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Obesity and Intelligence
1600 words
[Edit: My view here has changed, read my recent article Is Diet an IQ Test? It isn’t and it is, of course, much more nuanced than ‘IQ’ (which is a proxy for social class’ leading to obesity which would imply lack of funds and education on what and when to eat. Obesity is much more complex than ‘IQ’, numerous other variables come into play and since ‘IQ’ (which is just a proxy for general knowledge ‘is low then the individual in question won’t know what and when to eat and since this occurs in low income families more often than not who have low IQs then this effects them the most.]
The relationship between intelligence and obesity is often misinterpreted. Numerous studies have concluded that becoming obese leads to a drop in IQ. This mistake happens due to improper interpretation of cross-sectional studies. However, analyses of population-based, longitudinal data show that low intelligence from birth causes obesity. No credible evidence exists for obesity lowering intelligence. There are, however, mountains of evidence showing that low intelligence from childhood leads to obesity (Kanazawa, 2014).
Kanazawa (2014), reviewed the data on the research between obesity and IQ. What he found was that those studies that concluded that obesity causes lowered intelligence only observed cross-sectional studies. Longitudinal studies that looked into the link between obesity and intelligence found that those who had low IQs since childhood then became obese later in life and that obesity does not lead to low IQ. Those with IQs below 74 gained 5.19 BMI points, whereas those with IQs over above 126 gained 3.73 BMI points in 22 years, which is a statistically significant difference. Also noted, was that those at age 7 who had IQs above 125 had a 13.5 percent chance of being obese at age 51, whereas those with IQs below 74 at age 7 had a 31.9 percent chance of being obese. This data makes it clear: low IQ is correlated with obesity, so we, therefore, need to find sufficient measures to help those with lower IQs to learn how to manage their weight. Moreover, the lack of ability to delay gratification is also correlated with low IQ (Mischel, Ebbeson, and Zeiss, 1972).
Less intelligent individuals are more likely to become obese than those who are more intelligent. With what we know about low IQ people and how there is a strong relationship between low intelligence and lack of ability to delay gratification, we can see how this lack of thought for future problems for their actions in the present can manifest itself in obesity.
This study claims that there is a link between morbid obesity and a drop in IQ. The researchers compared 24 children who weighed 150 percent of their bodyweight before age 4 with 19 children and adults with Prader Willi’s Syndrome, using 24 siblings as controls as “they share the same socioeconomic environment and genetics”. Prader Willi’s Syndrome (PWS) is a chromosomal disorder in which chromosome 15 is deleted. They have an almost insatiable desire to eat,which can cause one suffering from PWS to eat themselves to death. Those with PWS were found to have an IQ of 63, while those who became obese were found to have an IQ of 78 with the control siblings having an IQ of 106. The researchers were surprised to see such a difference in IQ between siblings. They then state that this could be one facet of obesity that could be irreversible. MRI scans of the cohort discovered white matter lesions on the subjects with PWS and early-onset obesity. The researcher says that these lesions could affect food seeking centers in the brain leading to a want to gorge on food. Seeing how those with PWS eat when unsupervised, this is an interesting hypothesis.
This study compared 49 teens with metabolic syndrome and 62 peers without the disorder, while controlling for socioeconomics status. They found significantly lower scores in arithmetic, attention and attention span, spelling, mental flexibility and regions of the brain with lower volumes of matter in the hippocampus and white matter integrity.
There are a few problems with these two studies. In a population-representative birth cohort study of 1037 children, it was found that cohort members who became obese had a low IQ, as expected. But, contrary to what your study said, cohort members didn’t exhibit a decline in IQ from becoming obese, they instead had a lower IQ since childhood. There is no evidence of obesity contributing to a decline in IQ, even in obese individuals and those on the verge of metabolic syndrome. Another problem is that they wrongly conclude that obesity leads to lowered intelligence, completely misinterpreting the extremely strong negative correlation between obesity and intelligence.
This study shows how obese mothers give birth to less intelligent children. In an observational study (already garbage), the researchers took 3412 participants and found a strong relationship with pre-pregnancy obesity and math and reading scores in children. For math, a 3 percent reduction was observed. There was a 3-point drop in reading scores with math scores showing a decline of 2 points. These differences are within the normal variation between tests, so it’s nothing to take note of. Also, this is an observational study. I have shown above that longitudinal studies are superior for this, as well as researchers misinterpreting the results found from their studies.
So because of those factors involving the mother and child, that is what accounts for it. Not the environmental factors brought up.
This study claims that overweight parents are more likely to fail. This is all due to the fact that low IQ people are more likely to be obese or overweight, with heritability of BMI being .8, you can see how low IQ is the cause of both of those variables.
This shows that binge eating is linked to memory loss. I heard about a study a few months ago actually like this. Rats were fed high fat diets and they noticed that the brain microglia actually started to eat neuronal pathways actually leading to a decrease in cognitive ability. But they said that returning to a new diet will stop its effects. Researchers say the negative cognitive effects are reversible, but I already gave the citstion about obesity not being linked to decreased IQ. I should also note that this study was carried out on rats and while this may be a factor for humans as well, a few studies need to be done.
Binge eating, however, actually has a genetic component. Though this was only observed in girls. One reason I can think of for this is that women need higher body fat for a leptin release so puberty can begin so they can bear children.
This article purports to show 5 ways obesity affects the brain. Obesity does cause food addiction, however, those who lack the ability to delay gratification are more likely to not be able to control their impulse to overeat. I always link to the MRI scan showing the control, obese and cocaine user’s brain. Interesting to see that sugar is just as addictive as cocaine. Obesity doesn’t make us more impulsive. Check out the Marshmallow Experiment, as well as its follow-up studies. Those who are more impulsive are more likely to be obese, as well as have lower SAT scores.
Satoshi Kanazawa also noted that childhood IQ predicted whether or not one would become obese at the age of 51. General intelligence in childhood has a direct effect on weight gain, BMI, and obesity, net of parents education and SES, parents BMI, the child’s social class, and sex. More intelligent children grew up to make healthier choices, and therefore stayed leaner than those children who were less bright. The link between childhood obesity and intelligence also shows that the effect between childhood g is unmediated by education of income. Meaning, those with lower IQs in a higher socioeconomic bracket STILL have the same chance of becoming obese as those in the lower socioeconomic bracket. Finally, parental BMI itself is a consequence of parental general intelligence, which the parents pass on to their children. This shows the extremely high heritability of obesity as well as showing how intelligence plays a factor in the causes of obesity.
The known differences in ethnic obesity rates generally mirror the intelligence of those populations. All populations are showing a sharp dysgenic decline, which coincides with a more obese population as well. Sociologists and the like may say that those who are poor cannot afford the same types of food that those who have more wealth can. However, this is a false statement. Whole foods are not more expensive. The conclusion that was (obviously) reached is that there is expensive and non-expensive junk food as well as whole foods. Natural diets will not cost more, all things being equal. If you know how to eat and how to buy food, you will avoid spending too much money. This goes back to intelligence. One with a higher IQ will be able to think of what his present actions will lead to in the future while those with a lower IQ live in the now without a care for the future, which then manifests itself in their obesity.
There are numerous articles showing that the causality for low intelligence is not becoming obese, but that those who become obese have a lower IQ since childhood. Longitudinal studies show the relationship, while observational studies show that obesity drops intelligence. Clearly, observational studies are inferior for seeing the relationship between IQ and obesity. This then leads to researchers misinterpreting the data and drawing wrong conclusions.
** This is a great one. In a meta-analysis of twin and family studies, including mono and dizygotic twin studies, with a sample of 140,525 people, heritability of BMI was found to be between .75 and .82. Both extremely high correlations. Since the heritability of intelligence as well as height (another good predictor of intelligence), there is good evidence for the claim that becoming obese is due to lower childhood IQ, which is genetic in nature.
Neanderthals, Inbreeding, r/K Selection Theory and Eurasian Birthrates
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(Note, 6/24/17: Rushton’s r/K selection in applications to human races is dead. It’s been dead for almost 30 years after and ecologist critiqued his method and use of ecological theory in application to human races. Now, that doesn’t meant that everything written below—or even on my whole blog—is fully wrong, just that the attempted explanation is wrong. It still holds that Eurasians have worse fitness than Africans, which is partly due to deleterious Neanderthal variants, however, r/K theory does not explain it.)
Science Daily reported last week that Neanderthals left humans a genetic burden, which is having less offspring. Of course, these deleterious alleles only introgressed into non-African populations due to Africans not leaving Africa. This manifests itself today in birth rates within countries and between them based on the ethnic/racial mix. And (not) coincidentally, the areas with the highest rate of children are in sub-Saharan Africa.
The Neanderthals existed in small bands, so inbreeding was common. Due to this inbreeding, Neanderthals were more homogenous than we are today. When humans migrated out of Africa, they encountered the inbred Neanderthals who they interbred with. Harmful genetic variants acquired from Neanderthals are shown to reduce the fitness of populations with certain deleterious alleles. There are of course tradeoffs with everything in life. Increased intelligence and being better able to weather the Ice Age, among numerous other factors, were positive things gained from interbreeding with Neanderthals. Negative effects were the acquisition of deleterious alleles which still persist today in non-African hominids. These deleterious alleles decreased biological fitness which manifests itself in the birthrate of Eurasian populations throughout the world (the Germann and Japanese birthrate is 1.3 for reference).
Harris and Nielson also hypothesize that since Neanderthals existed in small bands that natural selection was less effective, allowing for weakly harmful mutations to pass on and not get weeded out over the generations. However, when introduced back into humans these effects become lost over time due to a large population with natural selection selecting against the deleterious Neanderthal alleles. Using a computer program, Harris and Nielson quantify how much of a negative effect the Neanderthal genome had on modern populations. The conclusion of the results was that Neanderthals are 40 percent LESS genetically fit than modern humans.
The researchers’ simulations also suggest that humans and Neanderthals mated more freely, which leads more credence to the idea that Neanderthals got absorbed into the Homo Sapien population and not mostly killed off. The estimation for Neanderthal DNA in modern hominids from the simulation was around 10 percent, which then continued to drop as the Neanderthal-Homo Sapiens hybrids interbred with those who hardly had any Neanderthal DNA. More evidence also shows that the percentage of Neanderthal DNA was higher in the past in Eurasians as well. Which makes sense since Asians have on average 20 percent more Neanderthal DNA than Europeans due to a second interbreeding event.
However, Harris and Nielson end up concluding that non-Africans historically had a 1 percent loss in biological fitness due to Neanderthal genetics. Moreover, a better immune system came from Neanderthal genetics. Skin color is another trait inherited from Neanderthals as well.
Along with the acquisition of deleterious Neanderthal alleles, early Eurasians also encountered the same environment as the Neanderthals. Those selection pressures, along with interbreeding due to small bands lead to a decrease in the number of children had. Fewer children are easier to care for as well as show more attention to. All of these variables in that environment lead to fewer children produced. It’s a better evolutionary strategy to have fewer children in more northerly climes than in more southerly ones due to the differing selection pressures. Environmental effects are also one reason why birthrates are lower for populations that evolved in northerly climes (Neanderthals and post-OoA hominids). Harsh winters lead to a decreased population size, as evidenced by the Inuit and Eskimoes, which their low population size didn’t allow for selection for high IQ despite having the same brain size as East Asians.
I couldn’t help but think that, yet again, for the second time in two weeks, one of JP Rushton’s theories was confirmed. This confirms one of the many variables of Rushton’s r/K Selection Theory. Just like I covered how Piantadosi and Kidd corroborated Rushton’s theory of brain size and earlier child birth. Neanderthals had bigger brains than we do today, and knowing what we know about the correlation between IQ, brain size and early childbirth, I would assume that Neanderthals also had earlier childbirths as well,.
Along with these deleterious gene variants from Neanderthals, other variables that contribute to the decline in Eurasian populations also include higher IQ as well, as JP Rushton says, is an extreme way to have control over their environment and individuality. These traits are seen in higher IQ populations in comparison to lower IQ populations. We could also make the inference that since Eurasian children have bigger heads, that multiple childbirths would be taxing on the Eurasian woman’s birth canal while it would be less taxing on the African woman’s.
This study also shows that Neanderthals also had less offspring due to being more intelligent. They had bigger brains than we do today, and since we know that higher IQ is correlated with fewer children conceived, we can say that they were pretty damn smart (they buried their dead 50,000 years ago. There was also a recent discovery of a 176,500-year-old Neanderthal constructions in a French cave). A main cause for the current trend in birthrates in Eurasian populations is due interbreeding with Neanderthals. These events also attributed more to the decline of the Neanderthals.
Deleterious Neanderthal alleles are yet another reason for lower Eurasian birthrates, which shows = that the current trend currently happening in the world with these populations is natural and evolutionarily based. I’ve said a few times that by showing positive things to women on television will increase the white birth rate, with Rushton cites National Socialist Germany as one example. By showing women happy with children, this lead to a massive boom in the German population. To ameliorate the effects of low natural birth rates, these positive things need to be shown on television to women to start to reverse the effects of low natural childbirths.
It’s been a great month for Rushton’s theories, with two of them being corroborated in one month. It’s only a matter of time before the denial of human nature is completely discarded from modern science. As the data piles up on human genetic diversity we will not be able to deny these clearly evident factors any longer.
Black American Men with More African Ancestry Less Likely to Be Obese
1300 words
Black American men are the least likely male ethnic group to be overweight or obese in America (69.2 percent) compared to ‘Hispanic’ men (78.6 percent) and white men (71.4 percent) (Ogden et al, 2014). As a result of being less likely to be obese, black men as a whole suffer from diabetes and other diseases that are correlated with higher body fat. Conversely, for women the rate for white women is 63.2 percent, 77.2 percent for ‘Hispanic’ women and 82.4 percent for black women. Why do black men have lower rates of obesity and chronic health diseases?
Klimentidis et al (2016) set out to find why black men have lower rates of obesity than black women despite having the same socioeconomic and environmental factors. Using 2814 self-identified African Americans from the Atherosclerosis Risk in Communities study, they estimated each individual’s degree of African ancestry using 3,314 genetic markers. They then tested whether sex modifies the association of West African genetic ancestry and body mass index, waist circumference, and waist to hip ratio. Also, they adjusted for income and education as well as examined associations of ancestry with the phenotypes of males and females separately. They recreated their results with the Multi-Ethnic Study of Atherosclerosis (n= 1611 AA).
They discovered that West African ancestry is negatively correlated with obesity as well as central obesity, which is obesity around the midsection, among black men but not black women. Also noted, was that black men with more African ancestry had a lower waist to hip ratio and less central adiposity than black men with less African ancestry. They conclude that their results suggest that a combination of male gender and West African ancestry is correlated with protection against central obesity and suggests that a portion of the difference in obesity (13.2 percent difference) may be due, in part to genetic factors. The study also suggests that there are specific genetic and physiologic differences in African and European Americans.
This study confirms two things. 1) Black women are more likely to be obese than black men as well as the general population. 2) Black men have less of a chance of becoming obese or overweight as well as less of a chance of incurring the risks that come along with being obese or overweight. The degree of African ancestry is the cause in both black men and black women for these differences in the rate of overweight and obese individuals in both populations. One of my theories also got confirmed. Since obesity is partly genetic in African Americans, and black girls have an earlier menarche (period) than white girls due to higher body fat which activates the hormone leptin, which precedes an increase in body fat to prepare for eventual menstruation, I theorize that black girls have earlier menarche than white girls due to r/K Selection Theory. It’s an evolutionary advantage to be able to have children earlier, as the population dies younger.
Evolutionarily speaking, black men needed to be more fit in order to protect the clan from predators. This is also why blacks evolved narrower hips (Rushton, 1995). Higher body fat allows for more protection for a baby in vitro, which is why an increase in leptin precedes an increase in body fat, which then causes black girls to have an earlier puberty.
One of the questions I would like answered is whether it’s the actual degree of African ancestry that is the cause of black men being less likely to be obese or it’s the cause of higher degree of European ancestry. European American men do have a slightly higher risk of being overweight or obese than African American men, so there is some credence to this hypothesis. Three SNPs were found to be correlated with obesity in African American populations as well as European American populations; this could be one cause.
Wagner and Heyward (2000) discovered biological differences exist between blacks and whites. They reviewed the literature on the differences between blacks and whites in fat-free body mass (water, mineral, and protein) fat patterning and body dimensions and proportions. Blacks, in general, have greater bone mineral density and body protein content than do whites, resulting in lower fat-free bone density. They also note racial differences in the differences of subcutaneous body fat, which is the body fat that’s just below the skin, as opposed to visceral body fat which is found in the peritoneal cavity, which can be measured with calipers to give a rough estimate of total body fat adiposity. The conclusion reached in the study was that differences in FFB (fat-free body) was statistically significant between blacks and whites. They also have a greater BMC (bone mineral content) and BMD (bone mineral density) than do whites. They also argue that for a given BMI (body mass index), blacks might have less adiposity because they tend to be more mesomorphic. Researchers push for the development of racial-specific equations to better see differences in FFB.
With the above study noting that there is a substantial difference between blacks and whites in FFB, there may be some truth to a negative effect of European ancestry on blacks in terms of obesity acquisition. However, lower FFB in black men is one reason why black men can’t swim as well as whites.
One of the causes for both racial and gender discrepancies in obesity is genetic in origin. The difference between black men and black women is 13.2 percent whereas for white men and white women the difference is 8.2 percent. There is a clear genetic difference between races that is the cause for this discrepancy. Black men and black women have the same socioeconomics status and live in the same environment, so some of the differences in obesity noticed in this population must be genetic in origin.
Freedman et al (2004) observed that, as expected, black men were more likely to choose heavier figures as an ideal body for women than white men. Also expected was that both groups would choose figures with a low waist to hip ratio, but black men would choose a lower waist to hip ratio as ideal. They also show weight to be a more important cue than waist to hip ratio in mate selection as well as supporting the theory that black men’s preferences may serve as a protective factor against eating and body image pathology in black women.
To give an example of the above study in action, we can look at Mauritania. They force feed their women up to 16,000 kcal a day in an effort to make them obese, as that’s what is seen as attractive in their society. Mauritanian love songs also describe the ideal woman as fat. Obesity is so celebrated in their society that parents beam at the fact that their daughters look obese, as they have a better chance of getting partners.
The higher the degree of West African ancestry in black men, the lower the chance they have for obesity. I do wonder, though, if it’s because they have less European ancestry or because they have more African ancestry. Black men with more African ancestry are less likely to be obese than black men with less African ancestry, so there is a correlation there that I would like to see explored in the future. Differences in fat-free body mass have been noticed between blacks and whites, but this is one of the first studies to my knowledge that shows that genetic differences between black men and black women may be part of the cause for obesity differences in that population. Cultural differences in perception of beauty, of course, come into play in regards to differences between black and white men, however, the cause of black women having higher rates of obesity is due in part to genetic factors, which then leads to black men liking that as their beauty standard.
Chinese IQ
1100 words
The Chinese have one of the highest IQs in the world. Their 100 and 108 in Hong Kong give them an average of 104. Chinese intelligence has been increasing from the 1940s all the way to today. This is the ‘Flynn Effect’ in action. Lui and Lynn (2013) reported that IQ scores are improving for 12-year-old Chinese children. The increases are as follows: 6.19 points for full-scale IQ, 6.55 points for performance IQ, and 1.91 points for verbal IQ. The Jintan Child Study is an ongoing longitudinal study to show the effects of health and cognitive ability.
They used 1656 6th graders (55.5 percent boys and 45.5 percent girls, “consisting of 24.3% of all children in this age range in the Jintan city region born in 1999.”) who either graduated or currently were in the grade with an average age of 12.2 years. The study from 86-87 used only individuals from urban areas, so Liu and Lynn did the same. They conclude that over the 26 years from the original study, between the two data sets that increases of 2.38 points on full-scale IQ, .73 points on verbal, and 2.52 points on performance IQ per decade. They theorize that economic development is a cause of rising IQ scores due to better nutrition. The study concludes a 105.89 IQ for the 12-year-olds in the study.
Liu and Lynn (2015) also observed the same sex differences in the same magnitude in Chinese and American boys and girls. In a study of 788 children aged 12 years old, boys obtained a higher IQ by 3.75 points on average. This exactly mirrors what Rushton and Jackson (2005) say about American men and women who are college aged. They state that males score 3.63 points higher than women. Liu and Lynn state that boys obtained 4.20 points higher in performance IQ, and 2.40 points higher in verbal IQ. This is what we would expect, evolutionarily speaking. The men need to be more intelligent to provide food, whereas women need to have a higher verbal IQ to be able to talk to and take care of children. The fact that the magnitude of sex differences in IQ between men and women has been noticed in the U.S. and China shows that sex differences in the brain do exist.
Better nutrition is a definite cause for the rise in IQ for the Chinese. Richard Lynn says that better nutrition is critical for increased cognitive functioning. This is one reason why Africa’s IQ is so low. Due to more Chinese getting better jobs and making more money, they were getting higher quality foods in order to be adequately nourished. Better nutrition explains most, if not all of the Flynn Effect. It’s what to expect if this phenomenon was not on g (it’s not), it’s on the intelligence that is affected by the environment, hence, bigger increases on that type of intelligence in comparison to the intelligence highly correlated with g.
The Chinese have the largest cranial capacity at 1492 ml. The bigger one’s brain, the more cortical neurons it has which allows for better cognitive processing. Piantadosi and Kidd (2016) corroborated one of Rushton’s theories on brain size and child rearing. Mainly that r/K selection theory explains Piantadosi and Kidd’s theory of earlier births being correlated with higher intelligence due to greater necessity to care for the more vulnerable child in comparison to those with smaller brains. Moreover, since East Asians have more myelin in the brain, this too adds to their higher cognition. Since the correlation between brain size and IQ is .35, a good amount of the variance in IQ can be explained by brain size.
We can also look at Chinese outside of China. Singapore, for instance, has an IQ of 108, the highest in the world. They’re also 74.4 percent Chinese. This is then mirrored in their IQ as well as their economy. Anywhere the Chinese go they are high achievers in both IQ as well as wealth attainment.
There are other measures to show that Chinese have higher IQs. In tests of reaction times, Rushton and Jensen (2005) say that East Asians beat whites while whites beat blacks. Since faster reaction times are associated with a more efficient brain, East Asians have a higher IQ as a result of that. Though, they are weak on verbal IQ, average 99 for Chinese in America and China, they are superior in visio-spatial IQ. This is due to their ancestors evolving in the harsh winters of Siberia which lead to being more K-selected and selecting for bigger brains which lead to children being born earlier and higher intelligence evolving to better care for defenseless children. Bigger brains also evolved due to colder temperatures, which is another cause for earlier childbirth and an even bigger increase in general intelligence to adapt.
IQ in China is higher in urban areas than in rural areas, which is seen in America as well. This is due to those with higher intelligence having the ability to be able to live in the city due to a better ability to attain wealth due to higher IQ. Those in rural areas have lower IQ, some having a lower IQ genetically, while others are depressed by bad nutrition. So with better nutrition, a lot of the rural Chinese would get an IQ boost. Nutrition is critical for brain development in vitro as well as in early childhood leading into young adulthood. This ensures the brain has adequate nutrients for growth and in turn grows to its full potential.
The Chinese are the best example of Rushton’s theory of intelligence and brain size. No matter where they go, if they have adequate nutrition, they have the biggest brains and highest IQs which shows in scholastic achievement as well as wealth attainment. The increase in full-scale IQ for China in the past 30 years is due to better nutrition as well as economic growth. Singapore has one of the world’s best economies and is 74.4 percent Chinese.
This extreme K-selection, though, is causing the Chinese birth rate to drop. This is the curse for high IQ peoples. They have a lower birth rate in comparison to those with lower IQs who have a higher birth rate. The current birth rate in China is 1.66. That is devastatingly low, almost as low as Germany and Japan (both at 1.3) and they have similar IQs as well. It seems that this intelligence increase is coming with a lower birth rate. Higher intelligence is correlated with a lower sex drive so this is another cause for the lower birth rate in East Asian countries as these slight IQ increases continue to occur. The same sex differences as seen in America were also seen in China, giving more evidence to the sexual selection theory of intelligence.
Blacks Are Less Violent Than Whites?
1850 words
I’ve read a lot of crazy things in my life, though this must be the craziest. Someone really believes that “blacks are less violent than whites“. To believe such a claim, you would have to close your eyes to all of the relevant data. From naming outright falsities to obscuring data to fit his narritive, this article will show and refute a distorted reality, one that the Left wishes to show, to one simply looking for the truth to interracial crime.
Don’t be modest, Caucasians. The Holocaust
The Holocaust is really beyond the scope of this blog, but check out the CODOH Library for the truth on this matter.
This is not unique to Europeans. The Rwandan Genocide (which was due to ethnocentrism) and the ethnic cleansing currently occurring in Central Africa aren’t real? Fact of the matter is, is that every ethnicity has participated in ‘ethnic cleansing’, which is really just protecting genetic interests. This is a non-factor as this has gone on before European colonialism.
Colonialism was good for the native inhabitants of Africa. Speaking of the Caribbean, how well did it end for the Haitians after they defeated Napolean?
Oh? You mean how a majority of the slaveholders were Sephardic Jews? Or how there are reports from New Orleans from their 1860 census that showed 3000 freed blacks owned slaves, accounting for 28 percent of the city’s population? In 1860 Louisiana, at least 6 blacks owned more than 65 slaves, with the biggest number of slaves being 165 slaves who worked on a sugar plantation. How about the Jews’ role in American slavery? Moreover, at the height of slavery, a paltry 6 percent of Southern whites owned slaves, when combined with the North it was 1.4 percent. An estimated 3000 blacks had about 20000 slaves in 1860. But tall is only about the whites who did slavery, and not about the Arabs and how they started enslaving Africans FIRST, in 650 AD.
The Alternative Hypothesis just had a post the other day about the non-genocide of American Indians. Basically, their population was anywhere between 1.5 and 2 million people. Population reduction for the Native Americans was only 0.22 percent!! Doesn’t seem like such a ‘genocide’ to me. If so, that’s the slowest genocide I’ve ever heard of.
People segregate naturally. We’re more segregated now than we were 50 years ago! Must be those residual effects from Jim Crow huh?
But somehow in the media it’s the black man who is portrayed as the savage.
It’s just not fair. We white folks are so much better at race-based aggression than our darker complected brothers.
More intelligent than them, that’s why.
Just this Wednesday a white guy walked into a historic African American Church in South Carolina, was accepted as part of the service, stayed for about an hour before shouting a spiteful message and gunning down several parishioners!
Now that’s some hate right there!
One person does something and that means….what exactly? It shows one person is hateful! Look at averages, not singular events.
Whenever anyone brings up race and violence, the first thing people mention is crime.
Because they are linked to each other. Why wouldn’t the two things be mentioned in the same breath?
There is more black-on-black crime than white-on-black crime, they say. And they’re correct!
According to a 2013 FBI Uniform Crime Report, when it comes to murder, 90 percent of black victims were killed by black offenders.
However, what people fail to mention is that according to the very same report, 83 percent of white victims were killed by white offenders, too.
These numbers don’t show black people are more violent than white people. They show that BOTH white and black people would rather kill within their own race.
Yea they show that both would rather kill within race, however you miss something very important here: Interracial crime!
First, we find that during the 2012/2013 period, blacks committed an average of 560,600 violent crimes against whites, whereas whites committed only 99,403 such crimes against blacks. This means blacks were the attackers in 84.9 percent of the violent crimes involving blacks and whites. This figure is consistent with reports from 2008, the last year DOJ released similar statistics. Perhaps not coincidentally, that was the year Mr. Obama was elected president.
In terms of raw numbers, black people and white people actually commit about the same number of murders. But you wouldn’t know that from the media.
Not really. Per capita rates are more important than raw numbers, luckily we have data on that!
As of 2008, young black men kill at a rate of 7 times higher than white men.
Homicide Trends in the United States, 1980-2008 Annual Rates for 2009 and 2010 pg 11
Is that the same number of murders?? I’m looking at the same stat in a completely different way than you are.
The FBI is charged under the Hate Crime Statistics Act with compiling statistics on spite-based legal transgressions. In its most recent report, for 2013, hate crimes based on race are far more numerous than any other kind.
- The FBI is biased towards blacks and ‘hate crimes’. How about all of the countless times we here about blacks attacking whites using racial epithets during the attack? Too many to count. They are, however, not counted as hate crimes by the FBI because it doesn’t fit their narritive.
- I wonder how those numbers would look if actual hate crimes were included in this data (black on white included).
According to the FBI statistics, 54.5 percent of the reported single-bias hate crimes that were racially motivated in 2013 targeted blacks. Only 16.3% target whites.
Want to talk bias? The amount of black on white hate crimes that are NOT categorized as such. This skews the statistics considerably.
Would our economy really have been so robust without the free labor of all those slaves?
Yes. Whites could have done it, but getting blacks to do it was cheaper and more efficient. Using brains to get ahead is what life is all about. Whites build great societies anywhere. Our economy would have been as robust as it is now without America never having a history of slavery.
Heck! Would we even have a country at all if we hadn’t murdered all those indigenous peoples in the first place?
Is a population decline of 0.22 percent per year ‘murdering all those indigenous peoples’?
So let’s put it to rest. When it comes to hate crimes, white folks kill! But don’t feel too bad, black folks. There are things you’re good at, too. Like nonviolent resistance.
This guy is delusional. Just because MLK preached non-violence doesn’t mean that blacks as a whole are non-violent. Look at crime stats from anywhere in the world.
After all this time, black people have very rarely used violence as a means to achieve their ends, to try to secure the rights and freedoms white America guards so jealously.
Is this guy living in the same America as I am?
In just the past year or so, unarmed black folks have been assaulted or killed for holding toy guns
Go ahead and pick out the real one, then do it from a distance when you get a phone call that there is a kid walking around while waving a gun.
He died due to asthma, obesity, and heart disease, that’s why he couldn’t breathe; he was 350 pounds. He also did not comply with the officers’ orders, which is why he needed to be taken down in such a fashion.
listening to music at a gas station
Dunn did say that Davis said he was going to kill him with a shotgun and that’s when he grabbed his gun out of his glove box.
asking for help after a car accident
Freak accidents happen that get blown up? Damn, that proves whites are more violent huh!?!?
Yea, people still believe that Trayvon got killed for ‘wearing a hoody’. We have jurys for a reason. We have trials for a reason. We have laws like Stand Your Ground for a reason. Trayvon was killed because if Zimmerman hadn’t of protected his life, he would be dead. All of these people complaining about the verdict, if you were put into that same situation, would you allow yourself to be killed for fear of being called ‘racist’?
Listen to police when they tell you to do something. Especially during an investigation. This shouldn’t even need to be said.
1) The Baltimore Six are going to get off for it. They caused no harm. 2) He threw himself around in the back of the van causing his own death.
and now just going to church!
Right. They were killed just for going to Church. eyeroll
And the response from the black community has been pretty darn nonviolent. Yeah there’s been some shouting and looting, but very little beating or killing.
‘Pretty darn non-violent. Which is why immediately after, blacks began false flagging Church burnings in an attempt to pin them on whites. So ‘peaceful’, right?
White folks, can you imagine having to undergo such indignity on a daily basis and NOT responding in kind!?
Can’t tell if serious. Just recently, a beta Trump supporter was thrown down on the ground by a ‘young black male’ and did not retaliate.
No wonder a blonde white girl from a Christian fundamentalist home darkened her skin, curled her hair and tried to pass as black! Sometimes – often really – it’s darn embarrassing to be white! Black folks have the moral high ground.
Because Dolezal is a moron. Blacks have the moral high ground? Please show me where this occurs.
Somehow they live in an American society that heaps hatred on their every move and they respond with dignity and perseverance.
There’s no reason at all for this right? Just good old fashioned ‘racism’?
So why are black people so nonviolent?
They aren’t. See the whole of sub-Saharan Africa to see how ‘non-violent’ they are.
Damned if I know! But I wish us white folks would take a lesson from them.
Yes!! The white man has tons to learn from the criminal black man! Much to learn about taking welfare and not working!!
Blacks have 2.5 to 4.9 percent higher testosterone than whites, which is causes more violence and crime. Beaver (2014) states that blacks who have the MAOA-L 2 repeat allele have significantly highier chances of being shot, stabbed or reporting shootings and stabbings than other genotypes. Blacks also have the highest rate of the 3 repeat allele (53 percent compared to 37 percent for whites) and 2 repeat allele (5 percent compared to .1 percent for whites). Moreover, he didn’t speak about how black violent crime is genetic in nature. This is mirrored in the crime rate and how violent blacks really are.
This is the age of the Internet where we have amassed tons of human knowledge which is readily available with a few hits of a few buttons. If people still want to be ignorant spewing falsities, it’s on them. But the truth is out there for those who seek it.
Blacks are not ‘non-violent’. Go to the nearest ghetto and see how ‘non-violent’ blacks are.
Individual and Racial Differences in IQ and Allele Frequencies
1300 words
In the past 100 years since the inception of the IQ test there have been racial differences in test scores. What causes these score differences? Genetics? Environment? Both? Recently it has come out that populations do differ in allele frequencies that affect intelligence. David Piffer’s “forbidden paper on population genetics and IQ” was rejected by the new editor of the journal Intelligence. In the paper, he shows how IQ alleles vary in frequency by population. One reviewer even said it should not be put up for review, which Piffer believes there was a hidden agenda or a closed minded attitude. He even puts reviewers comments and responds to them. He says science should be transparent, which is why he’s showing the researchers’ comments on his paper.
His December, 2015 paper titled: A review of intelligence GWAS hits: Their relationship to country IQ and the issue of spatial autocorrelation shows that there are differing allele frequencies in which IQ between populations that affect IQ which are then correlated highly with average IQ by country (r=.92, factor analysis showed a correlation of .86). There was also a “positive and significant correlation between the 9 SNPs metagene and IQ”(pg. 45). However, Piffer does conclude that since the 9 alleles are present within all populations (Africans, Latin Americans, Europeans, South Asians, and East Asians) that the intelligence polymorphisms don’t appear to be race-specific, but were already present in Homo Sapiens before the migration out of Africa. He then goes on to say that it’s extremely likely that the vast majority of alleles were subject do differential selection pressure which lead increases in cognitive abilities at different rates rates in different geographical areas (pg. 49). It’s of course known that differing populations faced differing selection pressures which then lead to genotypic changes which then affected the phenotype. It’s not surprising that genes that correlate strongly with intelligence have differing frequencies in different geographical populations; it’s to be expected with what we know about evolution and natural selection. Below is the scatter plot showing the relationship between polygenic score GWAS (Genome Wide Association Studies) hits and IQ:
The fact that these differences exist should not come as a shock to those who want to seek the truth, but as seen with how David Piffer didn’t even get consideration for a revision, this shows the bias in science to studies such as this that show racial differences in intelligence exist.
Piffer’s data also corroborates Lynn and Meisenberg’s (2010) finding of a correlation of .907 with measured and estimated IQ. This shows that the differing allele frequencies affect IQ, which then affect a countries GDP, GNP, and over all quality of life.
With a sample with a huge n (over 100,000 subjects) cognitive abilities tests were performed on verbal-numerical reasoning, memory and reaction time (a huge correlate for IQ itself, see Rushton and Jensen, 2005). Davies et al (2016) discovered that there were significant genome-wide SNP based associations in 20 genomic regions, with significant gene-based regions on 46 loci!! Once we find definitive proof that intelligence differences vary between individuals, as well as the loci and genomic regions responsible, we can then move on to difference in allele frequency in depth (which Piffer 2015 was one of the starts to this project).
Moreover, genes that influence intelligence determine how well axons are encased in myelin, which is the fatty insulation that coats our axons, allowing for fast signaling to the brain. Thicker myelin also means faster nerve impulses. The researchers used HARDI to measure water diffusion in the brain. If the water diffuses rapidly in one direction, that shows the brain has very fast connections. Whereas a more broad diffusion would indicate slower signaling, thus lower intelligence. It basically gives us a picture of an individuals mental speed. Thinking of reaction time tests where Asians beat whites who beat blacks, this could possibly show how differing process times between populations manifest itself in reaction time. Since myelin is correlated with fast connections, we can make the inference that Asians have more than whites who have more than blacks, on average. The researchers also say that it’s a long time from now, but we may be able to increase intelligence by manipulating the genes responsible for myelin. This leads me to believe that there must be racial differences in myelin as well, following Rushton’s Rule of Three.
Since the mother’s IQ is the best predictor of the child’s IQ, this should really end the debate on its own. Sure on average, intelligent black mothers would birth intelligent children, but due to regression to the mean, the children would be less intelligent than the mother. JP Rushton also says that regression works in the opposite way. Both blacks and whites who fall below their racial means will have children who regress to the means of 85 and 100 respectively, showing the reality of the genetic mean in IQ between the races.
Why would differing allele frequencies lead to the same cognitive processes in the brain in genetically isolated populations? I’ve shown that brain circuits vary by IQ genes, and populations do differ in this aspect, like all other differing genotypic/phenotypic traits.
East Asians have bigger brains, as shown by MRI studies. Rushton and Rushton (2001) showed that the three races differ in IQ, brain size, and 37 different musculoskeletal traits. We know that West Africans and West African-descended people have genes for fast twitch muscle fibers (Type II) (Nielson and Christenson, 2001). Europeans and East Asians have slow twitch muscle fibers (Type I) for strength and endurance. (East Africans have this as well, which allows for ability to run for distance, which fast twitch fibers do not allow for. The same is true for slow twitch fibers and sprinting events.) Bengt Saltin showed that European distance runners have up to 90 percent slow twitch fibers (see Entine, 2000)! So are genetic IQ differentials really that hard to believe? With all of these differing variables in regards to intelligence that all point to a strong genetic cause for individual differences in other genes that lead to stark phenotypic differences between the races, is it really not plausible that populations differ in intelligence, which is largely inherited?
Is it really plausible that differing populations would be the same cognitvely? That they would have the same capacity for intelligence? Even when evolution occurred in differing climates? The races/ethnicities differ on so many different variables with differing genes being responsible for it. Would IQ genes really be out of the question? Evolution didn’t stop from the neck up. Different populations faced different selection pressures, so different human traits then evolved for better adaption in that environment. Different traits clearly developed in genetically isolated populations that had no gene flow with each other for tens of thousands of years. These differing evolutionary environments for the races put different pressures on them, selecting some for high IQ alleles and others for low IQ alleles.
We are coming to a time where intelligence differences between populations will become an irrefutable fact. With better technology to see how differing genes or sets of genes affect our mind as well as physiology, we will see that most all human differences will come down to differing allele frequencies along with differing gene expression. Following Rushton’s simple rule based on over 60 variables, East Asians will have the most high IQ alleles followed by Europeans and then blacks. The whole battery of different cognitive abilities tests that have been conducted over the past 100 years show us that there are differences, yet we haven’t been able to fully explain it by GWAS and other similar techniques. Charles Murray says within the next 5 to 10 years we will have definitive proof that IQ genes exist. After that, it’s only a matter of time before it comes out that racial differences in IQ are due to differing allele frequency as well as gene expression.
Islam, Suicide Bombings, IQ and Consanguinity
1650 words
A lot of people seem to confuse causes between ‘Islam’ and behavior that’s just ‘low IQ’. Whenever these attacks like shootings, sexual assaults and rapes happen, that’s due to their low IQ; not religion. I wrote about this in IQ, Inbreeding and Clannishness. All of the behavior you see is due to low IQ. 1) being in an area with a hot climate and 2) cousin marriage has been going on there ever since Jews from the Levant introduced it to them around 200 BC. To quote myself:
Those innate behaviors which result in the favoring in all areas of life, themselves and their family, is a result of genetic similarity because of the closely related genes they share (the father’s brother’s daughter type is the most common in the Muslim world). Also, first and second cousin marriages are more common, which also result in increased altruism for their own family because of the close genetic similarity, but also those in their own group, which is mediated by the brain hormone oxytocin.
In a paper on the mean IQ of Muslims and non-Muslim countries, Donald Templer states that the Muslim world, which used to be have great intellectual achievements from the 7th to 12th centuries, has seen an underrepresentation in highly creative contributions in science journals. This is because of the inbreeding effect (2.5 to 10 point drop in IQ) of close cousin marriage. He ends up saying that genetic factors are more important than social/cultural/religious values (back to the inbreeding, causing defects and lowering IQ) in regards to IQ.
I also put a map of individualism and collectivism in Europe here. You can see that the collectivist countries are fighting back more. The countries/regions where it’s more red roughly matches up to the situation. You can see how in Central, Northern and Northwestern Europe they’re more individualistic, as well as more atheist, than those collectivist countries. So that leads to what we see with this ‘welcome refugees’ signs, as well as, I would assume, more oxytocin in the brain for Europeans, which leads to more altruism towards other peoples. Of course, 1000 years ago, the high altruism was fine due to being a mostly homogeneous society. But when others move in who are not from the area, and who do not have the same biology as you due to certain selection pressures, that’s when the ‘clash of cultures’ commences. Which it’s not really a clash of culture, more like a clash of biology, because 2 groups who shouldn’t live together are being forced to live together.
This also brings me to people who confuse the causality between Islam and blacks. As I said, it’s a low IQ religion (which I have provided enough evidence for my case). So blacks who become Muslim do so because of low IQ. Anything after that doesn’t mean that being a Muslim had them do it. Lets say that Islam never popped up and the same peoples were still there, continuing such close inbreeding, would that be Islam doing it? No. It’s their biology. **
Using environmental factors (Islam, culture) is what leftists do. In my post on behavior not equaling genes plus environment, I showed how people create their own environments based on their own genetics. The environment we put ourselves in is based on our genetics. We can clearly see that Islam is bringing their culture (genetics) to Europe and are incompatible with Europe as well as all Western societies around the world. Due to this, we can see that wherever any population goes, it will be the same from the original place they emigrated from if migration in large enough numbers occurs. A country is only as good as its majority population.
In Non-Western People are Abnormal to Our Societies, I showed how due to differing cultures (genetics), these third-world immigrants coming into our countries cannot readily assimilate due to differing average IQs and other hormones that lead to crime differentials with the native population. Though Arabs are Caucasian, evolving closer to the equator lead to higher levels of testosterone as more exposure to the sun increases vitamin D levels, which is not a vitamin but actually a steroid hormone. These differences in testosterone then lead to more sex attacks with high testosterone combined with low IQ. Lower IQ people are less likely to be virgins than higher IQ people. This shows that higher IQ people have less testosterone and can also hold back urges more than lower IQ people. This then translates over to an increase of sexual assaults by ‘migrants’ to European women. These ‘abnormalities’, though, would be abnormal anywhere. Putting differing cultures (genetics) in a place with a completely different culture will lead to strife due to genetic distance between the two populations.
I wrote in Evolutionary Reasons for Suicide Bombings that Muslims who suicide bomb do so to increase inclusive fitness. The increase in inclusive fitness comes about due to the suicide bomber having no prospects as well as no kids, so he/she is just taking up resources. By committing suicide, they are freeing resources for others who have a better chance to spread their genes. Many suicide bombers come from middle-class backgrounds, which further proves the case for genetic interests being the cause for this. The majority of Al-Qaeda members come from educated, middle-class backgrounds. Even for Palestinian suicide bombers, none of them were poor, uneducated, simple minded nor depressed.
The average IQ for a criminal is 85 adult offenders, 92 for juvenile offenders. What’s the average IQ in the Middle East? 81, around 1.3 SD lower than average, and 4 points lower for chronic adult offenders in America. The lower IQ comes from being more inbred, which then manifests itself in the crime rate. The strife in the middle east can also be traced back to IQ and consanguinity rates in those populations. How inbred a population is predicts IQ as well as how much strife occurs in those populations.
Germany has said they will begin IQ testing their ‘migrants’. If it works well (I highly doubt it will, and if it is, it won’t be implemented well) this could curb some attacks that happen. Since IQ differences between populations are one of the biggest causes for crime differentials (lower IQ is also correlated with higher testosterone) between them, screening for and only allowing high IQ ‘migrants’ in would curb some violent crime and sex attacks if implemented on a wide enough scale. IQ differences between populations are one of the biggest reasons for differences between any population you can think of.
For a comparison, we can use Christian Arabs. Christian Arabs are less inbred than Muslim Arabs, which shows in the amount of terror attacks committed by Christian Arabs, which I can’t find any data for. If anyone has found any, leave a comment. hbdchick then says this about consanguinity between Christian Arabs and Muslim Arabs:
so, the rate of cousin-marriage amongst lebanese christians was 16.5% while the rate for muslims approached double that at 29.6%.
christians married cousins more distant than first cousins at a slightly higher rate than they did first cousins: 8.6% (>1C) versus 7.9% (1C). muslims, on the other hand, favored first cousin marriage: 17.3% (1C) versus 12.3% (>1C). this is a similar pattern found elsewhere in the middle east/arab world. in egypt, for instance, copts tend to marry second cousins while muslims tend to marry first cousins (no, i can’t find the reference!).
there was also more fbd marriage amongst muslims (6.4%) versus christians (3%).
This is directly mirrored in how often we hear about Christian Arab attacks and crime (I haven’t heard of this), showing that consanguinity rates can predict crime rates. Due to this extreme inbreeding, they are more genetically similar, which leads to higher amounts of altruism for their own group, in turn leading to derogation of the out-group. Europeans are, on average, less inbred than Muslims. This is why it’s said that Muslims are incompatible with our societies. They are more clannish and altruistic for their own. Like JP Rushton said, groups will proliferate ideas that are good for their genetic interests.
Even more evidence can be shown with Chechen inbreeding. I can’t find any data on Chechen IQ, so lets use the closest country to Chechnya, which is Georgia with an average of 94. Since inbreeding can depress IQ 2.5 to 10 points, Chechnya’s average IQ should be somewhere around the mid-80s. This shows similarity with the consanguinity rate. hbdchick then concludes:
it’s no wonder, then, that they still engage in blood feuds (just like the albanians). you’d half expect them to build tower houses for protection during clan disputes like the albanians or the maniots.
oh, wait.
Muslim (Arab) populations are incompatible to Western societies due to how inbred they are. Their own societies are built on their genetics, which they then bring to the West and attempt to bring what they’re running from to their new host country.
In conclusion, whenever people say “it’s Islam doing it”, it’s low IQ behavior. Those with lower IQ are more likely to be drawn to Islam. Islam developed after 1300 years after the start of Arab inbreeding. We can draw, from IQ from American criminals, that 85 is the sweet spot for criminality, and since criminality is correlated with low IQ more so than any other variable you can think of. A good example of this is a low IQ person coaxed into committing a crime. It’s an obvious biological difference, the sociopolitical garbage is just that, garbage. The biology drives the politics. Consanguinity rates are one of the biggest factors. You should be concerned with the biology aspect.
Note: When I say “Muslim” I mean Arab. I am also not attempting to “apologize” for terror attacks. I’m simply looking at it through the lens of evolutionary psychology. Most people who read this blog know why Africans act the way they act, and African “migrants” are no different.
How to Use Current Knowledge to Effectively Treat the Symptoms of PWS Patients
2550 words
Abstract
Researchers have tried to manage those with Prader-Willi’s Syndrome for multiple decades. Though they have greatly curbed some of the implications of the disease, there are still numerous ways in which we can better use our knowledge of how the disease manifests in order to better help those suffering from PWS. Looking at research into how the extra chromosome 15 is linked to low IQ; IQ and its relationship to obesity; how the ability to delay gratification leads to obesity; growth hormone treatment to better treat low muscle mass and higher body fat; and finally using reinforcement theory to punish a response, where doing so will greatly diminish the probability of that response occurring again in the future; all of these factors can be used in conjunction to better mitigate problems from the disease. By examining all of these variables and thinking of better ways to handle them, we can then think of other, better ways to manage those with PWS. In doing so, we can better increase the quality of life of those suffering from PWS, as well as have less of a strain on healthcare workers who care for them. With new advances in technology with CRISPR Cas9, we can then edit the genomes and chromosomes of those suffering from this disease.
How to Use Current Knowledge to Effectively Treat and Manage the Symptoms of PWS Patients
How can we use the research on chromosomal differences, research on their IQ differences and their lack of ability to delay gratification that, in turn, would help those individuals with the disease? Seventy percent of PWS cases are attributed to the deletion of chromosome 15 (Ledbetter et al, 1981). Maternal uniparental disomy, which involves receiving an extra chromosome 15 from the mother, is yet another cause of PWS (Wang, 2004).
Whittington, Holland and Webb (2009) found that there was variation between families in deletion of chromosome 15. They found that the PWS and sibling IQ correlation was .3, a modest correlation. What was also noticed was that there were subtype differences which manifested itself in the familial differences in IQ. As they expected, the correlation with normal siblings and those with PWS was .5 in those who suffered from PWS due to unilateral disomy. But in the second subset (the chromosomal deletion subset), the correlation was negative at -0.07. Their research shows great promise in the role of chromosome 15 and IQ. They end up concluding that there needs to be an explanation for the small genetic differences between them. How can we use these differences in IQ to help people with PWS and what does this suggest for other symptoms of their disease?
Kanazawa (2014), reviewed the data on the research between obesity and IQ. What he found was that those studies that concluded that obesity causes lowered intelligence only observed cross-sectional studies. Longitudinal studies that looked into the link between obesity and intelligence found that those who had low IQs since childhood then became obese later in life and that obesity does not lead to low IQ. The average IQ for an individual suffering from PWS is 65 (Butler, Lee and Whitman 2006, p. 13), so that is one reason they have a tendency to be obese. He states that those with IQs below 74 gained 5.19 BMI points, whereas those with IQs over above 126 gained 3.73 BMI points in 22 years, which is a statistically significant difference. Also noted, was that those at age 7 who had IQs above 125 had a 13.5 percent chance of being obese at age 51, whereas those with IQs below 74 at age 7 had a 31.9 percent chance of being obese. This data makes it clear: low IQ is correlated with obesity, so we, therefore, need to find sufficient measures to help those with lower IQs who also suffer from PWS to better maintain their good health. Since we can better identify those PWS individuals who have lower IQs based on how they got the disease, we can then show them more attention in an effort to have them manage their gratification better. Moreover, the lack of ability to delay gratification is also correlated with low IQ (Mischel, Ebbeson, and Zeiss, 1972).
Schlam et al. (2013) observed in a follow-up study to the Marshmallow Experiment that found in a longitudinal study of individuals they found forty years later from the original Marshmallow Experiment, due to inability to delay gratification forty years previously, that was one cause of becoming obese forty years later. Due to PWS sufferers having lower average IQs, and, therefore, a lack of ability to delay their gratification, this is direct evidence that those PWS sufferers with low IQs need more stringent measures to be taken on them, which would then be helpful to those individuals who have a hard time delaying their gratification, which is partially caused by the drop in IQ due to the additional chromosome 15. We can see how those with PWS act; they want their gratification now and do not want to wait for it. This is why, when unsupervised, that those with PWS gorge on the food they understand they should not have, but do so, nevertheless, since their low IQ is correlated with lack of ability to delay gratification, which manifests itself in their obesity. We clearly need to find better methods in which to help those with low ability to delay gratification, which would strongly help those suffering from PWS.
Dykens et al (1997) note that those with PWS have hyperphagia, which correlates with their insatiable want for food. They state that the lack of fullness is due to an altered function of the hypothalamus, which is the part of the brain that is in control of feelings of satiety. Certain States gave restrictions to homes that take care of those with PWS and have come under fire because of this, mainly due to human rights violations. We must ask, then, should we limit their access to food if it will prolong their lives? Will doing so inhibit their freedom to do as they choose? PWS sufferers also have coronary heart problems; one could argue that given their free ability to choose what they want to do unfettered will lead to premature death due to obesity-related complications. Does their disease truly not allow them to learn the consequences of their behavior? Do they have the intellect to really understand the consequences of their actions of consuming too much food? There is no established or known way to control those with insatiable eating habits due to hyperphagia. So would the best course of action to take with those with PWS actually be to constantly monitor them and to lock access to easily attainable food? My answer is yes, however, there is a clear fine line in whether restricting access to food and constantly monitoring those with PWS infringes on their human rights, or that doing so actually will help them live better, healthier lives since they would have the constant supervision around them to better control their out of control eating habits. When negative actions occur, one idea that can be shown to them is constant positive reinforcement so that they may be better able to understand that what they are doing is harmful to their bodies. We can then use positive reinforcement when they do reach a healthy weight, so, in turn, they will have a higher chance of keeping a healthy weight. They may reap the benefits of positive reinforcement, and stick more closely to their program, and therefore, stay healthy.
The hormone ghrelin is secreted from the hypothalamus. With an altered hypothalamus, this would cause ghrelin levels to overload; then the individual suffering from PWS would feel the need to insatiably gorge on food due to this chemical imbalance in the brain. Ghrelin increased feeding in rats and ghrelin is the physiological mediator of feeding and probably has a function in growth regulation by stimulating feeding and release of growth hormone (Nakazato et al, 2001). There is a correlation between want of food, ghrelin release and growth hormone production. By attempting to mediate these variables, those who suffer from PWS will be able to better control their eating habits through positive reinforcement and better, more sustainable habits. Since whenever we eat we get a release of ghrelin that makes us hungry, people pretty much set their own eating times by eating multiple times a day. This affects PWS patients the same way. They can’t stop eating, due in part to constantly eating which constantly releases ghrelin in their body.
PWS sufferers have low muscle tone and, conversely, more body fat. Growth hormones may be a valid way of alleviating that problem, which in turn will give them a slightly higher resting metabolic rate so that they may burn slightly more calories, in an effort to stay healthier. Growth hormone therapy is great for those with PWS though they are largely inactive and lead a sedentary lifestyle, the growth hormone will allow them to have less body fat and more muscle mass. As noted earlier in my paper, those suffering from PWS have altered function in their hypothalamus, which is also where growth hormone is secreted. Aycan and Bas (2014) state that treatment with growth hormones should be strongly considered for those with PWS.
PWS sufferers are also quick to anger, which can be correlated with their sub-average IQ. They may, for instance, become irate at the fact that they do not have constant access to food, and may turn to emotional, angry and infantile outbursts in an attempt to receive what they want. This is one way that it’s tricky to treat those with the disease. How do you tell an individual with PWS who wants something “No”? Measures should be taken to show those with the disease what they are doing to their bodies in the simplest way possible as to better get the point across to them. We can help those sufferers of PWS who are quick to anger with by allowing them to discern between right and wrong ways to handle times when they don’t get what they want with positive reinforcement.
Since those who suffer from PWS have behavioral problems, there are better measures we can take to assure that they don’t have their violent outbursts. When positive reinforcement is consistently shown to an individual who has PWS, he will have more success with his program. When they do something wrong, they can then be shown positive reinforcement, and through being shown positive things with reinforcement theory, they can better learn that certain actions they take are dangerous and shouldn’t be done again, as Rushton (1980) states: “If one rewards a response, it will increase the probability of the future occurrence of that response. If one punishes a response, it will decrease the future probability of the occurrence of that response.” (p. 90).
Discussion
In this paper, I have presented causes for PWS as well as effective ways to manage the disease. To look at how IQ affects individuals in regards to obesity and because it is highly correlated with other measures as well, we can then better help those with the disease. By seeing which individuals have the parental disomy version of PWS, we can then monitor them and give them better care because of their lowered IQ and make sure they stay at a healthy weight. One of the best measures to take is to heavily restrict food, i.e., make sure ability to access food at all hours of the day is restricted along with constant supervision. Though, there are rights groups fighting for them saying that their human rights are being infringed on. In allowing them to have free reign over what, how and when they eat, they will gorge themselves to obesity, as well as lead themselves to horrible complications that come along with increased food consumption. When one is caught consuming food he or she shouldn’t be consuming, punishing them and letting them understand that the behavior they took was wrong will lead to better choices and outcomes from those choices, due in part to the main facet of reinforcement theory, that punishing a response will lead to a reduced outcome in that response that was punished happening in the future. Also, with the advent of CRISPR Cas9, we will be able to edit genomes, and therefore, eventually, put an end to PWS. It will enable us to fix the chromosomal deletion and uniparental disomy, which will eradicate this disease.
Conclusion
There are better, more helpful ways in which to help those suffering from PWS. By identifying and attempting to correct these abnormalities, those who suffer from the disease can, therefore, have a better quality of life due in part to the extra measures taken. By understanding that their lower average IQs lead to a lot of the problems associated with the disease, we can better structure methods for them to keep on a healthy track and reinforce positive behavior through reinforcement theory. Since obesity is correlated highly with low IQ, we can, therefore, use this information to better help those who suffer from PWS that have low IQs. Locking up food instead of providing free access, as well as understanding they do not have the ability to delay gratification, would be a big start to find better ways to treat sufferers of PWS. Treating negative actions with positive reinforcement through reinforcement theory will lead to better and increased prosocial behavior. It’s been shown that if you punish a response, then it will decrease the future probability of that response occurring. The advent of CRISPR Cas9 will then allow us to edit the chromosomes of those with this disease in the future. Should we use genome editing on individuals with this disease, as well as several other chromosomal/genetic diseases? I believe we should, in doing so, we will greatly increase the quality of life of those with the disease.
References
Aycan, Z., & Baş, V. N. (2014). Prader-Willi Syndrome and Growth Hormone Deficiency. Journal of Clinical Research in Pediatric Endocrinology Jcrpe, 62-67.
Butler, M. G., Lee, P. D., & Whitman, B. Y. (2006). Management of Prader-Willi syndrome (3rd ed.). New York: Springer-Verlag.
Dykens, E. M., Goff, B. J., Hodapp, R. M., Davis, L., Devanzo P., Moss, F. . . King, B. (1997). Eating Themselves to Death: Have “Personal Rights” Gone Too Far in Treating People With Prader-Willi Syndrome? Mental Retardation, 35(4), 312-314.
Kanazawa, S. (2014). Intelligence and obesity. Current Opinion in Endocrinology & Diabetes and Obesity, 21(5), 339-344.
Ledbetter, D. H., Riccardi, V. M., Airhart, S. D., Strobel, R. J., Keenan, B. S., & Crawford, J. D. (1981). Deletions of Chromosome 15 as a Cause of the Prader–Willi Syndrome. New England Journal of Medicine N Engl J Med, 304(6), 325-329.
Mischel, W., Ebbesen, E. B., & Zeiss, A. R. (1972). Cognitive and attentional mechanisms in delay of gratification. Journal of Personality and Social Psychology, 21(2), 204-218.
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White Men Can’t Jump? That’s OK; Black Men Can’t Swim
1200 words
Everyone sees how athletic blacks are, for instance in football, but why don’t blacks like swimming? Black children drown at a rate almost three times higher than white children. 70 percent of black children can’t swim, compared to 60 percent of ‘Hispanic’ children and 40 percent of white children. A combination of genetic and environmental factors are involved in this.
JP Rushton writes in Race, Evolution and Behavior (pg. 163):
Body structure differences likely account for the differential success of blacks at sporting events. Blacks are disproportionately successful in sports involving running and jumping but not at all successful at sports such as swimming. For example in the 1992 Olympic Games in Barcelona, blacks won every men’s running race. On the other hand, no black swimmer has ever qualified for the U.S. Olympic swim team. The bone density differences mentioned above may be a handicap for swimming.
Rushton noted above this paragraph that there are bone density differences between blacks and whites which have been noted at all ages and skeletal sites, remaining even after controlling for body mass. These differences even appear before birth, prenatally. Divergence in length and width of the bones in black and white fetus is followed by greater weight of black infants in comparison to white infants. These skeletal differences in weight obviously persist into adulthood, which then have implications for blacks with swimming, but give them advantages in other sports.
At the time of the writing of Race, Evolution, and Behavior in 1997, no black had ever qualified for the U.S. Olympic Swimming Team. However, Anthony Ervin became the first black swimmer to qualify for the Olympic swim team in the year 2000. The fact that it took so long for a black swimmer to make the Olympic swim team shows a genetic component.
Also contributing to this phenomenon is the fact that blacks have narrower chest cavities along with heavier skeletons (as mentioned above), this makes it harder for blacks to stay afloat and swim in the water. A smaller chest cavity brings with it less mobility while doing strokes in the water in comparison to one with a wider chest cavity.
Hochberg (2007) notes that fracture risk, particularly hip fractures, in whites is higher than for blacks in both sexes at 10.1 and 4.1 percent for white and black women respectively while the percentage is 4.3 and 3.1 percent for white and black men respectively. Other analyses of people aged 65 to 90 show 16.3 and 5.3 percent in white and black women while the percentage is 5.5 and 2.6 percent for white and black men respectively. Why do white women suffer more fractures? When women hit menopause, the drop in estrogen coincides with bone loss and osteoperosis. Moreover, in 2010, the Food Surveys Research Group published Fluid Milk Consumption in the United States, which shows women drink less milk then men, with seniors drinking less milk than all age groups (pg. 4). We can then infer from this that senior white women drink less milk, which is then another cause for more hip fractures and weaker bones. Though, there is of course a genetic component with blacks having stronger bones. However, stronger bones means heavier bones, which is a disadvantage when it comes to swimming.
Another funnier reason for blacks non-ability for swimming is that since blacks have a higher single mother rate (72 percent) they didn’t take them to the pool and teach their kids how to swim because they “didn’t want to get their hair wet”, as they put so much time in their hair. This may contribute to this slightly, but the physiological and biological differences mentioned above contribute a far greater amount of variance.
Blacks also wouldn’t be good swimmers due to them having more type II muscle fibers than type I, meaning their muscles fire off quicker and therefore tire quicker. This is why blacks are good sprinters, but would suffer in swimming events. Blacks also have a higher fat free bone density than whites, which leads to blacks not being able to float since fat floats.
For the same reasons why blacks wouldn’t be good swimmers, whites can’t jump, on average. Type II muscle fiber doesn’t allow for the explosive power needed to be able to jump as well as blacks. Even though blacks have a heavier skeletons than whites on average, they can still jump due to their muscle fiber typing. This is also why whites are underrepresented in the NBA (which is 74.4 percent black). Testosterone and musculoskeletal differences are the causes for racial differences in sports.
Whites drown more than blacks before 5 years of age, but after 5 years, more blacks drown in comparison to whites. Drowning in natural water settings was significantly higher for blacks than for whites, with blacks drowning more than whites at ages 7-8 through 17-18 years of age. Swimming pool drowning rates was also shown to be elevated for black children This data shows that after 5 years of age, blacks drown at a significantly greater amount than whites or ‘Hispanics’, which is attributed to the data above.
In our ‘post-racial society’, it’s become taboo to study, and even speak of differences so prevalent in our society, seeing as sports is a huge part of many peoples’ lives around the world. Most of the stars everyone speaks of are black. They must, subconsciously atleast, notice that these differences are there seeing the racial differences on the screen (except for baseball). Of course practice has a part in becoming elite in whatever sport is chosen, however, the best also have a higher inclination to want to do better as well as already being genetically gifted. Those who are more genetically different are, obviously, on the right side of the bell curve, thus, are extremely rare. Though, we can see these genetic disparities in racial differences in sports, with swimming being a huge tell.
It’s not just “a racist joke” that blacks can’t swim, there is biological and physical evidence that blacks, on average, have a more difficult time in the water due to difference in fat-free body mass as well as more narrow chest cavities and heavier skeletons than whites. The same reasons why blacks dominate other sports (football, soccer, boxing, basketball) is the same reason why blacks are underrepresented as swimmers: their biology. However, the trope that white men can’t jump is true as well, and also has it’s origin in genetic differences between the races.
The cause for racial disparities in drowning, as well as in swimming competitions is due considerably to genetic factors such as bone density and chest cavity narrowness. Other environmental factors include children not being taught how to swim due to them being more likely to be raised by single mothers. Bone density and heaviness also shows why blacks suffer less fractures than whites, with white women suffering from it the most. That same bone heaviness is also a reason why blacks are not good swimmers. Muscle fiber typing play a difference in racial differences in sports ability, which the races also vary in significantly. For these reasons, “White men can’t jump.” That’s OK, because “Black men can’t swim.”
Transvestic Disorder and Gender Dysphoria Identification and Prevention
2150 words
Abstract
Transvestic Disorder comes about in early childhood and manifests itself in sexually deviant actions. Men suffering from TD who aren’t homosexual, more likely than not, show attraction to themselves dressed in women’s clothing. The signs of TD are noticed at an early age when the individual begins to cross dress. TD is also correlated highly with numerous sexually deviant actions. Fluoxetine and serotonin reuptake blockers may be able to lessen TD since it is an impulsive disorder. With TD being co-morbid with OCD, by treating OCD we can better treat TD itself and give a better quality of life to the patient suffering from the disease. Since autogynephilia and transgenderism are related, measures taken to alleviate TD and autogynephilia could be taken to alleviate symptoms of gender dysphoria, since autogynephilia leads to transgenderism.
Transvestic Disorder and Gender Dysphoria Identification and Prevention
Transvestic disorder is a paraphilic disorder, classified by the American Psychological Association (2013), in which males dress up as women to gain sexual gratification. The individual suffering from TD suffers from compulsions to want to dress as a woman, which causes distress due to the individual not wanting their secret to come out. This then leads to the quality of life of the individual to decrease due to constantly being worried about his secret coming out. TD is diagnosed when a male has sexual feelings and gets sexual arousal from dressing in women’s clothing. It is only diagnosed when these activities are ongoing for at least six months. TD is also similar to another paraphilic disorder called ‘autogynephilia’ (Lawrence, 2011), in which the subject is aroused at the thought of himself being a female, so he, therefore, then begins to dress as a woman to fulfill his sexual desires. Blanchard (1989) proposed that most gender-dysphoric males who do not show sexual arousal to men, instead show sexual arousal to themselves dressed in the opposite sex’s clothing. He concludes that the hypothesis is supported that major types of those men who cross-dress are nonhomosexual, and do so because they become aroused at the thought of dressing as a woman. The DSM V says that autogynephilia is a specifier to transvestic disorder. This is because they are characterized by the same things (American Psychological Association, 2013).
The signs of TD are noticed at very early ages. Most notable are when children begin to cross-dress at or before puberty. This then continues into their adult lives where it begins to be a problem and cause dysfunction due to needing to keep their secret. Dr. Mark Griffiths (2012) states that all though children may engage in transvestic behavior, what differentiates it between an adult suffering from TD is that the child who cross-dresses does so for excitement and pleasure, not for sexual pleasure. Though some researchers say that the disorder is brought on through childhood trauma, i.e., accidental exposure to women’s clothing or exposure to a woman who is undressing. Numerous studies have also concluded that many men who suffer from TD have had to deal with parental separation during childhood.
The American Psychological Association (2013), reports that fewer than 3 percent of males are characterized as having transvestic disorder. TD is most always seen in males, though Moser (2009) noted that in his study using the Autogynephilia Scale for Women (ASW), that out of the 29 respondents that sent back questionnaires, 90 percent would be classified as having autogynephilia. Though, by using a more meticulous definition, only 28 percent were seen to be autogynephilic (Moser, 2009).
Langstrom and Zucker (2005) observed in a sample of 2,450 18 to 60-year-olds in Sweden that transvestic disorder was correlated significantly with being separated from their parents, homosexual relations, higher masturbatory frequency, being easily aroused sexually and pornography use. Also noticed, was a positive attitude in regards to sexual arousal from pain, exposing oneself to a stranger and voyeurism were all positively correlated with TD. Langstrom and Zucker observed how TD is co-morbid with many other paraphilic disorders as well as other deviant behavior. By attempting to treat what TD is correlated with, symptoms of TD can be lessened.
Men suffering from TD and autogynephilia are told that women are the standard of beauty. They then look at themselves in the mirror and see a male and not the standard of beauty they were told of growing up. They then turn to cross-dressing to finally see their “beauty standard” in the mirror but keep it a secret. This strong want to keep their disorder a secret then leads to dysfunction. Men suffering from TD will go to any lengths to hide their secret. This then causes extreme dysfunction in their lives, which leads to a lessened quality of life.
Less than three percent of males suffer from TD in the American population, as such, it is classified as a deviant lifestyle as it deviates from the norm of the population. It causes distress due to them not wanting their secret to be discovered. This, in turn, leads to dysfunction where the individual cannot live their daily lives to the fullest due to their abnormal disorder. It finally leads to danger due to their secret beginning to consume their lives so that they’re not discovered.
There are ways to treat TD. Usmani et al (2012) follow a case study in which a 17-year-old Indian male who had occurring desires to wear his mother’s clothes. He then would masturbate in his mother’s clothes to alleviate himself. This continued on for two years so he could pleasure himself. He was caught by his parents wearing his mother’s clothes and was beaten by them for it. He then said that it is a compulsive behavior and cannot be helped. This case also shows the obsessive compulsive side to TD. They have an urge so strong they cannot help but to do it compulsively to alleviate their sexual desires. He also said that the occurring thoughts then affected his schoolwork as he was so preoccupied with the thought of wearing women’s clothes. All of his brain scans were found to be normal, so what brought on this case in the individual in the case study? He was then diagnosed with TD and prescribed fluoxetine, an antidepressant SSRI. The dose was started at 20 mg and increased by 40 mg once a day for two weeks. In his six-month follow-up, he reported lessened desire to masturbate with women’s clothes (Usmani et al, 2012).
Paraphilias and other related disorders have been thought of as sexual addictions. Though it has been argued that they are not sexual addictions, but are sexual compulsions (Stein et al, S 1992). The researchers reviewed 13 patients who showed signs of TD and were administered serotonin reuptake blockers. The symptoms of those individuals were then divided into paraphilias, non-paraphilic sexual addictions, and sexual addictions. Stein et al discovered that paraphilias had the least improvement with the reuptake blockers whereas sexual compulsions showed the best improvement. They end up concluding that paraphilias and other related disorders are on the impulsive end of the spectrum compared to the compulsive end. These impulsions, then, have those men suffering from TD have the urge to dress in women’s clothes to fulfill their sexual impulsion.
TD is co-morbid with obsessive compulsive disorder (Abdo, Hounie, de Tubino Scanavino, and Miguel, 2001). They used longitudinal case studies in which they assessed two individuals who had OCD as well as TD. They conclude that some cases of TD may be OCD related and not always be caused by gender dysphoria. Since OCD and TD are co-morbid, by treating symptoms of OCD, the want to cross-dress will lessen, which will then lessen the symptoms of TD. Treatments could include SSRI and fluoxetine, as previously stated in the paper. Other treatment for TD should be looked at, such as treatment for OCD due to the co-morbidity between the two. By doing so, feelings of wanting to cross-dress may lessen due to one of the underlying causes (OCD) of TD being treated.
Autogynephlia could also explain transgenderism.Transvestism can be called both a paraphila and a sexual orientation. Lawrence (2004) says that it can explain mid-life MtF transitions, progression from transvestism to transgenderism, the prevalence of other paraphilias among MtF transsexuals and the late development of male intrest in MtF transsexuals. However, when Lawrence says that “Hormone therapy and sex reassignment surgery can be effective treatments in autogynephilic transsexualism”, that is incorrect. The prevalence of suicide attempts among transgenders is 41 percent according to the Williams Institute, UCLA School of Law, in comparison to 4.6 percent for the average population. That’s almost ten times higher than the national average. Clearly, surgery doesn’t do anything to alleviate the feelings of gender dysphoria, and as shown in this paper, therapy and drugs like Prozac can better help to alleviate feelings of gender dysphora in transsexuals due to them being extremely similar to eachother. These two disorders greatly mirror each other. Since Lawrence (2004) observed that there is a progression from transvestism to transgenderism, using similar techniques that work on those with TD may also work on those with gender dysphoria.
Discussion
TD can be helped with the correct therapy as well as right medication. With those, impulsions to wear women’s clothes, as well as impulsions to commit abnormal acts will be greatly lessened and quality of life will be restored to a somewhat normal level. Due to co-morbidity between TD and OCD, treating OCD will, in turn, better help the patient suffering from TD. When more studies are carried out on those suffering from TD, we can see whether or not SSRI drugs and fluoxetine will have the desired effect in alleviation of the symptoms of TD. The individual in the Usmani study reported lessened symptoms and impulsions of cross-dressing, so by identifying which parts of the brain are and were activated during the fluoxetine therapy, we can then better give better care and treatment to those suffering from TD. We can also use some data from TD cases for transgenders, as TD and transgenders have a lot of things in common. With therapy as well as, maybe even fluoxetine (which is just Prozac), and high doses of testosterone/estrogen, this could possibly help to alleviate ‘gender dysphoria’. It could also lower the suicide rate as it’s completely possible that these interventions could fix them mentally.
Conclusion
There is little current literature in treating TD, due to it being a shameful disorder and many men not speaking about what they suffer from. One major way in which to help those with TD is to administer SSRI drugs, in which compulsion to cross-dress, and other attitudes associated with TD lessened. Blanchard (1989) proposed the autogynephilia theory for those transgenders who are not attracted to men. With the obseration by Lawrence (2004) on how autogynephilia and transgenderism are related, this can better help those with transgenderism, as they can get correct help and the right hormones they need, instead of the opposite hormones. SSRI therapy is a good candidate in treating TD, as drastic changes in deviant behavior are seen while the patient is taking the SSRI drug. Seeing as most cases of TD begin in childhood before puberty, by better identifying warning signs of these disorder, we can better treat those children who are at risk of developing these disorders before they become a big problem later in life. As more men come out and say that they suffer from these disorders, more studies can be carried out that corroborate the findings in the studies laid out here. It is extremely promising that these disorders can be treated with common drugs already on the market. In those individuals suffering from TD as well as OCD, by treating the OCD first (which may be an underlying cause) the symptoms of TD may be lessened and the individual may eventually have the ability to lead a life without TD. In using these measures on those with transgenderism, this could possibly alleviate suicide rates and other negative variables associated with these paraphilic disorders and sexual orientations.
References
Abdo, C.H.N., Hounie, A., de Tubino Scanavino, M., & Miguel, E.C. (2001). OCD and transvestism: Is there a relationship?. ACTA Psychiatrica Scandinavica, 103(6
American Psychiatric Association. (2013). Diagnostic and statistical manual of mental disorders (5th ed.). Arlington, VA: American Psychiatric Publishing.
Blanchard, R. (1989). The Concept of Autogynephilia and the Typology of Male Gender Dysphoria. The Journal of Nervous and Mental Disease, 177(10), 616-623. doi:10.1097/00005053-198910000-00004
Griffiths, M. (2012, February 28) Dressed to thrill? A brief overview of transvestic fetishism. Retrieved from https://drmarkgriffiths.wordpress.com/2012/02/28/dressed-to-thrill-a-brief-overview-of-transvestic-fetishism/
Lawrence AA . Autogynephilia: An Underappreciated Paraphilia . In: Balon R, Hrsg . Sexual Dysfunction: Beyond the Brain-Body Connection. Adv Psychosom Med 2011 ; 135 – 148
Långström, N., & Zucker, K. J. (2005). Transvestic fetishism in the general population: Prevalence and correlates. Journal of Sex and Marital Therapy, 31, 87–95
Moser, C. (2009). Autogynephilia in Women. Journal of Homosexuality, 56(5), 539-547. doi:10.1080/00918360903005212
Stein DJ, Hollander E, Anthony DT, Schneier FR, Fallon BA, Liebowitz MR, Klein DF: Serotonergic medications for sexual obsessions, sexual addictions, and paraphilias. J Clin Psychiatry 1992; 53:267–271
Usmani et al, (2012) Treatment of Transvestic Fetishism With Fluoxetine: A Case Report. Iran J Psychiatry Behav Sci. 2012 Autumn-Winter; 6(2): 100–101
NotPoliticallyCorrect 
