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The Threat of Increasing Diversity: Why Many White Americans Supported Trump in the 2016 Presidential Election
Tl;dr: White Americans exposed to more diversity are more likely to support Trump, anti-PC speech and anti-immigration policies while showing less support and positivity towards Democratic candidates. In the racial shift group, whites with low racial identity, ethnic replacement didn’t seem to care about ethnic replacement and showed stronger support for Democratic candidates. To wake up more whites to anti-immigration sentiments and white identity politics, you need to show them the effects of diversity in the social context as well as what a demographic replacement will mean in the next two decades.
Why did so many white Americans support Donald Trump’s Presidency? The reasons are numerous, though there are some key reasons why he won. To look at the exact reasons why, we need to look at some evolutionary psychology as well as political psychology. I came across a paper today titled The threat of increasing diversity: Why many White Americans support Trump in the 2016 presidential election, it has many thought provoking things in it and pretty much confirms what the altright says about an increase in white identity occurring. An ‘ethnic awakening’ if you will. The authors state that white Americans high in racial identity will be more likely to derogate out-groups when white Americans realize they are becoming replaced in their own country.
Major, Blojorn and Blascovich (2016) state that reminding white Americans who are ‘high in ethnic identification’ (i.e., a white identitarian, an altrighters) that non-white populations will soon outnumber whites caused them to be more concerned about the future of whites in America, pushing them towards Trump and his anti-immigration policies. This also led to an increase in being politically incorrect. Moreover, whites low in ethnic identification (say, a progressive leftist) showed no greater chance in voting for Trump nor his anti-immigration policies. This did, however, decrease positivity towards Trump as well as decreased their opposition towards political correctness. The authors write:
The U.S. Census Bureau (2012) projects that the national population of non-White racial groups will exceed that of Whites before the middle of this century. Many White Americans in the US view race relations as “zero-sum,” in which status gains for minorities means status loss for Whites (Wilkins & Kaiser, 2014) and less bias against minorities means more bias against Whites (Norton & Sommers, 2011). The belief that Whites are losing out to ethnic minorities is particularly prevalent among Trump supporters (De Jonge, 2016).
This is noticed, anecdotally speaking and you can follow the citations to get more information. From an evolutionary perspective, this does make sense. Competition for resources between groups trigger evolutionary instincts. More non-whites in America will decrease the white population who has the lowest birth rate by ethnicity in the country and this will trigger more anti-immigration sentiments in whites high in ethnic identification. This ‘zero-sum game’, the ‘if your ethnic group has more than mine has less’ game will start to take hold in America in the next coming years if this paper is any indication of the future. The one particularly interesting point the authors bring up is that if there is “less ‘bias against minorities, there will be more minorities against whites”, and that, in turn, increased anti-immigration sentiments as well as drove people towards Trump and his anti-immigration views.
The more minorities that come into the country, the more whites in America will start to band together for their own ethnic genetic interests, move towards more conservative policies and begin to show more derogation towards the out-group.
The authors use the term ‘group status threat’, which is when one “worries that his group’s status, influence, and position in the hierarchy is under threat.” This threat then predicts out-group derogation. I wonder if oxytocin (a brain peptide that increases out-group derogation) increases when diversity occurs in the social context. I’d like to see that looked into one day.
There is also ‘integrated threat theory’ where increased diversity poses a threat to white Americans’ resources and American values. They also state, using social cognition theory, that increases in diversity will be ‘frightening’ and ‘confusing’ to whites, causing “uncertainty and fear”, which then drove whites towards more conservative anti-immigration policies.
When whites high in ethnic identification were shown a newspaper article stating that whites would be a minority by 2042, it led whites to be more concerned about whites’ social status in the country, leading them towards more conservative views and policies. It’s important to note that their views changed along with their policy recommendations.
In this study, the authors tested experimentally whether reminding white Americans that of the increasing diversity in the US affects their political leanings, whether or not group status is the cause of the political leanings when one hears about ethnic replacements, and whether or not ethnic identification or political alignment moderated the effects. They expected that reminding whites of ethnic replacement will cause them to lean towards conservative views and politicians (Trump, Kasich, Cruz) while decreasing support for Democrats (Clinton and Sanders).
People who experience ‘group status threat’ will be more likely to vote for Trump since he has more anti-immigration, antidiversity views than all politicians who ran for President. This, the researchers hypothesized, would come to fruition in their study. They also predicted that reminding white Americans of ethnic replacement would cause them to support more anti-immigration policies and be more resistant to political correctness, i.e., they would be more likely to be against positive policies for the out-group. They would become intolerant towards the out-group upon exposure to the reality of ethnic replacement in the country.
We also tested ethnic identification and political affiliation as potential moderators of the predicted effect of condition.1 Drawing on social identity theory (Tajfel & Turner, 1986), we expected that reminders of increasing ethnic diversity would be especially threatening to Whites whose race/ethnicity is a central aspect of their identity. Thus we expected them to report greater support for Republican candidates, anti-immigrant policies, and opposition to political correctness in response to reminders of the racial shift compared to Whites low in ethnic identification. In contrast, based on Craig and Richeson’s (2014b) finding that reminders of the racial shift increased support for conservative ideology irrespective of political leanings, we did not expect political affiliation to moderate effects.
Whites whose ‘race/ethnicity is a central aspect of their identity’, i.e. altrighters were predicted to be especially threatened at the reminder of ethnic replacement in their country of birth. However, as expected and what is seen in anecdotal accounts, whites low in ethnic identification, i.e., progressive leftists, antifas, etc, showed the opposite.
The researchers had a sample consisting of 450 white Americans with the following political beliefs: 262 Democrats, 114 Republicans, 50 Independents and 24 ‘other’. After removing the Independents and ‘others’ from the sample they had 376 white American participants (51.1 percent female).
They were given articles and were given two minutes to read them. One was an article talking about the ethnic replacement of whites and whites’ minority status in America that’s projected to occur by 2042 (aptly called ‘racial shift’) while the other article used “similar language to indicate geographic mobility is increasing (control condition).” It’s interesting to note that it seems like the only difference between the two articles is the wording. After reading the articles, they then completed tasks assessing group status threat, support for the current candidates running for office, anti-immigration sentiments, ethnic identification, and opposition to political correctness. After the completion of the tasks, they were then told the reason for the study and compensated their one dollar.
White Americans exposed to ‘racial shift condition’ reported greater group status threat than those in the control condition. This shows that white Americans who live in a diverse neighborhood will be more likely to be affected by the ‘racial shift condition’, leading them towards anti-immigration sentiment, a strong feeling towards white identity, and be more likely to hold more right-wing views. Whites high in ethnic identification showed greater group status threat than the control (.29) in the racial shift condition while whites low in ethnic identification did not. So, white identitarians showed a greater feeling of threat towards the group than did progressive leftists and antifas. Can’t say I’m too surprised. I did theorize in my article on the rise of the altright that either leftists have less oxytocin and altrighters have more, or that since political beliefs are heritable that high amounts of oxytocin will have one gravitate towards using their altruistic tendencies for the out-group or the in-group. This seems to be some evidence for my theory. For both right-wingers and left-wingers, ethnic identification was positively related to group status threat, but it was stronger in right-wingers. Even more evidence for my oxytocin/political beliefs theory.
White identitarians (whites high in ethnic identification) reported moderately greater positivity towards Trump as well as an even greater chance of voting for him in the racial shift scenario compared to the geographic movement scenario. Conversely, whites low in ethnic identification (progressive leftists, antifas, etc) showed less positivity towards Trump in the racial shift condition than in the geographic movement (control) condition,. However, in the racial shift condition, when one had high ethnic identification it led to increased positivity and a higher chance of voting for Trump. However, in the geographic condition, ethnic identification was unrelated to positivity towards Trump as well as voting for him.
Whites who showed less identification showed somewhat less support towards Sanders, being somewhat less likely to vote for him in the racial shift condition than in the geographic movement condition. In whites low in ethnic identity, neither condition (racial shift or geographic movement) had any effect on voting for Sanders or positivity towards him. Now here’s the good part: in the racial shift condition, whites high in ethnic identity showed somewhat less support and positivity towards Sanders in the racial shift condition compared to the geographic shift condition. Moreover, in the racial shift condition, ethnic identification was negatively correlated with positivity and chance of voting for Sanders, whereas in the control condition ethnic identification showed no effect.
In the racial shift condition, white identitarians were more supportive of anti-immigration policies than progressive leftists, while whites low in ethnic identification showed no difference, regardless of the condition. Ethnic identification was related to anti-immigration policies in both the racial shift and geographic movement conditions, but it was stronger in the racial shift condition.
White identitarians did not differ in outlook on political correctness by condition, while whites who show less ethnic identity reported less opposition to political correctness. Ethnic identification and anti-PC views were positively related in the racial shift condition but unrelated in the geographic shift condition.
Exposure to the racial shift condition vs. the geographic movement condition elicits different responses based on one’s political alignment and ethnic identification. Exposure to the racial shift condition increased group status threat, support for Trump and support for anti-immigration policies while somewhat decreasing support for Sanders, but only among whites high in ethnic identification. Conversely, for whites low in ethnic identification in exposure to the racial shift, there was no effect on group status threat, support for Sanders or anti-immigration sentiments and actually led to a decrease in positivity for Trump. That’s pretty powerful right there.
The support and election of Donald Trump is showing a paradigm shift in this country as ethnies in America start voting on racial lines. As diversity continues to increase and as more white Americans begin to realize the ethnic replacement will begin to impede on how many resources they have access to as well as the ‘racism being flipped on them’ with ‘less bias on minorities being more bias towards whites’, more and more whites will start voting not on party lines, but ethnic lines like all other ethnies in this country do. In the racial shift group, whites high in ethnic identification showed increased support for Trump and anti-immigration policies, increased opposition towards political correctness and decreased Sanders support through group status threat. Conversely, in the racial shift group, reminders of ethnic replacement in whites low in ethnic identification showed decreased Trump support and his policies and did not lead to group status threat. This can be termed ‘ethnic suicide’. Clearly, increased diversity is a threat to some but not all white Americans.
What boggles my mind is that when whites low in ethnic identification were reminded of the projected ethnic replacement by 2042, they decreased support for Trump and increased support for anti-immigration policies and their support for norms that prohibit bias in hate speech, which was not mediated by the group status threat. The authors put forth one theory why this may be the case. They say that whites low in ethnic identification were thinking of the changing racial demographics on the country as a whole, not just on their own ethnic group which may have led them to support a candidate who is tolerant of diversity and antibias norms. Reminding Americans high in racial identity of ethnic replacement increasingly shifted support to Trump and away from Sanders. Though this effect was not seen in relation to other candidates, the authors attributed this to Trump’s stance on immigration and political correctness relative to the other Republican candidates. To those white Americans with a high racial identity who experience group status threat, they would be drawn to Trump and his anti-immigration, anti-PC speech. The authors state:
Of all of the candidates, Trump has been most vocal in his opposition to “outsiders” such as Muslims and illegal immigrants from Latin America, and most openly critical of “political correctness” in both his rhetoric and his behavior. Trump’s rhetoric and policies thus appear to hold special appeal for White Americans highly in racial/ethnic identification who are concerned about the declining position of Whites in American society and who often perceive reverse discrimination as prevalent. In contrast, Sanders may have been perceived as the most inclusive candidate and thus most likely to exacerbate threats to White’s status as a group.
This sums up the 2016 election in one paragraph. White Americans high in racial identity showed a greater chance to vote for Trump, greater opposition to political correctness and were more likely to espouse anti-immigration sentiments.
Political leaning affiliation had a large and expected effect on candidate choice as well as policy preferences. Compared to Dems, Republicans reported much stronger support for Republican candidates than Democratic candidates while being more supportive of anti-immigration and “more un-PC attitudes”. However, when reminded of ethnic replacement, both Democrats and Republicans who showed high racial identification were more likely to lean right and vote Trump. This study shows important implications about group identity and intergroup process to voting preferences. In whites high in racial identity, increased racial diversity affects voting preferences amongst whites, with the strength of the racial/ethnic identity moderating the effect. I.e., the stronger a racial identity one has the more likely they are to support Trump and anti-immigration policies, irrespective of political leaning. Due to this study, psychologists and political scientists need to begin to pay attention to the increasing concerns of whites high in racial identification, while traditionally thinking that white Americans’ politics weren’t driven by white identity, deeming them to be unimportant to whites’ political outlooks. For example, one study showed that “racial identification, perceptions of discrimination, and linked fate were only weak predictors of White Americans’ attitudes on policies related to race and immigration. This led them to conclude that “Whites’ whiteness is usually likely to be no more noteworthy to them than is breathing the air around them” (Sears and Savelli, 2006, p. 901).
However, the current political climate shows that this no longer is the case. As more non-whites immigrate into America, whites who have high racial identity, irrespective of political leaning, will become more open to supporting Trump (or people like him) as well as anti-immigration policies. As the white majority in America shrinks, more and more white Americans will be open to white identity politics to get back their rightful resources in the country as well as the demographic majority. Eventually, with more and more unchecked immigration, white identity will start to become a central part in white American politics and voting blocks. White Americans who regard their identity as ‘white’ and an important part of their identity, future white American political preferences will be molded by group status threat as well as opposition to diversity. Trump has ‘tapped into’ the demographic of white Americans who feel looked down on in their own home country from mass immigration from the South (and soon from MENA countries). White Americans who feel that their numerical advantage is threatened are more likely to vote for Trump and support anti-immigration policies that will begin to benefit American whites.
It is, however, important to note that Trump may not be who he says he is (like most politicians). On election night last month I blogged on Donald Trump and Ethnic Genetic Interests. I showed that contrary to the average perception of him, his interests lie with Israel, not with his own racial group (due to his children marrying Jews). Moreover, he has already reneged on his wall, deporting illegals and his supposed moratorium on Muslim immigration into the US from threat countries. If anything, Trump is just a stepping stone towards more nationalistic attitudes in the US for whites. With the increased diversity, whites will start to see that they are becoming replaced by other ethnies and in whites with high racial identity, it will trigger nationalistic attitudes and responses to the impending threat on their unique genetic code. This will help to foster the awakening of more whites to identity politics, voting in their own ethnic interests and not for the interest of other ethnies.
I personally hope this leads to a renaissance of race-realism in America, but I may be aiming the bar too high. The conclusion of this study is hopeful for the status of whites in America, however. The more whites that get exposed to diversity AND have high racial identity will then lean more towards Trumpian policies. As whites decrease in number in the US, more and more whites will begin to vote for themselves and, in my opinion, once these nationalistic attitudes appear in the white consciousness in America, this demographic replacement can begin to be reversed. If it were not for the increased immigration, however, this would not have happened. The increased immigration is a main driver of these feelings towards political correctness and anti-immigration. The more anti-white sentiment that is heard in America, the more whites high in racial identity will move towards the right while leftists will continue to commit ‘ethnic suicide’.
The takeaway from this paper is this: Whites exposed to the racial shift high in racial identity were more likely to support Trump, anti-immigration policies and be anti-PC. Whites in the racial shift condition who showed low racial identity showed the opposite and were more likely to vote for Democratic candidates. This paper shows good news in the future for whites in America and voting in their interests. Whites in America are beginning to vote for their ethnic genetic interests and this is largely due to genetic similarity theory as immigration from MENA countries and South of the border increase into America. Moreover, with Trump’s allegiance to Israel, Trump is just a man to awaken more people to the realities of immigration. So Trump himself won’t do anything, but his anti-immigration rhetoric is having people notice the realities of immigration and ethnic replacement in America.
Today is the 4th anniversary of Jensen’s death, with the 4th anniversary of Rushton’s being three weeks ago on October 2nd. The fact that two of the biggest names in the IQ game, and race science game died in a 3-week span was crushing to the truth. When they were pushing their theories on racial differences in intelligence, they had their classes stormed in on and they pretty much couldn’t teach. All of this, in a country where so-called ‘freedom of speech exists’, we can have people shout others down when they speak uncomfortable truths when they don’t like what they’re hearing. BUT, just because those truths don’t want to be heard does not change the reality of them. Rushton and Jensen were pretty much explaining why black Americans have less academic achievement over whites. But when a genetic explanation is brought up instead of the Left’s want for there to be a fully environmental, ‘racism’ explanation they storm classrooms and protest against the so-called ‘fascists’ who are just reporting what they find.
In Rushton’s article The New Enemies of Evolutionary Science, he extensively talks about the derision he faced from students while he gave his lectures. It was so bad that he couldn’t teach:
Behind the scenes, however, I became the target of a witch hunt by some of the administrators. Dismayingly, my dean, a physical anthropologist, publicly declared that I had lost my scientific credibility and spearheaded an attack on me in the newspapers. She issued a series of preemptive statements making plain her negative opinion of me and my work. “What evidence is there for this ranked ordering of the evolution of the human races?” she wrote. “None.” Claiming that her views represented only her academic opinion she emphasized that she was not speaking in any administrative capacity. Her letter was nonetheless widely interpreted in the media as a refutation by my “boss.” Henceforth, in order to support me, a person would now have to go up against the dean in addition to prevailing opinion. Next, the chair of my department gave me an annual performance rating of “unsatisfactory” citing my “insensitivity.” This was a remarkable turnaround because it occurred for the same year in which I had been made a Fellow of the prestigious John Simon Guggenheim Foundation. My previous twelve years of annual ratings had been “good” or “excellent.” Indeed, my earlier non-controversial work had made me one of the most cited scholars at my university.
Some radical and black students mobilized and held rallies, even bringing in a member of the African National Congress to denounce me. In one demonstration, a mob of 40 people stormed through the psychology department, banging on walls and doors, bellowing slogans through bull horns, drawing swastikas on the walls, and writing on my door “Racists Pig Live Here.”
The administration responded by barring me from the classroom and ordering me to lecture by videotape on the pretext that they could not protect me from the lawlessness of students. Again I launched formal grievances. After a term of enforced teaching by videotape, I won the right to resume teaching in person, though then I was required to run a gauntlet of demonstrators shouting protests and threats. Only after several forced cancellations of my classes did the administration warn the demonstrators that further action would lead to suspension and legal action. That brought the protests to a halt.
This sounds just like today huh? Seems like much hasn’t changed in the past 30 years. The amount of derision that Rushton faced just for his areas of research interest speaks volumes. Clearly, people are scared of the truth about human nature, whether individually or racially/ethnically. The fact that Rushton couldn’t teach because his classes kept getting interrupted shows that people don’t care about factual data, especially when it hurts their feelings or conflicts with any type of egalitarian nonsense they have in their heads. Once challenged, they get mad, scream ‘racist’ and simply do anything in their power to make life a living hell for the person who dares to speak non-politically correct things. But, and a huge but, just because one stops, for the time being, this from being spoken of, DOES NOT make the racial differences between groups go away. People look for any and all types of environmental explanations to attempt to explain away racial IQ gaps and other racial differences both physically and mentally, but they don’t hold up to the genetic explanation, which was shouted down by people who don’t want their fragile reality broken.
The same thing, of course, happened to Arthur Jensen:
In the 1990s protesters in London pelted him with tomatoes at a lecture hall. “Jensenism” became a term of rebuke, used against those who championed theories about whites’ superiority. “Jensenism,” evolutionary biologist Stephen Jay Gould once declared, rested “on a rotten edifice.”
AR: Well, the sorts of things that you’ve been telling me, the sorts of things you’ve been doing research about, can you and do you freely teach these things in your classes?
Jensen: I sure do. I soft-pedaled things 20 years ago, and even then, there were great protests. I had students who would drop the course if these things were brought up even in a very mild way, in a hypothetical way. Students today wonder what all the shouting was about.
AR: Is that so?
Jensen: Yes, it’s rather hard to get students to believe that there were these protests and so on. They take a lot of this for granted. Oh, there’s been a great change in the students in that respect … But even in 1969-1970, I never saw a black in any of these demonstrations.
AR: Is that right?
Jensen: Not a one.
AR: They were SDS [Students for a Democratic Society]-types?
Jensen: All SDS and Progressive Labor Party, mainly. I tried to put them out when they tried to audit my course, because they were hecklers, and so some of the SDS people would sign up for the course. Of course, then they’d have to do the assignments and take the exams.
Interestingly enough, they usually were the top students in the course because they did so much outside reading to try and give me a bad time. They would go out and read everything Galton wrote! They were bright students. They just happened to be political radicals. “
Years ago, if I gave talks at the APA or the American Educational Research Association, the least little thing you’d say, people would get up on the floor and start denouncing you. I haven’t run into that for a long time, except in Canada and Australia. There’s about a ten year cultural lag in those places, I think, on this topic.
AR: I guess nowadays, as compared to fifteen or twenty years ago, you’re not a notorious presence on campus? People don’t say, “There goes Jensen!” You just don’t get that anymore?
Jensen: No, no. I used to. I used to have to be accompanied around campus by two campus policemen. In fact, they told me not to leave my office and go to the library, or any place, except to go to the men’s room around the corner, but not anywhere else without calling the campus police. They’d whiz across campus in a car and they’d be here in just a couple of minutes and walk with me wherever I wanted to go. One year I had two campus policemen, plain clothes men, in all my classes. They audited my courses.
He had to be escorted around campus by two campus police, all because of his research areas of interest. Why did Jensen have to be protected by two men in plain clothes just for talking about his scientific research? There is a clue in what he said: the Students for a Democratic Society (SDS) and the Labor Party, not coincidentally, leftists who like to censor free, true speech when it doesn’t match to their false views of egalitarianism. But as we know, just because the causes for racial differences aren’t spoken of, that doesn’t mean that they go away. Is race a causal factor in intelligence? Yes, it is. ‘Discrimination’, a so-called ‘stereotype threat’ is not the cause for lower black intelligence. Rushton and Jensen showed that the races differ by 1.2 SDs at the most, and that it was 80 percent genetic in nature (Rushton and Jensen, 2005: 279). Were the public to accept truths such as this and not any false ‘truths’ such as what the left puts out, Rushton and Jensen would not have received the derision they did when they were publishing and pushing their theories decades ago. I recall a specific poster that I saw on AmRen a few years back talking about how Jensen was a ‘racist’ and along with Shockley, both needed to be fired and lose their jobs because they just so happened to speak these uncomfortable truths.
When I met Phil in person for the first time the following year, I could not believe that a man so intensely hated in public (nearly always by idiots who did not know him personally and who did not know anything about science) could be so gentle, genial, and generous in person. His very kind and mild manners always impressed me, especially in stark contrast to how people thought and assumed he was.
There is one very small consolation in Phil’s tragically early death: Phil was not an artist, he was a scientist. When an artist dies, his art dies with him, which is why there has not been (and will never be) Beethoven’s 10th Symphony or Guernica II. Unlike art, however, science is cumulative. The rest of us can honor his memory and his scientific legacy by continuing his work. Phil was simultaneously a tremendous role model and a very tough act to follow. He was a model of scientific integrity. Unlike Galileo, he never recanted.
Science is cumulative. He, Jensen, Murray, Herrnstein, Gottfredson, Lynn et al are Modern Day Galileos. They spoke out against this leftist paradigm that has persisted in universities for decades, and even while shown extreme derision, they never recant their statements because they know they are just speaking the truth. Any allegations of so-called ‘racism’ are just that: allegations with no factual backing behind them. The fact that they need to be called childish words such as ‘racist’ and they need to have their lectures and classes interrupted by ‘protests’ speaks volumes on the research they were doing. The fact that the left has nothing to say except untrue vitriol shows who was right. When you have to attempt to silence someone because you don’t like what they’re saying or it ‘offends you’, that says volumes about your character than the one’s character you’re trying to spew vitriol at.
The fact of the matter is, no matter what anyone or any entity does to attempt to hide racial differences in intelligence, or any other trait for that matter, the truth will always come out. When answers one receives don’t line up to what one sees in his day-to-day life, one goes and looks for the truth. And that truth is that some races aren’t as intelligent as others which leads to differences in scholastic achievement and life success. These differences persist through the generations and when a downtrodden individual (no matter the race) realizes that those have a better social standing then they, they make mental leaps in their head that ‘they’re being held down’ or ‘the man is out to get them’ not even thinking that it’s their own innate ability that’s holding them down in comparison to the other group.
Rushton and Jensen will be fully vindicated one day. It’s only a matter of time. David Piffer showed that IQ alleles differ in frequency between races, meaning that a FULL vindication of Rushton, Jensen, Gottfredson, Murray, Herrnstein, Lynn, Kanazawa et al are going to come soon.
RIP to two great men who had a huge effect on my worldview and the reasons for differences around the world. I only hope that others rise up to continue their work and prove, once and for all, that they were right.
In the past 100 years since the inception of the IQ test there have been racial differences in test scores. What causes these score differences? Genetics? Environment? Both? Recently it has come out that populations do differ in allele frequencies that affect intelligence. David Piffer’s “forbidden paper on population genetics and IQ” was rejected by the new editor of the journal Intelligence. In the paper, he shows how IQ alleles vary in frequency by population. One reviewer even said it should not be put up for review, which Piffer believes there was a hidden agenda or a closed minded attitude. He even puts reviewers comments and responds to them. He says science should be transparent, which is why he’s showing the researchers’ comments on his paper.
His December, 2015 paper titled: A review of intelligence GWAS hits: Their relationship to country IQ and the issue of spatial autocorrelation shows that there are differing allele frequencies in which IQ between populations that affect IQ which are then correlated highly with average IQ by country (r=.92, factor analysis showed a correlation of .86). There was also a “positive and significant correlation between the 9 SNPs metagene and IQ”(pg. 45). However, Piffer does conclude that since the 9 alleles are present within all populations (Africans, Latin Americans, Europeans, South Asians, and East Asians) that the intelligence polymorphisms don’t appear to be race-specific, but were already present in Homo Sapiens before the migration out of Africa. He then goes on to say that it’s extremely likely that the vast majority of alleles were subject do differential selection pressure which lead increases in cognitive abilities at different rates rates in different geographical areas (pg. 49). It’s of course known that differing populations faced differing selection pressures which then lead to genotypic changes which then affected the phenotype. It’s not surprising that genes that correlate strongly with intelligence have differing frequencies in different geographical populations; it’s to be expected with what we know about evolution and natural selection. Below is the scatter plot showing the relationship between polygenic score GWAS (Genome Wide Association Studies) hits and IQ:
The fact that these differences exist should not come as a shock to those who want to seek the truth, but as seen with how David Piffer didn’t even get consideration for a revision, this shows the bias in science to studies such as this that show racial differences in intelligence exist.
Piffer’s data also corroborates Lynn and Meisenberg’s (2010) finding of a correlation of .907 with measured and estimated IQ. This shows that the differing allele frequencies affect IQ, which then affect a countries GDP, GNP, and over all quality of life.
With a sample with a huge n (over 100,000 subjects) cognitive abilities tests were performed on verbal-numerical reasoning, memory and reaction time (a huge correlate for IQ itself, see Rushton and Jensen, 2005). Davies et al (2016) discovered that there were significant genome-wide SNP based associations in 20 genomic regions, with significant gene-based regions on 46 loci!! Once we find definitive proof that intelligence differences vary between individuals, as well as the loci and genomic regions responsible, we can then move on to difference in allele frequency in depth (which Piffer 2015 was one of the starts to this project).
Moreover, genes that influence intelligence determine how well axons are encased in myelin, which is the fatty insulation that coats our axons, allowing for fast signaling to the brain. Thicker myelin also means faster nerve impulses. The researchers used HARDI to measure water diffusion in the brain. If the water diffuses rapidly in one direction, that shows the brain has very fast connections. Whereas a more broad diffusion would indicate slower signaling, thus lower intelligence. It basically gives us a picture of an individuals mental speed. Thinking of reaction time tests where Asians beat whites who beat blacks, this could possibly show how differing process times between populations manifest itself in reaction time. Since myelin is correlated with fast connections, we can make the inference that Asians have more than whites who have more than blacks, on average. The researchers also say that it’s a long time from now, but we may be able to increase intelligence by manipulating the genes responsible for myelin. This leads me to believe that there must be racial differences in myelin as well, following Rushton’s Rule of Three.
Since the mother’s IQ is the best predictor of the child’s IQ, this should really end the debate on its own. Sure on average, intelligent black mothers would birth intelligent children, but due to regression to the mean, the children would be less intelligent than the mother. JP Rushton also says that regression works in the opposite way. Both blacks and whites who fall below their racial means will have children who regress to the means of 85 and 100 respectively, showing the reality of the genetic mean in IQ between the races.
Why would differing allele frequencies lead to the same cognitive processes in the brain in genetically isolated populations? I’ve shown that brain circuits vary by IQ genes, and populations do differ in this aspect, like all other differing genotypic/phenotypic traits.
East Asians have bigger brains, as shown by MRI studies. Rushton and Rushton (2001) showed that the three races differ in IQ, brain size, and 37 different musculoskeletal traits. We know that West Africans and West African-descended people have genes for fast twitch muscle fibers (Type II) (Nielson and Christenson, 2001). Europeans and East Asians have slow twitch muscle fibers (Type I) for strength and endurance. (East Africans have this as well, which allows for ability to run for distance, which fast twitch fibers do not allow for. The same is true for slow twitch fibers and sprinting events.) Bengt Saltin showed that European distance runners have up to 90 percent slow twitch fibers (see Entine, 2000)! So are genetic IQ differentials really that hard to believe? With all of these differing variables in regards to intelligence that all point to a strong genetic cause for individual differences in other genes that lead to stark phenotypic differences between the races, is it really not plausible that populations differ in intelligence, which is largely inherited?
Is it really plausible that differing populations would be the same cognitvely? That they would have the same capacity for intelligence? Even when evolution occurred in differing climates? The races/ethnicities differ on so many different variables with differing genes being responsible for it. Would IQ genes really be out of the question? Evolution didn’t stop from the neck up. Different populations faced different selection pressures, so different human traits then evolved for better adaption in that environment. Different traits clearly developed in genetically isolated populations that had no gene flow with each other for tens of thousands of years. These differing evolutionary environments for the races put different pressures on them, selecting some for high IQ alleles and others for low IQ alleles.
We are coming to a time where intelligence differences between populations will become an irrefutable fact. With better technology to see how differing genes or sets of genes affect our mind as well as physiology, we will see that most all human differences will come down to differing allele frequencies along with differing gene expression. Following Rushton’s simple rule based on over 60 variables, East Asians will have the most high IQ alleles followed by Europeans and then blacks. The whole battery of different cognitive abilities tests that have been conducted over the past 100 years show us that there are differences, yet we haven’t been able to fully explain it by GWAS and other similar techniques. Charles Murray says within the next 5 to 10 years we will have definitive proof that IQ genes exist. After that, it’s only a matter of time before it comes out that racial differences in IQ are due to differing allele frequency as well as gene expression.
Researchers have tried to manage those with Prader-Willi’s Syndrome for multiple decades. Though they have greatly curbed some of the implications of the disease, there are still numerous ways in which we can better use our knowledge of how the disease manifests in order to better help those suffering from PWS. Looking at research into how the extra chromosome 15 is linked to low IQ; IQ and its relationship to obesity; how the ability to delay gratification leads to obesity; growth hormone treatment to better treat low muscle mass and higher body fat; and finally using reinforcement theory to punish a response, where doing so will greatly diminish the probability of that response occurring again in the future; all of these factors can be used in conjunction to better mitigate problems from the disease. By examining all of these variables and thinking of better ways to handle them, we can then think of other, better ways to manage those with PWS. In doing so, we can better increase the quality of life of those suffering from PWS, as well as have less of a strain on healthcare workers who care for them. With new advances in technology with CRISPR Cas9, we can then edit the genomes and chromosomes of those suffering from this disease.
How to Use Current Knowledge to Effectively Treat and Manage the Symptoms of PWS Patients
How can we use the research on chromosomal differences, research on their IQ differences and their lack of ability to delay gratification that, in turn, would help those individuals with the disease? Seventy percent of PWS cases are attributed to the deletion of chromosome 15 (Ledbetter et al, 1981). Maternal uniparental disomy, which involves receiving an extra chromosome 15 from the mother, is yet another cause of PWS (Wang, 2004).
Whittington, Holland and Webb (2009) found that there was variation between families in deletion of chromosome 15. They found that the PWS and sibling IQ correlation was .3, a modest correlation. What was also noticed was that there were subtype differences which manifested itself in the familial differences in IQ. As they expected, the correlation with normal siblings and those with PWS was .5 in those who suffered from PWS due to unilateral disomy. But in the second subset (the chromosomal deletion subset), the correlation was negative at -0.07. Their research shows great promise in the role of chromosome 15 and IQ. They end up concluding that there needs to be an explanation for the small genetic differences between them. How can we use these differences in IQ to help people with PWS and what does this suggest for other symptoms of their disease?
Kanazawa (2014), reviewed the data on the research between obesity and IQ. What he found was that those studies that concluded that obesity causes lowered intelligence only observed cross-sectional studies. Longitudinal studies that looked into the link between obesity and intelligence found that those who had low IQs since childhood then became obese later in life and that obesity does not lead to low IQ. The average IQ for an individual suffering from PWS is 65 (Butler, Lee and Whitman 2006, p. 13), so that is one reason they have a tendency to be obese. He states that those with IQs below 74 gained 5.19 BMI points, whereas those with IQs over above 126 gained 3.73 BMI points in 22 years, which is a statistically significant difference. Also noted, was that those at age 7 who had IQs above 125 had a 13.5 percent chance of being obese at age 51, whereas those with IQs below 74 at age 7 had a 31.9 percent chance of being obese. This data makes it clear: low IQ is correlated with obesity, so we, therefore, need to find sufficient measures to help those with lower IQs who also suffer from PWS to better maintain their good health. Since we can better identify those PWS individuals who have lower IQs based on how they got the disease, we can then show them more attention in an effort to have them manage their gratification better. Moreover, the lack of ability to delay gratification is also correlated with low IQ (Mischel, Ebbeson, and Zeiss, 1972).
Schlam et al. (2013) observed in a follow-up study to the Marshmallow Experiment that found in a longitudinal study of individuals they found forty years later from the original Marshmallow Experiment, due to inability to delay gratification forty years previously, that was one cause of becoming obese forty years later. Due to PWS sufferers having lower average IQs, and, therefore, a lack of ability to delay their gratification, this is direct evidence that those PWS sufferers with low IQs need more stringent measures to be taken on them, which would then be helpful to those individuals who have a hard time delaying their gratification, which is partially caused by the drop in IQ due to the additional chromosome 15. We can see how those with PWS act; they want their gratification now and do not want to wait for it. This is why, when unsupervised, that those with PWS gorge on the food they understand they should not have, but do so, nevertheless, since their low IQ is correlated with lack of ability to delay gratification, which manifests itself in their obesity. We clearly need to find better methods in which to help those with low ability to delay gratification, which would strongly help those suffering from PWS.
Dykens et al (1997) note that those with PWS have hyperphagia, which correlates with their insatiable want for food. They state that the lack of fullness is due to an altered function of the hypothalamus, which is the part of the brain that is in control of feelings of satiety. Certain States gave restrictions to homes that take care of those with PWS and have come under fire because of this, mainly due to human rights violations. We must ask, then, should we limit their access to food if it will prolong their lives? Will doing so inhibit their freedom to do as they choose? PWS sufferers also have coronary heart problems; one could argue that given their free ability to choose what they want to do unfettered will lead to premature death due to obesity-related complications. Does their disease truly not allow them to learn the consequences of their behavior? Do they have the intellect to really understand the consequences of their actions of consuming too much food? There is no established or known way to control those with insatiable eating habits due to hyperphagia. So would the best course of action to take with those with PWS actually be to constantly monitor them and to lock access to easily attainable food? My answer is yes, however, there is a clear fine line in whether restricting access to food and constantly monitoring those with PWS infringes on their human rights, or that doing so actually will help them live better, healthier lives since they would have the constant supervision around them to better control their out of control eating habits. When negative actions occur, one idea that can be shown to them is constant positive reinforcement so that they may be better able to understand that what they are doing is harmful to their bodies. We can then use positive reinforcement when they do reach a healthy weight, so, in turn, they will have a higher chance of keeping a healthy weight. They may reap the benefits of positive reinforcement, and stick more closely to their program, and therefore, stay healthy.
The hormone ghrelin is secreted from the hypothalamus. With an altered hypothalamus, this would cause ghrelin levels to overload; then the individual suffering from PWS would feel the need to insatiably gorge on food due to this chemical imbalance in the brain. Ghrelin increased feeding in rats and ghrelin is the physiological mediator of feeding and probably has a function in growth regulation by stimulating feeding and release of growth hormone (Nakazato et al, 2001). There is a correlation between want of food, ghrelin release and growth hormone production. By attempting to mediate these variables, those who suffer from PWS will be able to better control their eating habits through positive reinforcement and better, more sustainable habits. Since whenever we eat we get a release of ghrelin that makes us hungry, people pretty much set their own eating times by eating multiple times a day. This affects PWS patients the same way. They can’t stop eating, due in part to constantly eating which constantly releases ghrelin in their body.
PWS sufferers have low muscle tone and, conversely, more body fat. Growth hormones may be a valid way of alleviating that problem, which in turn will give them a slightly higher resting metabolic rate so that they may burn slightly more calories, in an effort to stay healthier. Growth hormone therapy is great for those with PWS though they are largely inactive and lead a sedentary lifestyle, the growth hormone will allow them to have less body fat and more muscle mass. As noted earlier in my paper, those suffering from PWS have altered function in their hypothalamus, which is also where growth hormone is secreted. Aycan and Bas (2014) state that treatment with growth hormones should be strongly considered for those with PWS.
PWS sufferers are also quick to anger, which can be correlated with their sub-average IQ. They may, for instance, become irate at the fact that they do not have constant access to food, and may turn to emotional, angry and infantile outbursts in an attempt to receive what they want. This is one way that it’s tricky to treat those with the disease. How do you tell an individual with PWS who wants something “No”? Measures should be taken to show those with the disease what they are doing to their bodies in the simplest way possible as to better get the point across to them. We can help those sufferers of PWS who are quick to anger with by allowing them to discern between right and wrong ways to handle times when they don’t get what they want with positive reinforcement.
Since those who suffer from PWS have behavioral problems, there are better measures we can take to assure that they don’t have their violent outbursts. When positive reinforcement is consistently shown to an individual who has PWS, he will have more success with his program. When they do something wrong, they can then be shown positive reinforcement, and through being shown positive things with reinforcement theory, they can better learn that certain actions they take are dangerous and shouldn’t be done again, as Rushton (1980) states: “If one rewards a response, it will increase the probability of the future occurrence of that response. If one punishes a response, it will decrease the future probability of the occurrence of that response.” (p. 90).
In this paper, I have presented causes for PWS as well as effective ways to manage the disease. To look at how IQ affects individuals in regards to obesity and because it is highly correlated with other measures as well, we can then better help those with the disease. By seeing which individuals have the parental disomy version of PWS, we can then monitor them and give them better care because of their lowered IQ and make sure they stay at a healthy weight. One of the best measures to take is to heavily restrict food, i.e., make sure ability to access food at all hours of the day is restricted along with constant supervision. Though, there are rights groups fighting for them saying that their human rights are being infringed on. In allowing them to have free reign over what, how and when they eat, they will gorge themselves to obesity, as well as lead themselves to horrible complications that come along with increased food consumption. When one is caught consuming food he or she shouldn’t be consuming, punishing them and letting them understand that the behavior they took was wrong will lead to better choices and outcomes from those choices, due in part to the main facet of reinforcement theory, that punishing a response will lead to a reduced outcome in that response that was punished happening in the future. Also, with the advent of CRISPR Cas9, we will be able to edit genomes, and therefore, eventually, put an end to PWS. It will enable us to fix the chromosomal deletion and uniparental disomy, which will eradicate this disease.
There are better, more helpful ways in which to help those suffering from PWS. By identifying and attempting to correct these abnormalities, those who suffer from the disease can, therefore, have a better quality of life due in part to the extra measures taken. By understanding that their lower average IQs lead to a lot of the problems associated with the disease, we can better structure methods for them to keep on a healthy track and reinforce positive behavior through reinforcement theory. Since obesity is correlated highly with low IQ, we can, therefore, use this information to better help those who suffer from PWS that have low IQs. Locking up food instead of providing free access, as well as understanding they do not have the ability to delay gratification, would be a big start to find better ways to treat sufferers of PWS. Treating negative actions with positive reinforcement through reinforcement theory will lead to better and increased prosocial behavior. It’s been shown that if you punish a response, then it will decrease the future probability of that response occurring. The advent of CRISPR Cas9 will then allow us to edit the chromosomes of those with this disease in the future. Should we use genome editing on individuals with this disease, as well as several other chromosomal/genetic diseases? I believe we should, in doing so, we will greatly increase the quality of life of those with the disease.
Aycan, Z., & Baş, V. N. (2014). Prader-Willi Syndrome and Growth Hormone Deficiency. Journal of Clinical Research in Pediatric Endocrinology Jcrpe, 62-67.
Butler, M. G., Lee, P. D., & Whitman, B. Y. (2006). Management of Prader-Willi syndrome (3rd ed.). New York: Springer-Verlag.
Dykens, E. M., Goff, B. J., Hodapp, R. M., Davis, L., Devanzo P., Moss, F. . . King, B. (1997). Eating Themselves to Death: Have “Personal Rights” Gone Too Far in Treating People With Prader-Willi Syndrome? Mental Retardation, 35(4), 312-314.
Kanazawa, S. (2014). Intelligence and obesity. Current Opinion in Endocrinology & Diabetes and Obesity, 21(5), 339-344.
Ledbetter, D. H., Riccardi, V. M., Airhart, S. D., Strobel, R. J., Keenan, B. S., & Crawford, J. D. (1981). Deletions of Chromosome 15 as a Cause of the Prader–Willi Syndrome. New England Journal of Medicine N Engl J Med, 304(6), 325-329.
Mischel, W., Ebbesen, E. B., & Zeiss, A. R. (1972). Cognitive and attentional mechanisms in delay of gratification. Journal of Personality and Social Psychology, 21(2), 204-218.
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Rushton, J. P. (1980). Altruism, socialization, and society. Englewood Cliffs, NJ: Prentice-Hall.
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Whittington, J., Holland, A., & Webb, T. (2009). Relationship between the IQ of people with Prader-Willi syndrome and that of their siblings: Evidence for imprinted gene effects. Journal of Intellectual Disability Research, 53(5), 411-418.
YM Wang, L Chuang, BT Wang, et al. Maternal uniparental disomy in a patient with Prader-Willi syndrome with an additional small inv dup(15) chromosome. J Formos Med Assoc, 103 (2004), pp. 943–947
Transvestic Disorder comes about in early childhood and manifests itself in sexually deviant actions. Men suffering from TD who aren’t homosexual, more likely than not, show attraction to themselves dressed in women’s clothing. The signs of TD are noticed at an early age when the individual begins to cross dress. TD is also correlated highly with numerous sexually deviant actions. Fluoxetine and serotonin reuptake blockers may be able to lessen TD since it is an impulsive disorder. With TD being co-morbid with OCD, by treating OCD we can better treat TD itself and give a better quality of life to the patient suffering from the disease. Since autogynephilia and transgenderism are related, measures taken to alleviate TD and autogynephilia could be taken to alleviate symptoms of gender dysphoria, since autogynephilia leads to transgenderism.
Transvestic Disorder and Gender Dysphoria Identification and Prevention
Transvestic disorder is a paraphilic disorder, classified by the American Psychological Association (2013), in which males dress up as women to gain sexual gratification. The individual suffering from TD suffers from compulsions to want to dress as a woman, which causes distress due to the individual not wanting their secret to come out. This then leads to the quality of life of the individual to decrease due to constantly being worried about his secret coming out. TD is diagnosed when a male has sexual feelings and gets sexual arousal from dressing in women’s clothing. It is only diagnosed when these activities are ongoing for at least six months. TD is also similar to another paraphilic disorder called ‘autogynephilia’ (Lawrence, 2011), in which the subject is aroused at the thought of himself being a female, so he, therefore, then begins to dress as a woman to fulfill his sexual desires. Blanchard (1989) proposed that most gender-dysphoric males who do not show sexual arousal to men, instead show sexual arousal to themselves dressed in the opposite sex’s clothing. He concludes that the hypothesis is supported that major types of those men who cross-dress are nonhomosexual, and do so because they become aroused at the thought of dressing as a woman. The DSM V says that autogynephilia is a specifier to transvestic disorder. This is because they are characterized by the same things (American Psychological Association, 2013).
The signs of TD are noticed at very early ages. Most notable are when children begin to cross-dress at or before puberty. This then continues into their adult lives where it begins to be a problem and cause dysfunction due to needing to keep their secret. Dr. Mark Griffiths (2012) states that all though children may engage in transvestic behavior, what differentiates it between an adult suffering from TD is that the child who cross-dresses does so for excitement and pleasure, not for sexual pleasure. Though some researchers say that the disorder is brought on through childhood trauma, i.e., accidental exposure to women’s clothing or exposure to a woman who is undressing. Numerous studies have also concluded that many men who suffer from TD have had to deal with parental separation during childhood.
The American Psychological Association (2013), reports that fewer than 3 percent of males are characterized as having transvestic disorder. TD is most always seen in males, though Moser (2009) noted that in his study using the Autogynephilia Scale for Women (ASW), that out of the 29 respondents that sent back questionnaires, 90 percent would be classified as having autogynephilia. Though, by using a more meticulous definition, only 28 percent were seen to be autogynephilic (Moser, 2009).
Langstrom and Zucker (2005) observed in a sample of 2,450 18 to 60-year-olds in Sweden that transvestic disorder was correlated significantly with being separated from their parents, homosexual relations, higher masturbatory frequency, being easily aroused sexually and pornography use. Also noticed, was a positive attitude in regards to sexual arousal from pain, exposing oneself to a stranger and voyeurism were all positively correlated with TD. Langstrom and Zucker observed how TD is co-morbid with many other paraphilic disorders as well as other deviant behavior. By attempting to treat what TD is correlated with, symptoms of TD can be lessened.
Men suffering from TD and autogynephilia are told that women are the standard of beauty. They then look at themselves in the mirror and see a male and not the standard of beauty they were told of growing up. They then turn to cross-dressing to finally see their “beauty standard” in the mirror but keep it a secret. This strong want to keep their disorder a secret then leads to dysfunction. Men suffering from TD will go to any lengths to hide their secret. This then causes extreme dysfunction in their lives, which leads to a lessened quality of life.
Less than three percent of males suffer from TD in the American population, as such, it is classified as a deviant lifestyle as it deviates from the norm of the population. It causes distress due to them not wanting their secret to be discovered. This, in turn, leads to dysfunction where the individual cannot live their daily lives to the fullest due to their abnormal disorder. It finally leads to danger due to their secret beginning to consume their lives so that they’re not discovered.
There are ways to treat TD. Usmani et al (2012) follow a case study in which a 17-year-old Indian male who had occurring desires to wear his mother’s clothes. He then would masturbate in his mother’s clothes to alleviate himself. This continued on for two years so he could pleasure himself. He was caught by his parents wearing his mother’s clothes and was beaten by them for it. He then said that it is a compulsive behavior and cannot be helped. This case also shows the obsessive compulsive side to TD. They have an urge so strong they cannot help but to do it compulsively to alleviate their sexual desires. He also said that the occurring thoughts then affected his schoolwork as he was so preoccupied with the thought of wearing women’s clothes. All of his brain scans were found to be normal, so what brought on this case in the individual in the case study? He was then diagnosed with TD and prescribed fluoxetine, an antidepressant SSRI. The dose was started at 20 mg and increased by 40 mg once a day for two weeks. In his six-month follow-up, he reported lessened desire to masturbate with women’s clothes (Usmani et al, 2012).
Paraphilias and other related disorders have been thought of as sexual addictions. Though it has been argued that they are not sexual addictions, but are sexual compulsions (Stein et al, S 1992). The researchers reviewed 13 patients who showed signs of TD and were administered serotonin reuptake blockers. The symptoms of those individuals were then divided into paraphilias, non-paraphilic sexual addictions, and sexual addictions. Stein et al discovered that paraphilias had the least improvement with the reuptake blockers whereas sexual compulsions showed the best improvement. They end up concluding that paraphilias and other related disorders are on the impulsive end of the spectrum compared to the compulsive end. These impulsions, then, have those men suffering from TD have the urge to dress in women’s clothes to fulfill their sexual impulsion.
TD is co-morbid with obsessive compulsive disorder (Abdo, Hounie, de Tubino Scanavino, and Miguel, 2001). They used longitudinal case studies in which they assessed two individuals who had OCD as well as TD. They conclude that some cases of TD may be OCD related and not always be caused by gender dysphoria. Since OCD and TD are co-morbid, by treating symptoms of OCD, the want to cross-dress will lessen, which will then lessen the symptoms of TD. Treatments could include SSRI and fluoxetine, as previously stated in the paper. Other treatment for TD should be looked at, such as treatment for OCD due to the co-morbidity between the two. By doing so, feelings of wanting to cross-dress may lessen due to one of the underlying causes (OCD) of TD being treated.
Autogynephlia could also explain transgenderism.Transvestism can be called both a paraphila and a sexual orientation. Lawrence (2004) says that it can explain mid-life MtF transitions, progression from transvestism to transgenderism, the prevalence of other paraphilias among MtF transsexuals and the late development of male intrest in MtF transsexuals. However, when Lawrence says that “Hormone therapy and sex reassignment surgery can be effective treatments in autogynephilic transsexualism”, that is incorrect. The prevalence of suicide attempts among transgenders is 41 percent according to the Williams Institute, UCLA School of Law, in comparison to 4.6 percent for the average population. That’s almost ten times higher than the national average. Clearly, surgery doesn’t do anything to alleviate the feelings of gender dysphoria, and as shown in this paper, therapy and drugs like Prozac can better help to alleviate feelings of gender dysphora in transsexuals due to them being extremely similar to eachother. These two disorders greatly mirror each other. Since Lawrence (2004) observed that there is a progression from transvestism to transgenderism, using similar techniques that work on those with TD may also work on those with gender dysphoria.
TD can be helped with the correct therapy as well as right medication. With those, impulsions to wear women’s clothes, as well as impulsions to commit abnormal acts will be greatly lessened and quality of life will be restored to a somewhat normal level. Due to co-morbidity between TD and OCD, treating OCD will, in turn, better help the patient suffering from TD. When more studies are carried out on those suffering from TD, we can see whether or not SSRI drugs and fluoxetine will have the desired effect in alleviation of the symptoms of TD. The individual in the Usmani study reported lessened symptoms and impulsions of cross-dressing, so by identifying which parts of the brain are and were activated during the fluoxetine therapy, we can then better give better care and treatment to those suffering from TD. We can also use some data from TD cases for transgenders, as TD and transgenders have a lot of things in common. With therapy as well as, maybe even fluoxetine (which is just Prozac), and high doses of testosterone/estrogen, this could possibly help to alleviate ‘gender dysphoria’. It could also lower the suicide rate as it’s completely possible that these interventions could fix them mentally.
There is little current literature in treating TD, due to it being a shameful disorder and many men not speaking about what they suffer from. One major way in which to help those with TD is to administer SSRI drugs, in which compulsion to cross-dress, and other attitudes associated with TD lessened. Blanchard (1989) proposed the autogynephilia theory for those transgenders who are not attracted to men. With the obseration by Lawrence (2004) on how autogynephilia and transgenderism are related, this can better help those with transgenderism, as they can get correct help and the right hormones they need, instead of the opposite hormones. SSRI therapy is a good candidate in treating TD, as drastic changes in deviant behavior are seen while the patient is taking the SSRI drug. Seeing as most cases of TD begin in childhood before puberty, by better identifying warning signs of these disorder, we can better treat those children who are at risk of developing these disorders before they become a big problem later in life. As more men come out and say that they suffer from these disorders, more studies can be carried out that corroborate the findings in the studies laid out here. It is extremely promising that these disorders can be treated with common drugs already on the market. In those individuals suffering from TD as well as OCD, by treating the OCD first (which may be an underlying cause) the symptoms of TD may be lessened and the individual may eventually have the ability to lead a life without TD. In using these measures on those with transgenderism, this could possibly alleviate suicide rates and other negative variables associated with these paraphilic disorders and sexual orientations.
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