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Yearly Archives: 2016
Differential K Theory, GFP, and the Evolution of Conscientousness
There is a link between higher IQ and higher ability to be more conscientiousness, which is then linked to the GFP or General Factor of Personality. Two meta-factors were identified beyond the Big Five Personality Traits and were described as ‘Plasticity’ and ‘Stability’ (Deyoung et al, 2002). To quote from the paper:
Stability subsumes Conscientiousness, Emotional Stability (the reverse ofNeuroticism), and Agreeableness, and refers to the extent to whichan individual is consistent in motivation, mood, and social interac-tions. Plasticity encompasses Extraversion and Openness to experi-ence, and refers to the extent to which a person actively searchesfor new and rewarding experiences, both intellectual and social.
Why did this evolve, especially so closely with high intellect? It evolved due to conscientiousness, which is defined as being thorough, careful, and vigilant.That, coincidentally enough, being needed to survive the harsh winters of Northern Europe and Siberia. Higher conscientiousness was is also another reason for the formation of European societies. With being able to be more conscientious, this, along with genetic pacification, is yet another cause for the cucking of Europe.
When our ancestors trekked out of Africa and into Siberia and Northern Europe, they needed differing abilities than those peoples who stayed in the more tropic climates. Those in the tropics, for the most part, could just lounge around all day. With food being readily available, there was really no pressing need to “save” or “partition” their findings (as Africa is full of mostly a hunter-gatherer societies). Conversely, in Europe and Asia, with harsh conditions in regards to their environment, which made food less plentiful than in the tropics, this meant that more cooperation was needed. Due to more cooperation being needed to survive, planning ahead (abstract thought) evolved to better help cope with the harsh environment.
The aforementioned factors in Europe and Asia then led to the higher rates of altruism seen today. Ancient Europeans needed to be thorough, vigilant and careful. Whether they needed to be careful with food storage, how much to eat, whether or not to help someone, etc, it’s clear that conscientiousness evolved with higher intelligence. Since higher intelligence is correlated with those three things involving conscientiousness, they evolved hand in hand, as selection only selects for good traits and discards the ones that aren’t useful.
These two environments that the three races evolved in then set the stage for what’s going on today. With Africans’ lack of conscientiousness, this leads to them not being vigilant, careful or thorough. This can be seen with how sloppy they are when they commit crimes. Their ancestral environment wasn’t conducive to conscientiousness, and in turn, higher IQ, so they evolved without the need of conscientiousness, as their societies (for what they are), function ‘well’ in their perception. This is yet another reason why that we cannot live together. Differing evolutionary strategies lead to these causes, yet we still think we can acclimate some peoples into society when their biology says otherwise.
JP Rushton proposed that the GFP (General Factor of Personality) and IQ were linked. It was found that the correlation between the GFP and IQ was -.23. The higher the g score, the higher the individual scores on the GFP. He noted that the correlation is so low, possibly due to the restricted range of the sample. Rushton provided an argument for the evolutionary process of cognitive ability and personality evolution. This is clear evidence of the GFP and IQ evolving hand-in-hand.
Lacking a high IQ, and therefore, lack of g, Africans (as well as other colored peoples), on average, have lower g and therefore lower conscientiousness, which then is a huge cause for crime. Increases criminality has been correlated, like I’ve said here before, to a lower verbal intelligence. Though this higher IQ and higher GFP is being taken advantage of. Ever since that fateful day in 1964, white pathological altruism has been taken advantage of. Those with lower conscientiousness know they can take advantage of those with higher IQ and higher conscientiousness as altruism is correlated highly with IQ and conscientiousness.
There is an altruistic personality; the altruist has a high IQ, is conscientious, and altruistic. Those on the opposite end of the spectrum, however, take advantage of that and this can be seen with the political climate around the world in regards to other races’ feelings towards whites.Conversely, those who are r-selected and have a low IQ and GFP tend to be more hostile and commit acts of aggression. This multiculturalism due to the altruistic personality on a large scale is one of the reasons for the mass immigration into Western societies. People, as a whole, become collectively altruistic. Then, those lower IQ, lower GFP, r-selected people then take advantage of the more altruistic people. They then turn into a parasitic entity, sucking the host dry before moving on to their next victim.
This is where a high GFP does not work, in multicultural societies. Of course, this is also due to ethnic dissimilarity, but the other thing I brought up along with that ethnic dissimilarity are all of the negative effects of those that are r selected, which, in their own societies is ‘fine’, but to who are K-selected and have complex societies, those behaviors are archaic.
There is a high correlation between low IQ, low conscientiousness, archaic actions, lack of abstract thought, and lack of society building. We know that those with lower IQs commit more crime on average than those with higher IQs.
This is why allowing non-Western people who are abnormal to our societies is a bad move, since they don’t share the same evolutionary track, and therefore, due to differing selections due to evolution, evolved differing behaviors to better adapt to the climate, environment, and surroundings.
Since skin color and IQ correlate at -.92, meaning the darker the skin the lower the IQ and vice versa, this is yet another great assessor on whether an individual has a high IQ, and is, therefore, conscientious. This is due to, as I alluded to earlier, the environment in sub-Saharan Africa being conducive to lack of ability to delay gratification. Due to that, we can infer, on average, whether someone will be conscientious or not. Since their ability to delay gratification is impeded due to certain evolutionary pressures not put on Asians and Europeans, they will, therefore, be less conscientious, which is a crucial building block in maintaining a successful society.
The research of Templer and Arikawa (2006) supports Rushton’s contention that higher intelligence develops in colder climates. They used 129 countries with primarily indigenous populations (Asia, Africa, and Europe) and correlated culture fair IQ (Lynn & Vanhanen, 2002) with temperature. In addition to temperature, Templer and Arikawa used skin color provided by an anthropology book (Biasutti, 1967). IQ correlated .92 with darker skin color, .76 with winter highs, .66 with winter lows, and .63 with per capita income. Previous research by Meisenberg (2004) reported a correlation of .89 between skin reflectance and IQ in a similar study. Templer (2010b) found a correlation of .96 between skin color measure used by the Templer and Arikawa and the one by Meisenberg’s skin reflectance. Because both measures were independently determined using different methodology, they both can be regarded as highly valid measures of skin color.
The cause of these two variables being correlated is evolution. They paired well with each other, so over tens of thousands of years, they got selected for with each other and persisted to today. Since we have modern societies and are much more civil (some of us), we can better see these differences in personality as well as behavior. There are This General Factor of Personality was able to evolve due to evolution in cold climates. The altered intelligence, which then altered personality. This r/K Life History Theory of Rushton’s blend’s beautifully with the GFP and intelligence. Evolution in cold climates along with genetic isolation caused differing evolutionary trajectories for each race/ethnicity which led to differences in crime, IQ, socioeconomic status, sexual maturity, cultures, and so on. There are clear genetic differences brought on by the environment (due to evolution), which leads to differing societies based on differing evolutionary trajectories. Conscientiousness and intelligence, the whole General Factor of Personality, evolved in Northern populations since it was more beneficial in that environment, and to survive, this is what done to adapt to the environment through natural selection, which then led to racial differences.
Germany Begins to (Slightly) Wise Up: Will Begin IQ Testing “Migrants”
800 words
Haaretz reported today that Germany was going to begin IQ testing on the ‘migrants’ to assess where talent and what occupational groups that they could put them in. This is a slightly positive change with all of the negativity this past year.
The mean IQ of Arab countries is 84 (Templer, 2010). With around 1.2 million ‘refugees’ coming from land and sea, assuming a SD of 15 (seeing as Arabs are Caucasian, I’ll assume a SD of 15), 50 percent of them fall at or below 84. So 600k at 84 or below. 16 percent fall at 100. 192k fall at 100. 12k at 120 and 1,680 fall at 130. 50 percent fall below 84. In America the average IQ for a repeat criminal is 85. With an IQ of 85, you can see that criminality begins to increase. This is due to lack of abstract thought(linked to verbal ability), which has them not think of the consequences of their actions before they act. At or below 85 is 1 in 6, 68% of the population is within 1 SD of 100, and 2.5% of people are 130 or more.
I can’t find any data on Arab testosterone at the moment, so I’ll just assume that it’s higher than Europeans due to the Arabs’ closer proximity to the equator (someone correct me if I’m wrong), as that’s why African’s testosterone is high. Due to higher average testosterone combined with low IQ, this leads to increased aggression along with increased sex crime, which is a cause for some of the sex assaults on European women by Muslim men. I can’t find anything on terrorist IQ, the closest I can find is how the FBI convinced a man with an IQ of 51 to attempt terrorist acts, though that’s an extreme case. Since low IQ is correlated highly with lack of abstract thought, it was easier for him to become convinced to do it. Like in most organizations, the more intelligent ones are at the top so they tell the lower IQ ones what to do. Though, by administering these tests, they will greatly lower their chances for another terrorist attack, seeing as those actions are correlated with low IQ.
In a study on prison inmates, IQ predicted inmate misconduct. Using a sample of 2500 inmates over 30 institutions from August 2004 to June 2006, it was found that those inmates who had higher IQs were involved in fewer incidents as well as being less likely to commit violent behavior. Verbal intelligence has been posited to be some of the cause for increased crime, seeing as verbal IQ is correlated with delinquent behavior, which is due to lack of abstract thought being correlated with lower IQ. With higher testosterone being correlated with low IQ and increased androgen sensitivity along with higher sperm counts (both are indicators of higher testosterone) being correlated negatively when measured by speed of neuronal transmission which causes a trade-off between g (general intelligence) and neuronal transmission, this shows that increased testosterone means decreased IQ. This is also seen with how higher IQ people have a lower sex drive.
I did say in my article Non-Western People are Abnormal for Our Society, that, as the title says, non-Western people are abnormal for our society due to not sharing our cultural values, which, we know is genetic. Though, higher IQ individuals will be better able to acclimate into society, as well as have a decreased proclivity to commit crime.
Since there are some evoultionary reasons for suicide bombings due to increased inbreeding this increased genetic similarity between them which led to increased altruism due to genetic similarity, by allowing those with higher IQs, this will lead to a greatly increased chance for attacks to happen as higher IQ people are better at controlling impulses.
This is a move I agree with. All countries should implement this procedure (obviously not enough to where it begins to displace the native population). With there being a cut-off limit on IQ, lets say 105 or even 110, that guarantees a high chance of those who are immigrating will be of value to the country and bring something to the table instead of the current situation with the benefits they currently receive (and lets be honest, you know these rules aren’t being followed). So by implementing this policy not only in Germany, but around the world, this would be a great thing for the West, to restrict immigration only to high-skilled workers, with a background check, intelligence test and someone with good credentials. Of course, only in sectors that really need the help. I of course advocate for the natives of any country to have first dibs when it comes to getting a job.
All in all, this is good move because a) rapes will be lessened and b) there won’t be as much individuals on welfare because there will be an (assumed) moratorium on those with lower IQs, leaving the higher IQ ones to find jobs and contribute to the economy.
Smoking and Race
1000 words
From the brands of cigarettes people use to the types of cigars people use, there are variations by race. There are also death rate differences and variations in how peoples of different race and ethnicity. I will explore causes for these points as well.
From the CDC’s Fact Sheet on tobacco use, we can see that Marlboro (41 percent), Newport (12 percent), Camel (8 percent), Pall Mall (8 percent) and Pyramid (2 percent) were the leading cigarette brands.The percentage of those older than 12 years old who smoked menthol was 19.1% black, 3.6% Asian, 7.8% ‘Hispanic’, and 6.5% white. You can see this looking in any majority black area how younger blacks most always smoke menthols, mainly Newport and sometimes Kools.
In my experience, that has been the case. For whites, the main brand was either Marlboro menthol, Marlboro lights or Marlboro Reds. For Asians it was most always Parliament Lights; they sure do love those.
Exposure to Newports in their environments may also be a factor for blacks smoking more menthol cigarettes, such as Newports. I have already written a bit about the media’s involvement in both a positive and negative way that affects behaviors, so by seeing more advertisements for a certain kind of cigarette, they’ll be more likely to smoke the brand that they constantly see, see their parents or siblings smoking (though, controlling for that in the study, they were still able to recognize Newports) smoking or ones that they can take that are lying around. Regardless of race, those students who were able to recognize Newports were more likely to begin at follow up even after controlling for other risk factors. In this sample of 1179 students (this was a longitudinal study), non-recognition of Camel and Marlboro did not predict smoking at follow-up. These factors also involve what I will discuss below.
Caraballo (1998) found that serum cotinine levels were higher than blacks than they were in whites. That is, blacks have higher levels of the metabolized form of nicotine in their bodies than do whites called ‘cotinine‘. This causes blacks’ bodies to absorb more nicotine than whites due to these biological factors. This may possibly explain why blacks have more cases of lung cancer and are less likely to quit smoking.
Non-‘Hispanic’ blacks also have two times the amount of cotinine than do Mexican and white Americans. This affects how much of the nicotine absorbed into the blood stream as well as the increase in getting cancer.
The differences in cancer disparity between blacks and whites cannot be explained by the preference for a certain brand of cigarette. Though, that study does confirm that those who smoke more mentholated cigarettes take deeper inhales. So in a way, in can be said to be explained by brand preference, since those who smoke menthol cigarettes inhale more deeply than those who smoke a non-mentholated brand.
Ross et al (2016) discovered that blacks take more pulls per cigarette and take deeper, longer pulls than do whites, which is the cause for the disparity in lung cancer between blacks and whites (along with higher cotinine levels in blacks, that’s the cause for more nicotine absorption). Deeper inhales means more nicotine is being absorbed into the body and therefore cotinine levels increase.I could also see personality differences playing a factor in who takes longer pulls and more pulls of a cigarette as well.
Higher testosterone levels may also be correlated with lung cancer. With blacks having more testosterone on average than whites, we can see how these disparities in hormone levels between the races are the cause for differences in acquisition of disease rate between the races. It seems that all cancers have high correlations with increased testosterone, not just prostate. Higher testosterone is associated with many negative variables, and of course, blacks have some of the most negative health effects. This is a combination of genes x environment. Their environment is more conducive to menthol cigarettes (mostly Newports), blacks also are more extroverted, which means they’re more sociable and therefore can coerce each other to do things, such as smoking (lower IQ also plays a part in coercion).
In teenage populations, whites showed a higher use for tobacco smoking and marijuana usage, but in the 20s, blacks and ‘Hispanics’ are more likely to pick up the habits, while whites drop off. The researchers conclude that we need to better understand why these substance abuse behaviors exist, which differing personality traits (due to testosterone, as well as that being a factor for smoking more) are part of the cause for it in my opinion.
Higher free and total testosterone was found in males, even after stratification of age, BMI, triglycerides and alcohol consumption were controlled for. This shows why more blacks and ‘Hispanics’ are represented more in the statistics after age 20, as that’s when genetics takes full effect, so they would be over-represented in these cases.
With the higher natural cotinine levels, this causes blacks to absorb more nicotine; they take more pulls per cigarette as well as deeper pulls. Along with smoking cigarettes with more nicotine and more tar in them, this is a cause for the disparity between blacks and whites and how it involves the acquisition of lung cancer. Higher testosterone individuals turn to smoking more than lower testosterone individuals.
I theorize that part of the reason for deeper pulls of a cigarette as well as more pulls is correlated with the amount of testosterone one has, which is then correlated with how extroverted they are. Extroversion leads to being around others more often, which, even with desegregation, there has been less integration, not more. Since they’re more likely to be around each other due to desegregation not ending people segregating by race, they then are exposed to others who are smoking as well as doing other thing that younger children with higher testosterone will be more likely to do, in part because of exposure to the new stimulus along with higher testosterone which leads to more impulsive behavior.. This leads to one of many social and biological variables that lead to blacks having higher rates of lung cancer due to smoking.
Rushton’s Differential K Theory and Intelligence
1250 words
This is my 50th post. I plan to cover altruism and ethnocentrism much more extensively, intelligence and race, psycological disease and race, personality and race and so on. A JayMan refutation on his belief of Ethnic Genetic Interests and Group Selection is coming soon.
Differential K Theory explains human behavior on countless variables. Rushton was one the first scientist to use r/K selection theory to explain human behavior. Albeit, it’s on a much smaller scale than what r/K selection theory is used for, it still greatly applies to human populations. On over 6o variables, Blacks (r selected) fall at one end of the continuum, whereas Orientals (K selected) fall at the other end, with Caucasians falling in the middle, consistently on over 60 variables.
Rushton’s theory, which of course is extremely controversial, drew the ire of many researchers because he chose to use r/K selection theory in regards to explaining differences between isolated human populations. But, what they don’t get is this: even if Rushton is wrong with his Differential K theory, you’d still need a way to explain how and why these 3 populations who evolved in genetic isolation came to have such stark and consistent differences between them no matter where in the world you look. Between IQ (Asians 103-4; Europeans 100 and African-Americans 85), rates of 2-egg twinning (Asians 4 for every 1000, Europeans 8 for every 1000, and Africans 16 for every 1000 [I wrote about some evolutionary causes for earlier menstration in black girls with leptin being the cause last week, it also explains how and why blacks have more rates of 2-egg twinning, because the life expectancy is so low it lead to evolutionary pressurses in which Africans evolved mechanisms that have them have more kids and earlier menstral cycle.]), average age of first walk (blacks one week before whites who are one week before Asians) and so on and so forth.
The more intelligent the woman, the less kids she will want to have, on average. One SD increase in childhood IQ leads to a 21-25 percent decrease in a woman’s odds to have children. This is a clear k strategy. Have less kids, but show more care for them. Conversely, blacks have more kids, but show less paternal involvement. This still continues today in America, even though we have an industrial society. The things involved in this discussion are, at its core, evolution in its process, but we don’t see it that way because of the society we live in. With intelligent women not having children, while the less intelligent, r selected women having more children, this will lead to an eventual dysgenisis and the average IQ in America will drop immensely. The birth rate for whites in America was 1.75 in 2013, 2.0 for blacks, 2.4 for Hispanics and 1.77 for Asians. The r/K selection theory perfectly explains this. It’s an evolutionary process that occurs as adaptations to the environment that their ancestors evolved in.
The more K selected peoples had to have less children in their ancestral environment due to food being scarce. This is where the higher intelligence evolved as well as high amounts of altruism, which I theorize that Europeans have a higher amount of the brain hormone oxytocin, which leads to more altruism, but is being taken advantage of by non-Western people who are abnormal to our societies and eventually leads to increased ethnocentrism.
Rushton postulates that Differential K Theory is a cause for group differences in intelligence. With the K selected having less children and the r selected having more, we can see this in birth trends as well as IQ trends in the country. More intelligent women can see that it’d be better to wait to have children as to be more financially able to take care of the baby. While the less intelligent women have more kids due to not thinking into the future and only thinking in the now, wanting their pleasure immediately. This is causing a dysgenic effect on America. The more the average intelligence drops, the more we can expect to see conditions begin to deteriorate, as a country is only as good as the majority of its population, as we can see looking at the average living conditions around the world.
With altruistic behavior being highly correlated with race due to evolution for tens of thousands of years, we can see that more intelligent people will be more law abiding, more altruistic, commit less crime, have less children, more cooperative and so on. Whereas less intelligent people will be less law abiding, less altruistic, commit more crime, have more children and be less cooperative. This is modeled in communities all over America in their crime rates, graduation rates, IQ scores and so on. One of the most noticeable r selected traits is criminality. This is due to lack of ability to delay gratification and higher testosterone. The reverse is true for lower testosterone Eurasians, as well as numerous other differing physiological factors between the races.
With differing crime rates no matter where you look, the racial disparities are all the same. This is no coincidence; social factors do not account for these differences. Evolution, does, though. Evolution is also the cause for the social factors that do arise within and between populations. Over time, these selection pressures caused these positive traits you see in those who evolved in the Northern, harsher, colder climates and the more negative traits in the more Southerly, hotter climates.
This is why importing more people from differing societies into our countries will not end well. They have differing evolutionary goals different evolutionary reasons for them. They are not normal for our societies; ethnic dissimilarity causes more strife between people due to genetic distance. The more r selected peoples are less intelligent and are also prone to higher rates of criminality as well due to higher testosterone.
These differing evolutionary goals would be fine; if not for forced mass immigrations into Western countries. Since birth rates never remain stable, a lower white birth rate would have been able to rebound eventually. With the introduction of mass immigration into Western Societies, this is causing the birth rate to continue to fall percentage wise, even if the birth rate increases due to the introduction of more r selected peoples. The more educated that women get, the less kids they have. It works in two ways: going for higher degrees and wanting success in life first, some women may be around their mid 30s before deciding to have children and when deciding to have children they then can’t have any due to being too old. Whereas those with lower intelligence and no career prospects would have been having more kids due to their r selected strategy and lack of foresight into the future due to lower intelligence.
Looking at things from an evolutionary lens will make what’s going on around the world clear. Everything you see is evolution in action, though you won’t see its effects and they won’t be noticeable for tens of thousands of years, it’s evolution on a macro level. Negative birth trends always do reverse, as they never remain stable. As more women get educated (and this holds for all women of every age, race, religion and ethnic group), birth rates drop due to less time in the fertile years attempting to get pregnant. If we’d only look at things in an evolutionary scientific way, we’d be better able to understand how and why human populations differ behaviorally cognitively, and a whole multitude of other factors. We’d also better understand how to, on average, deal with the average criminalistic personality. It would make the job for police easier if these evolutionary factors were known and life would be safer in America as a whole.
Leptin and its Role in the Sexual Maturity of Black Girls
1200 words
There are differences between the races in regards to sexual maturity. I’ve recently discovered that leptin plays a role in black female teenagers and how they reach puberty earlier than that of white women. This is one out of many variables that prove Rushton’s three-way rule; that the races all differ on certain variables, on average.
Buried in the middle of this article on black and white differences in body composition, it is observed that the mean concentration of leptin was significantly greater in black girls than that of white girls. The authors conclude that leptin may play an important role in the accelerated growth and sexual maturation of black girls:
In one study, the mean leptin concentration was significantly (P < 0.01) higher in 57 black girls (15.0 ± 10.1 μg/L) than in 79 white girls (8.4 ± 11.1 μg/L) aged 8–17 y (58). The authors concluded that this difference might play an important role in the accelerated growth and sexual maturation of black girls. Likewise, Nicklas et al (57) reported a strong correlation between leptin and %BF for both black (r = 0.71, P < 0.0001) and white (r = 0.61, P < 0.001) obese postmenopausal women, but a significant racial difference in leptin concentration (36.0 ± 4.8 compared with 45.8 ± 3.5 μg/L; P < 0.05), respectively. Additionally, leptin correlated with resting energy expenditure in black women (r = 0.58, P < 0.001) but not in white women (r = 0.08).
Also noticed, was that leptin correlated with REE (resting energy expenditure) in black women but not in white women.
Wong et al (1998), state that because African-American girls tend to be heavier and mature faster than white girls, then there must be a leptin difference between the 2 groups. He measured serum leptin concentrations in 12 hour fasted blood samples of 57 black and 79 white girls. Body comp was measured by x-ray absorptiometry, sexual maturity by doctor examination and physical fitness by treadmill testing. What was found was the serum leptin levels were positively correlated with body fat, maturation and insulin were higher in the black girls after controlling for age. Differences remained, but lessened, after controlling for differences in fat mass, maturation, and physical fitness. This is more proof that leptin is a cause for the role in earlier sexual maturity in black girls.
Kaplowitz (2008) observed in his paper Link Between Body Fat and the Timing of Puberty that several studies have shown that the increase in earlier sexual maturation may indeed be linked to the obesity epidemic which started to rise around 50 to 60 years ago when fat was demonized as “being the cause for fat gain”, all the while carbs were championed as a better macro over fat. Of course, we now know that’s not true, but the implications of this lie that we’ve been told are now coming out in the light 50 years later.
Kaplowitz states that the evidence currently out points to obesity being a cause for earlier puberty in girls, rather than earlier puberty causing an increase in body fat (hmmm.. where have I heard confusing one cause for another before? Oh yeah, the IQ/obesity cause).Many studies have come out that show, in both rats and humans, that leptin may be the critical link between body fat and earlier puberty. Both mice and humans who were deficient in leptin both failed to reach puberty unless leptin was administered. Though he says that leptin may end up playing a permissive, rather than a critical metabolic signal that initiates puberty. But with the data of black girls having higher leptin levels, leptin being correlated highly with body fat, and both being highly correlated with increased sexual maturation show the reason why black girls are a) more sexually active at a younger age and b) why black girls mature faster than white girls. He finally concludes that it makes evolutionary sense that body fat and reproduction in girls evolved as a mechanism to ensure that pregnancy won’t occur unless adequate levels of body fat are had.
This is yet more biological evidence for physiological differences between the races.
When fat mass increases, so do leptin levels. When leptin levels increase, puberty happens earlier. Black girls have higher rates of leptin, which correlates highly with body fat and sexual maturity, so it’s no surprise that we see these factors here.
What are evolutionary causes for this? Well, seeing Africa’s life expectancy, the rate of parasitic load as well as disease in Africa, and Rushton’s r/K Life History Theory, which states that those more r selected will have more children and show less care for them whereas those more k selected will have less progeny, but show more care, we can easily see how such a mechanism evolved. With IQ being highly correlated with life expectancy (at .95), we can see part of the reason why, for instance, black women evolved to have higher rates of 2 egg twinning in comparison to whites (16 per 1000 for blacks, 8 per 1000 for whites and 4 per 1000 for Orientals). To quote Rushton from Race, Evolution, and Behavior:
Black women, compared to white women, average a shorter period of ovulation and produce more eggs per ovulation in addition to all the other characteristics in Table 1.1. As mentioned, the rate of dizygotic twinning, a direct index of egg production, is less than 4 per 1,000 births among Mongoloids, 8 per 1,000 among Caucasoids, and 16 or greater per 1,000 among Negroids. (pg. 6)
This is a clear evolutionary strategy. Have more children where it’s less hospitable and there is a higher mortality rate, especially for young children and have fewer children where it is more stable and less environmental dangers are around.
Even more evidence to bolster my claim is how 82 percent of black women are overweight or obese (Ogden et al, 2014). Genes related to obesity have also been found in African-Americans, as well as European-Americans, which also bolster the claim seeing as the genes were found to do the same things in both populations. To quote myself from the above article:
In a study published back in 2013, researchers were looking for obesity genes in African Americans. The study, which involved more than 70,000 men and women of African descent, they were able to identify 3 SNPs that were associated with obesity and BMI in the sample population. What was also found, was that those same genetic sequences also heighten rates of obesity in peoples with no African ancestry, all of the genetic variants associated with obesity were also found in European populations. The same genes found in African populations did the same in European populations, and vice versa.
To quote the paper:
We provide evidence for a shared genetic influence on BMI across populations, as we found directionally consistent associations with the majority of known BMI risk variants. This observation suggests that bioloically functional alleles are ancient and probably arose before migrations out of Africa. In addition, we were able to refine the window of association of some of the previously established BMI loci, which may eventually help identify the biologically functional variant(s).
BMI is also between .75 and .82 heritable. These factors show yet another cause for black women’s high obesity rates, of course with eating garbage food all of the time, but I’m talking about evolutionary reasons.
There are reasons why black girls mature faster than white girls, it’s because of leptin, which precedes an increase in body fat to prepare for eventual menstruation and higher body fat helps to protect the baby in vitro. R/K Life History Theory then shows the evolutionary reasons why earlier sexual maturity happens for black girls over white girls: lower life expectancy.
Down-low Bruthas
1000 words
There is an epidemic: Down low bruthas. Down low bruthas spread diseases in all of the populations, which also spread them in our normal populations. Because black culture is “manly”, this forces a lot of those gay blacks to be down low. They then have secret relationships with men, who they then get diseases such as AIDS from, then pass it on to their wives or partners. When they lead these secret lives and don’t inform their partners about their real preferences, this then causes these degenerate diseases to come into our populations, and unknowing people may then become infected due to no previous knowledge that the individual has a disease.
A Gallup poll from 2012 shows that blacks represent the biggest part of the ‘LGBT’ community, with 4.6 percent of black Americans identifying with the label. Also noted, was that 4.2 percent of ‘Hispanics’, 4.3 percent of Asians and 3.2 percent of Caucasians identified as ‘LGBT’. They also say that poverty is involved:
the LGBT population has a larger proportion of non-white people and clearly is not overly wealthy,
We know that their IQs are innate and that when black and ‘Latino’ IQs are matched at 100, many racial differences disappear. So we can say, with reason, that if a black or ‘Latino’ is poor, they have a higher chance of being gay than whites.
This phenomenon has been noted in the recent years, and many are not telling their wives, which is having diseases slip into the normal population. Black males are over-represented in all STD cases around the country. “African Americans comprised approximately 6 percent of the population in CA, but represented about 19 percent of chlamydia cases and 28 percent of gonorrhea cases in CA.African American chlamydia rates were more than 5 times higher than those of non-Latino whites. African American gonorrhea rates were more than 6 times higher than those of non-Latino whites. African Americans had more cases of AIDS and syphilis, which affect predominately gay men and other men who have sex with men, than would be expected, based on their population size.These disparities are evident throughout most of the state, regardless of geographical location.” This is due to them not using condoms. OIder black men with HIV have sex more without condoms, which is yet another way these diseases get spread.
A number of blacks who go to prison come out gay. This is proof that it’s environmental, a choice, and not genetic. A meta-study was done on homosexuality, looking over 10,000 papers from 20 years and from both sides of the debate. What was found was, that homosexuality doesn’t have a genetic component, that significant environmental factors are attributed to homosexuality. Twin studies prove that it is environmental, seeing as identical twins share 100 percent of their genes, then if homosexuality were genetic, both twins would be gay 100 percent of the time. This is not the case, seeing as both twins are gay only 11 to 14 percent of the time. Quite the blow to the genetic hypothesis of homosexuality. Seeing as there is no ‘gay gene’, we can pretty much rule out a genetic hypothesis.
Blacks high rate of sexual encounters continues in prison as well. Even when confined to prison, their testosterone still urges them to fulfill their desires, and if there are no white women around to rape/sexually assault, they then do the same to the prisoners on the inside.
Then when they get released, these diseases then come out into the normal population due to the person not telling their wife or girlfriend of their escapades in prison.
“Bruthas” become down low because, in black “culture”, it’s seen as non-masculine to be gay (funny, seeing as they have the highest chance to be gay). Blacks contribute to the proliferation of diseases in two ways: a) by being down low bruthas and b) by sleeping with white women who then unknowingly spread diseases to the white population.
Rushton observed that skin melanin is highly correlated with many variables, HIV/AIDS included. This same pattern continues in America, showing that it’s not only environmental factors that lead to these diseases but that their promiscuity is genetic, as discussed in Race, Evolution and Behavior:
Testosterone may order many of the racial differences, for it has been related to self-concept, temperament, sexuality, aggression, and altruism, in women as well as in men (Baucom, Besch, & Callahan, 1985; Dabbs, Ruback, Frady, Hopper, & Sgoutas, 1988). In a study of 4,462 U.S. male veterans, where extensive archival records were available, Dabbs and Morris (1990) found testosterone correlated with reports of childhood delinquency, adult delinquency, drug use, alcohol abuse, military misconduct, and having many sex partners. Testosterone is also involved in the development of secondary sexual characteristics such as muscularity and depth of voice (Haeberle, 1978; Hudson & Holbrook, 1982) as well as the organization and structure of the brain. (p. 271)
Since these same factors exist anywhere blacks are in the world, as I have spoken about many times before, we chose our environment based on our genetics. Since blacks anywhere in the world have these high rates of disease, it’s obviously due to their sexual promiscuity. Being down low bruthas then further spreads it into the population.
***Always ask a man or woman you speak to if they have ever been with a black man or woman.*** Doing so could save your life as well as many other horrible implications from diseases. Seeing as those with higher IQs can better control sexual urges than those with lower IQs, and those with higher testosterone landed in prison more often, we can see this direct link in regards to testosterone in both IQ and ability to control sexual urges as well as those with higher testosterone going to prison more for crimes of sexual assault and rape.
Down low bruthas are yet another scourge we have to deal with. If it’s not blacks committing crimes, it’s blacks and their other deviant behaviors that then seep into our normal populations due to them not saying what they do on their spare time.
Always pre-screen partners for sex with blacks!!
Evolutionary Reasons for Suicide Bombings
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With all of these suicide bombings in the news recently, I figured I’d talk about some evolutionary reasons for suicide bombings. While reading JP Rushton’s paper Ethnic nationalism, evolutionary psychology and Genetic Similarity Theory, I came across a small part of the paper where he talks about evolutionary reasons for suicide bombings: mainly that it increases inclusive fitness. I know that biology doesn’t tell the whole story, but it tells a lot of it. Today I will argue that mainly, suicide bombings are driven by genetic similarity, as argued by Rushton in his paper. The data is there that this is a possibility and a worthwhile hypothesis to take note of.
Due to how inbred Muslims (Arabs) are, (as well as other Muslim populations, which are also inbred, such as the Chechens), they are more genetically similar to themselves than they are to other groups. The brain hormone oxytocin is conjectured to increase ethnocentrism, seeing as oxytocin is shown to increase in-group cooperation, and at the same time out-group derogation. This is also the case when two genetically distinct cultures meet up and live together. Their biology is so dissimilar, ethnic strife arises due to the far genetic distance between the two groups. So due to this increased genetic similarity, this causes those who are more similar to themselves, to favor those phenotypically similar to themselves, because if the phenotype is similar, more often than not, the genotype is as well. This is the basis for all ethnocentrism. To quote Rushton from the paper mentioned above:
Political issues are especially explosive when survival and reproduction are at stake. Consider the growth of Middle Eastern suicide bombers. Polls conducted among Palestinian adults from the Gaza Strip and the West Bank show that about seventy-five per cent support suicidal attacks, whereas only about twelve per cent are opposed (Margalit 2003). Many families state that they are proud of their kin who become martyrs.
Most analyses of the motives of suicide bombings emphasise unique aspects such as the Palestinian or Iraqi political situation, the teachings of radical Islam, or a popular culture saturated with the glorification of martyrs.
Political issues are especially explosive when survival and reproduction are at stake. Consider the growth of Middle Eastern suicide bombers. Polls conducted among Palestinian adults from the Gaza Strip and the West Bank show that about seventy-five per cent support suicidal attacks, whereas only about twelve per cent are opposed (Margalit 2003). Many families state that they are proud of their kin who become martyrs.
Most analyses of the motives of suicide bombings emphasise unique aspects such as the Palestinian or Iraqi political situation, the teachings of radical Islam, or a popular culture saturated with the glorification of martyrs.
These political factors play an indispensable role but from an evolutionary perspective aspiring to universality, people have evolved a ‘cognitive module’ for altruistic self-sacrifice that benefits their gene pool. In an ultimate rather than proximate sense, suicide bombing can be viewed as a strategy to increase inclusive fitness.
There is “altruistic self-sacrifice” for what suicide bombers do. Rushton then posits, that the self-sacrifice then, in turn, benefits their gene pool and that suicide bombing can be looked at as a strategy to increase inclusive fitness. Many people in the field have come to this conclusion. There is a reason, a genetic reason, for a lot of these suicide bombings. How could suicide bombings increase inclusive fitness if the individual is committing suicide? As I have said numerous times on my blog, evolution selects for genes, not individuals. So with selecting for genes, individuals who share similar genes with others who sacrifice themselves for other, more genetically similar people to themselves are actually increasing the proliferation of their genes. This is, yet again, is another answer to the people who argue that genetic similarity theory, which is predicated on self-sacrifice for those genetically similar to yourself, would select for selfishness, and not ethnic altruism. This is the case because those genes are being preserved. Individuals are basically just organisms to proliferate copies of their genes in to the next generation and nothing more.
Rushton then says:
What reasons do suicide bombers themselves give for their action? Many invoke the rhetoric of Islam while others appeal to political and economic grievances. Mahmoud Ahmed Marmash, a twenty-one-year-old bachelor from Tulkarm who blew himself up near Tel Aviv in May 2001 said in a videocassette recorded before he went on his mission (cited in Margalit, 2003):
I want to avenge the blood of the Palestinians, especially the blood of the women, of the elderly, and of the children, and in particular the blood of the baby girl Iman Hejjo, whose death shook me to the core. Many other national groups have produced suicide warriors. The term ‘zealot’ originates in a Jewish sect that existed for about 70 years in the first century CE. According to the classical historian Flavius Josephus (1981), an extreme revolutionary faction among them assassinated Romans and Jewish colla- borators with daggers; this likely reduced their chances of staying alive. A group of about 1,000 Zealots, including women and children, chose to commit suicide at the fortress of Masada rather than surrender to the Romans. Masada today is one of the Jewish people’s greatest symbols. Israeli soldiers take an oath there: ‘Masada shall not fall again’. Soldier armies – the Japanese kamikaze, or the Iranian basaji – have carried out suicide attacks against enemy combatants. Winston Churchill contemplated the use of suicide bombers against the Germans if they invaded Britain (see Cornwell 2003). Some of the Tamil Tigers of Sri Lanka, who are Hindus, have killed themselves in attacks on politicians and army installa- tions, and they have done so with utter disregard for the lives of civilians who happened to be around.
It’s clear that ethnic genetic interests were a main motivator for this attack. He also cites the Zealots, a Jewish sect from around 70 Ad, who committed suicide so that the Romans wouldn’t kill them. He cites the Japanese Kamikaze and the Iranian basaji, as well as saying that Churchill contemplated using suicide bombers against Germany if they invaded Britain, all of these examples serve as examples for genetic interests and altruistic self-sacrifice for you kin/co-ethnics. Rushton ends the paper as follows:
Genetic similarity, of course, is only one of many possible influences operating on political alliances. Causation is complex and there is no value in reducing relationships between ethnic groups to a single factor. Fellow ethnics will not always stick together, nor is conflict inevitable between groups any more than it is between genetically distinct individuals. In addition to reproductive success, individuals also work for motives such as economic success. However, as van den Berghe (1981) pointed out, from an evolutionary perspective, the ultimate measure of human success is not production but reproduction. Behavioural outcomes are always mediated by multiple causes. Nonetheless, genetic similarity can be expected to play a clear role in the social behaviour of small groups and even of large ones, both national and international. The hypothesis presented here is that because fellow ethnics carry copies of the same genes, ethnic consciousness is rooted in the biology of altruism and mutual reciprocity. Thus ethnic nationalism, xenophobia and genocide can become the ‘dark side’ of altruism. Moreover, shared genes can govern the degree to which an ideology is adopted (e.g. Rushton 1986 and 1989a). Some genes will replicate better in some cultures than in others. Religious, political and class conflicts become heated because they affect genetic fitness. Karl Marx did not take his analysis far enough: ideology may be the servant of economic interest, but genes influence both. Since individuals have a greater concentration of genetic interest (inclusive fitness) in their own ethnic group than they do in other ethnic groups, they can be expected to adopt ideas that proliferate their genes.
GST is a great argument that suicide bombers want to proliferate the genes of those genetically similar to themselves while at the same time getting rid of genes who didn’t pass kin on to the next generation, as well as getting rid of one individual who takes up resources without copulating kin to the next generation, by doing so this increases the fitness of his or her co-ethnics, and therefore, through altruistic self-sacrifice, spread on their genes in that manner. Because evolution is about reproduction, not production.
In this short paper, Suicide Bombers: Does an Evolutionary Perspective Make a Difference?, which is a review of a book called The Myth of Martyrdom, the author argues that suicide bombers have similarities to others who commit suicide as well as murder-suicide, he ends up positing that there is no altruistic self-sacrifice and that suicide bombings are a result of mental health issues and individual crisis. The linked paper expands the author of the book’s idea that suicide bombers are increasing the inclusive fitness of their people. Those who behave in ways to promote the reproductive success of close kin (kin selection), in turn, enhance their inclusive fitness. There is also evolutionary evidence that we humans have been programmed evolutionary history to promote reproductive success of their kin as well as those closely related to them (their co-ethnics).
Parents who sacrifice themselves for their children are doing so because of evolution. In saving their child, who shares 50 percent of their own genes, they are increasing the evolutionary success of their genes to continue to reproduce other generations. This is because the average similarity between people within a single population is on the magnitude of half-siblings. So co-ethnics are share 25 percent of their genes, on average. This is a cause for ethnocentrism, as I have argued many times here.
If an individual’s reproductive prospects are low, and they are not contributing to the welfare of those genetically similar to themselves, then removing their genes through suicide will not remove genes that already weren’t going to be removed due to not having any kin. The authors of the paper also argue that if the individual is taking up resources that could be better used by other kin to promote their best (ethnic) interests, then prolonging that individuals existence may diminish, rather than enhance, inclusive fitness for that group. Suicide is more common in those who are elderly as well as terminally ill, because those who are elderly or terminally ill have less of a chance of proliferating their genes, so they care less about their individual fitness, and in turn, care about inclusive fitness instead.
In the ASID (Adult Suicide Ideation Questionnaire), which is a 25 question self-report to measure suicide ideation and behavior in adults (Reynolds, 1991 b), those who participated in the study ranked feelings of suicide on a scale of zero to seven which include: “0 = Never had this thought; 1 = I had this thought before, but not in the last month; 2 = About once a month; 3 = Couple of times a month; 4 = About once a week; 5 = Couple of times a week; 6 = Almost every day). The ASIQ has extremely high, almost perfect correlations, .96, .96 and .97 in a sample of college students, community college students and a psychiatric sample using Cronbach’s Alpha Coefficients. Overall, the ASID correlates with depression (r=.60) and with hopelessness (r= .53) in a sample of college students (Reynolds, 1991 a).
There is also a positive correlation between suicide ideation and perceived burden to kin. The relationship was strengthened when participants were added for those with poor health as well as low interpersonal satisfaction, both of which indicate low inclusive fitness.
These reasons also show why Japanese Kamikaze Fighters did their suicide attacks: to protect their kin in their homeland as to better protect those genetically similar to themselves.
Many suicide bombers come from middle-class backgrounds, which further proves the case for genetic interests being the cause for this. The majority of Al-Qaeda members come from educated, middle-class backgrounds. Even for Palestinian suicide bombers, none of them were poor, uneducated, simple minded nor depressed. The myth of the suicide bomber being poor and destitute and, therefore, chooses to kill himself for the myth of 72 virgins, which a majority of Muslims don’t believe in and is pushed by the Jews, is just that, a myth. Most are driven for altruistic self-sacrifice for their co-ethnics, as all co-ethnics are around the world.
Satoshi Kanazawa argues that many suicide bombers are driven to suicide due to sexual repression. He also notes that most Western men who are tricked by porn movies, most Muslims are tricked by the Quran, which did not exist in their ancestral environment. He theorizes that in the same way that Western men who watch porn believe they can potentially copulate with the women they see in porn movies, the same reasoning can be said for Muslims who believe they can copulate with the 72 virgins in their Heaven. Kanazawa says:
If you are a likely reproductive loser in the United States, watching porn is your way of meeting women and having sex. If you are a likely reproductive loser in a Muslim society, committing suicide bombing is your ticket.
He also notes how most suicide bombers are slightly more wealthy as well as educated than the population they come from, which I have just referenced above:
Social scientists have recently noted that suicide bombers tend to be slightly more educated and wealthier than the general Muslim population from which they come (Atran, 2003; Berrebi, 2003; Krueger and Maleckova, 2003), in seeming contradiction to my suggestion here, because such men should have more reproductive opportunities on earth than their less educated and poorer competitors. Closer examination of these studies reveals, however, that they are not inconsistent with my evolutionary psychological explanation of suicide bombings. For example, a study of 129 Hezbollah shahids (martyrs), only three of whom were suicide bombers, shows that shahids are significantly more likely to have attended secondary school or higher, and significantly less likely to come from a poor family (Krueger & Maleckova, 2003, pp. 129-135). However, this is entirely because Hezbollah members are more likely to come from Beirut and South Lebanon, characterized by higher level of education and less poverty. Once the geographic origin is controlled, shahids are no more likely (albeit no less likely either) to come from privileged background. (emphasis his)
Though most Muslims don’t believe the hadiths involving 72 virgins, Kanazawa puts forth a great theory, which also goes along with what I’ve been talking about for this whole article: there is a subconscious thing in their brain, which motivates them to suicide bomb as a strategy for inclusive fitness. By doing so, they are not taking up any more resources, so their kin/co-ethnics can better use those resources in order to proliferate their genes to the next generation.
Ashkenazi Jews show the same nepotism as Arabs, but go about their goals in a different way. They are two different sides to ethnic genetic interests and genetic similarity theory, basically polar opposites. Looking into both groups’ motivations through history and learning why they do what they do shows a lot about how the world is today.
Inbreeding was introduced to the Arabs by the Jews around 200 BC near the Levant. With that much inbreeding happening for so long, this led to the aforementioned effect of lowered IQ by 2.5 to 10 points on average and increased clannishness.
Suicide bombings offer yet another window into the reality that is Ethnic Genetic interests, as well as Genetic Similarity Theory and Group-Selection. Without those drivers, suicide bombings would be less in number because a majority of suicide bombings happen to increase inclusive fitness in the group because many of the men/women are childless or terminally ill. So by stopping themselves from taking up resources, they also increase the inclusive fitness of their co-ethnics because they are not taking up any more resources. They are also eliminating their genes, which didn’t copulate more progeny to the next generation. By getting rid of genes that don’t make it to the next generation and strengthening the gene pool of those who reproduce.
Suicide bombings show yet more reasons for the existence of GST, because if they weren’t so genetically similar due to inbreeding, suicide attacks would be lessened.
Genetic Similarity Theory as a Cause for Ethnocentrism
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Genetic Similarity Theory evolved so we could better spread on shared genes in our immediate population, as well as those closest to ourselves. Meaning those of our race/ethnicity. People say “how could altruism evolve if it’s self-sacrifice, selfishness would win out”. Well, what’s being preserved is not the individual, obviously, but shared genes. To those who say (JayMan) that ethnic genetic interests don’t exist, there is a mountain of evidence that says otherwise.
Rushton and Nicholson (1988), tested predictions from genetic similarity theory and found that spouses select each other on the basis of more genetically influenced cognitive tests. It’s known since The Bell Curve came out in 1994 that spouses select each other based on IQ. What Rushton and Nicholson noted in the study was that estimates of genetic influence calculated on Koreans and Canadians predicted assortative mating in European Americans in Hawaii and California. Americans of mixed ancestry made up for ethnic dissimilarity by matching up on the more heritable traits, whereas the correlation is lower for those traits that are more influenced by the environment. The observations on genetic selection were weaker but still had a positive correlation, when the g factor was taken out of the equation. This suggests that we choose mates based on the general intelligence factor.
In studies on bereavement, it’s noted that those parents who believe their children resembled their side of the family grieved more than if they believed their child resembled the opposite side of their family (Littlefield and Rushton, 1986). This has huge implications for Genetic Similarity Theory.
Henry Harpending showed that against the background of worldwide genetic variance, the average similarity between people in a single population is on the order of magnitude of half siblings. To quote Rushton:
Political scientist Frank Salter calculated that compared to the Danes, any two random English people have a kinship of 1/32 of a cousin. Two English people become the equivalent of 3/8 of a cousin by comparison with people from the Near East, 1/2 cousin by comparison with people from India, half-siblings by comparison with people from China, and like full-siblings compared with people from sub-Saharan Africa.
Thus, the aggregate of genes people share with co-ethnics dwarfs those shared with extended families. Rather than being a poor relation of family nepotism, ethnic nepotism is virtually a proxy for it.
His conclusion being:
Conclusion: the reason people engage in ethnic nepotism, as well as marry similar others, and like, make friends with, and help the most similar of their neighbors, is that doing so benefits copies of their genes.
The sense of a common ethnicity remains a major focus of identification for individuals today. It is no more likely to diminish in the future than is that of the family.
Genetic similarity theory explains why.
In Rushton’s paper GENE-CULTURE COEVOLUTION AND GENETIC SIMILARITY THEORY: IMPLICATIONS FOR IDEOLOGY, ETHNIC NEPOTISM, AND GEOPOLITICS, two individuals will be, on average, more genetically similar to those of their own ethnicity than to those two from different ethnic groups. Therefore, it will be in the individual’s own self-interest to help one genetically similar to himself, and therefore, derogate the out-group, causing ethnic strife when two genetically dissimilar groups meet up and live together.
Jews that have been separated for thousands of years still show more genetic similarity to each other than to other populations. This shows in how ethnically nepotistic Ashkenazi Jews are to themselves. Jews from Iraq have more in common from a genetic viewpoint than do those 2 groups in comparison to other populations in the world. We can, therefore, expect Jews, as well as all populations in the world, to adopt ideologies that will proliferate their own genes, but come at the expense of derogating out-groups.
Genetic Similarity Theory may also explain how well and with how much tenacity the German military fought in WWII, as well as the lack of morale in the American Army during Vietnam.
He says that if genetic distance measures were calculated, that American liberals will be more genetically distant from the WASP average. The growth of white survivalism is also explained by genetic similarity theory. To quote Rushton:
The growth of “white survivalism” and militant “Christian Identity” groups such as the Aryan Nations, and the Covenant, the Sword, and the Arm of the Lord, represent a more extreme response to these perceived threats to the AngloSaxon gene pool. If this overall analysis is correct, one might expect similar correlations in deviations from both genetic and ideological norms in other groups. Preserving the “purity” of the ideology might be an attempt at preserving the “purity” of the gene pool. Are ideological “conservatives” typically more genetically homogeneous than the same ideology’s “liberals”?
This can be seen today, no matter where you look in the world. From the Rwandan genocide involving the Tutsis and the Hutus to La Raza in the American Southwest to Black Lives Matter to the KKK and other white interest groups, to even how East Asians and other Asian immigrants basically isolate themselves in areas with those who are culturally, as well as genetically like themselves. Genetic similarity manifests itself in our societies and makes itself evident every day.
We can also view conflicts from other parts of the world to see genetic causes for them as well. We can look at the Northern Ireland conflicts between the Irish Protestants and Catholics to see if it represents a thousand-year-old continuation of the war between the AngloSaxons and the Celts, to the conflict of the Babylonians and the Egyptians which could be manifesting itself today between the Jews and the Arabs, ethnic dissimilarity shows itself in world geopolitics, as well as showing that ethnic dissimilarity is a driving focus in most of our wars and problems.
Rushton then finally asks the question:
If the replication of genetically similar genes is as strong a biological imperative as sociobiological theorizing suggests, why are descendants of North European populations everywhere in the world currently experiencing negative growth, while concurrently allowing extensive immigration from genetically less similar gene pools? Why, at the same time have North European populations adopted an ideology of secular humanism which discourages racist attitudes and encourages antipathies toward religious sentiment proportional to the degree to which those ideologies combat the new orthodoxy?
Cultural and organic evolution are different, yet linked in many ways and may ultimately share certain properties. Both strive to replicate at the expense of other groups, so we can see how ideologies could evolve that dramatically decrease fitness for one group over another. This also goes back centuries. This is seen in classes, and sometimes race. Those at the top, i.e., the more intelligent, have fewer children than the people that they rule over. Then, it’s not too long until the ruled become the rulers and the cycle repeats itself. Rushton says:
There is indeed evidence that this trade off exists at a quite profound level and moreover is related to other characteristics, the whole complex being partly genetic in origin (Rushton, 1985). My own guess is that low fertility may be partly mediated by a psychological process in which the desire to be in control of both oneself and one’s environment is taken to an extreme.
This is one of the many reasons that Europeans today have such a low birth rate. I have written before on how to ameliorate this effect, i.e., positive things shown to women in the media such as being happy with babies. That was shown to increase the birth rate in pre-WWII Germany as well as having a positive benefit on the psyche of the German women seeing other women happy with children. The effects of media socialization, though, go both ways, which is one reason for low European birthrates.
Successful cultures ultimately arise in those that the top of the society limits its reproduction, which, in turn, didn’t give others more genetically similar the chance to replace them. This may be a cause, as Rushton says, for the fall of the Graeco-Roman Empire, stating that the Roman Empire and other similar cultures were, presumably, evolutionary dead ends. He then asks: “If this perspective is accurate, are North Europeans headed for the same fate as the ruling classes of ancient Greece and Rome?”
Rushton ends the paper as follows:
The question is: if that time comes, in whose image will it be shaped? People will differ in their moral prescriptions. The choices they make are likely to reflect both their genetic and their ideological interests.
This is why I say, that, on an individual level, morals are subjective. Society as a whole sets morals, but this says to me that on an individual level that morals are subjective, but that’s for another time.
There are many reasons why altruism and ethnocentrism evolved, as well as many reasons why that same altruism is being used against Europeans, as well as some more environmental factors. This is also seen in Non-Western people who are abnormal to our societies due to differing evolution and culture, which culture is a product of genetics.
It’s clear that we are more altruistic to people who look more phenotypically similar to ourselves, to pass on and benefit copies of our genes. This evolved in spite of the negative impact on behalf of the altruist. The altruist is helping copies of his shared genes survive so that they may be copied into the next generation of progeny. The tendency to favor co-ethnics is the tendency to attempt to help pass on shared genes, as if the phenotype is similar, more often than not, the genotype is as well. This is the basis for ethnocentrism.
Are There Genetic Causes for Obesity?
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We all know about the Healthy At Every Size Movement (HAES) and how they claim that genetics is the cause of them being overweight and or obese and that genetics is the cause that they cannot lose weight as well as other people. I’m not here today to defend that they are right and that’s why they can’t lose weight (because lets be honest, they have no idea what they’re talking about, nor can they reference any type of study that says it), nor am I here today to give any credence to the HAES movement. I’m here today to talk about genetic causes for obesity as well as causalities that people don’t talk about and believe that kcal in and kcal are the only factors in becoming obese (I have never, nor will I ever dispute that kcal in and out has been the biggest factor involving obesity, just there are underlying causes that people do not talk about, which is the a huge cause in keeping people obese). What people don’t understand is that there are underlying factors that no one talks about that lead to obesity.
When people say that there are no genetic underpinnings for obesity, they are speaking on a subject that they are extremely ignorant about. They always say “kcal in and out”. Right, which I have never disputed. Though, those same people cannot say a thing to the studies that I provide, because they cannot adapt to new information and just parrot the same things as if that disproves the studies that I link. Furthermore, obesity and diabetes (which there is a close relationship between the two), are both nowhere near close enough to being understood.
I have already covered here that ability to delay gratification has a genetic component, and that those with low ability to delay gratification, as noted in my post, had a higher chance of becoming obese than those with a better ability to delay gratification. Some people have said to me that the Marshmallow Experiment didn’t have anything to do with the ability to delay gratification, that it was something else entirely, but alas, the individual obviously said nothing more when I asked him to comment on the post so my readers can read the exchange.
You all know that I covered ethnicity and obesity, but I’m making this post to serve as a something to reference while in discussion with people, as well as educate people who don’t know about these studies.
As noted in my previous article on obesity, there are racial difference in obesity (that pretty much follow Rushton’s Rule). Of course there are socioeconomic factors that are involved there, but to say that there is no genetic component is intellectually dishonest. To believe that there are absolutely no genetic causes for obesity and that environmental factors means everything shows that that person has no idea what they are talking about.
According to a meta-study of twins and families, the heritability of BMI is between .75 and .82. This used mono and dizygotic twins, as well as having over 140 thousand participants. They observed 12 countries, all with differing racial/ethnic groups and the results were the same.
While in a discussion with someone, I got linked these studies: Obese toddlers have dramatically lowered IQ, Obese toddlers have dramatically lowered IQ 2 and Obesity lowers children’s IQ. This is hilarious. The causalities are completely reversed. I would love to hear the explanation for the physiological mechanism that has obesity lower IQ. Well, Satoshi Kanazawa tackled this in his study back in 2014, that low IQ leads to obesity, obesity doesn’t lead to low IQ.
A few of the highlights include:
- Cross-sectional studies conclude that obesity lowers IQ, whereas longitudinal studies conclude that those who become obese already have a low IQ since childhood
- Careful examination of longitudinal studies in Sweden, New Zealand and America clearly show that the casual direction goes from low IQ to obesity, not obesity to low IQ
- There is NO scientific evidence that shows that obesity leads to lowered IQ. There is, however, ample evidence, both in scientific theory as well as ample amounts of evidence that lower IQ people become obese
Individuals with IQs below 74 at 18 have BMI of 26.59 at 40, whereas those with IQs above 126 have BMI of 25.75 (P < 0.001). Similarly, there is a clear and monotonically negative association between intelligence at 18 and the BMI change from 18 to 40. Individuals with IQs below 74 gain 5.19 in BMI in 22 years, whereas those with IQs above 126 gain 3.73 (P < 0.001). Their conclusion remains identical even when they control for systolic and diastolic blood pressure, resting pulse rate, birth place, birth year, and education at conscription. Their results from a large population sample of Swedish men make it clear that it is adolescent intelligence that influences BMI in middle age, not the other way around
The fact of the matter is this: obesity does not lower IQ. Those with certain agendas would like you to believe that becoming obese drops IQ, whereas ample scientific data shows the opposite. Including this study which states that those in the cohort who became obese didn’t see a drop in IQ from childhood, instead, those individuals who became obese already had a lower IQ since childhood. You already know that I did not get a response to these studies. That’s because showing people that they’re wrong actually makes them believe their wrong beliefs more, especially if they have low self-confidence (how ironic).
There have been genes that have been found that are associated with binge eating. If a young adolescent has this particular variant on the FTO gene, they are 20 to 30 percent more likely to binge eat than those who don’t have the variant. This was observed in girls particularly, who were 30 percent more likely to binge eat if they had the variation.
Dr. Peter Atilla had a TED Talk on how obesity may be hiding an even more insidious problem. To quote from the transcript:
Yet when it came to a disease like diabetes that kills Americans eight times more frequently than melanoma, I never once questioned the conventional wisdom. I actually just assumed the pathologic sequence of events was settled science.
Three years later, I found out how wrong I was. But this time, I was the patient. Despite exercising three or four hours every single day, and following the food pyramid to the letter, I’d gained a lot of weight and developed something called metabolic syndrome. Some of you may have heard of this. I had become insulin-resistant.
Now, most researchers believe obesity is the cause of insulin resistance. Logically, then, if you want to treat insulin resistance, you get people to lose weight, right? You treat the obesity. But what if we have it backwards? What if obesity isn’t the cause of insulin resistance at all? In fact, what if it’s a symptom of a much deeper problem, the tip of a proverbial iceberg? I know it sounds crazy because we’re obviously in the midst of an obesity epidemic, but hear me out. What if obesity is a coping mechanism for a far more sinister problem going on underneath the cell? I’m not suggesting that obesity is benign, but what I am suggesting is it may be the lesser of two metabolic evils.
You can think of insulin resistance as the reduced capacity of our cells to partition fuel, as I alluded to a moment ago, taking those calories that we take in and burning some appropriately and storing some appropriately. When we become insulin-resistant, the homeostasis in that balance deviates from this state. So now, when insulin says to a cell, I want you to burn more energy than the cell considers safe, the cell, in effect, says, “No thanks, I’d actually rather store this energy.” And because fat cells are actually missing most of the complex cellular machinery found in other cells, it’s probably the safest place to store it. So for many of us, about 75 million Americans, the appropriate response to insulin resistance may actually be to store it as fat, not the reverse, getting insulin resistance in response to getting fat.
You can think of insulin as this master hormone that controls what our body does with the foods we eat,whether we burn it or store it. This is called fuel partitioning in the lingo. Now failure to produce enough insulin is incompatible with life. And insulin resistance, as its name suggests, is when your cells get increasingly resistant to the effect of insulin trying to do its job. Once you’re insulin-resistant, you’re on your way to getting diabetes, which is what happens when your pancreas can’t keep up with the resistance and make enough insulin.
But most important, I was left with these three burning questions that wouldn’t go away: How did this happen to me if I was supposedly doing everything right? If the conventional wisdom about nutrition had failed me, was it possible it was failing someone else? And underlying these questions, I became almost maniacally obsessed in trying to understand the real relationship between obesity and insulin resistance.
So what I’m suggesting is maybe we have the cause and effect wrong on obesity and insulin resistance.Maybe we should be asking ourselves, is it possible that insulin resistance causes weight gain and the diseases associated with obesity, at least in most people? What if being obese is just a metabolic response to something much more threatening, an underlying epidemic, the one we ought to be worried about?
Let’s look at some suggestive facts. We know that 30 million obese Americans in the United States don’t have insulin resistance. And by the way, they don’t appear to be at any greater risk of disease than lean people. Conversely, we know that six million lean people in the United States are insulin-resistant, and by the way, they appear to be at even greater risk for those metabolic diseases I mentioned a moment ago than their obese counterparts. Now I don’t know why, but it might be because, in their case, their cells haven’t actually figured out the right thing to do with that excess energy. So if you can be obese and not have insulin resistance, and you can be lean and have it, ******this suggests that obesity may just be a proxy for what’s going on.******
So what if we’re fighting the wrong war, fighting obesity rather than insulin resistance? Even worse, what if blaming the obese means we’re blaming the victims? What if some of our fundamental ideas about obesity are just wrong?
Personally, I can’t afford the luxury of arrogance anymore, let alone the luxury of certainty. I have my own ideas about what could be at the heart of this, but I’m wide open to others. Now, my hypothesis, because everybody always asks me, is this. If you ask yourself, what’s a cell trying to protect itself from when it becomes insulin resistant, the answer probably isn’t too much food. It’s more likely too much glucose: blood sugar. Now, we know that refined grains and starches elevate your blood sugar in the short run,and there’s even reason to believe that sugar may lead to insulin resistance directly. So if you put these physiological processes to work, I’d hypothesize that it might be our increased intake of refined grains, sugars and starches that’s driving this epidemic of obesity and diabetes, but through insulin resistance,you see, and not necessarily through just overeating and under-exercising.
The fact of the matter is this: we need to look at any and all causes to do with obesity. To fully understand this disease is to look at any and all factors involving it. To discard theories and make new ones, or just disregard what was looked at. People who say that we shouldn’t look at these types of things really have no idea what they’re talking about. To fully understand a problem, we need to look at any and all causes that may be underlying.
I’m currently writing a research paper on Prader-Willi’s Syndrome (which I will post here when I’m done with it), and as I was watching the documentary, a few things jumped out at me:
- They are infantile
- They clearly have a lack of ability to delay gratification
- Prader-Willi’s people have an IQ, on average of 70
- Due to being infantile, they have a lack of ability to delay gratification, so along with that, they have lower IQs which is correlated with lack of abstract thought
To say that these people “can control themselves” and they “just need to eat less” is dishonest, to say the least. Those people with disorders such as these really have no say in the matter.
In a follow-up to the Marshmallow Experiment, studies were done on those individuals they could still find 40 years later. What was found that those who lacked the ability to delay gratification in pre-school ended up becoming obese. We need to identify those children with low ability to delay gratification because it’s clear that those with the lack of ability to delay gratification end up becoming obese in adulthood.
This is a favorite of mine. People may say “Fat shaming is good!!! It leads to people thinking about what they’re doing and, in turn, they will lose weight!!!” How wrong that is. Present research indicates that in addition to poorer mental health outcomes, weight discrimination has implications for obesity. ******Rather than motivating people to lose weight, weight discrimination increases the risk for obesity.****** Why people think that making fun of people will lead to weight loss is beyond me.

Interestingly, a slightly different pattern emerged when the analyses were based on measured BMI. When the sample with measured weight and height was limited to participants who were overweight at baseline, the risk of obesity was a little stronger but essentially the same (OR = 2.18, 95% CI = 1.04–4.45). In contrast, when this sample was limited to normal weight participants at baseline, there was not enough data for the analysis: of the 14 participants in the normal weight category who reported weight discrimination, none became obese
Similar to weight gain, weight discrimination was associated with remaining obese over the period between the two assessments (see Table 1). That is, those who experienced discrimination based on their weight were over three times more likely to remain obese at follow-up, rather than drop below the obesity threshold, than those who did not experience such discrimination.
The evidence is clear: weight discrimination actually increases the problem that people actually laugh at and make fun of people for. How ironic is that?
In conclusion (I will add to this post as new research comes out), to say that there are no underlying causes of obesity is intellectually dishonest. There are clear underlying causes to this obesity epidemic, which we need to look at any and all of these causes to fully understand obesity better (which we are nowhere close to understanding this problem).
The Evolution of Jewish Nepotism and High IQ
1300 words
(I touched on the connection between Ashkenazi Jews and Italians. This will be about evolution of Jewish nepotism as well as another part of the puzzle to the high Jewish IQ.)
Ashkenazi Jews are over-represented in many facets in America, as well as around the world. What is the cause of Jewish nepotism? What makes them stick together so much while derogating other ethnicities?
The evolution of nepotism in Ashkenazi Jewish communities goes back a few thousand years. They constantly got kicked out of nations, 109 times to be exact, so therefore, they needed to be more clannish, which comes with increased genetic similarity. They needed to stick together and always have each other’s backs. This is due to inbreeding, which as noted above leads to increased genetic similarity and therefore, individuals who inbreed closely become more related to one another than non-co-ethnics. When two groups who are so genetically distant live in one society together, strife happens. Which is going on in Europe at the moment. But with Jews, it’s different. They are more in the background, so to speak. They hide in the shadows while giving more favoritism to their own kind, ethnic nepotism.
Ethnic nepotism in the Jewish community evolved due to persecution over the thousands of years by non-Jews on Jews for things such as usury, which is defined as the illegal action of borrowing money at extremely high interest rates. In the middle ages in Europe, the Catholic Church forbade money lending. This is where the Jews came in and became bankers, lending money to the populace of the countries. Abnormal amounts of interest were given to the people in the country. In turn, the Jews got driven out due to preying on the populace of the country and taking advantage of them.
So when they got driven out, they had to stick together. As I noted in the linked article on the connection between Ashkenazi Jews and Southern Italians, male Jews migrated from the Levant to Rome during Greco-Roman times, which mass conversions led to 6 million ( =^) Roman women who then began to practice Judaism. The genetic proximity of Ashkenazi Jews and Syrian Jews to Northern Italians, Sardinians and French populations suggest that there is non-Semitic ancestry in Ashkenazi Jews. The findings also say that any theories of Ashkenazi Jews having ancestry in Khazaria or from Slavs are incompatible with genetic studies. The close genetic similarity of Ashkenazi Jews and Southern Europeans has been noted in many studies. Any theories of Ashkenazi Jews being converts from the Khazar empire got put to rest by this paper. Anyway, that’s part of the reason for their higher average IQ, breeding with beautiful Roman women a few thousand years ago.
Combined with selection pressures selecting against those less smart Jews, as brought up by Cochran, Hardy and Harpending, this led to those less intelligent Jews to be culled from the population. Due to this, this led to them only being in occupations in which they had to have high intellect. Therefore, those less intelligent Jews couldn’t make the money needed to survive, and, therefore, their genes got taken out of the gene pool. The more intelligent Jews, in turn, then had more kids, increasing the chances for more genetic mutations to positively affect IQ. So because those rich Jews in the middle ages had more kids, this led to even more selection for higher IQs in the Ashkenazi population. So because those more intelligent Jews had more money, and obviously more intelligence, they could be more nepotistic to others in their in-group while derogating those in the out-group.
In Rushton’s paper, Ethnic nationalism, evolutionary psychology and Genetic Similarity Theory, he posits that since ethnic groups are repositories of shared genes, xenophobia, as well as out-group derogation, is the “dark side of human altruism”. Which makes sense. If you care more for your own group than for others, that will make for a better chance for individuals, as well as groups, to pass on shared genes. Due to very close inbreeding. All Ashkenazi Jews are 30th cousins. So with that increased genetic similarity, this leads to increased altruism as well as a higher chance to shun others not in the ethnic group. This is a sound evolutionary strategy to keep the close genetic similarities. Though, with whites, as I have alluded to a few times on this blog, that doesn’t happen due to media socialization (owned BY the Jews).
So because the individual is the carrier of the genes, the close relatedness (Rushton says in the paper linked above the co-ethnics from around the world are related to each other on the order of first cousins!), we can see that how we protect our close family and want nothing bad to happen to them as well as favor them over other peoples/groups, the same holds true for those ethnicities that are extremely genetically close due to inbreeding.
The close inbreeding, however, leads to an increased chance for recessive genes to be given to the child. Therefore, genetic diseases developed. In their paper, Cochran, Hardy and Harpending say that Gaucher, Tay-Sach’s and Niemann-Pick heterozygotes lead to increased IQ. To quote from the paper:
We do have strong but indirect evidence that one of these, Gaucher disease, does indeed increase IQ. Professor Ari Zimran, who heads the Gaucher Clinic at the Shaare Zedek Medical Centre in Jerusalem, furnished us a list of occupations of 302 Gaucher patients. Because of the Israeli medical care system, these are essentially all the Gaucher patients in the country. Of the 255 patients who are not retired and not students, 81 are in occupations that ordinarily average IQ’s greater than 120. There are 13 academics, 23 engineers, 14 scientists, and 31 in other high IQ occupations like accountants, physicians, or lawyers. The government of Israel states that 1.35% of Israeli’s working age population are engineers or scientists, while in the Gaucher patient sample 37/255 or 15% are engineers or scientists. Since Ashkenazim make up 60% of the workforce in Israel, a conservative base rate for engineers and scientists among Ashkenazim is 2.25% assuming that all engineers and scientists are Ashkenazim. With this rate, we expect 6 in our sample and we observe 37. The probability of 37 or more scientists and engineers in our sample, given a base rate of 2.25%, is approximately 4 x 10-19 . There are 5 physicists in the sample, while there is an equal number, 5, of unskilled workers. In the United States the fraction of people with undergraduate or higher degrees in physics is about one in one thousand. If this fraction applies even approximately to Israel the expected number of physicists in our sample is 0.25 while we observe 5. Gaucher patients are clearly a very high IQ subsample of the general population
So those certain genetic diseases, which came about due to such close inbreeding, have negative effects on Ashkenazi health, but clearly not their intellect.
The reasons for high Ashkenazi nepotism are persecutions for the past few thousand years (which led to them needing to stick together more), the need for them to go in to high IQ occupations such as banking, which led to the culling of those Jews who weren’t as intelligent, therefore leading to the culling of those genes out of the gene pool, and finally genetic diseases most likely, with some pretty solid evidence that there is a rise in a few IQ points due to certain diseases they have. The biggest reason for Jewish nepotism is, of course, increased genetic similarity due to such close inbreeding for thousands of years which basically make Ashkenazi Jews 30th cousins.