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The Bajau spleen size and better-living-through-evolution story is also a reminder that since the dawn of the theory of evolution, humans have been incredibly creative in coming up with evolutionary and hence genetic narratives and explanations for just about every human trait that can be measured. This effort recycles from time to time and is again reaching a peak in an era when it is possible to quickly and cheaply “genotype” human populations. (Joyner, Boros, and Fink, 2018: 524-525)
Yesterday on Twitter Matt Yglesias—Vox blogger—tweeted out a 2018 paper in which the authors argue that ‘natural selection’ explained the bigger spleens of the Bajau and, consequently, how they could hold their breaths for such a long time (Ilardo et al, 2018). The main claim of the paper is that ‘natural selection’ ‘selected-for’ larger spleens which then increases how much oxygen can be stored. The point of the study was to find out if their exceptional diving abilities had a ‘genetic basis.’ Thankfully, though, a group of physiologists looked into the claims of Ilardo et al (2018) and found their main claims wanting (Joyner, Boros, and Fink, 2018). That’s how easy it is for selectionists: Notice a trait; then work backward and attempt to formulate the best story you can (see Smith, 2016).
These diving capabilities are similar to high-altitude hypoxia—indeed the authors assume that there was an adaptation for breath-hold diving. They compared some Bajau people (n=59) and close neighbors who did not—the Saluan (n=34)—really interact with the water environment. So, using a portable ultrasound machine, they photographed the spleens of the two groups and noted a “clear visual difference” between Bajau spleens and Saluan spleens. The Bajau had spleens about 40-50 cc bigger than the Saluan.
These results suggest a physiological difference between the Bajau and the Saluan that is not solely attributable to a plastic response of the spleen to diving activity. While other unknown environmental factors could potentially explain the observed difference between the groups, genetic factors remain a possibility.
So big spleens mean big breath-diving ability. And big spleens were an object of selection. So breath-diving was therefore selected for—so the just-so story goes. But, “a slightly larger spleen is almost certainly not the most important [part of diving] from a physiological perspective” (Joyner, Boros, and Fink, 2018: 524). They then found ‘genes for’ big spleens and they then formulated their just-so story. Accompanying evidence was the fact that deep-diving animals have bigger spleens, so since the Bajau have bigger spleens, this then would mean that the bigger spleens then allows for better breath-diving. Joyner, Boros, and Fink (2018: 525) write:
The general idea is that, like blood doping in cyclists, bigger spleens that contract would give the Bajau divers a boost of oxygen-carrying red cells that would make them more successful at underwater-based hunting and gathering. Thus, spleen size was selected for via evolution over the last 1,000 years or so. A few simple back-of-the-envelope calculations about oxygen stores are instructive. A 40 cc bigger spleen that was all red cells and contracted completely with diving might give the Bajau 20 milliliters of extra oxygen. That is about 1% or less of the oxygen stored in the body and lungs of healthy humans.
Joyner and colleagues then go on to note other ethnies who perform similar breath-dives, while also noting that the Bajau routinely perform dives at 5-10 meters for 30 seconds, which while impressive, is something that fit people can do if they train their lung capacity.
Of note, in the famous Japanese and Korean breath-hold divers who have been studied for decades, repeated descents of this depth and duration don’t cause oxygen levels in the blood to fall much (Schagatay, Lodin-Sundstrom, and Abrahamsson 2011; Stanek et al. 1993). Second, repeated dives of this duration with brief periods of rest only cause whole-body oxygen consumption to rise to the level seen during a brisk walk or slow jog (Craig and Medd 1968). This is hardly the sort of maximum efforts made by deep-diving seals, competitive breath-hold divers, and blood-doping cyclists. The extra oxygen from a slightly larger spleen could easily be generated by a slightly larger breath prior to starting the dive.
It is incredibly easy to think up just-so stories for anything you notice (e.g., my just-so story on Mesoamerican human sacrifices). The Bajau learn to do breath-dive like that—it is an acquired ability. ALL human traits are experience-dependent and plastic, but obviously not to the same degrees.
The spleen is a blood filter. Being a blood filter, if it can filter MORE blood by being bigger, then it will give the individual whose spleen it is an advantage at certain tasks. It sits upper-left part of the abdomen and is protected by the rib cage while also being a place for blood cell storage (Pernar and Tavakkoli, 2019). The spleen acts as a reservoir of oxygen storing “‘thick blood’ rich in cells” so that when the diving animal the oxygen levels are stressed, the body has the reservoir of extra oxygen (Milton, 2004). Esperson et al (2002) also note how the spleen acts as a reservoir of red blood cells, while the contraction of the spleen causes extra hemoglobin production after exposure to diving events.
When a diver submerges their body in the water, two things happen: bradycardia—slower heart rate and vasoconstriction—the narrowing of arteries. The increase in water pressure further causes blood shift—an extension of vasoconstriction which allows us to dive deeply and the spleen effect—meaning that the spleen of divers shrinks which then releases the stored RBCs into the body, allowing for prolonged diving. So, if we look at this in regard to the Bajau, their large spleen shrinks and since their spleen is large due to the number of RBCs it has, it would then follow that as they deep-dive the spleen would shrink (known as splenic contraction) causing the release of the RBCs into the body, allowing for the deep-diving phenotype. Thus, without the blood shift and spleen effect, divers would not be able to dive to such deep depths. (See Gooden, 1994.) The mammalian dive response has, also, been hypothesized to be a reflex to preserve life (Panneton, 2013).
Bajau and, for example, Tibetan environments are similar in that the Bajau spend time in low-oxygen environments (the water) and so do the Tibetans (the mountains). But the Tibetans are constantly exposed to this effect; the Bajau need to be in the water for this effect to occur.
Lastly, the “HBDers” in the comments on Yglesias’ tweet said the same ol’ predictable things. Such as “human spleen growth is complex and due to what the stereotypes of a group’s spleen size are”; “of course it’s true!”; “variation in spleen size within groups is greater than variation in spleen size within groups”; “spleens are a social construct”; “evolution only happened in humans below the neck”; etc. These are the same ol’ sayings that come out from “HBDers” whenever a finding like this comes out. And, you can tell that they are ignorant to the anatomy/physiology of this stuff. They just jump on anything that they can say “HBD is real!!” as if anyone denies that humans are biologically diverse (like when the Rushton retraction came ‘it doesn’t mean that HBD isn’t true!). They move and weave different definitions of ‘HBD’ to fit their needs.
Such storytelling can be done for any trait, as noted by Smith (2016). One can think up selectionist stories and competing ideas for the evolution of traits, but without a way of ajudicating between two stories. One of the best definitions of just-so stories I am aware of is from Not in Our Genes (Lewontin, Rose, and Kamin, 1984: 258-262) cited in Smith (2016). Just-so stories:
predicate a genetically determined contrast in the past…unstated assumption that genes may arise with any arbitrarily complicated action needed by the theory…insulated from any possibility of being contradicted by fact…. If one is allowed to invent genes with arbitrary complicated effects on phenotype and then to invent adaptive stories about the unrecoverable past of human history, all phenomena, real and imaginary, can be explained.
So what happens when a physiologist looks into “HBD” claims? They are found very, very wanting.
I’ve been writing on this blog since June 15th, 2015. Back then, I held staunch hereditarian beliefs. Rushton’s views on testosterone were enticing to me, especially his theoretical article with Templer (Rushton and Templer, 2012). When I first read it I thought “Oh, justification for my prior beliefs about blacks and crime.” Then, when I started studying anatomy and physiology, I reread the paper and thought “Wow who gave the OK to publish this mess?”
So in February of 2018 I published a critique of the article, Do pigmentation and the melanocortin system modulate aggression and sexuality in humans as they do in other animals? A Response to Rushton and Templer (2012). I critiqued many of the claims in their article; its biggest weakness completely handwaving the warnings of Ducrest et al (2008) that human populations “are therefore not expected to consistently exhibit the associations between melaninbased coloration and the physiological and behavioural traits reported in our study.” Rushton and Templer dodged this by saying that we should compare African and European crime rates and that we do, indeed, see that Africans commit more crimes than whites and Africans have darker skin than whites so this is sort of “preliminary evidence” for the relationship in human races.
However, unfortunately for Rushton, Cernovsky and Litmann (2019) reanlyzed the INTERPOL crime data:
When all data from the same two Interpol Yearbooks are re-calculated, most of the statistically significant trends in the data are in the direction opposite to Rushton’s beliefs: Negroids had lower crime rates than Caucasoids with respect to sexual offenses, rapes, theft, and violent theft or robbery, with most correlation coefficients exceeding .60. While we do not place much credence in such Interpol statistics as they only reproduce information provided by government officials of different countries, our re-analysis indicated that Rushton excluded data that would discredit his theory.
So, if we accept the INTERPOL data—as Rushton did—then we would be justified in saying that the proposed relationships and causal pathways do not hold.
Then at the end of October the Twitter account @evopsychgoogle wrote a long thread with a strong, in-depth critique of the arguments and sources in Rushton and Templer (2012). He then published his critique as a letter to the editor of Personality and Individual Differences (PaID). In the letter, he exposes the shoddy logic of Rushton and Templer, while showing their misreadings of sources and misunderstanding of pleiotropy. Their main point in the paper is that LHT may explain why dark-skinned individuals are more violent, mature faster, etc while they also argue that the melanocortin system is a physiological coordinator of LH traits and skin color.
Testosterone production is a simple bodily process—to quote myself from November, 2017:
There are five simple steps to testosterone production: 1) DNA codes for mRNA; 2) mRNA codes for the synthesis of an enzyme in the cytoplasm; 3) luteinizing hormone stimulates the production of another messenger in the cell when testosterone is needed; 4) this second messenger activates the enzyme; 5) the enzyme then converts cholesterol to testosterone (Leydig cells produce testosterone in the presence of luteinizing hormone) (Saladin, 2010: 137). Testosterone is a steroid and so there are no ‘genes for’ testosterone.
Cells in the testes need to take cholesterol and then convert it into testosterone with the luteinizing hormone. According to Rushton and Templer, the result of this bodily process is part of the reason why blacks are more violent than whites—they have higher levels of testosterone and testosterone causes aggression/violence. Surely, Rushton and Templer were aware of Rohrmann et al (2007) who showed no difference in testosterone between blacks and whites. Surely Rushton and Templer were aware of all of the research that I cited in what I would call my ‘master article’ on race, testosterone, prostate cancer, and aggression. Rushton and Templer, quite obviously on a review of the literature, are misleading their readers.
Thankfully this paper has only been cited 15 times in the past 8 years since it was published. Evolution, presumably, accounts for the differences in death, AIDS rate, testosterone, lower life expectancy, etc. One recent citation can be found in the textbook Race and Crime (Gabiddon and Greene, 2018, 5th edition) in a discussion about Rushton’s r/K selection theory and causes for Rushton’s proposed relationship. They write (pg 122-123):
As with all theories, there have been several criticisms of the r/K selection theory. First, Rushton generally underemphasizes sociological factors. Most of his cross-national comparisons point strictly to numbers, without taking into account variables such as socioeconomic status, and other important sociological variables (Whihte, 2018). Second, in the 21st century, there are few “pure” races, especially in the United States, as noted in Chapter 1. White sexual aggression against Black females during the slave era produces countless mixed-race offspring. Therefore, the rigorous adherence to the Black-White-Asian split is problematic. Finally, if Rushton;s theory were true, what would explain White aggression as early colonizers and their current involvement in wars and violence across the globe? In contrast to Rushton’s theory, Bradley (1978) argued that, as a result of migration to colder regions, since the beginning of humanity, Whites have been the global aggressors.
So, testosterone does not cause aggression and it does not cause crime; the melanocortin system does not work in such a way that works for Rushton and Templer’s theory (Cone, 2006).
Hilliard (2012: 73) stated that “No evidence shows that racism motivated Rushton” while Dutton (2018) stated the same, interviewing Rushton’s ex-wife (whom he had an illegitimate black son with). But, knowing what we know about testosterone and Rushton’s views on blacks and whites, knowing about his (false) views on testosterone and race (while citing old, flawed studies like Ross et al) we can call into question the claim that Rushton had no racial animus. @evopsychgoogle also brought up the fact that COIs and funding sources were not brought up (this being Rushton’s final paper before his death and the fact that he was the head of the Pioneer Fund at the time means we don’t have to ask where the funding came from). Rushton and Templer should have never been published in the first place.
Lastly, Clark et al (2020) argued that ‘intelligence’ as ‘measured‘ by ‘IQ tests’ modulates the relationship between religiosity and crime at a country-wide level. They, of course, used Lynn’s global ‘IQ’ data. Though, there were many critiques of the paper and the data used in it (see here, here, and here) and Clark et al decided to finally retract their paper due to the problems of Lynn’s dataset. So, if they retracted due to the problems with Lynn’s dataset, does that mean that other papers that relied on Lynn’s shoddy work (e.g., Richardson, 2004; Morse, 2008) should be retracted too? Ebbeson (2020) notes the flawed ‘IQ estimates’ used in Clark (2020), noting the small, unrepresentative child samples used, along with showing that their dataset leads to “notions” which “are incompatible with psychological science” so “all conclusions drawn from these data are invalid.”
Clark et al “lost confidence in their findings” since “the homicide data have limitations that call [their] findings into question” while “The IQ data … have much more serious issues.” “Serious” is an understatement. To quote Ebbeson (2020)
For example, in the ‘NIQ_QNWSAS’ dataset, several African, South Asian and Central American countries have an average IQ below 50, such as Nepal (national IQ: 43.0), Sierra Leone (national IQ: 45.1), Guatemala (national IQ: 47.7) and Gambia (national IQ: 49.8) These estimates would seem to suggest that a majority of the population in these countries are moderately, severely, or profoundly cognitively impaired. (cf. Table 1). This notion is incompatible with psychological science and there is no doubt that these estimates are wrong.
All in all, this is not a good week for “HBD”—Rushton is being exposed for the know-nothing about physiology that he is, and Lynn’s ‘national IQ’ numbers are getting the scrutiny they finally deserve. So, I hold, if Clark et al was retracted for those reasons, we need to retract ALL papers that relied on Lynn’s ‘national IQ data.’ People may cry ‘censorship! HBD is dead!’, but ‘HBD’ (what I would term psychological hereditarianism) has been long dead. Rushton and Templer’s paper should have never been published but it’s better late than never that it is finally getting retracted. If you’re saying that Rushton and Templer got retracted for “political reasons” then it’s clear that you did not read either of the two existing critiques of the paper.
Sudden Infant Death Syndrome (SIDS) has a long history—almost as long as human civilization (Raven, 2018). The term was coined in 1969 to bring attention to children who died in the postnatal period (Kinney and Thach, 2012; Duncan and Byard, 2018). About 95 percent of SIDS cases occur within the first 6 months of life, happening around the 4-6 months mark (Fleming, Blair, and Pease, 2015). The syndrome is associated with the sleep period, presumed to have begun with the transition from sleep to waking (Kinney and Thach, 2012) The prone sleeping position, along with smoking, is said to increase the incidence of SIDS (Ramirez, Ramirez, and Anderson, 2018). Due to a campaign in the mid-90s, though (called the back-to-sleep campaign), it has been estimated that SIDS deaths have decreased by 50 percent, saving thousands of infant lives (Kinney and Thach, 2012).
But, those infants who die from SIDS may also have a problem with the part of their brain that controls waking/sleeping:
Infants who die from SIDS may have a problem with the part of the brain that helps control breathing and waking during sleep. If a baby is breathing stale air and not getting enough oxygen, the brain usually triggers the baby to wake up and cry to get more oxygen.
So, if a baby’s brain is not getting enough oxygen, its brain will have it wake up and cry in an attempt to rid itself of “stale oxygen”—this is one other purpose that crying serves—which then gets the baby more oxygen to its brain.
As can be imagined, acute rises in CO2 levels occur when an individual is unable to expel CO2, such as in the setting of an airway obstruction that might occur when an individual is lying prone in a crib or bed perhaps with a pillow and bedclothes covering the nose and mouth. It has been proposed that such a rise in CO2 would activate arousal circuitry in a normal baby to wake the baby up, cause them to cry out, summoning a caregiver who would come to their aid, and ostensibly correct the airway blockage to allow resumption of normal breathing [16,20,31]. It has been proposed, among other possibilities, that there is an impaired CO2-arousal system in SIDS-susceptible babies such that when they rebreathe CO2 as described above, they do not arouse, and thus do not cry out, and the blockage is not corrected [16,32]. They thus become acidotic and hypoxic and ultimately succumb.
So, if a babe’s airway gets blocked, for instance by a pillow or toy, they wake, cry out for attention and their caregiver comes to solve the problem or they change their laying position. But in SIDS cases, this does not occur. Why? Buchanan argues that those who succumb to sudden deaths like SIDS have screwy serotonin receptors—they ensure that blood oxygen and CO2 levels are healthy. But some of these infants may have brains that don’t allow them to detect the CO2 and blood oxygen levels—when the body may be suffocating. SIDS victims are usually found face-down in their cribs. But, there are no biomarkers for SIDS (Haynes, 2018). The SIDS diagnosis is only given after all other causes of death are ruled out—this is why SIDS is so mysterious. Genetic mutations have been posited as a cause (Männikkö et al, 2018), as has a pregnant mother smoking during pregancy, leading to a doubled risk of SIDS (Anderson et al, 2019).
But the best prevention against SIDS is nonprone sleeping—having the baby sleep on its back. The efficacy of this approach since the 1990ss has been noted (Gibson et al, 1992; de Luca and Hinide, 2016) while “Achieving recommended prenatal care and infant vaccinations, as well as reductions in maternal tobacco and substance use, has the potential to further reduce rates of SIDS and should be given as much attention as safe sleep advice in SIDS risk reduction campaigns” (Hauck and Tanabe, 2017: e289). The back-to-sleep program, though, has been associated with a decrease in motor development from the infant sending time in the supine position along with the strong possibility of developing plagiocephaly—which causes a “flat head” due to being placed in similar positions while the infant’s skull is soft and still developing (Miller et al, 2011). It has also been estimated that if it was known that the advice to place infants on their stomachs to sleep led to SIDS, then we “might have prevented over 10 000 infant deaths in the UK and at least 50 000 in Europe, the USA, and Australasia” (Gilbert et al, 2005: 884).
But the history of SIDS in America is a lot more sinister—rather than children dying from ‘natural causes’ (SIDS), in the 1970s, it was hypothesized by one SIDS researcher that SIDS was ‘genetic’ and ‘transmissible’ on the basis of one family who, unfortunately, had experienced this tragedy more than once.
This leads us to the story of Waneta Hoyt, who is the subject of this article.
Hoyt and Steinschneider: Genes vs environment
Horrible tragedies befell a woman from New York named Waneta Hoyt—five of her children had mysteriously died due to SIDS between the years of 1965-1971.
Waneta killed her first child, Eric who was three-months-old. SIDS is a diagnosis that is arrived at through a process of elimination—rule out all other causes at a young age (under 1) and the cause is then SIDS. But, the thing is, when an autopsy is performed on the infant, there is no difference between what would be said to be SIDS deaths and a light smothering.
After Waneta murdered her first child, she was cold and distant but it was not noticed. It was reported that she would never hold her children as a loving mother would, keeping them quite far from her. But it wasn’t until three years later that she, again, murdered. But this time it was two of her children—her two-year-old son and six-week-old daughter. The murders that Waneta were committing were wrongfully diagnosed as being due to SIDS.
This caught the attention of renowned SIDS researcher Alfred Steinschneider who had a clinic in which he specialized in caring for infants who were thought to be high-risk for SIDS. Steinschneider wanted to watch Waneta’s fourth child in his sleeping ward, in an attempt to prevent what he thought was due to SIDS. So, when he heard of Waneta’s story, he reached out to her to monitor her daughter, Molly.
The nurses at Steinschneider’s clinic, though, became suspicious of Waneta when she was at the clinic since she was cold and distant to Molly—she would not show her any affection. Steinschneider’s nurses emphatically told Steinschneider that it was Waneta who was murdering her children. Steinschneider shot back, and sent Molly home anyway. In an interview on the television program Deadly Women called Mothers Who Kill, one of the nurses who watched Molly before she was discharged by Steinschneider said:
And then about a quarter to eleven when we were getting ready to go off duty I said ‘Joyce, what do you think, do you think she’s still alive?’ Of course when I came on duty the next day she was dead.
Forty-eight hours later, on Thursday, June 4, Steinschneider scheduled Molly for her third discharge. By now, the nurses were speaking more openly about their suspicions. “I just know something’s going to happen,” Corrine Dower said to Thelma. “One of these times she’s going to do it.” Corrine was scornful of Steinschneider. “If he had any brains at all he would have seen that she didn’t want the baby,” she would say years later. “You can tell in the grocery store if a person cares about their child. We were just disgusted with Steinschneider.” (book excerpt from How Two Baby Deaths Led to a Misguided SIDS Theory)
Presumably, since this was Waneta’s fourth time experiencing the tragedy of SIDS, Steinschneider did not think that Waneta could be involved—but his nurses knew the truth. It was when Waneta had her fifth child that Steinschneider thought he would make his breakthrough in his research. Steinschneider was so convinced that the baby’s were dying due to SIDS, and he thought that if he could monitor Waneta’s new baby as much as possible, that he may figure out why babies die from SIDS.
Steinschneider believes that SIDS is hereditary—passed on through genes. The fifth child was watched at Steinschneider’s clinic and when Steinschneider discharged him—in an attempt to prove his theory—his nurses protested. Then, shortly after, Waneta called Steinschneider saying that it had happened again—her fifth child had mysteriously died.
After the death of Waneta’s fifth child, Steinschneider published his paper Prolonged Apnea and the Sudden Infant Death Syndrome: Clinical and Laboratory Observations arguing that SIDS was caused largely by hereditary sleep apnea (Steinschneider, 1972). By 1997, Steinschneider’s paper was the most-cited paper in the SIDS literature (Bergman, 1997). It was due to Steinschneider’s research, though, that parents began using sleep monitors to monitor their children’s sleep so they could be alerted in case their child had sleep apnea.
Steinschneider cared more about his research and theory of SIDS and sleep apnea over what was striking him right in the face—Waneta was responsible for the deaths of her five children. Steinschneider’s 1972 paper was cited and used for 22 years, until it was found upon an in-depth look into Steinschneider’s paper that what was clear to Steinschneider’s nurses and not him was true—Waneta was responsible for the deaths of her children. Steinschneider’s paper, in any case, concluded that SIDS is a genetic disorder and it was thusly inherited. And Waneta’s case, it seems, lent credence to his hypothesis. Steinschneider gave Waneta the perfect alibi—her woes were caused by a genetic disease and there was nothing that could have been done to prevent it.
Waneta was convicted in 1995 of five counts of murder and sentenced to 75 years in prison—therefore refuting Steinschneider’s theory. Three years after her sentence, though, Waneta died in prison of cancer. The case of Waneta Hoyt allowed mothers to kill their children in this specific way (a light smothering) for almost a quarter of a century.
Norton saw history repeating itself in the reluctance of many factors to face the fact that some deaths attributed to SIDS were homicides. She agreed with the bulk of SIDS research, which pointed to apnea, or the cessation of breathing, as the final pathway to death. But there were many causes of apnea, not all of them natural. An adult could place a hand or a pillow over an infant’s nose and mouth and stop the child from breathing. The pressure needed to smother an infant often left no telltale signs, Norton explained.
“There is no way for the pathologist at autopsy to distinguish between homicidal smothering and SIDS,” she concluded.
Norton worried that homicides were being passed off as SIDS because many doctors held the erroneous belief that SIDS ran in families. They ignored large-scale studies that had shown no genetic tendency toward SIDS. Flouting conventional wisdom, Norton warned that the sudden, unexplained death of a SIDS victim’s sibling should be treated as a possible homicide.
When Waneta was convicted, letters to the editor were sent about Steinschneider’s paper. The short correction to the paper chronicles, interestingly, a letter to the editor of the journal Pediatrics, who published the Steinschneider paper
“But the paper indicated a more sinister possibility to Dr. John F. Hick of Minnesota. In a letter to the journal, he wrote that the case offered “circumstantial evidence suggesting a critical role for the mother in the death of her children.” (See below.)
But his warning was dismissed, until Mr. Fitzpatrick read the paper 15 years later.
“The medical records described two happy, healthy, perfectly normal kids,” he said. “It convinced me that these children were murdered.”
Hick’s letter to Pediatrics says:
In reporting two siblings who succumbed to “sudden infant death syndrome,” Steinschneider exposes an unparalleled family chronicle of infant death.’ Of five children, four died in early infancy and the other died without explanation at age 28 months. Prolonged apnea is proposed as the common denominator in the deaths, yet the author leaves many questions relevant to the fate of these children unanswered.
In her signed confession, Waneta said that she smothered her five children because their screaming made her “feel useless”, though Waneta later stated that she only said that to stop the police from questioning her. Steinschneider, like another motivated-reasoner J.P. Rushton, ignored data that did not fit his theory of sleep-apnea-induced-SIDS—specifically how Waneta acted around her children while at his clinic and the thoughts of his own nursing staff telling him not to discharge the Hoyt infants.
Waneta recalled her strangling of her children—specifically Julie:
”They just kept crying and crying. . . . I just picked up Julie and I put her into my arm, in between my arm and my neck like this . . . and I just kept squeezing and squeezing and squeezing.”
Steinschneider’s testimony during Waneta’s trial, however, is very interesting. Reported by the New York Times, Steinschneider attempted to defend his patient Waneta against claims that she had murdered her children:
Autopsies were done,” he said, speaking of Molly. “They could not find a known cause of death.”
This, Dr. Steinschneider said, “by definition” is SIDS.
But under intense cross-examination. Dr. Steinschneider conceded that he could not remember — and did not record — crucial details from the medical histories of the two infants, whom he had hospitalized for observation soon after birth. In each case, the parents had reported that the baby was having difficulty breathing and that its older siblings had died mysteriously.
The doctor also acknowledged concluding that Molly and Noah had died of SIDS without knowing how thoroughly the authorities had probed the “death scene” for evidence of other causes, including murder.
It is said—even by the prosecutor on her case—that Waneta suffered from Munchausen by proxy (Firstman and Talan, 1996)—which is the intentional cause of illness, usually on children, in order for the mother to elicit sympathy for others (Gehlawat et al, 2015). In cases like this, mothers who have the Munchausen syndrome will suffocate their children and then rush them to the hospital—they get the satisfaction of inflicting pain and then the satisfaction of getting cared for for the so-called mysterious death of their baby. One study of 51 sleep apnea monitorings found that about 40 percent of the program treated infants who had apnea that seemed to be induced by the parent; this was inferred from the fact that once the infants were admitted to the hospital, the doctors found no signs of apnea (Light and Sheridan, 1990).
One doctor even took it upon himself to place cameras in his practice in order to monitor parents that were suspected of abusing their children. Thirty-nine infants were monitored—thirty-three infants were being abused by their parents, what’s more is that some of the infants in this study who were identified in the video also had a sibling who mysteriously died from SIDS (Southall et al, 1997). What the study shows is that these parents were suffocating their children, causing their breathing problems and that they most likely have gotten away with infanticide before. Another case involved a mother taking her daughter to eleven different hospitals, but none of them found anything wrong with the girl and she ended up dying under suspicious circumstances (Hassler, Zamorski, and Weirich, 2007).
We now know that Steinschneider ignored contrary evidence to his theory of genetically-induced sleep apnea causing SIDS which apparently ran in families, and since he brushed off his nursing staff telling him that Waneta was acting strangely around her two children that he had admitted into his clinic, he could have saved their lives. But Steinschneider’s genetic determinist theory was more important than seeing what was clear as day to his staff and even others who read his 1972 paper—a mother was strangling her own infants.
SIDS has a long history, dating back to biblical times. But, in the modern-era, erroneous theories on the causes of SIDS were pushed while other, more obvious causes were disregarded in favor of a grand genetic theory of SIDS causation. Waneta and Steinschneider both helped each other out: Steinschneider (unknowingly) helped Waneta evade detection for 22 years while Waneta lent credence to the hypothesis that Steinschneider was developing. The fact that, at the time of their first meeting, three of Waneta’s children had died in almost the same fashion pointed to a genetic, inherited cause in Steinschneider’s eyes.
At the time of publication of The Death of Innocents, Steinschneider still continues to defend his now-discredited theory and still lobbies for the use of infant sleep monitors. Of course, since he testified FOR Waneta, despite the mounting evidence against her, he could be seen as an accomplice, however weakly. But this case shows one thing that should be well-known—researchers become attached to their pet hypotheses/theories and will ignore contrary evidence if it is brought to their attention. Firstman and Talman estimate that between 5 and 10 percent of SIDS cases are actually homicides. (But see Milroy and Kepron, 2017.)
Steinschneider created the SIDS disease on the basis of Waneta’s story—and a multi-million dollar industry then appeared due to his paper—it’s all to save infants, buy these sleep apnea monitors. But there were two children that Steinschneider did not—could not—save: He could have saved those babies, if not for his genetic determinist beliefs on SIDS causation. Had Steinshneider looked at the more obvious answer to the problem which was right in front of his face, he may have seen that Waneta suffered from Munchausen by Proxy, and, as evidenced from the references above, those who suffer from the disease act out exactly how Waneta did—by strangling their children with the cause of death being blamed on SIDS.
The Hoyt-Steinschneider case is a warning—don’t jump so quickly to implicate heredity in the ontology of X, especially when other, more obvious, tells are right there in front of you.
The concept of “race” stretches back as long as human civilization. The concept of “racism” also stretches back just as far with it—they seem to be intertwined. There is a consensus, though, the term was constructed during the European Age of Exploration. This claim though, is false. The concept actually goes back at least 5,000 years. By looking at the art and reading the myths of these ancient civilizations, we can see that the social constructivist claim about race—that it is a recent creation—is false. They also described their physical features and also attempted to explain behavioral differences between races based on the limited knowledge they had in their day.
Sarich and Miele (2004) state that the PBS documentary on race—which is largely the main reason why they wrote their book Race: The Reality of Human Difference—claimed that race is a human invention and that since we create it we can then “unmake it.” We can look at art from ancient civilizations and see that they did sort people into groups based on their skin color and other physical characters. Each civilization, of course, thought itself and its racial features to be ‘superior’ to the others they encountered. The ancients used the set of observable features to describe what we now call “races.”
Our first trip on this long journey to understand the history of race is India. The earliest hints of what would become the caste system were written around 5kya. In the Rig Veda, a description of the Arya(n) invasion in the Indus valley where a dark-skinned people lived. The god of the Arya(n)s Indra “is described as “blowing away with supernatural might from earth and from the heavens the black skin which Indra hates” (Gossett, 1997: 3-4). They also called these dark-skinned people “Anasahs” which meant “the noseless people.” They then describe Indra killing all of the dark-skinned people and conquering the Indus for the Arya.
Sarich and Miele (2004: 47-48) note that the peoples that Indra hated were called Dasas—broad-nosed worshippers of the phallus. Even when Alexander the Great’s army reached India and described the Indians in the south of the country as some of the darkest people they have seen, they still made the distinction between Indians and Africans—by their hair type—so this tells us that race is more than ‘skin deep’ to the Greeks. Race, then, was known for thousands of years BEFORE the Age of Exploration.
We can look to ancient China, too, to see instances of racial descriptions and then racism along with it. For instance, a Chinese writer described yellow-haired, blue-eyed people from a distant province, “who greatly resembeled monkeys from whom they are descended” (Gossett, 1997: 4). Another Chinese legend describes differences between themselves and a brabarian tribe. A Chinese emperor stated that he would give his daughter to whomever slayed the chieftan he was having problems with. Then, the palace dog comes back with the head of the chieftan. The emperor did not go back on his word; he gave the dog his daughter and the resulting children were “fond of living in high altitudes and averse to plains” (Gossett, 1997: 4).
Like other civilizations, the ancient Han Chinese regarded other groups they came into contact with as barbarians. They were especially taken aback by the odd appearance of one group, the Yuehzi, because of their hairy, white, ruddy skin and their prominent noses, which the Chinese likened to those of monkeys.
The Han Chinese applied the term “Hu” to barbarians like the Yuehzi who had “deep eye sockets, prominent noses, and beards.” But they did not apply it to the Qiang, another barbarian group, who had a Mongoloid appearance and among whom some of the Yuezhi lived. Both groups were denigrated as uncivilized and inferior to the Chinese, but the Qiang were deemed to belong to the same racial stock, whereas the Yuezhi were viewed as being part of a very different stock, not only barbarian but ugly and monkey-like to boot.
The Egyptians used a color-coding system—red (themselves), yellow (for their eastern enemies), black (Africans), and white (those from the north). The Eygptians also accurately depicted Africans as early as the third century BCE, describing them exactly how 19th-century European anthropologists would. Below is a picture of how the Egyptians depicted these groups.
There is, also, an interesting bit about colorism—discrimination based on skin color—here:
Color prejudice, says one writer, depended on which ethnic group held sway. When the lighter-skinned Egyptians were dominant they referred to the darker group as “the evil race of Ish.” On the other hand, when the darker-skinned Egyptians were in power, they resorted to calling the lighter-skinned people “the pale, degraded race of Arvad.” (Gossett, 1997: 4)
The Jews are some of the oldest peoples on earth, so they should then have some stories about their encounters with different races. One of the oldest, thought to be first, racist sayings was asked by the prophet Jeremiah who said “Can the Ethiopian change his skin or the leopard his spots?” The Jews are said to have ‘invented’ anti-black racism (Gossett, 1997: 5; Sarich and Miele, 2004), but this has been contested (Goldenberg, 1998). Take the full text from Gossett on Ham:
The most famous example of racism among the Jews is found in the legends which greew up concerning Ham, the son of Noah. The account in Genesis tells us of Ham’s expressing contempt for his father because Noah had become drunk and was lying in a naked stupor. Noah’s other sons had covered their father’s nakedness, averting their eyes. Noah blessed the descendants of Shem and Japeth, his other sons, but cursed the descendants of Ham. There is some confusion in the account in Genesis because it is not clear whether the curse was to be visited upon Ham or upon Canaan, Ham being a later insertion. Nothng is said in Genesis about the descendants of either Ham or Canaan being Negroes. This idea is not found untl the oral traditions of the Jews were collected in the Babylonian Talmud from the second century to the sixth centry A.D. In this source, the descendants of Ham are said to be cursed by being black. In the Talmud, there are several contradictory legends concerning Ham—onoe that God forbade anyone to have sexual relations on the Ark and Ham disobeyed this command. Another story is that Ham was cursed with blackness because he resented the fact that his father desired to have a fourth son. To prevent the birth of a rival heir, Ham is said to have castrated his father. Elsewhere in the Talmud, Ham’s descendants are depicted as being led into captivity with their buttocks uncovered as a sign of degredation.
Greeks and Romans
The Greeks and the Romans are really interesting. Being near the intersection of the Medditerranean, they would have seen many different races of people—and this is reflected in their art and legends. The Greek myth of Phaethon, for example, shows that the Greeks knew that skin color was a function of climate.
In the story, Phaethon asked his father to drive the sun chariot, using it only for the day. He could not control the chariot so it came to close to the earth in some regions, burning the people there while for the people in the north he drove too far away from the earth, ligtening their skin. Greek and Roman myths, in fact, show exactly how things change and that if we had a different reference point—like the Greeks and Romans did—we would then create different theories of ‘intelligence’:
“The nations inhabiting the cold places and those of Europe are full of spirit but somewhat deficient in intelligence and skill, so that they continue comparatively free, but lacking in political organization and the capacity to rule their neighbors. The peoples of Asia on the other hand are intelligent and skillful in temperament, but lack spirit, so that they are in continuous subjection and slavery. But the Greek race participates in both characters, just as it occupies the middle position geographically, for it is both spirited and intelligent; hence it continues to be free and to have very good political institutions, and to be capable of ruling all mankind if it attains constitutional unity.” (Pol. 1327b23-33, my italics)
Views of direct environmental influence and the porosity of bodies to these effects also entered the military machines of ancient empires, like that of the Romans. Offices such as Vegetius (De re militari, I/2) suggested avoiding recruiting troops from cold climates as they had too much blood and, hence, inadequate intelligence. Instead, he argued, troops from temperate climates be recruited, as they possess the right amount of blood, ensuring their fitness for camp discipline (Irby, 2016). Delicate and effemenizing land was also to be abandoned as soon as possible, according Manilius and Caesar (ibid). Probably the most famous geopolitical dictum of antiquity reflects exactly this plastic power of laces: “soft lands breed soft men”, according to the claim that Herodotus attributed to Cyrus. (Meloni, 2017: 41-42)
The Roman historian Vitruvius “attributed the keen intelligence of his countrymen to the rarity of the atmosphere and to the heat. The less fortunate northern peoples, “being enveloped in a dense atmosphere, and chilled by moisture from the obstructing air … have but a sluggish intelligence”” (Gossett, 1997: 7). How convenient—people at the time thought they were ‘superior’ to others and then attempted to justify it on the basis of environmental—eventually evolutionary—differences. However, the Greek theory of humors. Such accounts, though, only speak to how the Greeks thought that the environment shaped individuals, not shared traits of the group. Such differences were thought to be almost immediately reversible. They believed that one could take a person who grew up in another environment who, therefore, had a different temperment which could be changed by switching his environment.
Thus if there were, say, a microregion of Germany where “Asiatic” environmental conditions prevailed, a person who settled in that microregion would end up with Asian attributes. Thus, humoral accounts of human diversity focused on the way environments shape individuals, rather than the way populations share traits. (Smith, 2016: 85)
The Greeks and the Romans, ironically, seemed to be really big on environmentalism—the thesis that environment drives the proliferation of traits and that changing the environment can change ones phenotypic traits. While this is not wholly true, there is a kernel of truth here.
Sarich and Miele (2004: 51) describe different ancient scholar’s writings on their observations of racial differences:
The most detailed surviving description of the racially denning characteristics of black Africans from the classical world appears in The Moretum, a poem attributed to Virgil (circa 1st century AD). A female character named Scybale. is described as “African in race—her hair tightly curled, lips thick, color dark, chest broad, breasts pendulous, belly somewhat pinched, legs thick, and feet broad and ample.” In his book Blacks in Antiquity: Ethiopians in the Greco-Roman Experience, Frank M. Snowden comared the description with portrayls by twentieth-century anthropologists E. A Hootn and M. J Herskovits. For example, Hootn described the “outstanding features of the ancient specialized Negro division of manking” as “narrow heads and wide noses, thick lips and thin legs, protruding jaws and receding chins, integument rich in pigment but poor in hairy growth, flat feet and round foreheads, tiny curls and big smiles.”
Snowden concluded: “While the author of The Moretum was writing poetry, not anthropology,” his description of the distinguishing racial characteristics of black Africans “is good anthropology; in fact, the ancient and modern phraseology is so similar that the modern might be considered a translation of the ancient” (emphasis added).
I’m sure most have heard the popular ‘myth’ that God burnt blacks by cooking them too long. Come to find out, there is a real basis for this myth. The Native Americans thought that white people weren’t baked enough, blacks were baked too much and they were—like Goldilocks—juuuuust right:
Earthmaker made the world with trees and fields, with rivers, lakes, and springs, and with hills and valleys. It was beautiful. However, there weren’t any humans, and so one day he decided to make some.
He scooped out a hole in a stream bank and lined the hole with stones to make a hearth, and he built a fire there. Then he took some clay and made a small figure that he put in the hearth. While it baked, he took some twigs and made tongs. When he pulled the figure out of the fire and had let it cool, he moved its limbs and breathed life into it, and it walked away. Earthmaker nonetheless realized that it was only half-baked. That figure became the white people.
Earthmaker decided to try again, and so he made another figure and put it on the hearth. This time he took a nap under a tree while the figure baked, and he slept longer than he intended. When he pulled the second figure out of the fire and had let it cool, he moved its limbs and breathed life into it, and it walked away. Earthmaker realized that this figure was overbaked, and it became the black people.
Earthmaker decided to try one more time. He cleaned the ashes out of the hearth and built a new fire. Then he scooped up some clay and cleaned it of any twigs or leaves, so that it was pure. He made a little figure and put it on the hearth, and this time he sat by the hearth and watched carefully as the figure baked. When this figure was done, he pulled it out of the fire and let it cool. Then he moved its limbs and breathed life into it, and it walked away. This figure was baked just right, and it became the red people. (A Potawatomi Story)
The first peoples to describe Africans in a racist manner was not Europeans, it was the Arabs—Islamics. They held slaves long before Europeans; they even castrated their slaves. Jahiz of Basra described Africans as “people of black color, flat noses, kinky hair.”…despite their dimness, their boundless stupidity, their crude prceptions and their evil dispositions” is how Jahiz of Basra described Africans. Ibn Khaldun stated “The only people who accept slavery are the Negroes, owing to their low degree of humanity and their proximity to the animal stage.” Nasir al-Din Tusi stated “Many have observed that the ape is more teachable than the Zanji [African].” (All quotes from Sarich and Miele, 2004: 60).
What this little tour of the concept of race throughout history tells us one thing: The concept ‘race’ is not a European invention—races were not socially constructed in 1492. They were constructed thousands of years in the past by many different peoples who had different explanations for the racial differences they had observed. While some of them, for their time, are great explanations for the observed differences, there was an element of racial prejudice, even all of those thousands of years ago. Yes, race is partly socially constructed (as evidenced here) but that social construction has a real, biological basis behind it.
It is obvious that the concept of ‘race’ and ‘racism’ went hand-in-hand all throughout antiquity. It is only today, it seems, that we can attempt to use the concept of race without having any ‘racist’ undertones. Though, the tour we went on proves one thing: race exists and was known to have existed for thousands of years.
The East Asian race has been held up as what a high “IQ” population can do and, along with the correlation between IQ and standardized testing, “HBDers” claim that this is proof that East Asians are more “intelligent” than Europeans and Africans. Lynn (2006: 114) states that the average IQ of China is 103. There are many problems with such a claim, though. Not least because of the many reports of Chinese cheating on standardized tests. East Asians are claimed to be “genetically superior” to other races as regards IQ, but this claim fails.
Chinese IQ and cheating
Differences in IQ scores have been noted all over China (Lynn and Cheng, 2013), but generally, the consensus is, as a country, that Chinese IQ is 105 while in Singapore and Hong Kong it is 103 and 107 respectively (Lynn, 2006: 118). To explain the patterns of racial IQ scores, Lynn has proposed the Cold Winters theory (of which a considerable response has been mounted against it) which proposes that the harshness of the environment in the ice age selected-for higher ‘general intelligence’ in East Asian and European populations; such a hypothesis is valid to hereditarians since East Asian (“Mongoloids” as Lynn and Rushton call them) consistently score higher on IQ tests than Europeans (eg Lynn and Dzobion, 1979; Lynn, 1991; Herrnstein and Murray, 1994). In a recent editorial in Psych, Lynn (2019) criticizes this claim from Flynn (2019):
While northern Chinese may have been north of the Himalayas during the last Ice Age, the southern Chinese took a coastal route from Africa to China. They went along the Southern coast of the Middle East, India, and Southeast Asia before they arrived at the Yangzi. They never were subject to extreme cold.
In response, Lynn cites Frost’s (2019) article where he claims that “mean intelligence seems to have risen during recorded history at temperate latitudes in Europe and East Asia.” Just-so storytelling about how and why such “abilities” were “selected-for”, the Chinese score higher on standardized tests than whites and blacks, and this deserves an explanation (the Cold Winters Theory fails; it’s a just-so story).
Before continuing, something must be noted about Lynn and his Chinese IQ data. Lynn ignores numerous studies on Chinese IQ—Lynn would presumably say that he wants to test those in good conditions and so disregards those parts of China with bad environmental conditions (as he did with African IQs). Here is a collection of forty studies that Lynn did not refer to—some showing that, even in regions in China with optimum living conditions, IQs below 90 are found (Qian et al, 2005). How could Lynn miss so many of these studies if he has been reading into the matter and, presumably, keeping up with the latest findings in the field? The only answer to the question is that Richard Lynn is dishonest. (I can see PumpkinPerson claiming that “Lynn is old! It’s hard to search through and read every study!” to defend this.)
Although the Chinese are currently trying to stop cheating on standardized testing (even a possible seven-year prison sentence, if caught cheating, does not deter cheating), cheating on standardized tests in China and by the Chinese in America is rampant. The following is but a sample of what could be found doing a cursory search on the matter.
One of the most popular ways of cheating on standardized tests is to have another person take the exam for you—which is rampant in China. In one story, as reported by The Atlantic, students can hire “gunmen” to sit-in on tests for them, though measures are being taken to fight back against that such as voice recognition and finger-printing. It is well-known that much of the cheating on such tests are being done by international students.
Even on the PISA—which is used as an “IQ” proxy since they correlate highly (.89) (Lynn and Mikk, 2009)—though, there is cheating. For the PISA, each country is to select, at random, 5,000 of their 15-year-old children around the country and administer the PISA—they chose their biggest provinces which are packed with universities. Further, score flucuations attract attention which indicates dishonesty. In 2000, more than 2000 people protested outside of a university to protest a new law which banned cheating on tests.
The rift amounted to this: Metal detectors had been installed in schools to route out students carrying hearing or transmitting devices. More invigilators were hired to monitor the college entrance exam and patrol campus for people transmitting answers to students. Female students were patted down. In response, angry parents and students championed their right to cheat. Not cheating, they said, would put them at a disadvantage in a country where student cheating has become standard practice. “We want fairness. There is no fairness if you do not let us cheat,” they chanted. (Chinese students and their parents fight for the right to cheat)
Surely, with rampant cheating on standardized tests in China (and for Chinese Americans), we can trust the Chinese IQ numbers in light of the news that there is a culture of cheating on tests in China and in America.
“Genetic superiority” and immigrant hyper-selectivity
Strangely, some proponents of the concept of “genetic superiority” and “progressive evolution” still exist. PumpkinPerson is one of those proponents, writing articles with titles like “Genetically superior: Are East Asians more socially intelligent too?, More evidence that East Asians are genetically superior, Oriental populations: Genetically superior, even referring to a fictional character on a TV show as a “genetic superior.” Such fantastical delusions come from Rushton’s ridiculous claim that evolution may be progressive and that some populations are, therefore, “more evolved” than others:
One theoretical possibility is that evolution is progressive and that some populations are more “advanced” than others. Rushton, 1992
Such notions of “evolutionary progress” and “superiority“—even back in my “HBD” days—never passed the smell test to me. In any case, how can East Asians be said to be “genetically superior”? What do “superior genes” or a “superior genome” look like? This has been outright stated by, for example, Lynn (1977) who prolcaims—for the Japanese—that his “findings indicate a genuine superiority of the Japanese in general intelligence.” This claim, though, is refuted by the empirical data—what explains East Asian educational achievement is not “superior genes”, but the belief that education is paramount for upward social mobility, and so, to preempt discrimination, this would then be why East Asians overperform in school (Sue and Okazaki, 1990).
Furthermore, the academic achievement of Asian cannot be reduced to Asian culture—the fact that they are hyper-selected is why social class matters less for Asian Americans (Lee and Zhou, 2017).
These counterfactuals illustrate that there is nothing essential about Chinese or Asian culture that promotes exceptional educational outcomes, but, rather, is the result of a circular process unique to Asian immigrants in the United States. Asian immigrants to the United States are hyper-selected, which results in the transmission and recreation of middle-class specific cultural frames, institutions, and practices, including a strict success frame as well as an ethnic system of supplementary education to support the success frame for the second generation. Moreover, because of the hyper-selectivity of East Asian immigrants and the racialisation of Asians in the United States, stereotypes of Asian-American students are positive, leading to ‘stereotype promise’, which also boosts academic outcomes
Inequalities reproduce at both ends of the educational spectrum. Some students are assumed to be low-achievers and undeserving, tracked into remedial classes, and then ‘prove’ their low achievement. On the other hand, others are assumed to be high-achievers and deserving of meeting their potential (regardless of actual performance); they are tracked into high-level classes, offered help with their coursework, encouraged to set their sights on the most competitive four-year universities, and then rise to the occasion, thus ‘proving’ the initial presumption of their ability. These are the spill-over effects and social psychological consequences of the hyper-selectivity of contemporary Asian immigration to the United States. Combined with the direct effects, these explain why class matters less for Asian-Americans and help to produce exceptional academic outcomes. (Lee and Zhou, 2017)
The success of second-generation Chinese Americans has, too, been held up as more evidence that the Chinese are ‘superior’ in their mental abilities—being deemed ‘model minorities’ in America. However, in Spain, the story is different. First- and second-generation Chinese immigrants score lower than the native Spanish population on standardized tests. The ‘types’ of immigrants that have emigrated has been forwarded as an explanation for why there are differences in attainments of Asian populations. For example, Yiu (2013: 574) writes:
Yet, on the other side of the Atlantic, a strikingly different story about Chinese immigrants and their offspring – a vastly understudied group – emerges. Findings from this study show that Chinese youth in Spain have substantially lower educational ambitions and attainment than youth from every other nationality. This is corroborated by recently published statistics which show that only 20 percent of Chinese youth are enrolled in post-compulsory secondary education, the prerequisite level of schooling for university education, compared to 40 percent of the entire adolescent population and 30 percent of the immigrant youth population in Catalonia, a major immigrant destination in Spain (Generalitat de Catalunyan, 2010).
… but results from this study show that compositional differences across immigrant groups by class origins and education backgrounds, while substantial, do not fully account for why some groups have higher ambitions than others. Moreover, existing studies have pointed out that even among Chinese American youth from humble, working-class origins, their drive for academic success is still strong, most likely due to their parents’ and even co-ethnic communities’ high expectations for them (e.g., Kao, 1995; Louie, 2004; Kasinitz et al., 2008).
The Chinese in Spain believe that education is a closed opportunity and so, they allocate their energy elsewhere—into entrepreneurship (Yiu, 2013). So, instead of Asian parents pushing for education, they push for entrepreneurship. What this shows is that what the Chinese do is based on context and how they perceive how they will be looked at in the society that they emigrate to. US-born Chinese immigrants are shuttled toward higher education whereas in the Netherlands, the second-generation Chinese have lower educational attainment and the differences come down to national context (Noam, 2014). The Chinese in the U.S. are hyper-selected whereas the Chinese in Spain are not and this shows—the Chinese in the US have a high educational attainment whereas they have a low educational attainment in Spain and the Netherlands—in fact, the Chinese in Spain show lower educational attainment than other ethnic groups (Central Americans, Dominicans, Morrocans; Lee and Zhou, 2017: 2236) which, to Americans would be seen as a surprise
Second-generation Chinese parents match their intergenerational transmission of their ethnocultural emphasis on education to the needs of their national surroundings, which, naturally, affects their third-generation children differently. In the U.S., adaptation implies that parents accept the part of their ethnoculture that stresses educational achievement. (Noam, 2014: 53)
So what explains the higher educational attainment of Asians? A mixture of culture and immigrant (hyper-) selectivity along with the belief that education is paramount for upward mobility (Sue and Okazaki, 1990; Hsin and Xie, 2014; Lee and Zhou, 2017) and the fact that what a Chinese immigrant chooses to do is based on national context (Noam, 2014; Lee and Zhou, 2017). Poor Asians do indeed perform better on scholastic achievement tests than poor whites and poor ‘Hispanics’ (Hsin and Xie, 2014; Liu and Xie, 2016). Teachers even favor Asian American students, perceiving them to be brighter than other students. But what are assumed to be cultural values are actually class values which is due to the hyper-selectivity of Asian immigrants to America (Hsin, 2016).
The fact that the term “Mongoloid idiot” was coined for those with Down syndrome because they looked Asian is very telling (see Hilliard, 2012 for discussion). But, the IQ-ists switched from talking about Caucasian superiority to Asian superiority right as the East began their economic boom (Liberman, 2001). The fact that there were disparate “estimates” of skulls in these centuries points to the fact such “scientific observations” are painted with a cultural brush. See eg table 1 from Lieberman (2001):
This tells us, again, that our “scientific objectivity” is clouded by political and economic prejudices of the time. This allows Rushton to proclaim “If my work was motivated by racism, why would I want Asians to have bigger brains than whites?” Indeed, what a good question. The answer is that the whole point of “HBD race realism” is to denigrate blacks, so as long as whites are above blacks in their little self-made “hierarchy” no such problem exists for them (Hilliard, 2012).
Note how Rushton’s long debunked- r/K selection theory (Anderson, 1991; Graves, 2002) used the current hierarchy and placed dozens of traits on a hierarchy where it was M > C > N (Mongoloids, Caucasoids, and Negroids respectively, to use Rushton’s outdated terminology). It is a political statement to put the ‘Mongoloids’ at the top of the racial hierarchy; the goal of ‘HBD’ is to denigrate blacks. But, do note that in the late 19th to early 20th century that East Asians were deemed to have small brains, large penises, and that Japanese men, for instance, would “debauch their [white] female classmates” (quoted in Hilliard, 2012: 91).
The “IQ” of China (along with scores on other standardized tests such as TIMMS and PISA), in light of the scandals occurring regarding standardized testing should be suspect. Richard Lynn has failed to report dozens of studies that show low IQ scores for China, thusly inflating their scores. This is, yet again, another nail in the coffin for the ‘Cold Winter Theory’, since the story is formulated on the basis of cherry-picked IQ scores of children. I have noted that if we have different assumptions that we would have different evolutionary stories. Thus, if the other data were provided and, say, Chinese IQ were found to be lower, we would just create a story to justify the score. This is illustrated wonderfully by Flynn (2019):
I will only say that I am suspicious of these because none of us can go back and really evaluate environment and mating patterns. Given free reign, I can supply an evolutionary scenario for almost any pattern of current IQ scores. If blacks had a mean IQ above other races I could posit something like this: they benefitted from exposure to the most rigorous environmental conditions possible, namely, competition from other people. Thanks to greater population pressures on resources, blacks would have benefitted more from this than any of those who left at least for a long time. Those who left eventually became Europeans and East Asians.
The hereditarians point to the academic success of East Asians in America as proof that IQ tests ‘measure’ intelligence, but East Asians in America are a hyper-selected sample. As the references I have provided show, second-generation Chinese immigrants show lower educational attainments than other ethnies (the opposite is true in America) and this is explained by the context that the immigrant family finds themselves in—where do you allocate your energy? Education or entrepreneurship? Such choices seem to be class-based due to the fact education is championed by the Chinese in America and not in Spain and the Netherlands—then dictate, and they also refute any claims of ‘genetic superiority’—they also refute, for that matter, the claim that genes matter for educational attainment (and therefore IQ)—although we did not need to know this to know that IQ is a bunk ‘measure’.
So if Chinese cheat on standardized tests, then we should not accept their IQ scores; the fact that they, for example, provide non-random children from large provinces speaks to their dishonesty. They are like Lynn, in a way, avoiding the evidence that IQ scores are not what they seem—both Lynn and the Chinese government are dishonest cherry-pickers. The ‘fact’ that East Asian educational attainment can be attributed to genes is false; it is attributed to hyper-selectivity and notions of class and what constitutes ‘success’ in the country they emigrate to—so what they attempt is based on (environmental) context.
In 1969, Arthur Jensen published a bombshell article in the Harvard Educational Review titled How Much Can We Boost IQ and Scholastic Achievement? in which he argued that compensatory education has failed (e.g., Headstart) and it, therefore, should be abandoned. Jensen was a big proponent against school integration due to his research on IQ (Tucker, 2002). Tucker (1998) also argued, “that the supposed significance of the genetic influence on IQ has invariably reflected a particular ideological view of the purpose of education and its relation to the state that is rooted in conservative political thought.” Such ideological leanings of the IQ-ists have been well-noted (Tucker, 2002; Saini, 2019).(Though it should be noted that school integration didn’t cause any negative effects for whites and had many positive effects for blacks, see Nazaryan and Johnson, 2019). Note how the revival of “racial differences in intelligence” in the mainstream occurred after the Civil Rights Act of 1964. Such ideological leanings have been incipient in ‘intelligence’ testing since its inception.
In any case, what was the ultimate goal of such research into racial/class differences in “intelligence”? The original application of what eventually became tests of “intelligence” were to (1) identify those with learning disabilities and (2) shoe-horn people into jobs “for” them—what Binet called his “ideal city.” When IQ tests were brought to America and translated from French to English by Henry Goddard in 1911 and then again by Lewis Terman in 1916. Goddard was hesitant to force sterilization, but he did believe that those his tests designated as “feeble-minded” should not be allowed to bear children.
Proponents of IQ emphatically state that it’s not a “measure of superiority” and that it’s only the critics who believe that, with no evidence for the claim. However, if one reads Jensen’s earliest writings on IQ, they would see that Jensen did, in fact, believe that heritability could estimate one’s “genetic standing” (Jensen, 1970) and that if we continue our welfare policy that we would lead a group toward “genetic enslavement” (Jensen, 1969). Jensen ran with racists, so there is a possibility that he himself held similar types of views to the people he ran with. The following quotes show Jensen’s eugenic thinking:
“Is there a danger that current welfare policies, unaided by eugenic foresight, could lead to the genetic enslavement of a substantial segment of our population?” – Jensen, 1969: 95, How Much Can We Boost IQ and Scholastic Achievement?
“What the evidence on heritability tells us is that we can, in fact, estimate a person’s genetic standing on intelligence from his score on an IQ test.” – Jensen, 1970, Can We and Should We Study Race Difference?
“… the best thing the black community could do would be to limit the birth-rate among the least-able members, which of course is a eugenic proposal.” – A Conversation with Arthur Jensen, American Reinnasance, 1992
In a review of Raymond Cattell’s Beyondism, Richard Lynn stated:
“What is called for here is not genocide, the killing off of the populations of incompetent cultures. But we do need to think realistically in terms of “phasing out” of such peoples.”
I don’t see how he’s not calling for genocide—genocide is the systemic killing of a specific group of people. Eugenic methods are one way to accomplish this. Richard Lynn’s father was a eugenicist, signing his name to a manifesto which asked the question of how the genetic constitution of the world could be improved, per Lynn (see Interview with a pioneer, American Reinassance). Lynn continues:
My father’s interests did give me an early appreciation of the importance of genetics, although I think I would have adopted this position anyway since the evidence is irrefutable for a strong genetic determination of intelligence and educational attainment and a moderate genetic determination of personality. More importantly, my father served as a role model for scientific achievement and has given me the confidence to advance theories that have sometimes been controversial.
Lynn stated that he is “very pessimistic” about the future of the West, due to the immigration of individuals from low IQ countries who have a higher birthrate than Westerners along with the supposed dysgenic fertility that American white women are facing. (See Lynn, 1996, 2001 for a discussion and look into these views.) In Dysgenics, Lynn (1996: 2) writes that he hopes “To make the case that in the repudiation of eugenics an important truth has been lost, and to rehabilitate the argument that genetic deterioration is occurring in Western populations and in most of the developed world.”
Raymond Cattell was also one who believed that certain people should (voluntarily) be sterilized. He created a religion called “Beyondism” in an attempt to accomplish this goal, his research, in fact, served his eugenic and political beliefs (Tucker, 2009). Compassion was seen as evil to Cattell, which is one major way it strays from other religions. Presumably, one is compassionate to those less fortunate and, therefore, the compassion would help one who Cattell deems “genetically inferior” and so compassion is evil since it leads to the propagation of those Cattell deems less fit. Cattell also stated that, from the perspective of Beyondism, the propagation of ‘genetic failures’ is “positively evil” (Tucker, 2009: 136). He also coined the term ‘genthanasia’ which was “phasing out” a “moribund culture … by educational and birth measures, without a single member dying before his time” (Cattell, quoted in Tucker, 2009: 146).
William Shockley “reasoned” that if the problems that blacks face in America are hereditary, then by attempting to halt the reproduction of blacks, there would be less racism against them. Well if there are few people to be racist against, then there would be less racism against those people. Shocking. Further, Shockley wanted to enstate what he called a “Voluntary Sterilization Bonus Plan” where individuals with IQs below 100 would, with each single point below 100, be given $1000—although the plan was never implemented (Hilliard, 2012: 50). He also wanted to institute a sperm bank of ‘geniuses’ (whatever that means) but, he was never told, women did not want the sperm of Shockley’s short, balding self (he was 5’6” weighing 150 pounds)—although he had a ‘high IQ’ (he was rejected as being one of Terman’s Termites)—they wanted the sperm of taller, good-looking men, regardless of their IQ (Hilliard, 2012: 20).
It is worth noting that Shockley precedes Jensen’s thinking on race and IQ—Jensen was in the audience of one of Shockley’s talks in the late ’60s hearing him talk about racial differences in IQ. Psychology was Jensen’s second choice; his first was to be a symphony conductor. Hilliard (2012: 51) describes this:
“When Shockley addressed a meeting of the Center for Advanced Study in the Behavioral Sciences at Stanford in the late 1960s, one member of the audience drawn to his discourse was Arthur R. Jensen, a psychologist who taught at the University of California–Berkeley. Jensen, who had described himself as a “frustrated symphony conductor,” may have had his own reasons for reverencing Shockley’s every word. The younger psychologist had been forced to abandon a career in music because his own considerable talents in that area nevertheless lacked “soul,” or the emotional intensity needed to succeed in so competitive a profession. He decided on psychology as a second choice, carrying along with him a grudge against those American subcultures perceived as being “more expressive” than the white culture from which he sprang. Jensen received his bachelor’s degree in that field from the University of California– Berkeley in 1945.”
Shockley even disowned his son for dating a Costa Rican woman since it would “deteriorate their white gene pool” while describing his children as a “considerable regression”, even though they had advanced degrees. He blamed this ‘genetic misfortune’ on his wife who did not have as high educational attainment as he did (Hilliard, 2012: 49). This man greatly influenced Jensen—and it seems to show in his first writings on IQ—what eventually kicked off the ‘IQ debate’ (which is frivolous) back in the late 1960s. (James Thompson has said that Shockley wouldn’t talk to anyone if he didn’t know their IQ—presumably, because he did not want to talk to anyone ‘lower’ than he. The idiotic ‘thinking’ of eugenic IQ-ists.
Shockley was involved in a car accident, and received a head injury, with colleagues noting that his views on race and eugenics came about after his car accident (Hilliard, 2012: 48). So, it can rightly be argued that if Shockley would never have gotten into a car accident then he would have never had the views he did on race and IQ, meaning that he would not be speaking for Jensen to be in the audience to then eventually write his infamous 1969 paper. So, the current revival of the race-and-IQ debate can be said to be due to Shockly’s influence on Jensen, which is due (in some way) to head injuries sustained during a car accident.
IQ-ists speak of a “genetic deterioration”, what is termed “dysgenics” (the opposite of eugenics). Professor Seymour Itzkoff published The Decline of Intelligence in America (Itzkoff, 1994), arguing that the decline in our country’s “intelligence” is the cause of our economic and political woes. And, while he does not outright discuss eugenics in the book, he states that higher IQ people are not having children and so the national IQ is decreasing—a dysgenic effect. He is also a recipient of funding from the Pioneer Fund, published in Mankind Quarterly, and was one of the 52 signatories of Mainstream Science on Intelligence (Gottfredson, 1997).
The Decline of Intelligence in America was The Bell Curve before The Bell Curve. Itzkoff argued policy proscriptions, including encouraging certain people to breed and certain people not to breed (eugenics without calling it eugenics). Itzkoff stated that welfare policy is one reason why our “intelligence”—as a nation—has declined (see also Jensen, 1969; Lynn, 2001). Itzkoff (1994: 195) states that “Those at the bottom should be humanely persuaded, with generous gifts if deemed appropriate but for one generation only to refrain from conceiving and having children.” So his views are a mixture of Jensen’s, Shockley’s and others. Itzkoff advocates for both positive and negative eugenics for black Americans. I have not seen any IQ-ist discuss Itzkoff’s writings; I will do so in the future.
Philosopher and IQ-ist Jonathon Anomaly (see Winegard, Winegard, and Anomaly, 2020) has a paper in which he ‘defends eugenics’, even stating what we ‘should’ (cautiously) do about public policy in relation to eugenic ideas. He speaks of “undesirable genetic endowment“, while couching his “moral obligation to produce children with the best chance of the best life” (Anomaly, 2018) “through mechanisms of prenatal screening, enshrined in the principle of procreative beneficence and our responsibility to not pass along an “undesirable genetic endowment” (Love, 2018: 4). (See my arguments to discourage such research here and here.) Presumably, like Itzkoff (1994), such policies will be concentrated on the lower classes, of which minority populations are the majority. Robert Wilson (2019), author of The Eugenic Mind Project writes that Anomaly (2018) fails to argue for eugenics, mischaracterizes eugenics, mischaracterizes the scientific consensus, simplifies and misleads on the history, is careless about race and IQ, appealing for moral principles, and no substance linking demography, eugenics and policy recommendations. Anomaly could hardly contain his negative eugenist views; his views being akin to more traditional, negative forms of eugenics” (Wilson, 2019: 74).
In any case, IQ tests were used as a vehicle for sterilization and barring immigrants into America in the 1920s (Swanson, 1995; Gould, 1996; Wilson, 2017; Dolmage, 2018). In his book The Eugenic Mind Project, Wilson (2017) discusses standpoint eugenics—how eugenic policies affected people and their own personal experiences with eugenic policies. In the book, Wilson argues that, to the eugenicists, there are different ‘sorts’ of people that can be distinguished from others. Wilson (2017: 48) writes:
This was not, however, the way of human betterment favored by the applied science of eugenics and that continues to forms [sic] a key part of The Eugenic Mind. Instead, historically eugenicists typically followed Galton in emphasizing that quality was not equally distributed in the kinds of human populations that are regulated by governmental policies and jurisdictional legislation. More specifically, they thought of such populations as being composed of fundamentally distinct kinds of people, with some kinds being of higher quality than others. Some of these sorts of people were to be improved through eugenic policies that encouraged their own reproduction; others were to be eliminated over generational time. The goal of intergenerational human improvement within the eugenics movement was thus achieved by increasing the proportion of higher-quality people in future generations, and this could be achieved in two ways under eugenic logic. Thus, eugenicists historically advocated ideas, laws, policies, and practices either that aimed to maximize the reproduction of higher-quality people—positive eugenics—or that aimed to minimalize the reproduction of lower-quality people. Or both.
A hallmark of The Eugenic Mind, says Wilson, is the distinction between the ‘fit’ and ‘unfit’. Thus, those deemed ‘unfit’ would be sterilized as they are different ‘kinds’ of people. Eugenics is seen as an applied science and so, it attempts to achieve certain goals—the propagation of the ‘fit’ and elimination of the ‘unfit.’ The first IQ tests were constructed with class, so that the test scores mirrored current racial/class divisions, justifying the social hierarchy (Mensh and Mensh, 1991). Thus, using the ‘science of IQ’, we could then identify ‘feeble-minds’ and therefore select them out of the gene pool. What really would be going on here is not selecting out ‘low IQ people’ but selecting out those of lower classes—one of the main reasons these types of views sprang up. A trait was ‘eugenic’ if it fit “folk knowledge characteristic of people” (Wilson, 2017: 70).
Eugenic-type thinking also had its beginnings in criminality, right when the first IQ tests were being constructed by Binet in 1905 (Kuhar and Fatović-Ferenčić, 2012). Lombroso’s thesis of hereditary criminality also gave American eugenicists the platform for sterilization of criminals (Applegate, 2018: 438). But early eugenicists were more concerned over white female ‘morons’ and white lower-status, promiscuous white women being coerced and segregated in order to prevent them from breeding, it even being suggested by “one or two scientists” that the women should live on a farm performing menial tasks and should be sterilized; the eugenicists wanted state control of heredity (Applegate, 2018: 439). Mexican-American men and women were even sterilized in the 1900s (Lira, 2015). Such beliefs seem to be baked-in from political and social prejudices; not any basis in ‘science.’
Eugenicists wished for state control over the “propagation of the mentally incompetent,” whether through mental illness or disability. Ultimately, these beliefs would lead not only to forced detention and isolation, but also to regular affronts to human life and dignity. (Applegate, 2018: 442)
But Dr. Sullivan, the medical officer of Holloway Prison in The Eugenics Review stated that “Criminals, looked at from the eugenic standpoint, cannot be put into any single category; some of them, probably most of them, are of average stock, and become criminal under the influence of their milieu; they do not directly interest the eugenist” (Sullivan, 1909: 119-120). The “hyperincarceration of blacks” is also argued to be eugenic in nature (Oleson, 2016; Jones and Seabrook, 2017). Such race-based segregation, argues Oleson, significantly depresses the birthrate of affected groups—racialized minorities (socialgroups taken to be races, e.g., ‘Hispanics‘ and blacks). So since minority populations are overrepresented in prison and they are less likely to procreate, it then follows that such arguments like Oleson’s (2016) has some weight to it. So this then would have eugenic effects over generational time. Even today, America is still sterilizing prisoners, so, it seems, the legacy of the 20th century has yet to let up.
… the penal code is a eugenic instrument, although until today, it has been without consciousness of this function. And following the results of eugenic science, it can tomorrow widen or narrow the circle of crimes in the end of conducing to the physical and psychic improvement of the race. (Battaglini, 1914)
Hitler, noticing the American sterilization laws and the Immigration Act of 1924, instituted eugenic policies on this basis—yes, the Nazi eugenic movement was largely taken from the then-existing social policy in America at the time. Pioneer Fund president Henry Laughlin, who used data from IQ tests in front of Congress to bar certain immigrants from the United States (Swanson, 1995; Dolmage, 2018). The Nazis and Americans had extensive contact with each other, while Germany modeled their sterilization law after Henry Laughlin’s laws for sterilization in US states (Cornwell, 2001; Black, 2003; see Allen, 2004; Lelliot, 2004; and Weikart, 2006 for reviews; Wittmann, 2004). But it is worth noting that Hitler was not a Darwinian (Richards, 2012, 2013). Hitler’s laws in the early 1930s on eugenics “may have had some resemblance to the most extreme of American state’s laws” (Wittmann, 2004: 19), since he was observing the eugenic programs implemented by certain states (eugenic laws were never federally mandated).
The IQ-ist thinking that IQ tests ‘measure’ intelligence led to eugenic policies and the sterilization of criminals and those with low IQs. Jensen and Shockley were the forefronts of bringing IQ-ism back into the picture, and they both had eugenic views (Shockley being way more radical than Jensen, but it is clear that Shockley was his influence here and that, without Shockley, IQ-ism may not have had the sway it does today. The IQ-ist ideology that has led to eugenic thinking and social policies, race and ‘intelligence’ has been there since its inception and it is clear that it still exists today (Chitty, 2007). Most of the big-name IQ-ists have, either explicitly or implicitly, stated things that can be construed in a eugenic way, and thusly, the main goal of the IQ-ist program is revealed: limit the birthrates of the lower classes, which are mostly minorities.
The eugenics movement in America—which then influenced Nazi policy—was not built on science, nor was it political (even though it aimed to be “applied science”; Wilson, 2017), it was a political movement which was erected to control social groups thought to be inferior to the higher-ups (Quigley, 1995). The link between eugenic thinking and IQ-ism in its history go hand-in-hand and some IQ-ists, even today, still advocate for such social policy based on the results from IQ tests (Herrnstein and Murray, 1994; Itzkoff, 1994).
Such prescriptions from IQ-ists for what ‘should be’ done with low IQ people speak to their bias in the matter. One of the great IQ-ists, so revered, Arthur Jensen was very implicit in his views in the late 1960s and early 70s about what should be done for the black population in America. His predecessors, too, had the same type of eugenic beliefs which then influenced their thoughts and values on crime and ‘intelligence.’ This game that IQ-ists have been playing has been going on for over 100 years; and with the advent of new genetic technology, the IQ-ists can continue their eugenic games, attempting to prevent ‘certain people’ from having children.
These tests, originally devised to correlate group’s places on the social hierarchy, cannot be ‘used for good’, as the point of its inception was to justify current class hierarchies as ‘genetic and immutable.’ These psychologists and criminologists leave their fields of inquiry and then attempt to influence public policy using clearly biased tests, as the history of the field has shown since its inception in the early 1900s. These are yet more reasons why IQ testing should be banned, as no good can come from believing that a group or individual is ‘less intelligent’ than another. The eugenic thinking of IQ-ists and criminologists feed off each other, with the IQ-ist ideas being the catalyst for the eugenic policies that followed.
In its essence the traditional notion of general intelligence may be a secularised version of the Puritan idea of the soul. … perhaps Galtonian intelligence had its roots in a far older kind of religious thinking. (John White, Personal space: The religious origins of intelligence testing)
In chapter 1 of Alas, Poor Darwin: Arguments Against Evolutionary Psychology, Dorothy Nelkin identifies the link between the founder of sociobiology E.O. Wilson’s religious beliefs and the epiphany he described when he learned of evolution. A Christian author then used Sociobiology to explain and understand the origins of our own sinfulness (Williams, 2000). But there is another hereditarian-type research program that has these kinds of assumptions baked-in—IQ.
Philosopher of education John White has looked into the origins of IQ testing and the Puritan religion. The main link between Puritanism and IQ was that of predestination. The first IQ-ists conceptualized IQ—‘g’ or general intelligence—to be innate, predetermined and hereditary. The predetermination line between both IQ and Puritanism is easy to see: To the Puritans, it was predestined whether or not one went to Hell before they even existed as human beings whereas to the IQ-ists, IQ was predestined, due to genes.
John White (2006: 39) in Intelligence, Destiny, and Education notes the parallel between “salvation and success, damnation and failure”:
Can we usefully compare the saved/damned dichotomy with the perceived contribtion of intelligence or the lack of it to success and failure in life, as conventionally understood? One thing telling against this is that intelligence testers claim to identify via IQ scores a continuous gamut of ability from lowest to highest. On the other hand, most of the pioneers in the field were … especially interested in the far ends of this range — in Galton’s phrase ‘the extreme classes, the best and the worst.’ On the other hand there were the ‘gifted’, ‘the eminent’, ‘those who have honourably succeeded in life’, presumably … the most valuable portion of our human stock. On the other, the ‘feeble-minded’, the ‘cretins’, the ‘refuse’ those seeking to avoid ‘the monotony of daily labor’, democracy’s ballast, not always useless but always a potential liability’.
A Puritan-type parallel can be drawn here—the ‘cretins and ‘feeble-minded’ are ‘the damned’ whereas ‘the extreme classes, the best and worst’ were ‘the saved.’ This kind of parallel can still be seen in modern conceptualizations of the debate and current GWASs—certain people have a certain surfeit of genes that influence intellectual attainment. Contrast with the Puritan “Certain people are chosen before they exist to either be damned or saved.” Certain people are chosen, by random mix-ups of genes during conception, to either be successful or not, and this is predetermined by the genes. So, genetic determinism when speaking of IQ is, in a way, just like Puritan predestination—according to Galton, Burt and other IQ-ists in the 1910s-1920s (ever since Goddard brought back the Binet-Simon Scales from France in 1910).
Some Puritans banned the poor from their communities seeing them as “disruptors to Puritan communities.” Stone (2018: 3-4) in An Invitation to Satan: Puritan Culture and the Salem Witch Trials writes:
The range of Puritan belief in salvation usually extended merely to members of their own communities and other Puritans. They viewed outsiders as suspicious, and people who held different beliefs, creeds, or did things differently were considered dangerous or evil. Because Puritans believed the community shared the consequences of right and wrong, often community actions were taken to atone for the misdeed. As such, they did not hesitate to punish or assault people who they deemed to be transgressors against them and against God’s will. The people who found themselves punished were the poor, and women who stood low on the social ladder. These punishments would range from beatings to public humiliation. Certain crimes, however, were viewed as far worse than others and were considered capital crimes, punishable by death.
Could the Puritan treatment of the poor be due to their beliefs of predestination? Puritan John Winthrop stated in his book A Model of Christian Charity that “some must be rich, some poor, some high and eminent in power and dignity, others mean and in subjection.” This, too, is still around today: IQ sets “upper limits” on one’s “ability ceiling” to achieve X. The poor are those who do not have the ‘right genes’. This is, also, a reason why IQ tests were first introduced in America—to turn away the poor (Gould, 1996; Dolmage, 2018). That one’s ability is predetermined in their genes—that each person has their own ‘ceiling of ability’ that they can reach that is then constrained by their genes is just like the Puritan predestination thesis. But, it is unverifiable and unfalsifiable, so it is not a scientific theory.
To White (2006), the claim that we have this ‘innate capacity’ that is ‘general’ this ‘intelligence’ is wanting. He takes this further, though. In discussing Galton’s and Burt’s claim that there are ‘ability ceilings’—and in discussing a letter he wrote to Burn—White (2006: 16) imagines that we give instruction to all of the twin pairs and that, their scores increase by 15 points. This, then, would have a large effect on the correlation “So it must be an assumption made by the theorist — i.e. Burt — in claiming a correlation of 0.87, that coaching could not successfully improve IQ scores. Burt replied ‘I doubt whether, had we returned a second time, the coaching would have affected our correlations” (White, 2006: 16). Burt seems to be implying that a “ceiling of ability” exists, which he got from his mentor, Galton. White continues:
It would appear that Galton nor Burt have any evidence for their key claim [that ability ceilings exist]. The proposition that, for all of us, there are individually differing ceilings of ability seems to be an assumption behind their position, rather than a conclusion based on telling grounds.
I have discussed elsewhere (White, 1974; 2002a: ch. 5) what could count as evidence for this proposition, and concluded that it is neither verifiable nor falsifiable. The mere fact that a child appears not able to get beyond, say, elementary algebra is not evidence of a ceiling. The failure of this or that variation in teaching approach fares no better, since it is always possible for a tracher to try some different approach to help the learner get over the hurdle. (With some children, so neurologically damaged that they seem incapable of language, it may seem that the point where options run out for the teacher is easier to establish than it is for other children. But the proposition in question is supposed to applu to all of us: we are all said to have our own mental ceiling; and for non-brain-damaged people the existence of a ceiling sems impossible to demonstrate.) It is not falsifiable, since for even the cleverest person in the world, for whom no ceiling has been discovered, it is always possible that it exists somewhere. As an untestable — unverifiable and unfalsifiable — proposition, the claim that we each have a mental ceiling has, if we follow Karl Popper (1963: ch. 1), no role in science. It is like the proposition that God exists or that all historical events are predetermined, both of which are equally untestable. As such, it may play a foundational role, as these two propositions have played, in some ideological belief system of belief, but has no place in empirical science. (White, 2006: 16)
Burt believed that we should use IQ tests to shoe-horn people into what they would be ‘best for’ on the basis of IQ. Indeed, this is one of the main reasons why Binet constructed what would then become the modern IQ test. Binet, influenced by Galton’s (1869) Hereditary Genius, believed that we could identify and help lower-‘ability’ children. Binet envisioned an ‘ideal city’ in which people were pushed to vocations that were based on their ‘IQs.’ Mensh and Mensh (1991: 23) quote Binet on the “universal applications” of his test:
Of what use is a measure of intelligence? Without doubt, one could conceive many possible applications of the process in dreaming of a future where the social sphere would be better organized than ours; where everyone would work according to his known apptitudes in such a way that non particle of psychic force should be lost for society. That would be the ideal city.
So, it seems, Binet wanted to use his test as an early aptitude-type test (like the ones we did in grammar school which ‘showed us’ which vocations we would be ‘good at’ based on a questionnaire). Having people in Binet’s ‘ideal city’ work based on their ‘known aptitudes’ would increase, not decrease, inequality so Binet’s envisioned city is exactly the same as today’s world. Mensh and Mensh (1991: 24) continue:
When Binet asserted that everyone would work to “known” aptitudes, he was saying that the individuals comprising a particular group would work according to the aptitudes that group was “known” to have. When he suggested, for example, that children of lower socioeconomic status are perfectly suited for manual labor, he was simply expressing what elite groups “know,” that is, that they themselves have mental aptitudes, and others have manual ones. It was this elitist belief, this universal rationale for the social status quo, that would be upheld by the universal testing Binet proposed.
White (2006: 42) writes:
Children born with low IQs have been held to have no hope of a professional, well-paid job. If they are capable of joining the workforce at all, they must find their niche as the unskilled workers.
Thus, the similarities between IQ-ist and religious (Puritan) belief comes clear. The parallels between the Puritan concern for salvation and the IQ-ist belief that one’s ‘innate intelligence’ dictated whether or not they would succeed or fail in life (based on their genes); both had thoughts of those lower on the social ladder, their work ethic and morals associated with the reprobate on the one hand and the low IQ people on the other; both groups believed that the family is the ‘mechanism’ by which individuals are ‘saved’ or ‘damned’—presuming salvation is transmitted based one’s family for the Puritans and for the IQ-ists that those with ‘high intelligence’ have children with the same; they both believed that their favored group should be at the top with the best jobs, and best education, while those lower on the social ladder should also get what they accordingly deserve. Galton, Binet, Goddard, Terman, Yerkes, Burt, and others believed that one was endowed with ‘innate general intelligence’ due to genes, according to the current-day IQ-ists who take the same concept.
White drew his parallel between IQ and Puritanism without being aware that one of the first anti-IQ-ists—and American Journalist named Walter Lippman—who also been made in the mid-1920s. (See Mensh and Mensh, 1991 for a discussion of Lippman’s grievances with the IQ-ists). Such a parralel between Puritanism and Galton’s concept of ‘intelligence’ and that of the IQ-ists today. White (2005: 440) notes “that virtually all the major players in the story had Puritan connexions may prove, after all, to be no more than coincidence.” Though, the evidence that White has marshaled in favor of the claim is interesting, as noted many parallels exist. It would be some huge coincidence for there to be all of these parallels without them being causal (from Puritanistic beliefs to hereditarian IQ dogma).
This is similar to what Oyama (1985: 53) notes:
Just as traditonal though placed biological forms in the mind of God, so modern thought finds many ways of endowing the genes with ultimate formative power, a power bestowed by Nature over countless milennia.
But this parallel between Puritanism and hereditarianism doesn’t just go back to the early 20th century—it can still be seen today. The assumption that genes contain a type of ‘information’ before activated by the physiological system for its uses still pervades our thought today, even though many others have been at the forefront to change that kind of thinking (Oyama, 1985, 2000; Jablonka and Lamb, 1995, 2005; Moore, 2002, 2016; Noble, 2006, 2011, 2016).
The links between hereditarianism and religion are compelling; eugenic and Puritan beliefs are similar (Durst, 2017). IQ tests have now been identified as having their origins in eugenic beliefs, along with Puritan-like beliefs have being saved/damned based on something that is predetermined, out of your control just like your genetics. The conception of ‘ability ceilings’—using IQ tests—is not verifiable nor is it falsifiable. Hereditarians believe in ‘ability ceilings’ and claim that genes contain a kind of “blueprint” (which is still held today) which predestines one toward certain dispositions/behaviors/actions. Early IQ-ists believed that one is destined for certain types of jobs based on what is ‘known’ about their group. When Binet wrote that, the gene was yet to be conceptualized, but it has stayed with us ever since.
So not only did the concept of “IQ” emerge due to the ‘need’ to ‘identify’ individuals for their certain ‘aptitudes’ that they would be well-suited for in, for instance, Binet’s ideal city, it also arose from eugenic beliefs and religious (Puritan) thinking. This may be why IQ-ists seem so hysterical—so religious—when talking about IQ and the ‘predictions’ it ‘makes’ (see Nash, 1990).
The history of standardized testing—including IQ testing—has a contentious history. What causes score distributions between groups of people? I stated at least four reasons why there is a test gap:
(1) Differences in genes cause differences in IQ scores;
(2) Differences in environment cause differences in IQ scores;
(3) A combination of genes and environment cause differences in IQ scores; and
(4) Differences in IQ scores are built into the test based on the test constructors’ prior biases.
I hold to (4) since, as I have noted, the hereditarian-environmentalist debate is frivolous. There is, as I have been saying for years now, no agreed-upon definition of ‘intelligence’, since there are such disparate answers from the ‘experts’ (Lanz, 2000; Richardson, 2002).
For the lack of such a definition only reflects the fact that there is no worked-out theory of intelligence. Having a successful definition of intelligence without a corresponding theory would be like having a building without foundations. This lack of theory is also responsible for the lack of some principled regimentation of the very many uses the word ‘intelligence’ and its cognates are put to. Tao many questions concerning intelligence are still open, too many answers controversial. Consider a few examples of rather basic questions: Does ‘intelligence’ name some entity which underlies and explains certain classes of performances1, or is the word ‘intelligence’ only sort of a shorthand-description for ‘being good at a couple of tasks or tests’ (typically those used in IQ tests)? In other words: Is ‘intelligence’ primarily a descriptive or also an explanatorily useful term? Is there really something like intelligence or are there only different individual abilities (compare Deese 1993)? Or should we turn our backs on the noun ‘intelligence’ and focus on the adverb ‘intelligently’, used to characterize certain classes of behaviors? (Lanz, 2000: 20)
Nash (1990: 133-4) writes:
Always since there are just a series of tasks of one sort or another on which performance can be ranked and correlated with other performances. Some performances are defined as ‘cognitive performances’ and other performances as ‘attainment performances’ on essentially arbitrary, common sense grounds. Then, since ‘cognitive performances’ require ‘ability’ they are said to measure that ‘ability’. And, obviously, the more ‘cognitive ability’ an individual posesses the more that individual can acheive. These procedures can provide no evidence that IQ is or can be measured, and it is rather besides the point to look for any, since that IQ is a metric property is a fundamental assumption of IQ theory. It is imposible that any ‘evidence’ could be produced by such procedures. A standardised test score (whether on tests designated as IQ or attainment tests) obtained by an individual indicates the relative standing of that individual. A score lies within the top ten perent or bottom half, or whatever, of those gained by the standardisation group. None of this demonstrates measurement of any property. People may be rank ordered by their telephone numbers but that would not indicate measurement of anything. IQ theory must demonstrate not that it has ranked people according to some performance (that requires no demonstration) but that they are ranked according to some real property revealed by that performance. If the test is an IQ test the property is IQ — by definition — and there can in consequence be no evidence dependent on measurement procedures for hypothesising its existence. The question is one of theory and meaning rather than one of technique. It is impossible to provide a satisfactory, that is non-circular, definition of the supposed ‘general cognitive ability’ IQ tests attempt to measure and without that definition IQ theory fails to meet the minimal conditions of measurement.
These is similar to Mary Midgley’s critique of ‘intelligence’ in her last book before her death What Is Philosophy For? (Midgley, 2018). The ‘definitions’ of ‘intelligence’ and, along with it, its ‘measurement’ have never been satisfactory. Haier (2016: 24) refers to Gottfredson’s ‘definition’ of ‘intelligence, stating that ‘intelligence’ is a ‘general mental ability.’ But if that is the case, that it is a ‘general mental ability’ (g) then ‘intelligence’ does not exist because ‘g’ does not exist as a property in the brain. Lanz’s (2000) critique is also like Howe’s (1988; 1997) that ‘intelligence’ is a descriptive, not explanatory, term.
Now that the concept of ‘intelligence’ has been covered, let’s turn to race and test bias.
Test items are biased when they have different psychological meanings across cultures (He and van de Vijver 2012: 7). If they have different meanings across cultures, then the tests will not reflect the same ‘ability’ between cultures. Being exposed to the knowledge—and correct usage of it—on a test is imperative for performance. For if one is not exposed to the content on the test, how are they expected to do well if they do not know the content? Indeed, there is much evidence that minority groups are not acculturated to the items on the test (Manly et al, 1997; Ryan et al, 2005; Boone et al, 2007). This is what IQ tests measure: acculturation to the the tests’ constructors, school cirriculum and school teachers—aspects of white, middle-class culture (Richardson, 1998). Ryan et al (2005) found that reading and and educational level, not race or ethnicity, was related to worse performance on psychological tests.
Serpell et al (2006) took 149 white and black fourth-graders and randomly assigned them to ethnically homogeneous groups of three, working on a motion task on a computer. Both blacks and whites learned equally well, but the transfer outcomes were better for blacks than for whites.
Helms (1992) claims that standardized tests are “Eurocentric”, which is “a perceptual set in which European and/ or European American values, customs, traditions and characteristics are used as exclusive standards against which people and events in the world are evaluated and perceived.” In her conclusion, she stated that “Acculturation
and assimilation to White Euro-American culture should enhance one’s performance on currently existing cognitive ability tests” (Helms, 1992: 1098). There just so happens to be evidence for this (along with the the studies referenced above).
Fagan and Holland (2002) showed that when exposure to different kinds of information was required, whites did better than blacks but when it was based on generally available knowledge, there was no difference between the groups. Fagan and Holland (2007) asked whites and blacks to solve problems found on usual IQ-type tests (e.g., standardized tests). Half of the items were solvable on the basis of available information, but the other items were solveable only on the basis of having acquired previous knowledge, which indicated test bais (Fagan and Holland, 2007). They, again, showed that when knowledge is equalized, so are IQ scores. Thus, cultural differences in information acquisition explain IQ scores. “There is no distinction between crassly biased IQ test items and those that appear to be non-biased” (Mensh and Mensh, 1991). This is because each item is chosen because it agrees with the distribution that the test constructors presuppose (Simon, 1997).
How do the neuropsychological studies referenced above along with Fagan and Holland’s studies show that test bias—and, along with it test construction—is built into the test which causes the distribution of the scores observed? Simple: Since the test constructors come from a higher social class, and the items chosen for inclusion on the test are more likely to be found in certain cultural groups than others, it follows that the reason for lower scores was that they were not exposed to the culturally-specific knowledge used on the test (Richardson, 2002; Hilliard, 2012).
The [IQ] tests do what their construction dictates; they correlate a group’s mental worth with its place in the social hierarchy. (Mensh and Mensh, 1991)
This is very easily seen with how such tests are constructed. The biases go back to the beginning of standardized testing—the first one being the SAT. The tests’ constructors had an idea of who was or was not ‘intelligent’ and so constructed the tests to show what they already ‘knew.’
…as one delves further … into test construction, one finds a maze of arbitrary steps taken to ensure that the items selected — the surrogates of intelligence — will rank children of different classes in conformity with a mental hierarchy that is presupposed to exist. (Mensh and Mensh, 1991)
Garrison (2009: 5) states that standardized tests “exist to assess social function” and that “Standardized testing—or the theory and practice known as “psychometrics” … is not a form of measurment.” The same way tests were constructed in the 1900s is the same way they are constructed today—with arbitrary items and a presuppossed mental hiearchy which then become baked into the tests by virtue of how they are constructed.
IQ-ists like to say that certain genes are associated with high intelligence (using their GWASes), but what could the argument possibly be that would show that variation in SNPs would cause variation in ‘intelligence’? What would a theory of that look like? How is the hereditarian hypothesis not a just-so story? Such tests were created to justify the hierarchies in society, the tests were constructed to give the results that they get. So, I don’t see how genetic ‘explanations’ are not just-so stories.
1 Blacks and whites are different cultural groups.
2 If (1), then they will have different experiences by virtue of being different cultural groups.is
3 So blacks and whites, being different cultural groups, will score differently on tests of ability, since they are exposed to different knowledge structures due to their different cultures and so, all tests of ability are culture-bound. Knowledge, Culture, Logic, and IQ
Rushton and Jensen (2005) claim that the evidence they review over the past 30 years of IQ testing points to a ‘genetic component’ to the black-white IQ gap, relying on the flawed Minnesota study of twins “reared apart” (Joseph, 2018)—among other methods—to generate heritability estimates and state that “The new evidence reviewed here points to some genetic component in Black–White differences in mean IQ.” The concept of heritability, however, is a flawed metric (Bailey, 1997; Schonemann, 1997; Guo, 2000; Moore, 2002; Rose, 2006; Schneider, 2007; Charney, 2012, 2013; Burt and Simons, 2014; Panofsky, 2014; Joseph et al, 2015; Moore and Shenk, 2016; Panofsky, 2016; Richardson, 2017). That G and E interact means that we cannot tease out “percentages” of nature and nurture’s “contribution” to a “trait.” So, one cannot point to heritability estimates as if they point to a “genetic cause” of the score gap between blacks and whites. Further note that the gap has closed in recent years (Dickens and Flynn, 2006; Smith, 2018).
And now, here is another argument based on the differing experiences that cultural groups experience which then explains IQ score differences (eg Mensh and Mensh, 1991; Manly et al, 1997; Kwate, 2001; Fagan and Holland, 2002, 2007; Cole, 2004; Ryan et al, 2005; Boone et al, 2007; Au, 2008; Hilliard, 2012; Au, 2013).
(1) If children of different class levels have experiences of different kinds with different material; and
(2) if IQ tests draw a disproportionate amount of test items from the higher classes; then
(c) higher class children should have higher scores than lower-class children.
Nature vs nurture can be said to be a debate on what is ‘innate’ and what is ‘acquired’ in an organism. Debates about how nature and nurture tie into athletic ability and race both fall back onto the dichotomous notion. “Athleticism is innate and genetic!”, the hereditarian proclaims. “That blacks of West African ancestry are over-represented in the 100m dash is evidence of nature over nurture!” How simplistic these claims are.
Steve Sailer, in his response to Birney et al on the existence of race, assumes that because those with ancestry to West Africa consistently have produced the most finalists (and winners) in the Olympics that race, therefore, must exist.
I pointed out on Twitter that it’s hard to reconcile the current dogma about race not being a biological reality with what we see in sports, such as each of the last 72 finalists in the Olympic 100-meter dash going all the way back to 1984 nine Olympics ago being at least half sub-Saharan in ancestry.
the abundant data suggesting that individuals of sub-Saharan ancestry enjoy genetic advantages.
For example, it’s considered fine to suggest that the reason that each new Dibaba is fast is due to their shared genetics. But to say that one major reason Ethiopians keep winning Olympic running medals (now up to 54, but none at any distance shorter than the 1,500-meter metric mile because Ethiopians lack sprinting ability) is due to their shared genetics is thought unthinkable.
Sailer’s argument seems to be “Group X is better than Group Y at event A. Therefore, X and Y are races”, which is similar to the hereditarian arguments on the existence of ‘race’—just assume they exist.
The outright reductionism to genes in Sailer’s view on athleticism and race is plainly obvious. That blacks are over-represented in certain sports (e.g., football and basketball) is taken to be evidence for this type of reductionism that Sailer and others appeal to (Gnida, 1995). Such appeals can be said to be implicitly saying “The reason why blacks succeed at sport is due to genes while whites succeed due to hard work, so blacks don’t need to work as hard as whites when it comes to sports.”
There are anatomic and physiological differences between groups deemed “black” and “white”, and these differences do influence sporting success. Even though this is true, this does not mean that race exists. Such reductionist claims—as I myself have espoused years ago—do not hold up. Yes, blacks have a higher proportion of type II muscle fibers (Caesar and Henry, 2015), but this does not alone explain success in certain athletic disciplines.
Current genetic testing cannot identify an athlete (Pitsiladis et al, 2013). I reviewed some of the literature on power genotypes and race and concluded that there are no genes yet identified that can be said to be a sufficient cause of success in power sports.
Just because group A has gene or gene networks G and they compete in competition C does not mean that gene or gene networks G contribute in full—or in part—to sporting success. The correlations could be coincidental and non-functional in regard to the sport in question. Athletes should be studied in isolation, meaning just studying a specific athlete in a specific discipline to ascertain how, what, and why works for the specific athlete along with taking anthropomorphic measures, seeing how bad they want “it”, and other environmental factors such as nutrition and training. Looking at the body as a system will take us away from privileging one part over another—while we also do understand that they do play a role but not the role that reductionists believe.
No evidence exists for DNA variants that are common to endurance athletes (Rankinen et al, 2016). But they do have one thing in common (which is an environmental effect on biology): those born at altitude have a permanently altered ventilatory response as adults while “Peruvians born at altitude have a nearly 10% larger forced vital capacity compared to genetically matched Peruvians born at sea level” (Brutasaert and Parra, 2009: 16). Certain environmental effects on biology are well-known, and those biological changes do help in certain athletic events (Epstein, 2014). Yan et al (2016) conclude that “conclude that the traditional argument of nature versus nurture is no longer relevant, as it has been clearly established that both are important factors in the road to becoming an elite athlete.”
Georgiades et al (2017) go the other way and what they argue is clear in the title of their paper “Why nature prevails over nurture in the making of the elite athlete.” They continue:
Despite this complexity, the overwhelming and accumulating evidence, amounted through experimental research spanning almost two centuries, tips the balance in favour of nature in the “nature” and “nurture” debate. In other words, truly elite-level athletes are built – but only from those born with innate ability.
They use twin studies as an example stating that since heritability is greater than 50% but lower than 100% means “that the environment is also important.” But this is a strange take, especially from seasoned sports scientists (like Pitsiladis). Attempting to partition traits into a ‘nature’ and ‘nurture’ component and then argue that the emergence of that trait is due more to genetics than environment is an erroneous use of heritability estimates. It is not possible—nor is it feasible—to separate traits into genetic and environmental components. The question does not even make sense.
“… the question of how to separate the native from the acquired in the responses of man does not seem likely to be answered because the question is unintelligible.” (Leonard Carmichael 1925, quoted in Genes, Determinism and God, Alexander, 2017)
Tucker and Collins (2012) write:
Rather, individual performance thresholds are determined by our genetic make-up, and training can be defined as the process by which genetic potential is realised. Although the specific details are currently unknown, the current scientific literature clearly indicates that both nurture and nature are involved in determining elite athletic performance. In conclusion, elite sporting performance is the result of the interaction between genetic and training factors, with the result that both talent identification and management systems to facilitate optimal training are crucial to sporting success.
Tucker and Collins (2012) define training as the realization of genetic potential, while DNA “control the ceiling” of what one may be able to accomplish. “… training maximises
the likelihood of obtaining a performance level with a genetically controlled ‘ceiling’, accounts for the observed dominance of certain populations in specific sporting disciplines” (Tucker and Collins, 2012: 6). “Training” would be the environment here and the “genetically controlled ‘ceiling'” would be genes here. The authors are arguing that while training is important, training is just realizing the ‘potential’ of what is ‘already in’ the genes—an erroneous way of looking at genes. Shenk (2010: 107) explains why:
As the search for athletic genes continues, therefore, the overwhelming evidence suggests that researchers will instead locate genes prone to certain types of interactions: gene variant A in combination with gene variant B, provoked into expression by X amount of training + Y altitude + Z will to win + a hundred other life variables (coaching, injuries, etc.), will produce some specific result R. What this means, of course, What this means, of course, is that we need to dispense rhetorically with thick firewall between biology (nature) and training (nurture). The reality of GxE assures that each person’s genes interacts with his climate, altitude, culture, meals, language, customs and spirituality—everything—to produce unique lifestyle trajectories. Genes play a critical role, but as dynamic instruments, not a fixed blueprint. A seven- or fourteen- or twenty-eight-year-old is not that way merely because of genetic instruction.
The model proposed by Tucker and Collins (2012) is pretty reductionist (see Ericsson, 2012 for a response), while the model proposed by Shenk (2010) is more holistic. The hypothetical model explaining Kenyan distance running success (Wilbur and Pitsiladis, 2012) is, too, a more realistic way of assessing sport dominance:
The formation of an elite athlete comes down to a combination of genes, training, and numerous other interacting factors. The attempt to boil the appearance of a certain trait to either ‘genes’ or ‘environment’ and partition them into percentages is an unsound procedure. That a certain group continuously wins a certain event does not constitute evidence that the group in question is a race, nor does it constitute evidence that ‘genes’ are the cause of the outcome between groups in that event. The holistic model of human athletic performance in which genes contribute to certain physiological processes along with training, and other biomechanical and psychological differences is the correct way to think about sport and race. Actually seeing an athlete in motion in his preferred sport is (and I believe always will be) superior to just genetic analyses. Genetic tests also have “no role to play in talent identification” (Webborn et al, 2015).
One emerging concept is that there are many potential genetic pathways to a given phenotype . This concept is consistent with ideas that biological redundancy underpins complex multiscale physiological responses and adaptations in humans . From an applied perspective, the ideas discussed in this review suggest that talent identification on the basis of DNA testing is likely to be of limited value, and that field testing, which is essentially a higher order ‘bioassay’, is likely to remain a key element of talent identification in both the near and foreseeable future . (Joyner, 2019; Genetic Approaches for Sports Performance: How Far Away Are We?)
Athleticism is irreducible to biology (Louis, 2004). Holistic (nature and nurture) will beat the reductionist (nature vs nurture) views; with how biological systems work, there is no reason to privilege one level over another (Noble, 2012), so there is no reason to privilege the gene over the environment, environment over the gene. The interaction of multiple factors explains sport success.
Mary Midgley (1919-2018) is a philosopher perhaps most well-known for her writing on moral philosophy and rejoinders to Richard Dawkins after his publication of The Selfish Gene. Before her passing in October of 2018, she published What Is Philosophy For? on September 21st. In the book, she discusses ‘intelligence’ and its ‘measurement’ and comes to familiar conclusions.
‘Intelligence’ is not a ‘thing’ like, say, temperature and weight (though it is reified as one). Thermometers measure temperature, and this was verified without relying on the thermometer itself (see Hasok Chang, Inventing Temperature). Temperature can be measured in terms of units like kelvin, celsius, and Fahrenheit. The temperature is the available kinetic energy of heat; ‘thermo’ means heat while ‘meter’ means to measure, so heat is what is being measured with a thermometer.
Scales measure weight. If energy balance is stable, so too will weight be stable. Eat too much or too little, then weight gain or loss will occur. But animals seem to have a body set weight which has been experimentally demonstrated (Leibel, 2008). In any case, what a scale measures is the overall weight of an object which is done by measuring how much force exists between the weighed object and the earth.
The whole concept of ‘intelligence’ is hopelessly unreal.
Prophecies [like those of people who work on AI] treat intelligence as a quantifiable stuff, a standard, unvarying, substance like granulated sugar, a substance found in every kind of cake — a substance which, when poured on in larger quantities, always produces a standard improvement in performance. This mythical way of talking has nothing to do with the way in which cleverness — and thought generally — actually develops among human beings. This imagery is, in fact, about as reasonable as expecting children to grow up into steamrollers on the ground that they are already getting larger and can easily be trained to stamp down gravel on roads. In both cases, there simply is not the kind of continuity that would make any such progress conceivable. (Midgley, 2018: 98)
We recognize the divergence of interests all the time when we are trying to find suitable people for different situations. Thus Bob may be an excellent mathematician but is still a hopeless sailor, while Tim, that impressive navigator, cannot deal with advanced mathematics at all. which of them then should be considered the more intelligent? In real life, we don’t make the mistake of trying to add these people’s gifts up quantitatively to make a single composite genius and then hope to find him. We know that planners wanting to find a leader for their exploring expedition must either choose between these candidates or send both of them. Their peculiar capacities grow out of their special interests in topics, which is not a measurable talent but an integral part of their own character.
In fact, the word ‘intelligence’ does not name a single measurable property, like ‘temperature’ or ‘weight’. It is a general term like ‘usefulness’ or ‘rarity’. And general terms always need a context to give them any detailed application. It makes no more sense to ask whether Newton was more intelligent than Shakespeare than it does to ask if a hammer is more useful than a knife. There can’t be such a thing as an all-purpose intelligence, any more than an all-purpose tool. … Thus the idea of a single scale of cleverness, rising from the normal to beyond the highest known IQ, is simply a misleading myth.
It is unfortunate that we have got so used today to talk of IQs, which suggests that this sort of abstract cleverness does exist. This has happened because we have got used to ‘intelligence tests’ themselves, devices which sort people out into convenient categories for simple purposes, such as admission to schools and hospitals, in a way that seems to quantify their ability. This leads people to think that there is indeed a single quantifiable stuff called intelligence. But, for as long as these tests have been used, it has been clear that this language is too crude even for those simple cases. No sensible person would normally think of relying on it beyond those contexts. Far less can it be extended as a kind of brain-thermometer to use for measuring more complex kinds of ability. The idea of simply increasing intelligence in the abstract — rather than beginning to understand some particular kind of thing better — simply does not make sense. (Midgley, 2018: 100-101)
IQ researchers, though, take IQ to be a measure of a quantitative trait that can be measured in increments—like height, weight, and temperature. “So, in deciding that IQ is a quantitative trait, investigators are making big assumptions about its genetic and environmental background” (Richardson, 2000: 61). But there is no validity to the measure and hence no backing for the claim that it is a quantitative trait and measures what they suppose it does.
Just because we refer to something abstract does not mean that it has a referent in the real world; just because we call something ‘intelligence’ and say that it is tested—however crudely—by IQ tests does not mean that it exists and that the test is measuring it. Thermometers measure temperature; scales measure weight; IQ tests….don’t measure ‘intelligence’ (whatever that is), they measure acculturated knowledge and skills. Howe (1997: 6) writes that psychological test scores are “an indication of how well someone has performed at a number of questions that have been chosen for largely practical reasons” while Richardson (1998: 127) writes that “The most reasonable answer to the question “What is being measured?”, then, is ‘degree of cultural affiliation’: to the culture of test constructors, school teachers and school curricula.”
But the word ‘intelligence’ refers to what? The attempt to measure ‘intelligence’ is a failure as such tests cannot be divorced from their cultural contexts. This won’t stop IQ-ists, though, from claiming that we can rank one’s mind as ‘better’ than another on the basis of IQ test scores—even if they can’t define ‘intelligence’. Midgley’s chapter, while short, gets straight to the point. ‘Intelligence’ is not a ‘thing’ like height, weight, or temperature. Height can be measured by a ruler; weight can be measured by a scale; temperature can be measured by a thermometer. Intelligence? Can’t be measured by an IQ test.