still outperform all negro families in all other income brackets except in negro families making over 200,000 dollars per year by only 3 points. Not even worth talking about.

20 percent of the achievement gap is apparently explained by developmental lags in the front and temporal lobes of the brain. Blacks have a smaller PFC (prefrontal cortex) which may explain it.

Socioeconomic disparities in school readiness and academic performance are well documented but little is known about the mechanisms underlying the influence of poverty on children’s learning and achievement.

It’s well documented that even in poor whites and poor blacks, poor whites are still on average more intelligent and have higher standardized test scores than do blacks.

Seth D. Pollak, Ph.D., of the University of Wisconsin-Madison, and colleagues analyzed magnetic resonance imaging (MRI) scans of 389 typically developing children and adolescents ages 4 to 22 with complete sociodemographic and neuroimaging data. The authors measured children’s scores on cognitive and academic achievement tests and brain tissue, including gray matter of the total brain, frontal lobe, temporal lobe and hippocampus.

I wonder what the breakdown was. Will revise when the full paper comes out. Hippocampal differences are also explained by whites having larger brains than blacks as well as the other parts they state.

The authors found regional gray matter volumes in the brains of children below 150 percent of the federal poverty level to be 3 to 4 percentage points below the developmental norm, while the gap was larger at 8 to 10 percentage points for children below the federal poverty level. On average, children from low-income households scored four to seven points lower on standardized tests, according to the results. The authors estimate as much as 20 percent of the gap in test scores could be explained by developmental lags in the frontal and temporal lobes.

On frontal lobes from this Rushton paper decimating Gould’s garbage *Mismeasure of Man*, which also states they have smaller frontal lobes than whites:

“Bean also reported that the 103 Negro brains were less convoluted than were 49 White brains and that Whites had a proportionately larger genus to splenium ratio (front to back part of corpus callosum), implying that Whites may have more activity in the frontal lobes which were thought to be the seat of intelligence. Consider the following statistically significant comparisons (sexes combined) from recently conducted studies using the four techniques mentioned above. Using brain mass at autopsy, Ho et al. (1990) summarized data for 1,261 individuals. They reported a mean brain weight of 1,323 grams for White Americans and 1,223 grams for Black Americans. Using endocranial volume, Beals et al. (1984) analyzed about 20,000 skulls from around the world and found that East Asians, Europeans, and Africans averaged cranial volumes of 1,415, 1,362, and 1,268 cm3 respectively. Using external head measurements from a stratified random sample of 6,325 U.S. Army personnel, Rushton (1992) found that Asian Americans, European Americans, and African Americans averaged 1,416, 1,380, and 1,359 cm3, respectively. Using external head measures from tens of thousands of men and women from around the world collated by the International Labour Office, Rushton (1994) found that Asians, Europeans, and Africans averaged 1,308, 1,297, and 1,241 cm3, respectively. Finally, an MRI study in Britain found that people of African and of Caribbean background averaged a smaller brain volume than did those of European background (Harvey et al., 1994). Contrary to most purely environmental theories, racial differences in brain size show up early in life. Data from the U.S. National Collaborative Perinatal Project on 19,000 Black children and 17,000 White children showed that Black children had a smaller head perimeter at birth and, although Black children were born shorter in stature and lighter in weight than White children, by age 7 ‘catch-up growth’ led Black children to be larger in body size than White children. However, Blacks remained smaller in head perimeter (Broman et al., 1987). Further, head perimeter at birth, 1 year, 4 years, and 7 years correlated with IQ scores at age 7 in both Black and White children (r = 0.13 to 0.24).”

On temporal lobes, from the scientist that Rushton cited above:

“The size of the pole of the temporal lobe is less in the Negro than in the white, and less in the Negro female than in the Negro male…The shape of the pole of the temporal lobe is different in the two races, being slightly more slender in the Negro, and almost the same size in the two races antero-posteriorly. The differences are not only absolute but are also relative to the to the weight and size of the entire cerebral hemispheres.”

.”Development in these brain regions appears sensitive to the child’s environment and nurturance. These observations suggest that interventions aimed at improving children’s environments may also alter the link between childhood poverty and deficits in cognition and academic achievement,” the study concludes.

“Appears sensitive”. I doubt it. See the table on test scores and income above.

In a related editorial, Joan L. Luby, M.D., of the Washington University School of Medicine, St. Louis, writes: “Building on a well-established body of behavioral data and a smaller but expanding body of neuroimaging data, Hair et al provide even more powerful evidence of the tangible detrimental effects of growing up in poverty on brain development and related academic outcomes in childhood. … In developmental science and medicine, it is not often that aspects of a public health problem’s etiology and solution become clearly elucidated. It is even less common that feasible and cost-effective solutions to such problems are discovered and within reach. Based on this, scientific literature on the damaging effects of poverty on child brain development and the efficacy of early parenting interventions to support more optimal adaptive outcomes represent a rare roadmap to preserving and supporting our society’s most important legacy, the developing brain. This unassailable body of evidence taken as a whole is now actionable for public policy.”

I’m assuming they have never seen the SAT score gaps and how whites in families making less than 20,000 dollars a year score the same as blacks in families making over 200,000 dollars a year.

Source

It’s well-known that IQ is the best predictor of success in life. Blacks have a lower a lower average IQ, and smaller brains than whites and Asians which explains the achievement test score gaps. I would like to see a study that separates rich blacks, rich whites and poor blacks and poor whites with controls and see how they differ. I’m assuming it’ll be the same as the SAT score gaps which I have linked above.

The difference in brain size between blacks and whites perfectly describe what is being shown above. More blacks live in poverty because they have lower IQ. IQ is correlated with poverty, crime, illegitimacy, and chronic welfare status. G, or general intelligence, is highly correlated with most things in life. Excerpt from THE g FACTOR The Science of Mental Ability by Arthur Jensen:

The well-established correlation of IQ and similar cognitive measures with a number of social variables, such as poverty, crime, illegitimacy, and chronic welfare status, makes it almost a certainty that g itself is the major cognitive component in the relationship. However, I have not found a study that directly addresses the extent to which just g itself, rather than IQ or other highly gloaded measures, is related to social variables. The repeated finding that verbal test scores are somewhat more highly correlated with delinquent and criminal behavior than are nonverbal performance tests (generally loaded on the spatial factor) suggests that other cognitive factors in addition to g are probably responsible for the correlation of IQ with these most common forms of antisocial behavior. pg 294

In conclusion, they need to have studies that have poor whites and poor blacks, rich blacks and rich whites, rich whites and poor blacks, poor whites and rich blacks and controls to see what the differences really are, and we know there will be differences between the above-mentioned groups, and that poor whites still perform better academically than poor blacks.

Dailymail: Mixed-race relationships are making us taller and smarter: Children born to genetically diverse parents are more intelligent than their ancestors.

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The Dailymail came out with an article today about how mixed race relationships are making us taller and smarter. It also says that children born to genetically diverse parents are more diverse. While this is true, they fail to realize certain other things when it comes to mixed race relationships/mixed race kids.

A study has found humans today are taller and more intelligent than their ancestors, and the cause has been linked to the rise in more genetically diverse populations.

Wrong. People are taller and more intelligent today because of better nutrition. That has NO link to genetically diverse populations. Of course they’re more intelligent than their ancestors. Back in 1945, only 70 years ago, in America the average white IQ was 85. We are smarter than out ancestors back then, is it genetic diversity? No, it’s not.

Miscegenation lowers IQ in the white but highers it for the black. It’s a net loss for whites to miscegenate but a net gain for a lower race to miscegenate with whites. People don’t realize the host of problems that come with being mixed-race which I will explain later in this post.

And those born to parents from different races and cultures also tend to have higher levels of education.

I don’t know where they got their information from so I can’t quite comment on this part of the article. I’m assuming it’s in the paper, but I can only find the abstract, not the PDF. With some anecdotal examples, I know some mixed race black and whites with a white mother, they both were high achievers in high-school as well as in sports. The brother plays for the NFL while the sister plays pro women’s basketball in Europe. That’s just one anecdote, though, I live in a small rich town so of course we would have a few outliers. I wouldn’t use that as a basis to say emphatically that yes, mixed race kids do tend to have higher levels of education. We know that Asians have a higher level of education slightly more than whites who have a level of education way more than blacks. As said in the Minnesota Transracial Adoption Study, mixed black and white kids have a better prenatal environment.

According to Rushton and Jensen in THIRTY YEARS OF RESEARCH ON RACE DIFFERENCES IN COGNITIVE ABILITY, at the end of the study the researchers came to the conclusion that in their breakdowns of the children at age 17, that the mother’s IQ was the single best predictor of the adopted child’s IQ when all other variables are accounted for, but then go on to say:

“the social environment maintains a dominant role in determining the average IQ level of Black and interracial children and that both social and genetic variables contribute to individual variations among them” [pg 259]

Levin and Lynn (1994) then disputed Weinberg et al’s conclusion with a hereditarian alternative. That the average IQ and school achievement scores of the black children directly reflected their amount of African ancestry. At both age 7 and 17, the adopted children with 2 black parents had lower average IQs and worse school achievement tests than those with one black parent and one white parent. So right here, in the MTAS, it shows that mixed-race people DO score better than just blacks, which is attributed to their white ancestry. Weinberg et al responds to their claims with this:

Waldman, Weinberg, and Scarr (1994) responded to Levin (1994) and Lynn (1994) with further regression analyses that indicated the children’s preadoptive experience was confounded with racial ancestry, and so an unambiguous interpretation of the results was not possible. [pg 259]

So we have evidence of mixed race children being better with IQ as well as school achievement tests than full black children. That’s all I’ll say on this matter for now until this full paper comes out.

Where few instances of this occur in a person’s genes, it indicated greater genetic diversity in their heritage and the two sides of their family are unlikely to be distantly related.

Sure, that’s right. But I don’t believe it’s worth the downsides to doing it.

The team found that greater genetic diversity is linked to increased height. It is also associated with better cognitive skills, as well as higher levels of education.

Funnily enough, they just described mono-racial children. As I said earlier, miscegenation lowers IQ, so along with that, it would lower education levels as well.

The only traits they found to be affected by genetic diversity are height and the ability to think quickly.

Yes. “Height” doesn’t really have to do with genetic diversity. Intelligence is negatively affected, if it’s not a white mother. White mother’s have a better prenatal environment than minority mother’s which leads to a higher IQ, as well as the white mother’s IQ being one of the best tells for a child as I said earlier in the piece. “Ability to think quickly” is directly linked to cognitive ability.

However, genetic diversity had no effect on factors such as high blood pressure or cholesterol levels, which affect a person’s chances of developing heart disease, diabetes and other complex conditions.

Wrong. Let’s take a mixed race black and white. Blacks have a higher propensity to have hypertension (elevated blood pressure), diabetes and heart disease. That’ll obviously be passed on to a mixed race kid.

The findings suggest that over time, evolution is favouring people with increased stature and sharper thinking skills but does not impact on their propensity for developing a serious illness.

But it’s not mixed race people who this is talking about, it’s mono-racial people. Talking about the propensity for developing serious illness, it’s true that mixed race people are more heterozygous than non-mixed, but the small boost to disease immunity isn’t worth the slew of health effects.

Source

In Brazil, there’s evidence that ancestry is becoming independent from skin color due to so much mixing. Rio de Janeiro is one of the most dangerous cities in the world with its murder rate. The article, of course, doesn’t mention that, nor does it mention this.

For years now, the Brazilian city of Rio de Janeiro has made just about every short list for the world’s most violent and dangerous cities. Plagued by violent gun crime, assassinations and drug-trafficking, nearly 50,000 people have died of crime-related violence in Rio between 1978 and 2000.

The city’s crime problem was put on display once again this year for the entire world to see during the annual Carnaval celebration in Rio. Despite the deployment of nearly 10,000 police officers, the festivities were still marred by unusally high instances of robbery, assault and violence. Crime has been an embarrassment for Rio, placing the city’s bid to host the 2016 Summer Olympics in jeopardy.  Source

Let’s get to some health problems that plague mixed race kids, which along with for instance the dangers of so much mixing as seen in places like Rio De Janerio, that this article didn’t mention.

Mixedrace children suffer from more health problems

A study on Black-White mixes in agreement found that ”When it comes to engaging in risky/anti-social adolescent behavior, however, mixed race adolescents are stark outliers compared to both blacks and whites.” This holds true despite being raised in similar environments to mono-racial children.

The more people of different backgrounds who produce offspring = the more types that are harder to match. Multiracial patients have uncommon profiles and since there can be many possible racial and ethnic combinations in multiracial societies, finding a match can still be extremely difficult.

If you become ill with a blood cancer or other disease that requires a stem cell transplant, here’s an uncomfortable fact: Your race matters. Diversity is a strength in much of life, [citation needed!] but it’s a curse when finding a stem cell donor match.

Why things like this weren’t put into the article and only one side was shown, the “good side” of mixing races, beats me. Oh, yea, they have an agenda they’re pushing and want all of us to believe that there are no racial differences.

It’s funny. Far leftists will adamantly say that race doesn’t exist or IQ isn’t real or that IQ tests are biased, yet when things come out that are “positive” for minorities, these same people who were saying so much that those things don’t exist, they all of a sudden start to say things like that. I thought race doesn’t exist, why would they point to examples like this? Things like that boggle my mind how you can say somethings like that so much but then switch up your tune when the opposite is shown to be proven. We need to recognize racial differences.

Without doing so, we will lose our country. I hope we realize this soon before it’s too late and we lost the land we call home. I have hope though that a paradigm shift is coming soon and it will be a rude awakening to those cultural Marxists. The lies they spread in the name of “equality” and “egalitarianism” are simply lies to any intelligent person willing to educate themselves and always ask questions no matter what they’re told. Everyone should always ask questions. That’s how you learn and how you to not become like the mindless drones who just repeat like a parrot what they’re taught.

Always ask questions, especially about issues such as this. The fact that the author of this article didn’t even put one mention of the negatives of being mixed race shows that there is an ideological bias behind the piece and that they don’t want you to know any more than what they’re telling you.